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Human Immunology, ISSN 0198-8859, 10/2012, Volume 73, p. 14
Journal Article
Human Immunology, ISSN 0198-8859, 2012, Volume 73, pp. 14 - 14
Aim HCV mediated liver diseases is a major health problem both in the United States and worldwide. This study is to identify the role of HCV induced changes in... 
Allergy and Immunology
Journal Article
Nucleus, ISSN 1949-1034, 11/2012, Volume 3, Issue 6, pp. 508 - 515
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e50826
Liver disease due to hepatitis C virus (HCV) infection is an important health problem worldwide. HCV induced changes in microRNAs (miRNA) are shown to mediate... 
TRANSCRIPTION FACTORS | FIBROSIS | C/EBP-ALPHA | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | CELL-CYCLE ARREST | INFECTION | MICRORNAS | HUMAN HEPATOCELLULAR-CARCINOMA | NF-KAPPA-B | EXPRESSION | Tumor Necrosis Factor-alpha - metabolism | Transcription, Genetic - drug effects | Humans | Middle Aged | Molecular Sequence Data | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | Hepatocytes - metabolism | Promoter Regions, Genetic - genetics | Chitinase-3-Like Protein 1 | Inflammation - complications | Lectins - metabolism | Adipokines - genetics | Base Sequence | Adult | Female | Hepacivirus - physiology | Hepatitis C - complications | Liver Cirrhosis - genetics | Hepatocytes - drug effects | Biomarkers - metabolism | CCAAT-Enhancer-Binding Proteins - metabolism | Liver Cirrhosis - complications | Hepatitis C - genetics | Up-Regulation - genetics | Adipokines - metabolism | Signal Transduction - genetics | Receptor, Notch1 - metabolism | Down-Regulation - drug effects | Mutation - genetics | Down-Regulation - genetics | Up-Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Signal Transduction - drug effects | Models, Biological | Hepatitis C - virology | Inflammation - genetics | Liver Cirrhosis - pathology | Lectins - genetics | MicroRNAs - genetics | Infection | MicroRNA | Analysis | Transplantation of organs, tissues, etc | Organ transplant recipients | Inflammation | Hepatitis C virus | Gene expression | Health aspects | Liver cirrhosis | Protein binding | Transcription factors | Transplants & implants | Transcription | Syngeneic grafts | Liver | Viruses | Infections | CCAAT/enhancer-binding protein | Hepatitis | Liver cancer | Signal transduction | Cell growth | Allografts | Rodents | Hepatology | Surgery | Cell cycle | Bioindicators | Localization | Deoxyribonucleic acid--DNA | Liver diseases | Cytokines | Nucleotide sequence | MiRNA | Tumor necrosis factor-α | Ribonucleic acid--RNA | Patients | Medicine | Signaling | Hepatocytes | Biopsy | MicroRNAs | Fibrosis | Regulatory mechanisms (biology) | Biomarkers | Notch protein | Hepatitis C | Prostate cancer | Binding sites | Viral infections | Liver transplantation | RNA | Deoxyribonucleic acid | Ribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e70744
Hepatitis C virus (HCV) induced liver disease is the leading indication for liver transplantation (LTx). Reinfection and accelerated development of fibrosis is... 
TGF-BETA | CIRRHOSIS | MULTIDISCIPLINARY SCIENCES | LIVER FIBROSIS | KINASE | POSTTRANSCRIPTIONAL GENE-REGULATION | HCV | GROWTH-FACTOR | MICRORNA EXPRESSION | REPRESSORS ZEB1 | PROGRESSION | Hepatitis C, Chronic - metabolism | Liver Cirrhosis - etiology | Gene Expression | Signal Transduction | Humans | Middle Aged | Protein Tyrosine Phosphatase, Non-Receptor Type 13 - metabolism | Gene Expression Regulation | Transcriptome | Male | MicroRNAs - metabolism | Hepatitis C, Chronic - complications | Gene Regulatory Networks | Transforming Growth Factor beta - biosynthesis | Transfection | RNA Interference | src-Family Kinases - metabolism | Adult | Female | Hepacivirus - physiology | MicroRNAs - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 13 - genetics | Enzyme Activation | Hepatitis C, Chronic - genetics | Tyrosine | Medical research | Phosphatases | Genes | Liver | Transplantation | B cells | Infection | MicroRNA | Analysis | Fibrosis | Medicine, Experimental | Fibroblast growth factors | Hepatitis C virus | Health aspects | Protein kinases | GTP-binding protein | Target recognition | Syngeneic grafts | Hormones | Phosphatase | Proteins | Cyclin-dependent kinase | Hepatitis | Liver cancer | Surgery | Fibroblasts | Physiology | Growth factors | Liver diseases | MiRNA | MAP kinase | Gene expression | Src protein | Patients | Polymerase chain reaction | Pathology | Protein phosphatase | Liver transplantation | Fibroblast growth factor | Transplants & implants | Viruses | Infections | Kinases | Allografts | Protein folding | Hepatology | Cell cycle | Extracellular matrix | Inflammation | Ribonucleic acid--RNA | Phospholipase C | Medicine | Phospholipase | Signaling | Cyclin-dependent kinase inhibitor p21 | Protein kinase | Biopsy | MicroRNAs | Collagen | Hepatitis C | Combinatorial analysis | Protein-tyrosine-phosphatase | RNA | Ribonucleic acid
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e67032
Journal Article
Biophysical Journal, ISSN 0006-3495, 01/2013, Volume 104, Issue 2, pp. 687a - 687a
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2010, Volume 285, Issue 21, pp. 16248 - 16257
NUP98 is a nucleoporin that plays complex roles in the nucleocytoplasmic trafficking of macromolecules. Rearrangements of the NUP98 gene in human leukemia... 
IMPORT | GENE | PORE COMPLEX | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEMATOPOIETIC STEM-CELLS | NUCLEOPORIN NUP98 | ACUTE MYELOID-LEUKEMIA | ONCOGENE NUP98-HOXA9 | CRM1 | NF-KAPPA-B | BINDING | Antigens, CD34 | Oncogene Proteins, Fusion - metabolism | Leukemia - pathology | ran GTP-Binding Protein - genetics | Homeodomain Proteins - metabolism | Humans | NF-kappa B - metabolism | Guanosine Triphosphate - metabolism | Leukemia - metabolism | rev Gene Products, Human Immunodeficiency Virus - metabolism | Promoter Regions, Genetic - genetics | Nuclear Pore Complex Proteins - genetics | Cell Nucleus - metabolism | Cell Nucleus - pathology | Active Transport, Cell Nucleus - genetics | Leukemia - genetics | HIV-1 - metabolism | Nuclear Pore Complex Proteins - metabolism | NFATC Transcription Factors - metabolism | ran GTP-Binding Protein - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | HIV-1 - genetics | Homeodomain Proteins - genetics | Amino Acid Motifs | Karyopherins - metabolism | Cell Nucleus - genetics | NF-kappa B - genetics | Oncogene Proteins, Fusion - genetics | K562 Cells | Karyopherins - genetics | Guanosine Triphosphate - genetics | rev Gene Products, Human Immunodeficiency Virus - genetics | Mutation | Transcription, Genetic - genetics | NFATC Transcription Factors - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Molecular Bases of Disease | Nucleus | Leukemia | Nuclear Export | Nucleoporin | Cell Biology | Diseases | Acute Myeloid Leukemia | Transcription Factors | NUP98 | Nuclear Pore | Nuclear Transport | Cancer
Journal Article
The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation, ISSN 1053-2498, 7/2013, Volume 32, Issue 7, pp. 714 - 722
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 11, p. e27117
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2005, Volume 102, Issue 16, pp. 5749 - 5754
The recruitment model for gene activation presumes that DNA is a platform on which the requisite components of the transcriptional machinery are assembled. In... 
Yeasts | Biological Sciences | Nuclear pore | Chromatin | Nuclear pore complex proteins | Transcriptional activation | Genes | DNA |