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1. Wissenschaft und Kultur in Bibliotheken, Museen und Archiven
: Klaus-Dieter Lehmann zum 65. Geburtstag
by Schneider-Kempf, Barbara and Saur, Klaus G. (Klaus Gerhard) and Schuster, Peter-Klaus and Lehmann, Klaus-Dieter
2005, ISBN 9783598117299, 574
Books and reading | Libraries | Museums | Germany Cultural policy | Lehmann, Klaus-Dieter | Protection | Cultural property
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2. Full Text Abstract IA22: Modeling and targeting the tumor microenvironment of pancreatic cancer
by Saur, Dieter
Cancer Research, ISSN 0008-5472, 08/2016, Volume 76, Issue 15 Supplement, pp. IA22 - IA22
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3. Full Text Interstitial cells of Cajal integrate excitatory and inhibitory neurotransmission with intestinal slow-wave activity
by Klein, Sabine and Seidler, Barbara and Kettenberger, Anna and Sibaev, Andrei and Rohn, Michael and Feil, Robert and Allescher, Hans-Dieter and Vanderwinden, Jean-Marie and Hofmann, Franz and Schemann, Michael and Rad, Roland and Storr, Martin A and Schmid, Roland M and Schneider, Günter and Saur, Dieter
Nature Communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, pp. 1630 - 1630
The enteric nervous system contains excitatory and inhibitory neurons, which control contraction and relaxation of smooth muscle cells as well as... 
MOTOR-NEURONS | SMOOTH-MUSCLE | V MUTANT MICE | ENTERIC NERVOUS-SYSTEM | DEPENDENT PROTEIN-KINASES | MULTIDISCIPLINARY SCIENCES | GASTROINTESTINAL-TRACT | IN-VIVO | LOWER ESOPHAGEAL SPHINCTER | NITRIC-OXIDE | CHOLINERGIC NEUROTRANSMISSION | Animals | Interstitial Cells of Cajal - physiology | Cells, Cultured | Cyclic GMP-Dependent Protein Kinase Type I - genetics | Mice, Transgenic | Mice | Enteric Nervous System - physiology | Synaptic Transmission | Index Medicus
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4. Full Text Nitrergic signalling via interstitial cells of Cajal regulates motor activity in murine colon
by Lies, Barbara and Beck, Katharina and Keppler, Jonas and Saur, Dieter and Groneberg, Dieter and Friebe, Andreas
The Journal of Physiology, ISSN 0022-3751, 10/2015, Volume 593, Issue 20, pp. 4589 - 4601
Key points Dysregulation of nitric oxide (NO) signalling is associated with GI motility dysfunctions like chronic constipation, achalasia or Hirschsprung's... 
INHIBITORY NEUROTRANSMISSION | SMOOTH-MUSCLE | PHYSIOLOGY | MECHANICAL-ACTIVITY | GASTROINTESTINAL-TRACT | NITRIC-OXIDE | C-KIT | RELAXATION | GASTRIC ANTRUM | RAT ANOCOCCYGEUS MUSCLE | GUINEA-PIG | NEUROSCIENCES | Interstitial Cells of Cajal - physiology | Colon - physiology | Signal Transduction | Mice, Inbred C57BL | Myocytes, Smooth Muscle - physiology | Male | Nitric Oxide - physiology | Receptors, Cytoplasmic and Nuclear - physiology | Soluble Guanylyl Cyclase | Receptors, Cytoplasmic and Nuclear - genetics | Mice, Knockout | Animals | Guanylate Cyclase - physiology | Muscle Contraction - physiology | Guanylate Cyclase - genetics | Muscle, Smooth - physiology | Nitric oxide | Smooth muscle | Colon | Rodents | Index Medicus | Alimentary | Research Paper
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5. Full Text Interstitial cells of Cajal mediate nitrergic inhibitory neurotransmission in the murine gastrointestinal tract
by Lies, Barbara and Gil, Víctor and Groneberg, Dieter and Seidler, Barbara and Saur, Dieter and Wischmeyer, Erhard and Jiménez, Marcel and Friebe, Andreas
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 07/2014, Volume 307, Issue 1, pp. G98 - G106
Nitric oxide (NO) is a major inhibitory neurotransmitter in the gastrointestinal (GI) tract. Its main effector, NO-sensitive guanylyl cyclase (NO-GC), is... 
CGMP | Knockout mice | Inhibitory junction potential | Nitric oxide | Gastrointestinal smooth muscle | JUNCTION POTENTIALS | nitric oxide | GASTRIC FUNDUS | PHYSIOLOGY | knockout mice | COLONIC CIRCULAR MUSCLE | cGMP | PROXIMAL COLON | SENSITIVE GUANYLYL CYCLASE | gastrointestinal smooth muscle | inhibitory junction potential | SMOOTH-MUSCLE | PURINERGIC NEUROMUSCULAR-TRANSMISSION | NITRIC-OXIDE | INTERNAL ANAL-SPHINCTER | GASTROENTEROLOGY & HEPATOLOGY | LOWER ESOPHAGEAL | Gastric Fundus - innervation | Neural Inhibition | Mice, Inbred C57BL | Nitrergic Neurons - metabolism | Colon - innervation | Guanylate Cyclase - metabolism | Synaptic Transmission | Mice, Knockout | Interstitial Cells of Cajal - metabolism | Animals | Time Factors | Guanylate Cyclase - genetics | Mice | Inhibitory Postsynaptic Potentials | Nitric Oxide - metabolism | Myocytes, Smooth Muscle - metabolism | Stomach | Neurotransmitters | Colon | Rodents | Cells | Index Medicus
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6. Full Text Dominant role of interstitial cells of Cajal in nitrergic relaxation of murine lower oesophageal sphincter
by Groneberg, Dieter and Zizer, Eugen and Lies, Barbara and Seidler, Barbara and Saur, Dieter and Wagner, Martin and Friebe, Andreas
The Journal of Physiology, ISSN 0022-3751, 01/2015, Volume 593, Issue 2, pp. 403 - 414
Oesophageal achalasia is a disease known to result from reduced relaxation of the lower oesophageal sphincter (LES). Nitric oxide (NO) is one of the main... 
INHIBITORY NEUROTRANSMISSION | SMOOTH-MUSCLE | ACTIVATION | PHYSIOLOGY | V MUTANT MICE | ACHALASIA | EXCITABILITY | NITRIC-OXIDE SYNTHASE | MEDIATOR | HYPERTENSION | SENSITIVE GUANYLYL CYCLASE | NEUROSCIENCES | Esophageal Sphincter, Lower - physiology | Interstitial Cells of Cajal - physiology | Mice, Inbred C57BL | Myocytes, Smooth Muscle - physiology | Guanylate Cyclase - metabolism | Soluble Guanylyl Cyclase | Receptors, Cytoplasmic and Nuclear - genetics | Muscle Relaxation | Esophageal Sphincter, Lower - metabolism | Interstitial Cells of Cajal - metabolism | Animals | Guanylate Cyclase - genetics | Isometric Contraction | Mice | Esophageal Sphincter, Lower - cytology | Nitric Oxide - metabolism | Myocytes, Smooth Muscle - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Gastrointestinal system | Smooth muscle | Nitric oxide | Rodents | Index Medicus | Alimentary
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7. Bitte recht unscharf! Rolf Dieter Brinkmanns 'Postkartenkulisse' und W. G. Sebalds 'nomadische' Fotografien
by Sauer-Kretschmer, Simone
2017
"Rom, Blicke" | prose | travel literature | photographs | German literature | Brinkmann, Rolf Dieter | Die Ringe des Saturn | 1900-1999 | Sebald, Winfried Georg | The Rings of Saturn
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8. Bitte recht unscharf! Rolf Dieter Brinkmanns 'Postkartenkulisse' und W. G. Sebalds 'nomadische' Fotografien
by Sauer-Kretschmer, Simone
01/2017, ISBN 9783503171309
Book Chapter
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9. Full Text Reply
by Klein, Sabine and Saur, Dieter
Gastroenterology, ISSN 0016-5085, 12/2013, Volume 145, Issue 6, pp. 1480 - 1481
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10. Full Text Reply
by Klein, Sabine and Saur, Dieter
Gastroenterology, ISSN 0016-5085, 12/2013, Volume 145, Issue 6, p. 1480
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11. Full Text Reply
by Klein, Sabine and Saur, Dieter
Gastroenterology, ISSN 0016-5085, 12/2013, Volume 145, Issue 6, pp. 1480 - 1481
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12. Full Text Reply
by Klein, Sabine and Saur, Dieter
Gastroenterology, ISSN 0016-5085, 2013, Volume 145, Issue 6, pp. 1480 - 1481
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13. Full Text Reply
by Klein, Sabine and Saur, Dieter
Gastroenterology, ISSN 0016-5085, 2013, Volume 145, Issue 6, pp. 1480 - 1481
Gastroenterology and Hepatology
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14. Full Text Retinoic acid-induced nNOS expression depends on a novel PI3K/Akt/DAX1 pathway in human TGW-nu-I neuroblastoma cells
by Florian Nagl and Katrin Schönhofer and Barbara Seidler and Jörg Mages and Hans-Dieter Allescher and Roland M. Schmid and Günter Schneider and Dieter Saur
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 11/2009, Volume 297, Issue 5, pp. 1146 - 1156
Neuronal nitric oxide synthase (nNOS)-derived nitric oxide (NO) acts as a neurotransmitter and intracellular signaling molecule in the central and peripheral... 
Phosphatidylinositol 3-kinase/Akt signaling | DAX1 | Neuronal nitric oxide synthase | PHYSIOLOGY | NITRIC-OXIDE SYNTHASE | PHOSPHOINOSITIDE 3-KINASE/AKT PATHWAY | PANCREATIC-CANCER | neuronal nitric oxide synthase | CELL BIOLOGY | NUCLEAR RECEPTOR DAX1 | SIGNALING PATHWAY | MOUSE MODEL | GENE-EXPRESSION | TRANSCRIPTION FACTOR | NONGENOMIC ACTIONS | phosphatidylinositol 3-kinase/Akt signaling | NEURONAL NO SYNTHASE | Immunoprecipitation | Humans | Nitric Oxide Synthase Type I - biosynthesis | Phosphatidylinositol 3-Kinases - metabolism | DNA-Binding Proteins - metabolism | Transfection | DAX-1 Orphan Nuclear Receptor | Phosphatidylinositol 3-Kinases - drug effects | Antineoplastic Agents - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Repressor Proteins - metabolism | Tretinoin - pharmacology | Receptors, Retinoic Acid - drug effects | Gene Expression | DNA-Binding Proteins - drug effects | Gene Expression Regulation - physiology | Receptors, Retinoic Acid - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Gene Expression Regulation - drug effects | Signal Transduction - drug effects | Cell Line, Tumor | Repressor Proteins - drug effects | Signal Transduction - physiology | Neuroblastoma - metabolism | Nitric Oxide Synthase Type I - drug effects | Proto-Oncogene Proteins c-akt - drug effects | Analysis | Physiological aspects | Cellular signal transduction | Research | Neuroblastoma | Gene expression | Protein kinases | Risk factors | Signal transduction | Protein synthesis | Nitric oxide | Physiology | Kinases | Cells | Index Medicus
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15. Full Text Single-Nucleotide Promoter Polymorphism Alters Transcription of Neuronal Nitric Oxide Synthase Exon 1c in Infantile Hypertrophic Pyloric Stenosis
by Dieter Saur and Jean-Marie Vanderwinden and Barbara Seidler and Roland M. Schmid and Marc-Henri De Laet and Hans-Dieter Allescher and Solomon H. Snyder
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2004, Volume 101, Issue 6, pp. 1662 - 1667
Infantile hypertrophic pyloric stenosis (IHPS), characterized by enlarged pyloric musculature and gastric-outlet obstruction, is associated with altered... 
Polymerase chain reaction | Stomach | Biological Sciences | Messenger RNA | Exons | Neurons | DNA | Alleles | Promoter regions | Genetic mutation | Hypertrophy | VARIANTS | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | PROGRESS | MICE | MUSCLE-CELL-PROLIFERATION | HUMAN GASTROINTESTINAL-TRACT | EXPRESSION | GENE-TRANSCRIPTION | Promoter Regions, Genetic | Humans | Molecular Sequence Data | Infant | Pyloric Stenosis - genetics | Nitric Oxide Synthase - genetics | Nitric Oxide Synthase Type I | Base Sequence | Cell Line, Tumor | Transcription, Genetic | Polymorphism, Single Nucleotide | Infant, Newborn | Nucleotides | Research | Genetics | Nitric oxide | Birth defects | PGP9.5 gene | Infantile hypertrophic pyloric stenosis | Index Medicus
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16. Full Text siRNA-coupled nanoparticles for improved therapeutic targeting of pancreatic cancer
by Lange, Sebastian and Saur, Dieter and Rad, Roland
Gut, ISSN 0017-5749, 11/2016, Volume 65, Issue 11, pp. 1780 - U6
  PC treatment is further complicated by a dense tumour stroma and low vessel density, leading to impaired drug delivery and high adverse effects of systemic... 
GASTROENTEROLOGY & HEPATOLOGY | Chemotherapy | Transplants & implants | Pancreatic cancer | Cell cycle | Clinical trials | Lipids | Kinases | Cancer therapies | Nanotechnology | Tumors | Index Medicus | Abridged Index Medicus
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17. Full Text IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality
by Zhang, Hong and Neuhöfer, Patrick and Song, Liang and Rabe, Björn and Lesina, Marina and Kurkowski, Magdalena U and Treiber, Matthias and Wartmann, Thomas and Regnér, Sara and Thorlacius, Henrik and Saur, Dieter and Weirich, Gregor and Yoshimura, Akihiko and Halangk, Walter and Mizgerd, Joseph P and Schmid, Roland M and Rose-John, Stefan and Algül, Hana and Kirurgi and Lund University and Surgery and Lunds universitet
Journal of Clinical Investigation, ISSN 0021-9738, 03/2013, Volume 123, Issue 3, pp. 1019 - 1031
Acute lung injury (ALI) is an inflammatory disease with a high mortality rate. Although typically seen in individuals with sepsis, ALI is also a major... 
C-REACTIVE PROTEIN | INFLAMMATORY-RESPONSE | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | Acute lung injury (ALI) is an inflammatory disease with a high mortality rate. Although typically seen in individuals with sepsis | of STAT3 and production of the neutrophil attractant CXCL1 in pancreatic acinar cells. Examination of human samples revealed expression of IL-6 in combination with soluble IL-6 receptor was a reliable predictor of ALI in SAP. These results demonstrate that IL-6 trans-signaling is an essential mediator of ALI in SAP across | INTERLEUKIN-6 | RESPIRATORY-DISTRESS-SYNDROME | PREDICTION | species and suggest that therapeutic inhibition of IL-6 may prevent SAP-associated ALI | ACUTE-PHASE RESPONSE | ALI is also a major complication in severe acute pancreatitis (SAP). The pathophysiology of SAP-associated ALI is poorly understood, but elevated serum levels of IL-6 is a reliable marker for disease severity. Here, we used a mouse model of acute pancreatitis-associated (AP-associated) ALI to determine the role of IL-6 in ALI lethality. Il6-deficient mice had a lower death rate compared with wild-type mice with AP, while mice injected with IL-6 were more likely to develop lethal ALI. We found that inflammation-associated NF-kappa B induced myeloid cell secretion of IL-6, and the effects of secreted IL-6 were mediated by complexation with soluble IL-6 receptor, a process known as trans-signaling. IL-6 trans-signaling stimulated phosphorylation | BLOCKADE | INHIBITOR | SEVERITY | Phosphorylation | Humans | NF-kappa B - metabolism | Pancreatitis - complications | Receptors, Interleukin-8B - antagonists & inhibitors | Benzenesulfonates - pharmacology | Interleukin-6 - physiology | Pancreatitis - immunology | Acute Lung Injury - metabolism | Chemokine CXCL1 - metabolism | STAT3 Transcription Factor - metabolism | Acinar Cells - metabolism | Aminosalicylic Acids - pharmacology | Signal Transduction | Mice, Inbred C57BL | Pancreas - pathology | Mice, Transgenic | Acute Lung Injury - pathology | Interleukin-6 - secretion | Animals | Myeloid Cells - secretion | Receptors, Interleukin-8B - metabolism | Myeloid Cells - metabolism | Acute Lung Injury - immunology | Pancreatitis - pathology | Mice | Phenylurea Compounds - pharmacology | Protein Processing, Post-Translational | Pancreatitis - metabolism | STAT3 Transcription Factor - antagonists & inhibitors | Acute Lung Injury - etiology | Receptors, Interleukin-6 - metabolism | Lung diseases | Pancreatitis | Genetic aspects | Research | Health aspects | Risk factors | Interleukin-6 | Index Medicus | Abridged Index Medicus | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Surgery | Kirurgi
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18. Full Text Nitrergic signalling via interstitial cells of Cajal regulates motor activity in murine colon
: NO and colonic pacemaker activity
by Lies, Barbara and Beck, Katharina and Keppler, Jonas and Saur, Dieter and Groneberg, Dieter and Friebe, Andreas
The Journal of Physiology, ISSN 0022-3751, 10/2015, Volume 593, Issue 20, pp. 4589 - 4601
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19. Full Text Quo vadis, Kras?
by Schneider, Günter and Saur, Dieter
Oncotarget, ISSN 1949-2553, 2013, Volume 4, Issue 9, pp. 1336 - 1337
ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Animals | Neoplasms - genetics | Humans | Proto-Oncogene Proteins - genetics | Mice | Mutation | Genes, ras | Index Medicus
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20. Full Text The role of Pdk1-dependent signalling pathways in pancreatic cancer
by Hesse, Felix and Schönhuber, Nina and Saur, Dieter
Pancreatology, ISSN 1424-3903, 2017, Volume 17, Issue 3, pp. S2 - S2
The deletion of the 3-phosphoinositide dependent protein kinase 1 (Pdk1) entirely blocks the formation of precursor lesions and PDAC in a pancreatic cancer... 
Endocrinology & Metabolism | Gastroenterology and Hepatology | Pancreas transplantation | Signal transduction | Kinases | Pancreatic cancer
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