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1. Full Text Metabolic alterations by indoxyl sulfate in skeletal muscle induce uremic sarcopenia in chronic kidney disease
by Sato, Emiko and Mori, Takefumi and Mishima, Eikan and Suzuki, Arisa and Sugawara, Sanae and Kurasawa, Naho and Saigusa, Daisuke and Miura, Daisuke and Morikawa-Ichinose, Tomomi and Saito, Ritsumi and Oba-Yabana, Ikuko and Oe, Yuji and Kisu, Kiyomi and Naganuma, Eri and Koizumi, Kenji and Mokudai, Takayuki and Niwano, Yoshimi and Kudo, Tai and Suzuki, Chitose and Takahashi, Nobuyuki and Sato, Hiroshi and Abe, Takaaki and Niwa, Toshimitsu and Ito, Sadayoshi
Scientific reports, ISSN 2045-2322, 11/2016, Volume 6, Issue 1, pp. 36618 - 36618
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Tricarboxylic acid cycle | Metabolomics | Pentose phosphate | Mass spectroscopy | Metabolism | Morbidity | Skeletal muscle | Sarcopenia | Musculoskeletal system | Mitochondria | Rodents | Phosphate | Glycolysis | Pentose phosphate pathway | Kidney diseases | Sulfate | Kidney transplantation | Index Medicus
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2. Full Text Impact of the oral adsorbent AST-120 on organ-specific accumulation of uremic toxins: LC-MS/MS and MS imaging techniques
by Sato, Emiko and Saigusa, Daisuke and Mishima, Eikan and Uchida, Taeko and Miura, Daisuke and Morikawa-Ichinose, Tomomi and Kisu, Kiyomi and Sekimoto, Akiyo and Saito, Ritsumi and Oe, Yuji and Matsumoto, Yotaro and Tomioka, Yoshihisa and Mori, Takefumi and Takahashi, Nobuyuki and Sato, Hiroshi and Abe, Takaaki and Niwa, Toshimitsu and Ito, Sadayoshi
Toxins, ISSN 2072-6651, 01/2018, Volume 10, Issue 1, p. 19
p-cresyl sulfate | Chronic kidney disease | Mass spectrometry | Indoxyl sulfate | Uremic toxin | Food Science & Technology | Toxicology | Life Sciences & Biomedicine | Science & Technology | Oxides - therapeutic use | Carbon - therapeutic use | Administration, Oral | Kidney Failure, Chronic - drug therapy | Mice, Inbred C57BL | Kidney Failure, Chronic - diagnostic imaging | Male | Sulfuric Acid Esters - metabolism | Cresols - metabolism | Kidney Failure, Chronic - metabolism | Tandem Mass Spectrometry | Animals | Adsorption | Toxins, Biological - metabolism | Indican - metabolism | Chromatography, Liquid | Uremia | Immunohistochemistry | Neuroimaging | Brain | Sulfates | Accumulation | Adsorbents | Atrophy | Sarcopenia | Mouse devices | Bioaccumulation | Animal tissues | Spectroscopy | Kidneys | Organs | Imaging techniques | Muscles | Adenine | Mass spectroscopy | Liquid chromatography | Skeletal muscle | Musculoskeletal system | Renal failure | Toxins | Mice | Scientific imaging | Index Medicus | mass spectrometry | chronic kidney disease | uremic toxin | indoxyl sulfate
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3. Full Text Nrf2 Suppresses Oxidative Stress and Inflammation in App Knock-In Alzheimer's Disease Model Mice
by Uruno, Akira and Matsumaru, Daisuke and Ryoke, Rie and Saito, Ritsumi and Kadoguchi, Shiori and Saigusa, Daisuke and Saito, Takashi and Saido, Takaomi C and Kawashima, Ryuta and Yamamoto, Masayuki
Molecular and cellular biology, ISSN 0270-7306, 03/2020, Volume 40, Issue 6
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Index Medicus
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4. Full Text Ferroptosis is controlled by the coordinated transcriptional regulation of glutathione and labile iron metabolism by the transcription factor BACH1
by Nishizawa, Hironari and Matsumoto, Mitsuyo and Shindo, Tomohiko and Saigusa, Daisuke and Kato, Hiroki and Suzuki, Katsushi and Sato, Masaki and Ishii, Yusho and Shimokawa, Hiroaki and Igarashi, Kazuhiko
The Journal of biological chemistry, ISSN 0021-9258, 01/2020, Volume 295, Issue 1, pp. 69 - 82
cardiovascular disease | ferroptosis | cell death | iron sequestration | myocardial infarction | iron | transcription repressor | (BTB domain and CNC homolog 1) BACH1 | glutathione peroxidase | ischemia | Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Basic-Leucine Zipper Transcription Factors - metabolism | Oxidative Stress | Glutathione - metabolism | Transcriptional Activation | Amino Acid Transport System y+ - genetics | Ferroptosis | Amino Acid Transport System y+ - metabolism | Ferritins - metabolism | Cation Transport Proteins - metabolism | Ferritins - genetics | Myocardial Infarction - pathology | Cation Transport Proteins - genetics | Glutamate-Cysteine Ligase - metabolism | Fibroblasts - metabolism | Oxidoreductases - metabolism | Oxidoreductases - genetics | Mice, Inbred C57BL | Cells, Cultured | Basic-Leucine Zipper Transcription Factors - genetics | Myocardial Infarction - metabolism | Iron - metabolism | Animals | Myocytes, Cardiac - metabolism | Glutamate-Cysteine Ligase - genetics | Mice | Index Medicus | Cell Biology
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5. Full Text Impacts of NRF2 activation in non–small‐cell lung cancer cell lines on extracellular metabolites
by Saigusa, Daisuke and Motoike, Ikuko N and Saito, Sakae and Zorzi, Michael and Aoki, Yuichi and Kitamura, Hiroshi and Suzuki, Mikiko and Katsuoka, Fumiki and Ishii, Hirofumi and Kinoshita, Kengo and Motohashi, Hozumi and Yamamoto, Masayuki
Cancer science, ISSN 1347-9032, 02/2020, Volume 111, Issue 2, pp. 667 - 678
NRF2 | metabolites | non–small‐cell lung cancer | metabolome | culture supernatant | Life Sciences & Biomedicine | Oncology | Science & Technology | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Humans | Lung Neoplasms - metabolism | Carcinoma, Non-Small-Cell Lung - metabolism | Gene Expression Regulation, Neoplastic | Gene Expression Profiling | Gene Regulatory Networks | Metabolomics - methods | Whole Exome Sequencing | Cullin Proteins - genetics | Kelch-Like ECH-Associated Protein 1 - genetics | NF-E2-Related Factor 2 - metabolism | Cell Line, Tumor | NF-E2-Related Factor 2 - genetics | Mutation | Adenocarcinoma | Cell culture | Fourier transforms | Squamous cell carcinoma | Cytokines | Toxicity | Lung cancer | Genes | Non-small cell lung carcinoma | Tumor cell lines | Gene expression | Metabolism | Cell activation | Databases | Metabolites | Correlation analysis | Nutrients | Scientific imaging | Mass spectrometry | NRF2 protein | Index Medicus | Original
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6. Full Text Nrf2 Improves Leptin and Insulin Resistance Provoked by Hypothalamic Oxidative Stress
by Yagishita, Yoko and Uruno, Akira and Fukutomi, Toshiaki and Saito, Ritsumi and Saigusa, Daisuke and Pi, Jingbo and Fukamizu, Akiyoshi and Sugiyama, Fumihiro and Takahashi, Satoru and Yamamoto, Masayuki
Cell reports (Cambridge), ISSN 2211-1247, 02/2017, Volume 18, Issue 8, pp. 2030 - 2044
leptin resistance | Nrf2 | proopiomelanocortin | pancreatic beta-cells | Trsp | selenoproteins | insulin resistance | oxidative stress | hypothalamus | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Antioxidants - metabolism | Leptin - metabolism | Oxidative Stress - physiology | Diabetes Mellitus - metabolism | RNA, Transfer, Amino Acid-Specific - metabolism | Mice, Knockout | Insulin - metabolism | Insulin-Secreting Cells - metabolism | Animals | Hypothalamus - metabolism | Promoter Regions, Genetic - physiology | Insulin Resistance - physiology | NF-E2-Related Factor 2 - metabolism | Signal Transduction - physiology | Mice | Index Medicus
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7. Full Text Transcription factor Nrf2 hyperactivation in early-phase renal ischemia-reperfusion injury prevents tubular damage progression
by Nezu, Masahiro and Souma, Tomokazu and Yu, Lei and Suzuki, Takafumi and Saigusa, Daisuke and Ito, Sadayoshi and Suzuki, Norio and Yamamoto, Masayuki
Kidney international, ISSN 0085-2538, 02/2017, Volume 91, Issue 2, pp. 387 - 401
ischemia reperfusion | transcription regulation | chronic kidney disease | acute kidney injury | oxidative stress | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Reactive Oxygen Species - metabolism | Kidney Tubular Necrosis, Acute - genetics | Oxidative Stress | Glutathione - metabolism | Antioxidants - metabolism | Humans | Kidney Tubular Necrosis, Acute - prevention & control | Kidney Tubular Necrosis, Acute - metabolism | Kelch-Like ECH-Associated Protein 1 - genetics | Transfection | RNA Interference | Time Factors | NF-E2-Related Factor 2 - genetics | Kidney Tubules - pathology | NADP - metabolism | Kidney Tubules - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Disease Models, Animal | Cell Line | Reperfusion Injury - pathology | Signal Transduction | Genotype | Kelch-Like ECH-Associated Protein 1 - metabolism | NF-E2-Related Factor 2 - deficiency | Disease Progression | Mice, Knockout | Gene Expression Regulation, Enzymologic | Phenotype | Animals | Reperfusion Injury - prevention & control | NF-E2-Related Factor 2 - metabolism | Glucosephosphate Dehydrogenase - metabolism | Glucosephosphate Dehydrogenase - genetics | Kidney Tubular Necrosis, Acute - pathology | Index Medicus
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8. Full Text Maternity Log study: a longitudinal lifelog monitoring and multiomics analysis for the early prediction of complicated pregnancy
by Sugawara, Junichi and Ochi, Daisuke and Yamashita, Riu and Yamauchi, Takafumi and Saigusa, Daisuke and Wagata, Maiko and Obara, Taku and Ishikuro, Mami and Tsunemoto, Yoshiki and Harada, Yuki and Shibata, Tomoko and Mimori, Takahiro and Kawashima, Junko and Katsuoka, Fumiki and Igarashi-Takai, Takako and Ogishima, Soichi and Metoki, Hirohito and Hashizume, Hiroaki and Fuse, Nobuo and Minegishi, Naoko and Koshiba, Seizo and Tanabe, Osamu and Kuriyama, Shinichi and Kinoshita, Kengo and Kure, Shigeo and Yaegashi, Nobuo and Yamamoto, Masayuki and Hiyama, Satoshi and Nagasaki, Masao
BMJ open, ISSN 2044-6055, 02/2019, Volume 9, Issue 2, pp. e025939 - e025939
complicated pregnancy | prediction | lifelog | multi-omics analysis | Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Life Style | Prospective Studies | Humans | Japan | Middle Aged | Computational Biology | Transcriptome | Metabolome | Proteome | Pregnancy Complications - diagnosis | Pregnancy | Young Adult | Microbiota | Adult | Female | Hypertension | Urine | Time series | Systematic review | Gene expression | Obstetrics | Smartphones | Hospitals | Womens health | Consent | Questionnaires | Data collection | Physiology | Software | Diabetes | Bioinformatics | Index Medicus
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9. Full Text Epithelial Fli1 deficiency drives systemic autoimmunity and fibrosis: Possible roles in scleroderma
by Takahashi, Takehiro and Asano, Yoshihide and Sugawara, Koji and Yamashita, Takashi and Nakamura, Kouki and Saigusa, Ryosuke and Ichimura, Yohei and Toyama, Tetsuo and Taniguchi, Takashi and Akamata, Kaname and Noda, Shinji and Yoshizaki, Ayumi and Tsuruta, Daisuke and Trojanowska, Maria and Sato, Shinichi
The Journal of experimental medicine, ISSN 0022-1007, 04/2017, Volume 214, Issue 4, pp. 1129 - 1151
Immunology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Autoimmunity | Th17 Cells - physiology | Transcription Factors - physiology | Th2 Cells - physiology | Humans | Transcriptome | Keratin-14 - analysis | Proto-Oncogene Protein c-fli-1 - physiology | Transcription Factors - genetics | Epithelial Cells - physiology | Esophagus - pathology | Animals | Scleroderma, Systemic - etiology | Keratinocytes - metabolism | Fibrosis | Mice | Homeodomain Proteins - physiology | Skin - pathology | Disease Models, Animal | Transcription factors | Pathogenesis | Epithelial cells | Leukemia | Viruses | Mimicry | Thymus | Keratin | Vascular diseases | Rodents | Fibroblasts | Scleroderma | Immune system | Lung diseases | Organs | Keratinocytes | Esophagus | Gene silencing | Lungs | Systemic sclerosis | Skin | AIRE protein | Autoimmune diseases | Auditory defects | Index Medicus | 300
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