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Studies in the Age of Chaucer, ISSN 0190-2407, 2014, Volume 36, Issue 1, pp. 320 - 323
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 76 - 76
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, pp. e0164897 - e0164897
Nemo-like kinase (NLK) is an evolutionary conserved serine/threonine protein kinase implicated in development, proliferation and apoptosis regulation. Here we... 
DROSOPHILA NEMO | HYPERTROPHY | ACTIVATION | PROTEIN | CANCER CELLS | GENETIC INHIBITION | INDUCED APOPTOSIS | PATHWAY | MULTIDISCIPLINARY SCIENCES | IN-VIVO | EXPRESSION | Humans | Myosin Heavy Chains - genetics | Male | Cardiomyopathies - etiology | STAT1 Transcription Factor - metabolism | Myocardium - metabolism | HEK293 Cells | Female | Promoter Regions, Genetic | Echocardiography | Myocytes, Cardiac - cytology | Signal Transduction | Cells, Cultured | Rats | Mice, Transgenic | Myocardium - pathology | STAT1 Transcription Factor - genetics | Rats, Sprague-Dawley | Heart - diagnostic imaging | Mitogen-Activated Protein Kinases - deficiency | Animals | Myocytes, Cardiac - metabolism | Protein Binding | Mitogen-Activated Protein Kinases - genetics | Mice | Mitogen-Activated Protein Kinases - metabolism | Heart failure | Heart | Heart enlargement | Genes | Myosin | Genetic engineering | Muscle proteins | Protein kinases | Heart attack | Apoptosis | Myocardial infarction | Pediatrics | Animal models | Phosphorylation | Transcription factors | Chains | Kinases | Proteins | Clonal deletion | Transgenic animals | Evolutionary conservation | Rodents | Surgery | Deletion | Stat1 protein | Heart diseases | Threonine | Transgenic mice | Cardiomyocytes | Tamoxifen | Gene expression | NLK gene | Coronary artery disease | Signaling | Insects | Protein kinase | Fibrosis | Infarction | Protein-serine/threonine kinase | Prostate | Hypertrophy | Index Medicus
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 08/2017, Volume 70, Issue 8, p. 958
Cardiac fibroblasts are a critical cell population responsible for myocardial extracellular matrix homeostasis. Upon injury or pathological stimulation, these... 
Heart failure | Medical colleges | Development and progression | G proteins | Ischemia | Membrane proteins | Ablation (Surgery) | Analysis | Myocardial ischemia | Transforming growth factors
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 12/2014, Volume 371, Issue 26, pp. 2457 - 2466
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, pp. e0130520 - e0130520
Journal Article
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 08/2017, Volume 136, Issue 6, pp. 549 - 561
Journal Article