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1991, Nihon gendaishi bunko, ISBN 9784886252807, Volume 45, 158
Book
STEM CELLS, ISSN 1066-5099, 11/2016, Volume 34, Issue 11, pp. 2613 - 2624
The network of stemness genes and oncogenes in human patient‐specific reprogrammed cancer stem cells (CSCs) remains elusive, especially in liver cancer.... 
OCT4 | c‐JUN | Liver cancer | Reprogramming | Cancer stem cells | Positive feedback | Pluripotency | c-JUN | CELLS | DEFINED FACTORS | OVARIAN-CANCER | CELL & TISSUE ENGINEERING | CELL BIOLOGY | BREAST-CANCER | HEPATOCELLULAR-CARCINOMA | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | IN-VIVO | EPIGENETIC REGULATION | DIMERIZATION PROTEIN-2 | HEMATOLOGY | EXPRESSION | Neoplastic Stem Cells - drug effects | Humans | Middle Aged | Transcriptional Activation | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Cellular Reprogramming | Octamer Transcription Factor-3 - genetics | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Female | Liver Neoplasms - pathology | Antineoplastic Agents - pharmacology | Cell Differentiation | Spheroids, Cellular - drug effects | JNK Mitogen-Activated Protein Kinases - genetics | Induced Pluripotent Stem Cells - metabolism | Induced Pluripotent Stem Cells - pathology | Induced Pluripotent Stem Cells - drug effects | Liver Neoplasms - genetics | Signal Transduction | Spheroids, Cellular - metabolism | Liver Neoplasms - drug therapy | Cisplatin - pharmacology | Mice, SCID | Hep G2 Cells | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Feedback, Physiological | Animals | Tumor Burden - drug effects | Octamer Transcription Factor-3 - metabolism | Liver Neoplasms - metabolism | Aged | Fluorouracil - pharmacology | Mice | Doxorubicin - pharmacology | Development and progression | Stem cells | Transcription factors | Liver | Oct-4 protein | c-Myc protein | Chemoresistance | Stem cell transplantation | Transplantation | Feedback loops | Myc protein | Allografts | Cell fate | Feedback | Xenografts | Tumorigenesis | Tumor markers | Colonies | c-Jun protein | Gene expression | Hepatocytes | Tumors | Cancer
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 2010, Volume 246, Issue 1, pp. 8 - 17
Acetaminophen (APAP) overdose, which causes liver injury in animals and humans, activates c-jun N-terminal kinase (JNK). Although it was shown that the JNK... 
Mitochondria | Acetaminophen | JNK | Bax | Hepatotoxicity | Oxidant stress | SP600125 | BAX TRANSLOCATION | CELL INJURY | INDUCED LIVER-INJURY | TUMOR-NECROSIS-FACTOR | MITOCHONDRIAL PERMEABILITY TRANSITION | CULTURED MOUSE HEPATOCYTES | ONCOTIC NECROSIS | PROTECTIVE ROLE | IN-VIVO | PHARMACOLOGY & PHARMACY | TOXICOLOGY | NH2-TERMINAL KINASE | Liver - pathology | Phosphorylation | Liver - enzymology | Apoptosis - drug effects | Analgesics, Non-Narcotic - pharmacology | Nitric Oxide Synthase - drug effects | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Alanine Transaminase - blood | Analgesics, Non-Narcotic - toxicity | Liver - drug effects | Acetaminophen - pharmacology | Enzyme Induction - drug effects | Nitric Oxide Synthase - biosynthesis | JNK Mitogen-Activated Protein Kinases - drug effects | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Subcellular Fractions - drug effects | Acetaminophen - toxicity | Mice, Inbred C57BL | bcl-2-Associated X Protein - metabolism | Enzyme Activation - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Anthracenes - pharmacology | Blotting, Western | Mitochondria, Liver - drug effects | Peroxynitrous Acid - biosynthesis | Animals | JNK Mitogen-Activated Protein Kinases - physiology | Mice | Oxidative Stress - drug effects | Translocation, Genetic - genetics | Messenger RNA | Glutathione transferase | Nitric oxide | Mitochondrial DNA | Superoxide | Permeability | Index Medicus | OXYGEN COMPOUNDS | NITRIC OXIDE | DIGESTIVE SYSTEM | MITOCHONDRIA | 60 APPLIED LIFE SCIENCES | STRESSES | PHOSPHORUS-GROUP TRANSFERASES | GLANDS | OXIDIZERS | NITROGEN OXIDES | ENZYMES | DRUGS | VERTEBRATES | MICE | NITROGEN COMPOUNDS | RADIOPROTECTIVE SUBSTANCES | MAMMALS | ANIMALS | INJURIES | RODENTS | RESPONSE MODIFYING FACTORS | ORGANIC COMPOUNDS | TRANSFERASES | NITRITES | POLYPEPTIDES | CHALCOGENIDES | ORGANS | DISEASES | PEPTIDES | GLUTATHIONE | LIVER | PHOSPHOTRANSFERASES | OXIDES | PROTEINS | BODY | CELL CONSTITUENTS | Oxidant Stress
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2004, Volume 24, Issue 36, pp. 7879 - 7887
The c-Jun N-terminal protein kinase (JNK) signaling pathway is implicated in neuronal apoptosis. The mechanism by which activated JNK induces neuronal... 
Bax | c-Jun N-terminal kinase | Cerebral ischemia | Bim | Apoptosis | SP600125 | HIPPOCAMPAL CA1 NEURONS | BCL-2 FAMILY MEMBERS | apoptosis | TRANSDUCTION | DEPENDENT APOPTOSIS | INDUCE APOPTOSIS | RELEASE | NEUROSCIENCES | CELL-DEATH | CYTOCHROME-C | JNK PHOSPHORYLATION | cerebral ischemia | BH3-ONLY PROTEINS | Neurons - pathology | Phosphorylation | Proto-Oncogene Proteins c-bcl-2 - physiology | Mitogen-Activated Protein Kinase Kinases - physiology | Male | MAP Kinase Signaling System | MAP Kinase Kinase 4 | Bcl-2-Like Protein 11 | Membrane Proteins - physiology | Neurons - metabolism | Protein-Serine-Threonine Kinases - metabolism | Carrier Proteins - physiology | Dyneins - metabolism | Proto-Oncogene Proteins - metabolism | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Molecular Motor Proteins | Rats | Mitochondria - metabolism | Infarction, Middle Cerebral Artery - pathology | bcl-2-Associated X Protein | Rats, Sprague-Dawley | Anthracenes - pharmacology | Protein Interaction Mapping | Protein Transport | Proto-Oncogene Proteins c-akt | Animals | Apoptosis Regulatory Proteins | JNK Mitogen-Activated Protein Kinases - physiology | Proto-Oncogene Proteins - physiology | Signal Transduction - physiology | Protein Processing, Post-Translational | Apoptosis - physiology | Neurobiology of Disease
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2005, Volume 25, Issue 9, pp. 1831 - 1836
Journal Article
TUMOR BIOLOGY, ISSN 1010-4283, 07/2015, Volume 36, Issue 7, pp. 5249 - 5254
Identification of the key molecules that mediate susceptibility to anticancer treatments would be highly desirable. Based on clinical and cell biological... 
SURVIVAL | Radiosensitivity | PROTEIN | PHOSPHORYLATION | JNK | ALPHA | MAPK | c-Jun | GENE | ONCOLOGY | DIFFERENTIATION | DEFINITIVE CHEMORADIOTHERAPY | EXPRESSION | CARCINOMA | REG I | ERK | KINASES
Journal Article
Pulmonary Pharmacology & Therapeutics, ISSN 1094-5539, 2015, Volume 32, pp. 29 - 36
Journal Article
Journal Article
Tumor Biology, ISSN 1010-4283, 07/2015, Volume 36, Issue 7, p. 5249
Identification of the key molecules that mediate susceptibility to anticancer treatments would be highly desirable. Based on clinical and cell biological... 
Studies | Proteins | Cellular biology | Kinases | Esophageal cancer
Journal Article