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rats (50) 50
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foods, foodstuffs, or non-alcoholic beverages, not covered bysubclasses a23b - a23j (47) 47
mice (47) 47
preservation of foods or foodstuffs, in general (47) 47
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disease (17) 17
japan (17) 17
liver - metabolism (16) 16
specific use of cosmetics or similar toiletpreparations (16) 16
abridged index medicus (15) 15
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mice, inbred balb c (12) 12
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by Yamagami, Keitaro and Kurogi, Ryota and Kurogi, Ai and Nishimura, Kunihiro and Onozuka, Daisuke and Ren, Nice and Kada, Akiko and Nishimura, Ataru and Arimura, Koichi and Ido, Keisuke and Mizoguchi, Masahiro and Sakamoto, Tetsuya and Kayama, Takamasa and Suzuki, Michiyasu and Arai, Hajime and Hagihara, Akihito and Iihara, Koji and Masayoshi, Takigami and Kenji, Kamiyama and Kiyohiro, Houkin and Shougo, Nishi and Tetsuyuki, Yoshimoto and Sadao, Kaneko and Koji, Oka and Hiroshi, Ooyama and Kyousuke, Kamada and Kenichi, Makino and Naoki, Tokumitsu and Kazuhiro, Sako and Susumu, Suzuki and Nozomi, Suzuki and Naoto, Izumi and Kazumi, Nitta and Masahumi, Ootaki and Masanori, Isobe and Mikio, Nishiya and Takaaki, Yamazaki and Syouji, Mabuchi and Kuniaki, Ogasawara and Naohiko, Kubo and Yukihiko, Shimizu and Keiichi, Saito and Tatumi, Yamanome and Atsuo, Yoshino and Mitsuyuki, Fujitsuka and Masaaki, Takami and Hirotoshi, Ohtaka and Teruyuki, Hirano and Yosiaki, Shiokawa and Takaharu, Okada and Ichiro, Suzuki and Michihiro, Kohno and Jou, Haraoka and Yoshinori, Arai and Noriyoshi, Kawamura and Akira, Isoshima and Masaharu, Yasue and Mitsuhiko, Hokari and Takayoshi, Kobayashi and Kensuke, Kawai and Taketoshi, Maehara and Hajime, Arai and Makoto, Noguchi and Haruhiko, Hoshino and Hirofumi, Hiyama and Kensaku, Yoshida and Osamu, Utsugi and Yasuaki, Takeda and Kouichi, Tamaki and Hirohide, Karasudani and Takao, Urabe and Shiro, Kobayashi and Michio, Nakamura and Yorio, Koguchi and Junichi, Ono and Sumio, Suda and Hiromu, Hadeishi and Toshio, Fukutake and Kenji, Wakui and Hirokazu, Tanno and Naoki, Ishige and Takashi, Ohasi and Hideki, Sakai and Yasuaki, Nishimura and Takayuki, Watanabe and Takashi, Matsumoto and Naoki, Koketsu and Yuichi, Hirose and Manabu, Doyu and Toshinori, Hasegawa and Naoto, Kuwayama and Shinichi, Terao and Nobuhiko, Mizutani and Noriyuki, Suzaki and Satoshi, Okuda and Keizo, Yasui and Yukio, Seki and Yasuhiro, Hasegawa and Akira, Ikeda and Youtarou, Takeuchi and ... and J-ASPECT Study Collaborators
World Neurosurgery, ISSN 1878-8750, 2019
Background: The epidemiology of patients with traumatic brain injury (TBI) has changed dramatically over recent decades as a result of rapid advances in aging... 
Aging | Hospital mortality | Prognosis | Traumatic brain injury | Subdural hematoma | Index Medicus
Journal Article
Nihon Geka Gakkai zasshi, ISSN 0301-4894, 03/2014, Volume 115, Issue 2, p. 56
Journal Article
Surgery Today, ISSN 0941-1291, 9/2011, Volume 41, Issue 9, pp. 1171 - 1181
Journal Article
Nihon Geka Gakkai zasshi, ISSN 0301-4894, 2014, Volume 115, Issue 2, pp. 56 - 57
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 04/2016, Volume 310, Issue 8, pp. E643 - E651
Extracellular signal-regulated kinase (ERK) has been implicated in the development of insulin resistance associated with obesity and type 2 diabetes mellitus.... 
Adipocytokine | Type 2 diabetes | Adipocyte | MEK inhibitor | Extracellular signal-regulated kinase pathway | adipocytokine | PHYSIOLOGY | DOWN-REGULATION | RECEPTOR SUBSTRATE-1 | TUMOR XENOGRAFT MODELS | type 2 diabetes | THERAPEUTIC-EFFICACY | adipocyte | ENDOCRINOLOGY & METABOLISM | PPAR-GAMMA | ADIPOCYTE DIFFERENTIATION | extracellular signal-regulated kinase pathway | HISTONE DEACETYLASE INHIBITORS | ACTIVATED PROTEIN-KINASES | REGULATED KINASE PATHWAY | ADIPOSE-TISSUE | Tumor Necrosis Factor-alpha - metabolism | Diphenylamine - pharmacology | Lipolysis - drug effects | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Immunoblotting | Male | Adipocytes - drug effects | Diabetes Mellitus, Type 2 - metabolism | Diphenylamine - analogs & derivatives | Fatty Acids, Nonesterified - metabolism | Diet, High-Fat | Adiponectin - metabolism | Benzamides - pharmacology | Chemokine CCL2 - metabolism | Interleukin-6 - metabolism | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Glucose Tolerance Test | Hyperlipidemias - metabolism | Signal Transduction | Insulin Resistance | Adipokines - metabolism | 3T3-L1 Cells | Reverse Transcriptase Polymerase Chain Reaction | Triglycerides - metabolism | Blood Glucose - drug effects | Receptors, Leptin - deficiency | Insulin - metabolism | Animals | MAP Kinase Signaling System - drug effects | Tumor Necrosis Factor-alpha - drug effects | Adipocytes - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | Blood Glucose - metabolism | In Vitro Techniques | Chemokine CCL2 - drug effects | Tissue | Obesity | Insulin resistance | Diabetes | Pathogenesis | Rodents | Index Medicus
Journal Article
Helvetica Chimica Acta, ISSN 0018-019X, 04/2019, Volume 102, Issue 4
Octadehydrodibenzo[12]annulenes (DBAs), readily available by the oxidative acetylenic coupling of 1,2‐diethynylbenzene derivatives, were reacted with organic... 
Azides (organic) | Side reactions | Fluorescence | Polymerization | Derivatives | Molecular weight | Weight | Coupling (molecular) | Magnetic resonance spectroscopy | Cycloaddition | Triazoles | Computer applications | NMR spectroscopy | Azide | Reaction mechanisms | Alkynes | Crystal structure | Structural analysis
Journal Article
Journal of Pharmacological Sciences, ISSN 1347-8613, 11/2016, Volume 132, Issue 3, pp. 205 - 209
The two-pore domain K channel K 5.1 has been implicated in the pathogenesis of autoimmune diseases. We investigated the changes in K 5.1 activity caused by a... 
K+ channel | Pre-mRNA splicing inhibitor | K2P5.1 | 5.1 | channel | Index Medicus
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 12/2016, Volume 17, Issue 12, p. 2083
  Vitamin D (VD) reduces the risk of breast cancer and improves disease prognoses. Potential VD analogs are being developed as therapeutic agents for breast... 
Protein degradation | 1.1 | Transcription | Vitamin D receptor | channel | Breast cancer | K | Proteins | Vitamin D | Cellular biology | Index Medicus | K+ channel | breast cancer | vitamin D receptor | KCa1.1 | transcription | protein degradation
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 04/2019, Volume 9, Issue 1, pp. 6166 - 9
No optimal assay for assessing isolated hepatocytes before hepatocyte transplantation (HTx) has been established, therefore reliable and rapid assays are... 
APOPTOSIS | LIVER-TRANSPLANTATION | RAT | ISLETS | MULTIDISCIPLINARY SCIENCES | DISEASE | TISSUE | DEATH | INHIBITOR | OKADAIC ACID | Ammonia | Allografts | Transplants & implants | Hepatocytes | Syngeneic grafts | Quality control | Okadaic acid | Deoxyribonucleic acid--DNA | Apoptosis | Necrosis | Liver transplantation
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 08/2012, Volume 53, Issue 4, pp. 817 - 827
The Keap1-Nrf2 system plays a critical role in cellular defense against electrophiles and reactive oxygen species. Keap1 possesses a number of cysteine... 
Keap1 | Reactive cysteine | Stress response | Nrf2 | OXIDATIVE STRESS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CDDO-IM | PHASE-2 RESPONSE | ANTIOXIDANT | COVALENT MODIFICATION | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | NITRIC-OXIDE | CYSTEINE RESIDUES | Oxidants - pharmacology | Maleates - pharmacology | Cytoskeletal Proteins - genetics | Humans | Oleanolic Acid - analogs & derivatives | Transcriptional Activation - drug effects | Oleanolic Acid - pharmacology | NAD(P)H Dehydrogenase (Quinone) - genetics | Hydroquinones - pharmacology | Organoselenium Compounds - pharmacology | HEK293 Cells | Cytoskeletal Proteins - metabolism | NF-E2-Related Factor 2 - genetics | Glutamate-Cysteine Ligase - metabolism | Dimethyl Fumarate | Kelch-Like ECH-Associated Protein 1 | Macrophages, Peritoneal - drug effects | Isothiocyanates - pharmacology | Gene Expression | Mice, Transgenic | Imidazoles - pharmacology | Antioxidants - pharmacology | Azoles - pharmacology | Gene Expression Regulation - drug effects | Animals | Fumarates - pharmacology | NF-E2-Related Factor 2 - metabolism | Adaptor Proteins, Signal Transducing - genetics | NAD(P)H Dehydrogenase (Quinone) - metabolism | Glutamate-Cysteine Ligase - genetics | Mice | Oxidative Stress - drug effects | Adaptor Proteins, Signal Transducing - metabolism | Macrophages, Peritoneal - metabolism | Amino Acid Substitution | Purines | Cysteine | Dimethylformamide | Ligases | Quinone | Transplantation of organs, tissues, etc | Universities and colleges | Macrophages | Cystine | Index Medicus | stress response | reactive cysteine
Journal Article
by Chong, Pin Fee and Kira, Ryutaro and Mori, Harushi and Okumura, Akihisa and Torisu, Hiroyuki and Yasumoto, Sawa and Shimizu, Hiroyuki and Fujimoto, Tsuguto and Hanaoka, Nozomu and Kusunoki, Susumu and Takahashi, Toshiyuki and Oishi, Kazunori and Tanaka-Taya, Keiko and Toyofuku, Etsushi and Fukuyama, Tetsuhiro and Sato, Tatsuharu and Takahashi, Yuya and Kanazawa, Akane and Hiyane, Masato and Fukushima, Takao and Toki, Taira and Hayashi, Ryoko and Kubota, Sonoko and Ishii, Wakako and Akasaka, Manami and Miyazawa, Haruna and Motobayashi, Mitsuo and Asaoka, Mari and Shiihara, Takashi and Miyoshi, Yoshitaka and Tsuru, Tomohiko and Ikeda, Kenta and Matsukura, Masaru and Nakamura, Ryoko and Moriyama, Kengo and Sugawara, Yuji and Takami, Yuichi and Fujita, Takako and Yano, Tamami and Kasai, Mariko and Uchida, Takashi and Fujita, Masashi and Uematsu, Mitsugu and Hata, Atsuko and Ogata, Hideto and Miyamoto, Tomoyuki and Sumi, Kataharu and Ishida, Yu and Takeshita, Eri and Kawazoe, Tomoya and Kawabata, Takayoshi and Miyatake, Chiharu and Yakuwa, Akiko and Kakimoto, Yu and Terashima, Hiroshi and Kubota, Masaya and Abe, Yuichi and Nagura, Michiaki and Yamanouchi, Hideo and Mori, Satomi and Konishi, Yukihiko and Ikegami, Mariko and Tomonaga, Yuko and Takashima, Yumiko and Ichikawa, Kazushi and Moriyama, Nobuko and Oba, Chizu and Kashiwagi, Mitsuru and Yoshikawa, Sosuke and Tanaka, Kenichi and Ohta, Genrei and Hattori, Ayako and Ieda, Daisuke and Ono, Sahoko and Tanimura, Tomoshige and Ban, Kyoko and Sugiyama, Nobuyoshi and Kouzan, Nozomi and Yamada, Yuki and Inoue, Mika and Sakajiri, Kenichi and Ohyama, Ken and Yamamuro, Miho and Ishigaki, Hidetoshi and Seino, Azusa and Igarashi, Shuichi and Nakamoto, Takahito and Sugimoto, Kanae and Ochi, Mitsuhiro and Hamanaka, Eri and Ohi, Kazuki and Kawasaki, Hidefumi and Nishitani, Masahiko and Uno, Hiroshi and Inoue, Masaru and Okuyama, Mai and Yamamoto, Ayako and Sato, Ryota and Azuma, Norihiko and Mabuchi, Sakiko and ... and Acute Flaccid Myelitis Collabo and Acute Flaccid Myelitis Collaborative Study Investigators and for the Acute Flaccid Myelitis Collaborative Study Investigators
Clinical Infectious Diseases, ISSN 1058-4838, 03/2018, Volume 66, Issue 5, pp. 653 - 664
Journal Article
Nippon Geka Gakkai zasshi, ISSN 0301-4894, 01/2011, Volume 112, Issue 1, pp. 2 - 3
Journal Article
Nihon Geka Gakkai zasshi, ISSN 0301-4894, 01/2011, Volume 112, Issue 1, p. 2
Journal Article