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PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 9, p. e73426
Glioblastoma (GBM) is the most common and aggressive form of brain tumor, characterized by high migratory behavior and infiltration in brain parenchyma which... 
PATHWAYS | ACTIVATION | ANGIOGENESIS | TRANSACTIVATION | MULTIDISCIPLINARY SCIENCES | EPIDERMAL-GROWTH-FACTOR | PROLIFERATION | HYPOXIA | FACTOR RECEPTOR | EXPRESSION | FACTOR-ALPHA | Glioblastoma - pathology | Humans | Cell Line, Tumor | Receptors, CXCR4 - genetics | Tyrphostins - pharmacology | Chemotaxis - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Receptor, Platelet-Derived Growth Factor beta - genetics | Receptor Cross-Talk - drug effects | Receptor, Platelet-Derived Growth Factor beta - metabolism | Receptors, CXCR4 - metabolism | Platelet-derived growth factor | Physiological aspects | Development and progression | Genetic aspects | Research | Glioblastoma multiforme | Chemokine receptors | Brain | CXCL12 protein | Phosphorylation | Brain tumors | Brain cancer | Crosstalk | Glioblastoma | Parenchyma | Smooth muscle | Biology | Kinases | Metastases | Cell adhesion & migration | Receptors | Epidermal growth factor | Conditioning | Growth factors | Tyrosine | Aggressive behavior | Cytokines | Growth factor receptors | Chemotaxis | CXCR4 protein | Signaling | Molecular modelling | Glioma | Glioblastoma cells | Infiltration | Cell migration | Life Sciences | Receptor Cross-Talk | Neurons and Cognition | Gene Expression Regulation, Neoplastic | Receptor, Platelet-Derived Growth Factor beta | Tyrphostins | Receptors, CXCR4
Journal Article
The Journal of Nutritional Biochemistry, ISSN 0955-2863, 01/2014, Volume 25, Issue 1, pp. 81 - 90
Insulin resistance (IR) has been identified as crucial pathophysiological factor in the development and progression of non-alcoholic fatty liver disease... 
Glucose tolerance | Gut permeability | Insulin resistance | Inflammation | Non-alcoholic fatty liver disease | Toll-like receptor | INTESTINAL MICROBIOTA | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROBIOTICS | ALPHA | NONALCOHOLIC STEATOHEPATITIS | GUT MICROBIOTA | LIVER-DISEASE | NUTRITION & DIETETICS | RESISTANCE | PPAR-GAMMA | MICE | Toll-Like Receptor 2 - genetics | Glucose Transporter Type 4 - metabolism | Toll-Like Receptor 9 - genetics | Fatty Liver - therapy | Galactans - pharmacology | Male | Insulin Receptor Substrate Proteins - metabolism | PPAR gamma - metabolism | RNA, Messenger - metabolism | Intestinal Mucosa - drug effects | Synbiotics | Oligosaccharides - pharmacology | Adipose Tissue - metabolism | Liver - drug effects | Adiponectin - genetics | Diet, High-Fat | Adiponectin - metabolism | Insulin Receptor Substrate Proteins - genetics | Lactobacillus | PPAR gamma - genetics | Glucose Transporter Type 4 - genetics | Signal Transduction | Down-Regulation | Liver - metabolism | RNA, Messenger - genetics | Insulin Resistance | Rats | Suppressor of Cytokine Signaling Proteins - genetics | Toll-Like Receptor 4 - genetics | Intestinal Mucosa - microbiology | Toll-Like Receptor 2 - metabolism | Rats, Sprague-Dawley | Toll-Like Receptor 4 - metabolism | Suppressor of Cytokine Signaling 3 Protein | Animals | Enterobacteriaceae - drug effects | Suppressor of Cytokine Signaling Proteins - metabolism | Toll-Like Receptor 9 - metabolism
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 11/2018, Volume 9, p. 2750
Microglia are patrolling cells that sense changes in the brain microenvironment and respond acquiring distinct phenotypes that can be either beneficial or... 
Neuroinflammation | CXCR6 | Glioma | Tumor microenvironment | CXCL16 | Microglia | TUMOR PROGRESSION | TRANSMEMBRANE CHEMOKINE CXCL16 | CELL INVASION | RECEPTOR | IMMUNOLOGY | OSTEOPONTIN | neuroinflammation | GROWTH | NEURONS | glioma | tumor microenvironment | microglia | EXPRESSION | Phenotype | Gliomas | Development and progression | Cellular signal transduction | Research | Macrophages | Gene expression
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, p. e0171276
Osteoporosis is a metabolic skeletal disease characterized by an imbalance between osteoclast-mediated bone resorption and osteoblast-mediated bone formation.... 
MINERAL DENSITY | THERAPY | ADIPONECTIN | CATHEPSIN-K | MULTIDISCIPLINARY SCIENCES | ESTROGEN | FRACTURES | BODY-COMPOSITION | DIFFERENTIATION | POSTMENOPAUSAL WOMEN | OSTEOPOROSIS | Bone and Bones - pathology | Ovariectomy - adverse effects | Raloxifene Hydrochloride - pharmacology | Osteoporosis - drug therapy | High-Energy Shock Waves | PPAR gamma - metabolism | Osteoporosis - etiology | Bone and Bones - drug effects | Bone Resorption - genetics | Adiponectin - genetics | Bone and Bones - metabolism | Bone Resorption - metabolism | Adiponectin - metabolism | Female | Transcription, Genetic | Bone and Bones - radiation effects | Osteogenesis - genetics | Disease Models, Animal | Bone Resorption - etiology | Osteogenesis - drug effects | Rats | Osteogenesis - radiation effects | Bone Density Conservation Agents - pharmacology | Bone Resorption - prevention & control | Animals | Osteoporosis - pathology | Biomarkers | Shock waves | Usage | Growth | Bones | Raloxifene | Dosage and administration | Research | Drug therapy | Health aspects | Osteopenia | Alkaline phosphatase | Neurosciences | Femur | Mesenchyme | Spine | Estrogen receptors | Veterinary medicine | Bone surgery | Adiponectin | Osteoprotegerin | Bone resorption | Osteoporosis | Biomedical materials | Bone growth | Rodents | Surgery | Computer architecture | Bone marrow | Biocompatibility | Bone loss | Ultrasound | Public health | Vertebrae | Cathepsin K | Ovariectomy | Bone remodelling | Metabolism | Gene expression | Osteoblastogenesis | Tumor necrosis factor | Parathyroid hormone | Womens health | Pharmacy | Weight reduction | Stem cells | Bone mineral density | Clinical medicine | Synergistic effect | Osteogenesis
Journal Article
Oncotarget, ISSN 1949-2553, 05/2016, Volume 7, Issue 21, pp. 30781 - 30796
Malignant gliomas are among the most frequent and aggressive cerebral tumors, characterized by high proliferative and invasive indexes. Standard therapy for... 
Malignant glioma | channels | Migration | Cell cycle | Apoptosis | activated K | SURVIVAL | cell cycle | PHASE-III | TUMOR | apoptosis | CA2+-ACTIVATED K+ CHANNEL | MODEL | malignant glioma | CANCER | CELL BIOLOGY | ARREST | GLIOBLASTOMA | migration | CELL-CYCLE | ION CHANNELS | Ca2+ activated K+ channels | Microglia - metabolism | Phosphorylation | Pyrazoles - therapeutic use | Intermediate-Conductance Calcium-Activated Potassium Channels - genetics | Apoptosis - drug effects | Dacarbazine - therapeutic use | Neoplastic Stem Cells - drug effects | Humans | CDC2 Protein Kinase - metabolism | G2 Phase Cell Cycle Checkpoints - drug effects | Dacarbazine - pharmacology | Dacarbazine - analogs & derivatives | Intermediate-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Brain Neoplasms - mortality | Neoplasms, Experimental - mortality | Drug Therapy, Combination | Pyrazoles - pharmacology | Cell Survival - drug effects | Glioma - mortality | Microglia - drug effects | Mice, Inbred C57BL | Kaplan-Meier Estimate | Brain Neoplasms - drug therapy | Antineoplastic Agents, Alkylating - therapeutic use | Drug Synergism | Cell Movement - drug effects | Animals | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Primary Cell Culture | Neoplasms, Experimental - drug therapy | Glioma - drug therapy
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 06/2017, Volume 174, Issue 11, pp. 1484 - 1496
Background and Purpose Butyrate has shown benefits in inflammatory bowel diseases. However, it is not often administered orally because of its rancid smell and... 
INTESTINAL INFLAMMATION | MONOCARBOXYLATE TRANSPORTER | CONCISE GUIDE | CROHNS-DISEASE | SUBSTRATE-INDUCED REGULATION | METABOLITE BUTYRATE | PHARMACOLOGY & PHARMACY | DISTAL ULCERATIVE-COLITIS | NUCLEAR HORMONE-RECEPTORS | COLONIC-MUCOSA | BOWEL-DISEASE | Inflammation - pathology | NF-kappa B - antagonists & inhibitors | Administration, Oral | Anti-Inflammatory Agents - pharmacology | Butyrates - pharmacology | Colitis - pathology | Male | Dextran Sulfate - toxicity | Up-Regulation - drug effects | Animals | Time Factors | Inflammation - drug therapy | Anti-Inflammatory Agents - administration & dosage | Butyrates - administration & dosage | Colitis - drug therapy | Mice | Mice, Inbred BALB C | Colitis - prevention & control | Neutrophil Infiltration - drug effects | Disease Models, Animal | PPAR gamma - genetics | Dextran | Esters | Colitis | Transforming growth factors | Proteases | Gastrointestinal diseases | Drinking water | Histone deacetylase | Inflammatory bowel diseases | Sodium sulfates | Recruitment | Interleukin 6 | Intestine | Rodents | Histones | Acetylation | Food quality | Colon | Inhibition | Localization | Nutraceuticals | NF-κB protein | Signs and symptoms | Cytokines | Neutrophils | Oral administration | Nitric-oxide synthase | Smell | Inflammatory bowel disease | Sodium butyrate | Interleukin 10 | Olfaction | Peroxisome proliferator-activated receptors | Ly-6 antigen | Transporter | Monocyte chemoattractant protein 1 | Rancidity | Themed Section | Research Papers | Research Paper
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e68626
Background & Aims: Nonalcoholic fatty liver disease (NAFLD) is the most common form of chronic liver disease. Insulin resistance (IR) appears to be critical in... 
INHIBITION | METABOLISM | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | PPAR-GAMMA | ACTIVATED RECEPTOR-ALPHA | MICE | DYSFUNCTION | HEPATIC STEATOSIS | GROWTH-FACTOR 21 | Inflammation - pathology | Liver - pathology | Fatty Liver - pathology | Diet, High-Fat - adverse effects | Male | NF-kappa B - metabolism | Adipose Tissue - metabolism | Inflammation - metabolism | Liver - drug effects | Non-alcoholic Fatty Liver Disease | Butyric Acid - pharmacology | Toll-Like Receptors - metabolism | Disease Models, Animal | Amides - pharmacology | Fatty Liver - metabolism | Liver - metabolism | Insulin Resistance | Rats | Butyric Acid - administration & dosage | Enzyme Activation - drug effects | Fatty Liver - drug therapy | Animals | Glucose - metabolism | Adipose Tissue - drug effects | Amides - administration & dosage | Fatty Liver - etiology | Diet therapy | Liver diseases | Insulin resistance | Esters | Inflammation | Glucose | Insulin | Dextrose | Adipose tissue | Pathogenesis | Weaning | Liver | Homeostasis | Lipids | Kinases | Veterinary medicine | High fat diet | Proteins | Disease resistance | Receptors | Fatty liver | Rodents | Animal tissues | Toll-like receptors | Growth factors | NF-κB protein | Tumor necrosis factor-α | Gene expression | Fatty acids | Steatosis | Glucose tolerance | Sodium butyrate | Palatability | Diet | Sodium | Neural networks | Transaminases | Pharmacy | Diabetes | Metabolic disorders | Veins & arteries
Journal Article
Cancers, ISSN 2072-6694, 03/2019, Volume 11, Issue 3, p. 279
Glioblastoma (GBM) is a deadly brain tumor, with fast recurrence even after surgical intervention, radio- and chemotherapies. One of the reasons for relapse is... 
IL-4 | KCa3.1 | Invasion | Glioblastoma | Radiation | GLIOMA-CELLS | MIGRATION | ACTIVATE | TRANSCRIPTION | PROLIFERATION | CA2+-ACTIVATED K+ CHANNEL | IRRADIATION | invasion | ONCOLOGY | radiation | INVADOPODIA ACTIVITY | POTASSIUM CHANNEL
Journal Article