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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2001, Volume 98, Issue 24, pp. 13681 - 13686
Jun N-terminal kinase (JNK) is a stress-activated protein kinase that can be induced by inflammatory cytokines, bacterial endotoxin, osmotic shock, UV... 
T lymphocytes | Enzymes | Biological Sciences | Phosphorylation | Cell growth | Nervous system diseases | Messenger RNA | Cytokines | Thymocytes | Inhibitory concentration 50 | Apoptosis | SIGNAL-TRANSDUCTION | TUMOR-NECROSIS-FACTOR | MESSENGER-RNA | ANTICANCER AGENTS | MULTIDISCIPLINARY SCIENCES | C-JUN | CELL-DIFFERENTIATION | TRANSCRIPTION FACTOR | EXPRESSION | AP-1 | T-CELLS | Gene Expression - drug effects | Monocytes - cytology | Humans | Monocytes - metabolism | Structure-Activity Relationship | Pyrazolones | CD4-Positive T-Lymphocytes - immunology | Adenosine Triphosphate - metabolism | Enzyme Inhibitors - chemistry | JNK Mitogen-Activated Protein Kinases | Female | Molecular Structure | Binding, Competitive | Cytokines - metabolism | Enzyme Inhibitors - metabolism | Anthracenes - chemistry | Jurkat Cells | Pyrazoles | CD4-Positive T-Lymphocytes - cytology | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Anthracenes - pharmacology | Protein Kinase Inhibitors | Animals | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Anthraquinones | Cell Differentiation - drug effects | Lymphocyte Activation - drug effects | Anthracenes - metabolism | Mice | Tumor Necrosis Factor-alpha - biosynthesis | CD4-Positive T-Lymphocytes - drug effects | Physiological aspects | Protein kinases | Enzyme inhibitors
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 1996, Volume 271, Issue 22, pp. 12762 - 12766
Aggregation of the high-affinity Fc receptors for immunoglobulin E (IgE) (Fc epsilon RI) on the surface of mast cells initiates intracellular signal... 
ACTIVATION DOMAIN | AFFINITY IGE RECEPTOR | RBL-2H3 CELLS | TYROSINE PHOSPHORYLATION | PROTEIN-KINASE | PHOSPHATIDYLINOSITOL 3-KINASE | MAP KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSFECTED CELLS | ANTIGEN RECEPTOR | SIGNAL TRANSDUCTION
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 1/2013, Volume 33, Issue 1, pp. 106 - 114
Rosiglitazone, a synthetic peroxisome proliferator-activated receptor-γ (PPARγ) agonist, prevents cell death after cerebral ischemia in animal models, but the... 
c-Jun N-terminal kinase | rosiglitazone | dual-specificity phosphatase 8 | p38 mitogen-activated protein kinase | cerebral ischemia | RECEPTOR-GAMMA | BRAIN-INJURY | N-TERMINAL KINASE | PROTEIN-TYROSINE PHOSPHATASES | RATS | REPERFUSION | EXPERIMENTAL STROKE | NEUROSCIENCES | INHIBITION | SIGNALING PATHWAY | ENDOCRINOLOGY & METABOLISM | HEMATOLOGY | ISCHEMIA/REPERFUSION | Neuroprotective Agents - therapeutic use | Neurons - pathology | Phosphorylation | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Ischemic Attack, Transient - enzymology | Thiazolidinediones - administration & dosage | Oxygen - metabolism | Thiazolidinediones - therapeutic use | Neuroprotective Agents - pharmacology | Cell Death | Neurons - drug effects | Neuroprotective Agents - administration & dosage | Thiazolidinediones - pharmacology | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Ischemic Attack, Transient - pathology | Ischemic Attack, Transient - drug therapy | Dual-Specificity Phosphatases - metabolism | Mice, Inbred C57BL | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Animals | PPAR gamma - antagonists & inhibitors | Neurons - enzymology | PPAR gamma - agonists | Glucose - metabolism | Mice | Enzyme Activation | Brain | Neuroprotection | Animal models | Oxygen | Neurons | Cortex | MAP kinase | mRNA | Glucose | Western blotting | MAP kinase phosphatase | Reperfusion | Ischemia | c-Jun amino-terminal kinase | Cell death | Cerebral blood flow | Peroxisome proliferator-activated receptors | Original
Journal Article
Journal Article
Journal Article
Neurogastroenterology & Motility, ISSN 1350-1925, 06/2015, Volume 27, Issue 6, pp. 875 - 884
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 01/2018, Volume 71, Issue 4, pp. 414 - 425
Recent studies suggested that perivascular components, such as perivascular adipose tissue (PVAT) and adventitial vasa vasorum (VV), play an important role as... 
coronary adventitia | perivascular adipose tissue | Rho-kinase | coronary spasm | cardiac CT | FDG PET | PLAQUE INFLAMMATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ARTERY SPASM | HYPERCONSTRICTING RESPONSES | MULTICENTER REGISTRY | PIGS IN-VIVO | VASA VASORUM FORMATION | VASOMOTION ABNORMALITIES | ELUTING STENT IMPLANTATION | SPASM ASSOCIATION | Prospective Studies | Panniculitis - diagnostic imaging | Humans | Middle Aged | Adventitia - diagnostic imaging | Coronary Vasospasm - diagnostic imaging | Male | Angina Pectoris - etiology | Panniculitis - complications | Case-Control Studies | Positron Emission Tomography Computed Tomography | Coronary Angiography | Coronary Vasospasm - complications | Angina Pectoris - diagnostic imaging | Female | Fluorodeoxyglucose F18 | Aged | Adipose Tissue | Radiopharmaceuticals | Heart | Adipose tissue | Angiography | Pathogenesis | Emission analysis | Medical services | Cardiovascular disease | Leukocytes | Angina | Computed tomography | Tomography | Lesions | Stents | EKG | Medical imaging | Cytokines | Medical treatment | Coronary artery | Positron emission | Inflammation | Angina pectoris | Patients | Rho-associated kinase | White blood cells | Optical Coherence Tomography | Coronary vessels | Acetylcholine | Acute coronary syndromes | Cardiovascular diseases | Positron emission tomography
Journal Article
Journal Article