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2016, Second edition., ISBN 0128011343
Web Resource
Nature, ISSN 0028-0836, 11/2012, Volume 491, Issue 7422, pp. 66 - 71
Journal Article
2010, 1st ed., ISBN 012374430X, x, 323 p., [8] p. of plates
Since the beginning of the current century, endocrine disease diagnosis and treatment have moved beyond the standard hormone measurements. While, indeed... 
Endocrine glands | Pre-clinical Medicine: Basic Sciences | Genetic aspects | Treatment | Diagnosis | Diseases
Book
2010, 1st ed., ISBN 012374430X
Since the beginning of the current century, endocrine disease diagnosis and treatment have moved beyond the standard hormone measurements. While, indeed... 
Treatment | Endocrine glands | Genetic aspects | Diagnosis | Diseases
Web Resource
Journal of Clinical Investigation, ISSN 0021-9738, 12/2011, Volume 121, Issue 12, pp. 4700 - 4711
Advanced human thyroid cancers, particularly those that are refractory to treatment with radioiodine (RAI), have a high prevalence of BRAF (v-raf murine... 
MEDICINE, RESEARCH & EXPERIMENTAL | GROWTH-INHIBITION | BRAF(V600E) MUTATION | CARCINOMA CELLS | GENE | RAS | ANTISENSE RNA | KINASE | SENSITIVITY | PROLIFERATION | EXPRESSION | MAP Kinase Signaling System - physiology | Proto-Oncogene Proteins B-raf - physiology | Apoptosis - drug effects | Humans | Antineoplastic Agents - therapeutic use | Mutation, Missense | Indoles - administration & dosage | Benzamides - administration & dosage | Neoplasm Proteins - genetics | MAP Kinase Kinase 1 - antagonists & inhibitors | Benzamides - toxicity | Diphenylamine - therapeutic use | Carcinoma, Papillary - drug therapy | Mice, Transgenic | Diphenylamine - toxicity | Sulfonamides - pharmacology | Antineoplastic Combined Chemotherapy Protocols - toxicity | Protein Kinase Inhibitors - administration & dosage | Indoles - therapeutic use | Mice | DNA Damage | Thyroid Gland - metabolism | Sulfonamides - administration & dosage | Thyroid Neoplasms - metabolism | Carcinoma, Papillary - genetics | Diphenylamine - pharmacology | Neoplasm Proteins - physiology | Carcinoma, Papillary - metabolism | Diphenylamine - analogs & derivatives | Benzamides - therapeutic use | Indoles - pharmacology | Benzamides - pharmacology | Diphenylamine - administration & dosage | Genes, Synthetic - drug effects | Indoles - toxicity | Enzyme Activation - drug effects | Iodine Radioisotopes - pharmacokinetics | Carcinoma, Papillary - pathology | Point Mutation | Thyroid Neoplasms - genetics | Animals | MAP Kinase Signaling System - drug effects | Sulfonamides - therapeutic use | Thyroid Neoplasms - drug therapy | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Proto-Oncogene Proteins B-raf - genetics | Protein Kinase Inhibitors - toxicity | Sulfonamides - toxicity | Protein Kinase Inhibitors - pharmacology | Doxorubicin - pharmacology | Drug Screening Assays, Antitumor | Thyroid Neoplasms - pathology | Care and treatment | Thyroid cancer | Cancer cells | Genetic aspects | Diagnosis | Properties | Isotopes | Health aspects | Mitogen-activated protein kinases | Iodine | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2011, Volume 108, Issue 4, pp. 1615 - 1620
Mutations of BRAF are found in ~45% of papillary thyroid cancers and are enriched in tumors with more aggressive properties. We developed mice with a... 
Cell growth | Thyroid cancer | Papillary carcinoma | Cholangiography | Developmental biology | Cell lines | Genetic mutation | Papillae | Oncogenes | Tumors | Pax8 | Ras | G protein | pax8 | MULTIDISCIPLINARY SCIENCES | DEVELOPMENTAL DEFECTS | CRE RECOMBINASE | DNA-SYNTHESIS | CANCER | BRAF(V600E) MUTATION | RAS MUTATIONS | HIGH PREVALENCE | TARGETED EXPRESSION | GROWTH-FACTOR-I | TRANSGENIC MICE | Immunohistochemistry | Receptors, Thyrotropin - genetics | Carcinoma | Humans | Thyroxine - blood | Ki-67 Antigen - metabolism | Male | Radioimmunoassay | Iodide Peroxidase - metabolism | Female | Proto-Oncogene Proteins B-raf - metabolism | Thyrotropin - blood | Gene Expression | Signal Transduction | Receptors, Thyrotropin - metabolism | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Thyrotropin - metabolism | Mice, Knockout | Thyroid Cancer, Papillary | Thyroid Neoplasms - genetics | Animals | Carcinoma, Papillary | Proto-Oncogene Proteins B-raf - genetics | Thyroid Gland - pathology | Thyroxine - metabolism | Mice | Iodide Peroxidase - genetics | Thyroid Gland - metabolism | Thyroid Neoplasms - metabolism | Thyroid Neoplasms - pathology | Physiological aspects | Care and treatment | Genetic aspects | G proteins | Rodents | Thyroid diseases | Growth hormones | Oncology | Models | Biosynthesis | Mutation | Gene expression | Index Medicus | Biological Sciences
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 10/2017, Volume 102, Issue 10, pp. 3775 - 3782
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2017, Volume 127, Issue 12, pp. 4326 - 4337
Deficiency in Kruppel-like zinc finger transcription factor GLI-similar 3 (GLIS3) in humans is associated with the development of congenital hypothyroidism.... 
MEDICINE, RESEARCH & EXPERIMENTAL | SODIUM/IODIDE SYMPORTER | NEONATAL DIABETES-MELLITUS | CYCLIC-AMP | MICE DEFICIENT | CONGENITAL HYPOTHYROIDISM | ZINC-FINGER | IN-VIVO | GENE-EXPRESSION | TRANSCRIPTION FACTOR | MTOR | Receptors, Thyrotropin - genetics | Thyrotropin - genetics | Cell Proliferation | Ribosomal Proteins - metabolism | Goiter - genetics | Mechanistic Target of Rapamycin Complex 1 - genetics | Trans-Activators - genetics | Goiter - metabolism | Thyroid Gland - cytology | Repressor Proteins - metabolism | Sulfate Transporters - metabolism | Sulfate Transporters - genetics | Promoter Regions, Genetic | Thyroid Hormones - biosynthesis | Receptors, Thyrotropin - metabolism | Ribosomal Proteins - genetics | Repressor Proteins - genetics | Goiter - prevention & control | Thyroid Hormones - genetics | Thyrotropin - metabolism | Mechanistic Target of Rapamycin Complex 1 - metabolism | Symporters - metabolism | Mice, Knockout | Animals | Symporters - genetics | Trans-Activators - metabolism | Mice | Thyroid Gland - metabolism | Cell proliferation | Analysis | Physiological aspects | Development and progression | Genetic aspects | Thyroid hormones | Hypothyroidism | Health aspects | TOR protein | Neonates | Animal models | Transcription factors | Biosynthesis | Thyroid gland | Kinases | Proteins | Cell growth | Goiter | Rodents | Transcription activation | Zinc finger proteins | Thyroid | Nutrient deficiency | Congenital diseases | Cell division | Ribosomal protein S6 | Inflammation | Gene expression | Thyroid-stimulating hormone receptors | Thyroid-stimulating hormone | Mutation | Index Medicus | Abridged Index Medicus
Journal Article
18.