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Neuron, ISSN 0896-6273, 04/2013, Volume 78, Issue 2, pp. 256 - 268
Journal Article
Journal Article
PLoS genetics, ISSN 1553-7390, 12/2018, Volume 14, Issue 12, p. e1007791
Journal Article
PLOS ONE, ISSN 1932-6203, 07/2019, Volume 14, Issue 7, p. e0218111
Journal Article
PLOS ONE, ISSN 1932-6203, 07/2019, Volume 14, Issue 7, p. e0218111
Late onset Alzheimer's disease is the most common form of dementia for which about 30 susceptibility loci have been reported. The aim of the current study is... 
COMMON VARIANTS | IMMUNITY | PGC-1-ALPHA | ANNOTATION | MULTIDISCIPLINARY SCIENCES | ALPHA | GENERATION | DIFFERENTIATION | ZNF423 | BRAIN | CD2AP | Genome-wide association studies | Genes | Genetic aspects | Single nucleotide polymorphisms | Alzheimer's disease | Health aspects | Identification and classification
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 10/2019, Volume 72, Issue 1, pp. 301 - 318
Most of the loci identified by genome-wide association studies (GWAS) for late-onset Alzheimer’s disease (LOAD) are in strong linkage disequilibrium (LD) with... 
Target recognition | Statistical analysis | Downstream effects | Genes | Genomics | Linkage disequilibrium | Genetic diversity | Ribonucleic acid--RNA | Gene expression | Tissues | Loci | Biological activity | Genetic variance | Enhancers | Biological effects | Algorithms | Molecular modelling | Apolipoprotein E | Regulatory mechanisms (biology) | Localization | Bayesian analysis
Journal Article
BRAIN, ISSN 0006-8950, 09/2019, Volume 142, Issue 9, pp. 2581 - 2589
Autopsy measures of Alzheimer's disease neuropathology have been leveraged as endophenotypes in previous genome-wide association studies (GWAS). However,... 
DIAGNOSIS | GUIDELINES | tau | RISK | NEUROSCIENCES | CLINICAL NEUROLOGY | beta-amyloid | NATIONAL INSTITUTE | COGNITIVE DECLINE | genome-wide association study | DISEASE | HAPLOTYPE | Alzheimer's disease | EXPRESSION | neuropathology | GENOME-WIDE ASSOCIATION | Neurosciences | Neurovetenskaper
Journal Article
ACTA NEUROPATHOLOGICA, ISSN 0001-6322, 12/2018, Volume 136, Issue 6, pp. 857 - 872
Cerebrospinal fluid (CSF) levels of amyloid- 42 (A42) and tau have been evaluated as endophenotypes in Alzheimer's disease (AD) genetic studies. Although there... 
SURFACE-BASED ANALYSIS | Alzheimer disease | Neuropathology | Tau | COGNITIVE IMPAIRMENT | RISK | HYPOTHETICAL MODEL | PATHOLOGY | APOLIPOPROTEIN-E GENOTYPE | CEREBROSPINAL-FLUID | Sex difference | NEUROSCIENCES | CLINICAL NEUROLOGY | MEMORY | COMPOSITE SCORE | Cerebrospinal fluid biomarkers | Amyloid | APOE | EXPRESSION | GENOME-WIDE ASSOCIATION | Neurosciences | Neurovetenskaper
Journal Article