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Particle and Fibre Toxicology, ISSN 1743-8977, 10/2010, Volume 7, Issue 1, pp. 28 - 28
Journal Article
Particle and fibre toxicology, ISSN 1743-8977, 2011, Volume 8, Issue 1, pp. 21 - 21
Journal Article
Journal Article
Particle and fibre toxicology, ISSN 1743-8977, 2013, Volume 10, Issue 1, pp. 5 - 5
Journal Article
Toxicologic pathology, ISSN 0192-6233, 1/2018, Volume 46, Issue 1, pp. 28 - 46
Assessing the potential health risks for newly developed nanoparticles poses a significant challenge. Nanometer-sized particles are not generally detectable... 
toxicology | nanotechnology | inhalation | fibrosis | nanoparticles | pathology | PULMONARY | CARDIOVASCULAR-RESPONSES | LUNG | TRANSLOCATION | OXIDATIVE STRESS | RATS | WALLED CARBON NANOTUBES | OXIDE NANOPARTICLES | CERIUM OXIDE | Microscopy - methods | Tissue Distribution | Animals | Inhalation Exposure | Lung - drug effects | Mice, Inbred C57BL | Rats | Male | Mice | Nanoparticles - toxicity | Rats, Sprague-Dawley
Journal Article
Journal of nanomaterials, ISSN 1687-4129, 2012, Volume 2012, pp. 1 - 17
Silicon nanowires (Si NWs) are being manufactured for use as sensors and transistors for circuit applications. The goal was to assess pulmonary toxicity and... 
RESPONSES | WALLED-CARBON-NANOTUBES | INFLAMMATION | DISPERSION | BEHAVIOR | MATERIALS SCIENCE, MULTIDISCIPLINARY | TISSUE | NANOMATERIALS | NANOSCIENCE & NANOTECHNOLOGY | MICE | ASBESTOS | Studies | Nanomaterials | Occupational safety | Toxicity | Rodents | Nanotechnology
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 2012, Volume 13, Issue 12, pp. 13781 - 13803
Journal Article
Environmental health perspectives, ISSN 0091-6765, 9/2006, Volume 114, Issue 9, pp. 1367 - 1373
Background: Miners inhaling respirable coal dust (CD) frequently develop coal workers' pneumoconiosis, a dust-associated pneumoconiosis characterized by lung... 
Keratins | Pulmonary alveoli | Certificates of deposit | Lungs | Epithelial cells | Coal | Fluorescent antibody techniques | Inflammation | Coal mining | Research | Apoptosis | Pneumoconiosis | Polycyclic aromatic hydrocarbons | Bax | Caspase | CYP1A1 | CYP2B1 | Xenobiotic metabolism | Coal dust | Modifiers | coal dust | INJURY | apoptosis | LIGAND PATHWAY | DEATH | MODEL | modifiers | polycyclic aromatic hydrocarbons | xenobiotic metabolism | caspase | ENVIRONMENTAL SCIENCES | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | EPITHELIAL-CELLS | INFLAMMATION | DISEASE | MINERS | pneumoconiosis | PULMONARY-FIBROSIS | Coal - toxicity | Lung - pathology | Polycyclic Aromatic Hydrocarbons - toxicity | Apoptosis - drug effects | Rats | Male | Cytochrome P-450 CYP1A1 - metabolism | Caspase Inhibitors | Rats, Sprague-Dawley | Aryl Hydrocarbon Hydroxylases - metabolism | Dose-Response Relationship, Drug | Gene Expression Regulation - drug effects | Dust | Animals | Caspases - metabolism | Pneumonia - chemically induced | Lung - drug effects | Pulmonary Alveoli - metabolism | Pulmonary Alveoli - cytology | Cytochrome P-450 CYP1B1 | Apoptosis - physiology | beta-Naphthoflavone - toxicity | Coal industry | Lung diseases | CYTOCHROMES | INHIBITION | METABOLISM | 01 COAL, LIGNITE, AND PEAT | LUNGS | DUSTS | COAL | RATS | PNEUMOCONIOSES | POLYCYCLIC AROMATIC HYDROCARBONS | COAL MINERS
Journal Article
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