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Circulation, ISSN 0009-7322, 05/2013, Volume 127, Issue 21, pp. 2114 - 2124
BACKGROUND—CCR5 plays an important role in atherosclerosis and ischemic cardiovascular diseases, as well as in HIV replication and diffusion. HIV infection is... 
Receptors | Inflammation | Maraviroc | CCR5 | Acquired immunodeficiency syndrome | Atherosclerosis | atherosclerosis | maraviroc | CARDIAC & CARDIOVASCULAR SYSTEMS | ENDOTHELIAL DYSFUNCTION | MYOCARDIAL-INFARCTION | receptors, CCR5 | acquired immunodeficiency syndrome | HIV-INFECTED PATIENTS | inflammation | PPAR-GAMMA | PERIPHERAL VASCULAR DISEASE | CHEMOKINE RECEPTORS | PROTEASE INHIBITOR RITONAVIR | CORONARY-ARTERY-DISEASE | CARDIOVASCULAR RISK | Tumor Necrosis Factor-alpha - metabolism | Apolipoproteins E - deficiency | Male | Plaque, Atherosclerotic - chemically induced | Cyclohexanes - therapeutic use | Ritonavir - adverse effects | Chemokine CCL2 - metabolism | Chemokine CCL5 - metabolism | CCR5 Receptor Antagonists | Disease Models, Animal | Triazoles - therapeutic use | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Atherosclerosis - chemically induced | Mice, Inbred C57BL | Plaque, Atherosclerotic - prevention & control | Anti-Retroviral Agents - adverse effects | Treatment Outcome | Atherosclerosis - metabolism | Mice, Knockout | Interleukin-17 - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Animals | Apolipoproteins E - genetics | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Atherosclerosis - prevention & control | Cell Movement | Antagonists (Biochemistry) | Animal experimentation | Complications and side effects | Usage | Care and treatment | Atherosclerotic plaque | Ritonavir | Development and progression | Health aspects
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3150 - 3161
From viral binding to the hepatocyte surface to extracellular virion release, the replication cycle of the hepatitis C virus (HCV) intersects at various levels... 
hepatitis C virus | proprotein convertase subtilisin/kexin type 9 | cholesterol | lipids | lipoprotein | LIFE-CYCLE | MEDICINE, RESEARCH & EXPERIMENTAL | EVOLOCUMAB AMG 145 | CARDIOVASCULAR EVENTS | CELLULAR DETERMINANTS | RANDOMIZED-TRIAL | APOLIPOPROTEIN-E | CELL BIOLOGY | LIPOVIRAL PARTICLES | STATIN INTOLERANCE | HETEROZYGOUS FAMILIAL HYPERCHOLESTEROLEMIA | DENSITY-LIPOPROTEIN-RECEPTOR | Hepatitis C, Chronic - metabolism | Hepatitis C, Chronic - pathology | Receptors, Virus - metabolism | Humans | Virulence | Hepacivirus - genetics | Hepatitis C, Chronic - virology | Hepacivirus - pathogenicity | Hepatocytes - metabolism | Receptors, Virus - genetics | Viral Proteins - metabolism | Hepacivirus - metabolism | Receptors, Lipoprotein - metabolism | Lipid Metabolism - genetics | Signal Transduction | Gene Expression Regulation | Receptors, Lipoprotein - genetics | Viral Proteins - genetics | Genotype | Proprotein Convertase 9 - genetics | Proprotein Convertase 9 - metabolism | Host-Pathogen Interactions | Virus Replication | Hepatocytes - virology | Hepatitis C, Chronic - genetics | Physiological aspects | Hepatitis C virus | Transcription factors | Liver | Viruses | Lipids | Subtilisin | Metabolism | Proteins | Hepatitis | Receptors | Kexin | Human immunodeficiency virus--HIV | Infectivity | Lipid metabolism | Hepatitis C | kexin type 9 | Reviews | proprotein convertase subtilisin | Review
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2019, Volume 14, Issue 3, p. e0213160
Journal Article
Scientific Reports, ISSN 2045-2322, 06/2016, Volume 6, Issue 1, p. 28741
Endothelial dysfunction, a marker of cardiovascular (CV) risk, is common in human immunodeficiency virus (HIV)-infected patients. Microalbuminuria is frequent... 
DIABETES-MELLITUS | INDIVIDUALS | METABOLIC SYNDROME | INDICATORS | MICROALBUMINURIA | MULTIDISCIPLINARY SCIENCES | CARDIOVASCULAR-DISEASE | MARKERS | RISK | PROTEINURIA | HYPERTENSION
Journal Article
Journal Article
BMC INFECTIOUS DISEASES, ISSN 1471-2334, 06/2019, Volume 19, Issue 1, pp. 555 - 9
BackgroundAmong people living with HIV (PLWH), the prevalence of non-HIV related co-morbidities is increasing. Aim of the present study is to describe... 
POPULATION | INFECTIOUS DISEASES | MANAGEMENT | GUIDELINES | PATTERNS | PREVALENCE | MULTIMORBIDITY | Co-morbidity | Disease-disease interactions | INDIVIDUALS | HIV | DISEASE | Multi-morbidity | INFECTED PATIENTS | HYPERTENSION | Clusterization | Hypertension | Usage | Comorbidity | HIV patients | Research | Cardiovascular diseases | Observational studies | Health aspects | Risk factors
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e35924
Background: Long-term remission of HIV-1 disease can be readily achieved by combinations of highly effective antiretroviral therapy (HAART). However, a... 
PATHOGENESIS | ACTIVATION | DENDRITIC CELLS | METABOLISM | MACROPHAGES | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | CD4(+) | INDUCTION | CD36 | Inflammation - pathology | HIV Antigens - pharmacology | Humans | Vaccination | Neutralization Tests | Janus Kinases - metabolism | Neoplasm Proteins - metabolism | PPAR gamma - metabolism | gag Gene Products, Human Immunodeficiency Virus - pharmacology | Lipopolysaccharide Receptors - metabolism | HIV Infections - immunology | Inflammation - complications | STAT1 Transcription Factor - metabolism | HIV Infections - pathology | Monocytes - pathology | Receptors for Activated C Kinase | Lipid Metabolism - genetics | Phosphorylation - drug effects | Thiazolidinediones - pharmacology | Cell Line | Atherosclerosis - pathology | Macrophages - pathology | Cell Separation | HIV-1 - drug effects | Receptors, Cell Surface - metabolism | Atherosclerosis - complications | Signal Transduction - genetics | Vidarabine - analogs & derivatives | Gene Expression Regulation - drug effects | Isoxazoles - pharmacology | Monocytes - drug effects | Monocytes - enzymology | Vidarabine - pharmacology | Phenotype | Signal Transduction - drug effects | HIV Infections - complications | Lipid Metabolism - drug effects | PPAR gamma - agonists | Macrophages - drug effects | GTP-Binding Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Highly active antiretroviral therapy | Antiviral agents | AIDS vaccines | Viral proteins | Physiological aspects | HIV patients | Development and progression | Genetic aspects | HIV (Viruses) | Macrophages | Therapy | CD40 antigen | CD86 antigen | Pathogenesis | Genes | Gene regulation | Lipids | Infections | Activation | Vaccines | Kinases | Proteins | Genotype & phenotype | Signal transduction | Receptors | Cell activation | Ischemia | Acquired immune deficiency syndrome--AIDS | Antiretroviral agents | Human immunodeficiency virus--HIV | Atherosclerosis | Remission | Lipid metabolism | Matrix protein | Stat1 protein | Recombinant | Immune response | Polypeptides | Intercellular adhesion molecule 1 | Exposure | Inflammation | CD36 antigen | Metabolism | Gene expression | Nuclear receptors | Cholesterol | Signaling | Monocytes | Cell lines | Monocyte chemoattractant protein 1 | Acquired immune deficiency syndrome | AIDS | HIV | Human immunodeficiency virus
Journal Article
BMC Nephrology, ISSN 1471-2369, 07/2017, Volume 18, Issue 1, pp. 255 - 8
Journal Article
Scientific Reports, ISSN 2045-2322, 10/2015, Volume 5, Issue 1, p. 15403
Liver disease is the second most common cause of mortality in HIV-infected persons. Exactly how HIV infection per se affects liver disease progression is... 
GAMMA | MITOCHONDRIAL ABNORMALITIES | ACTIVATION | COACTIVATOR | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | FATTY LIVER | LIVER X-RECEPTORS | NONALCOHOLIC STEATOHEPATITIS | ACTIVE-ANTIRETROVIRAL-THERAPY | LXR-ALPHA | Humans | Transcriptional Activation - drug effects | AIDS Vaccines - immunology | STAT Transcription Factors - metabolism | Peptide Fragments - pharmacology | env Gene Products, Human Immunodeficiency Virus - metabolism | Hepatocytes - metabolism | Recombinant Proteins - biosynthesis | Orphan Nuclear Receptors - metabolism | Hepatocytes - cytology | env Gene Products, Human Immunodeficiency Virus - genetics | Liver X Receptors | Trans-Activators - genetics | Cholesterol - analysis | HIV - metabolism | Peptide Fragments - genetics | Sterol Regulatory Element Binding Protein 1 - metabolism | Cholesterol - analogs & derivatives | Peptide Fragments - metabolism | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Mediator Complex Subunit 1 - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Recombinant Proteins - pharmacology | Orphan Nuclear Receptors - genetics | Hep G2 Cells | env Gene Products, Human Immunodeficiency Virus - pharmacology | Orphan Nuclear Receptors - antagonists & inhibitors | Animals | Recombinant Proteins - immunology | Signal Transduction - drug effects | Sterol Regulatory Element Binding Protein 1 - genetics | Lipid Metabolism - drug effects | STAT Transcription Factors - chemistry | Trans-Activators - metabolism | Mice | Mediator Complex Subunit 1 - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Proteins | Signal transduction | Liver diseases | Hepatocytes | Human immunodeficiency virus--HIV | Transcription activation | Vaccination | Intracellular signalling | Transduction | Matrix protein | Gene expression | Nuclear receptors
Journal Article
Dermatopathology (Basel, Switzerland), ISSN 2296-3529, 01/2018, Volume 5, Issue 1, pp. 38 - 40
Journal Article
Atherosclerosis, ISSN 0021-9150, 05/2018, Volume 272, pp. 101 - 107
Elevated serum uric acid (SUA) levels may be associated with endothelial dysfunction. Increased rates of metabolic syndrome (MS) and elevated SUA levels were... 
Uric acid | Endothelial function | HIV | Metabolic syndrome | HAART | MORTALITY | URATE | CARDIAC & CARDIOVASCULAR SYSTEMS | RISK | PREVALENCE | HUMAN-IMMUNODEFICIENCY-VIRUS | ALLOPURINOL | ASYMPTOMATIC HYPERURICEMIA | DISEASE | PERIPHERAL VASCULAR DISEASE | INFECTED PATIENTS
Journal Article