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Bulletin de l'Academie Nationale de Medecine, ISSN 0001-4079, 04/2017, Volume 201, Issue 4-6, pp. 775 - 784
Journal Article
Circulation, ISSN 0009-7322, 11/2016, Volume 134, Issue Suppl_1 Suppl 1, pp. A15123 - A15123
IntroductionPreliminary results from our group suggested a protective role of cardiac QSOX1 induction during acute cardiac stress. QSOX1, a sulfhydryl oxidase,... 
Journal Article
Circulation, ISSN 0009-7322, 11/2016, Volume 134, Issue Suppl_1 Suppl 1, pp. A14069 - A14069
IntroductionQSOX1, a sulfhydryl oxidase involved in the protein folding, is upregulated in acute heart failure.HypothesisWe hypothesized that QSOX1 could play... 
Journal Article
Circulation, ISSN 0009-7322, 11/2016, Volume 134, Issue Suppl_1 Suppl 1, pp. A19794 - A19794
IntroductionThe Notch3 receptor plays an important role in differentiation, migration and signal transduction of vascular smooth muscle cells (VSMCs). We... 
Journal Article
Nature Reviews Cardiology, ISSN 1759-5002, 03/2010, Volume 7, Issue 3, pp. 125 - 127
Journal Article
Nature Reviews Cardiology, ISSN 1759-5002, 03/2010, Volume 7, Issue 3, p. 125
Journal Article
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 06/2018, Volume 119, pp. 75 - 86
QSOX1, a sulfhydryl oxidase, was shown to be upregulated in the heart upon acute heart failure (AHF). The aim of the study was to unravel QSOX1 roles during... 
Endoplasmic reticulum oxidases | QSOX1 | Dilated cardiomyopathy | Isoproterenol | Acute heart failure | Endoplasmic reticulum stress | Oxidases | Heart failure | Proteins | Heart | Actors | Actresses
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 02/2014, Volume 171, Issue 3, pp. 555 - 566
Journal Article
Hypertension, ISSN 0194-911X, 06/2012, Volume 59, Issue 6, pp. 1179 - 1187
The renin-angiotensin-aldosterone system is involved in the arterial hypertension-associated cardiovascular remodeling. In this context, the development of... 
BMP4 | Hypertension | Cardiac fibrosis | BNP | Inflammation | Aldosterone | Angiotensin II | FIBROSIS | BRAIN NATRIURETIC PEPTIDE | ACTIVATED PROTEIN-KINASE | MYOCARDIAL-INFARCTION | RAT | aldosterone | ANGIOTENSIN-II | FAILURE | CARDIAC FIBROBLASTS | cardiac fibrosis | inflammation | angiotensin II | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | hypertension | TRANSGENIC MICE | Blood Pressure | Renin - metabolism | Gene Expression - drug effects | Bone Morphogenetic Protein 4 - genetics | Natriuretic Peptide, Brain - metabolism | Galectin 3 - metabolism | Male | Hyperaldosteronism - metabolism | Antigens, CD - genetics | Bone Morphogenetic Protein 4 - metabolism | Antigens, CD - metabolism | Aldosterone - metabolism | Renin - genetics | Spironolactone - pharmacology | Hyperaldosteronism - physiopathology | Myocardium - metabolism | Mineralocorticoid Receptor Antagonists - pharmacology | Female | Hypertension - genetics | Spironolactone - analogs & derivatives | Cytochrome P-450 CYP11B2 - metabolism | Animals, Newborn | Natriuretic Peptide, Brain - genetics | Cells, Cultured | Hyperaldosteronism - genetics | Organ Size | Aldosterone - pharmacology | Mice, Transgenic | Myocardium - pathology | Galectin 3 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Antigens, Differentiation, Myelomonocytic - genetics | Blotting, Western | Hypertension - physiopathology | Hypertension - metabolism | Animals | Antigens, Differentiation, Myelomonocytic - metabolism | Fibrosis | Mice | Cytochrome P-450 CYP11B2 - genetics | Index Medicus
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 02/2017, Volume 24, Issue 2, pp. 343 - 356
Over the past decade, endoplasmic reticulum (ER) stress has emerged as an important mechanism involved in the pathogenesis of cardiovascular diseases including... 
APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CARDIAC MYOCYTES | CARDIOMYOPATHY | DISEASE | MYOCARDIAL INJURY | ENDOPLASMIC-RETICULUM STRESS | DYSFUNCTION | SIRTUINS | ISOPROTERENOL | ISCHEMIA/REPERFUSION | CELL BIOLOGY | Index Medicus | Original Paper
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2015, Volume 10, Issue 10, pp. e0139858 - e0139858
Myocardial fibrosis contributes to the remodeling of heart and the loss of cardiac function leading to heart failure. SRF is a transcription factor implicated... 
TGF-BETA | MYOCARDIAL FIBROSIS | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | SERUM RESPONSE FACTOR | GENE-EXPRESSION | ACTIN DYNAMICS | DILATED CARDIOMYOPATHY | MATRIX-METALLOPROTEINASE ACTIVITY | MYOFIBROBLAST DIFFERENTIATION | TRANSGENIC MICE | Serum Response Factor - genetics | Cell Line | Heart Diseases - metabolism | Serum Response Factor - metabolism | Extracellular Matrix Proteins - genetics | Gene Expression Regulation | Rats | Mice, Transgenic | MicroRNAs - metabolism | Myocardium - pathology | Fibrosis - metabolism | Animals | Myocardium - metabolism | Myocytes, Cardiac - metabolism | Connective Tissue Growth Factor - genetics | Mice | MicroRNAs - genetics | Fibrosis - pathology | Connective Tissue Growth Factor - metabolism | Extracellular Matrix Proteins - metabolism | Heart Diseases - pathology | Physiological aspects | Transcription factors | Genetic aspects | MicroRNA | Research | Fibrosis | Heart | Cardiomyopathy | Genes | Biology | Experiments | Fibronectin | Proteins | Connective tissues | Cell growth | Transgenic animals | Rodents | Fibroblasts | Aging | Extracellular matrix | Heart diseases | Growth factors | VP16 protein | Heart failure | Hypertension | Procollagen | MiRNA | Connective tissue growth factor | Cardiomyocytes | Tamoxifen | Gene expression | Ribonucleic acid--RNA | Low level | MicroRNAs | Structure-function relationships | Index Medicus | Life Sciences | Human health and pathology | Cardiology and cardiovascular system | RNA | Ribonucleic acid
Journal Article
Circulation, ISSN 0009-7322, 11/2015, Volume 132, Issue Suppl_3 Suppl 3, pp. A13993 - A13993
IntroductionCD146 is a component of the endothelial junction. We previously showed that its soluble form (sCD146) is as powerful as plasma natriuretic peptides... 
Journal Article
Conference Proceeding
PLoS ONE, ISSN 1932-6203, 11/2015, Volume 10, Issue 11, pp. e0142237 - e0142237
Journal Article