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Biochemistry, ISSN 0006-2960, 12/2017, Volume 56, Issue 50, pp. 6515 - 6516
Journal Article
Nature Chemical Biology, ISSN 1552-4450, 11/2015, Volume 11, Issue 12, pp. 909 - 916
Analysis of genomes, transcriptomes and proteomes reveals the existence of hundreds to thousands of translated, yet non-annotated, short open reading frames... 
OPEN READING FRAMES | RNA | CELLS IN-VITRO | FUNCTIONAL PROTEINS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEIN-PROTEIN INTERACTIONS | RECEPTOR | SMALL ORFS | GENOME-WIDE SEARCH | PEPTIDE | LIVING CELLS | Animals | Peptides - metabolism | Peptides - chemistry | Open Reading Frames - genetics | Humans | Peptides - genetics | Polypeptides | Molecular biology | Genomics | Biological activity
Journal Article
Journal Article
Trends in pharmacological sciences, ISSN 0165-6147, 08/2019, Volume 40, Issue 10, pp. 716 - 718
Enzymes with multiple substrates pose a unique challenge for drug development because of an increased potential for on-target side effects. Maianti and... 
Enzyme inhibitors
Journal Article
Nature Chemical Biology, ISSN 1552-4450, 01/2013, Volume 9, Issue 1, pp. 59 - 64
Journal Article
Molecular Cell, ISSN 1097-2765, 05/2015, Volume 58, Issue 4, pp. 699 - 706
Journal Article
BBA - Molecular and Cell Biology of Lipids, ISSN 1388-1981, 10/2013, Volume 1831, Issue 10, pp. 1542 - 1554
Journal Article
Nature, ISSN 0028-0836, 01/2018, Volume 553, Issue 7688, pp. 351 - 355
The circadian clock imposes daily rhythms in cell proliferation, metabolism, inflammation and DNA damage response(1,2). Perturbations of these processes are... 
CIRCADIAN CLOCK | TARGET | STEM-CELLS | METABOLISM | DNA-DAMAGE RESPONSE | BEHAVIOR | MULTIDISCIPLINARY SCIENCES | ALPHA | AUTOPHAGY | MODULATION | Apoptosis - drug effects | Humans | Male | Circadian Clocks - genetics | Autophagy - drug effects | Pyrrolidines - pharmacology | Neoplasms - genetics | Female | Membrane Proteins - metabolism | Nevus - drug therapy | Nuclear Receptor Subfamily 1, Group D, Member 1 - metabolism | Cell Survival - drug effects | Oncogenes - genetics | Nuclear Receptor Subfamily 1, Group D, Member 1 - agonists | Membrane Proteins - genetics | Mice, Inbred C57BL | Thiophenes - pharmacology | Neoplasms - drug therapy | Animals | GTP Phosphohydrolases - metabolism | Signal Transduction - drug effects | GTP Phosphohydrolases - genetics | Nevus - pathology | Glioblastoma - pathology | Lipogenesis - drug effects | Cell Line, Tumor | Glioblastoma - drug therapy | Neoplasms - pathology | Circadian Clocks - physiology | Physiological aspects | Development and progression | Genetic aspects | Cancer | Cell proliferation | Oxidative stress | Regulators | Senescence | Toxicity | p53 Protein | DNA damage | Glioblastoma | Cytotoxicity | Genomes | Autophagy | Cancer therapies | Neoplasms | Anticancer properties | Receptors | Antitumor agents | Metabolites | Animal tissues | Cell cycle | Biocompatibility | Lipogenesis | Deoxyribonucleic acid--DNA | Circadian rhythms | Enzymes | Pharmacology | Circadian rhythm | Gene expression | Nuclear receptors | Metabolism | Fatty acids | Stem cells | Hypoxia | Antitumor activity | Phagocytosis | Tumors | Apoptosis | Index Medicus
Journal Article
Biochemistry, ISSN 0006-2960, 02/2019, Volume 58, Issue 8, pp. 1022 - 1023
Journal Article