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Nature medicine, ISSN 1078-8956, 11/2014, Volume 20, Issue 11, pp. 1289 - 1300
Pulmonary hypertension (PH) is characterized by increased proliferation and apoptosis resistance of pulmonary artery smooth muscle cells (PASMCs). Forkhead box... 
SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL PROLIFERATION | NUCLEAR EXPORT | PACLITAXEL | CELL BIOLOGY | MAINTENANCE | ARTERIAL-HYPERTENSION | MOLECULAR PATHOGENESIS | GROWTH-FACTOR | INHIBITORS | MORPHOGENETIC PROTEIN-RECEPTOR | Small Molecule Libraries - pharmacology | Paclitaxel - pharmacology | Muscle, Smooth, Vascular - metabolism | Apoptosis - drug effects | Humans | Hypertension, Pulmonary - physiopathology | Apoptosis - genetics | Male | Quinolones - pharmacology | RNA, Messenger - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Bone Morphogenetic Proteins - metabolism | Forkhead Transcription Factors - metabolism | Gene Deletion | Adult | Female | Bromodeoxyuridine - metabolism | Vascular Remodeling - drug effects | Muscle, Smooth, Vascular - drug effects | Lung - pathology | Cytokines - metabolism | RNA, Messenger - genetics | Rats | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Lung - physiopathology | Signal Transduction - genetics | Forkhead Transcription Factors - genetics | Gene Expression Regulation - drug effects | Muscle, Smooth, Vascular - pathology | Animals | Signal Transduction - drug effects | Vascular Remodeling - genetics | Cell Proliferation - drug effects | Mice | Lung - blood supply | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Transcription factors | Research | Analysis | Pulmonary hypertension | Smooth muscle | Cell growth | Pulmonary arteries | Apoptosis
Journal Article
EUROPEAN RESPIRATORY JOURNAL, ISSN 0903-1936, 01/2019, Volume 53, Issue 1
Clinical and translational research has played a major role in advancing our understanding of pulmonary hypertension (PH), including pulmonary arterial... 
ADVENTITIAL FIBROBLASTS | MICROVASCULAR DISEASE | TO-MESENCHYMAL TRANSITION | GROWTH-FACTOR-BETA | RESPIRATORY SYSTEM | ARTERIAL-HYPERTENSION | ENDOTHELIAL-CELLS | VASCULAR-LESIONS | MITOCHONDRIAL-FUNCTION | SMOOTH-MUSCLE-CELLS | PROTEIN-RECEPTOR 2
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 02/2013, Volume 187, Issue 3, pp. 294 - 302
Journal Article
Oncogene, ISSN 0950-9232, 02/2013, Volume 32, Issue 9, pp. 1121 - 1134
Lung cancer is the leading cause of cancer death worldwide. Recent data suggest that cyclic nucleotide phosphodiesterases (PDEs) are relevant in various cancer... 
lung cancer | phosphodiesterases | hypoxia | proliferation | angiogenesis | MIGRATION | CELLS | APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | COMPONENTS | CELL BIOLOGY | INHIBITION | ONCOLOGY | GENETICS & HEREDITY | TARGETS | LEUKEMIA | TUMOR-GROWTH | EXPRESSION | Humans | Lung Neoplasms - metabolism | RNA, Small Interfering - pharmacology | Cyclic AMP | Transplantation, Heterologous | Phosphodiesterase 4 Inhibitors - pharmacology | Animals | Basic Helix-Loop-Helix Transcription Factors - metabolism | Signal Transduction - drug effects | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Mice, Nude | Cyclic Nucleotide Phosphodiesterases, Type 4 - physiology | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Cyclic Nucleotide Phosphodiesterases, Type 3 - metabolism | Cyclic Nucleotide Phosphodiesterases, Type 4 - metabolism | Care and treatment | Lung cancer | Physiological aspects | Development and progression | Genetic aspects | Research | Gene expression | Vascular endothelial growth factor | Angiogenesis | Pathology | Adenocarcinoma | Drugs | Protein kinase A | Carcinoma | Immunocytochemistry | Pyruvic acid | Regulatory sequences | Drug development | dehydrogenase | Western blotting | Reporter gene | Xenografts | Hypoxia-inducible factors | 3',5'-Cyclic-nucleotide phosphodiesterase | Colonies | Tumor cells | Data processing | siRNA | Tumor cell lines | phosphodiesterase IV | L-Lactate dehydrogenase | Polymerase chain reaction | Hypoxia | Mutation | phosphodiesterase | Tumors
Journal Article
European Respiratory Journal, ISSN 0903-1936, 04/2009, Volume 33, Issue 4, pp. 785 - 792
Journal Article
Intensive Care Medicine, ISSN 0342-4642, 8/2011, Volume 37, Issue 8, pp. 1368 - 1377
The calcium-sensitizing drug levosimendan increases myocardial contractility and, by activating K+-channels, dilates pulmonary vessels. In the acute setting,... 
Vascular remodeling | Pediatrics | Pain Medicine | Emergency Medicine | Pneumology/Respiratory System | Medicine & Public Health | Levosimendan | Intensive / Critical Care Medicine | Pulmonary arterial hypertension | Anesthesiology | Potassium channel | VASODILATING MECHANISMS | CELL-PROLIFERATION | CARDIAC-MUSCLE | NICORANDIL | HEART | SMOOTH-MUSCLE | INHIBITION | ARTERIAL-HYPERTENSION | ENDOTHELIAL-CELLS | CHANNELS | CRITICAL CARE MEDICINE | Myocardial Contraction - drug effects | Pyridazines - pharmacology | Hypertension, Pulmonary - chemically induced | Nicorandil - pharmacology | Glyburide - pharmacology | Hypoglycemic Agents - administration & dosage | Pyridazines - administration & dosage | Hypertension, Pulmonary - drug therapy | Glyburide - administration & dosage | Disease Models, Animal | Vasodilator Agents - pharmacology | Hydrazones - administration & dosage | Rats | Cardiotonic Agents - pharmacology | Nicorandil - administration & dosage | Potassium Channels - drug effects | Rats, Sprague-Dawley | Hydrazones - pharmacology | Hypoglycemic Agents - pharmacology | Animals | Cardiotonic Agents - administration & dosage | Pulmonary Circulation - drug effects | Airway Remodeling - drug effects | Vasodilator Agents - administration & dosage | Germany | Complications and side effects | Patient outcomes | Physiological aspects | Respiratory agents | Dosage and administration | Potassium channels | Research | Drug therapy | Pulmonary hypertension
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2009, Volume 54, Issue 1, pp. S10 - S19
Inflammatory processes are prominent in various types of human and experimental pulmonary hypertension (PH) and are increasingly recognized as major pathogenic... 
Cardiovascular | Internal Medicine | growth factors | inflammation | pulmonary vascular remodeling | CARDIAC & CARDIOVASCULAR SYSTEMS | IMMUNODEFICIENCY-VIRUS-INFECTION | SARCOMA-ASSOCIATED HERPESVIRUS | ENDOTHELIAL-CELL APOPTOSIS | SYSTEMIC-SCLEROSIS | HEPATITIS-C VIRUS | ACTIVE ANTIRETROVIRAL THERAPY | ARTERIAL-HYPERTENSION | PLEXIFORM LESIONS | 5-HYDROXYTRYPTAMINE TRANSPORTER GENE | CONNECTIVE-TISSUE DISEASES | Chemokine CCL5 | Inflammation - pathology | Humans | Scleroderma, Systemic - pathology | Hypertension, Pulmonary - physiopathology | NFATC Transcription Factors - physiology | Chemokines, CX3C - physiology | Cytokines - physiology | Vascular Resistance - physiology | Animals | Scleroderma, Systemic - physiopathology | Antineoplastic Agents - pharmacology | Hypertension, Pulmonary - virology | Hypertension, Pulmonary - etiology | Hypertension, Pulmonary - pathology | Inflammation - physiopathology | Hypertension | Tyrosine | Medical colleges | Lung diseases, Obstructive | Pharmacogenetics | Micropollutants | Dendritic cells | Antibodies | Development and progression | Inflammation | HIV (Viruses) | T cells | Macrophages | Endothelium | Polymerase chain reaction | Viral antibodies | Messenger RNA | Viral proteins | Hepatitis C | Vascular endothelial growth factor | Growth factors | Tumors | Platelet-derived growth factor | Endothelial growth factors | B cells | Pulmonary hypertension | Biomedical research | Cytokines | Pulmonary arteries | Migration | Recruitment | Proteins | Angiogenesis | Lymphocytes | Human immunodeficiency virus--HIV | Medical prognosis | Cell cycle | Chemokines
Journal Article