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Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7519, pp. 564 - 568
Journal Article
Immunity, ISSN 1074-7613, 2010, Volume 33, Issue 2, pp. 279 - 288
Mutations in the gene are linked to inflammatory bowel disease susceptibility. Experimental models have shown that interleukin-23 (IL-23) orchestrates innate... 
CELLIMMUNO | Cellimmuno | DEPENDENT COLITIS | AUTOIMMUNE INFLAMMATION | SCID MICE | HELPER-CELLS | IN-VIVO | DIFFERENTIATION | IMMUNOLOGY | IL-23 | GENOME-WIDE ASSOCIATION | BOWEL-DISEASE | TH17 CELLS | Inflammation | Interleukins | T cells | Analysis | Gastrointestinal diseases | Inflammatory bowel disease | Cytokines | Pathogenesis | Rodents | Colon | Gene expression | Immune system | Index Medicus
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 07/2015, Volume 43, Issue 1, pp. 187 - 199
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 08/2018, Volume 49, Issue 2, pp. 353 - 362.e5
The epithelium and immune compartment in the intestine are constantly exposed to a fluctuating external environment. Defective communication between these... 
intestinal epithelial cell | goblet cells | inflammation | colon cancer | crypt stem cell | Wnt-β-catenin | IBD | AHR | diet | gut barrier | indole-3-carbinol | TRYPTOPHAN | PATHWAYS | ACTIVATION | LIGANDS | ARYL-HYDROCARBON RECEPTOR | COLORECTAL-CANCER | SUSCEPTIBILITY | DIOXIN RECEPTOR | INDOLE-3-CARBINOL | NUTRITION | IMMUNOLOGY | Cell Proliferation | Humans | Wnt Signaling Pathway - physiology | Stem Cells - cytology | Enterobacteriaceae Infections - immunology | Intestinal Mucosa - cytology | Epithelial Cells - physiology | Tight Junctions - physiology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Intestinal Mucosa - immunology | HEK293 Cells | Receptors, Aryl Hydrocarbon - metabolism | Cell Line | Basic Helix-Loop-Helix Transcription Factors - genetics | Citrobacter rodentium - immunology | Enterobacteriaceae Infections - microbiology | Mice, Inbred C57BL | Receptors, Aryl Hydrocarbon - genetics | Ubiquitin-Protein Ligases - biosynthesis | Mice, Knockout | Carcinogenesis - pathology | Cell Differentiation - immunology | Animals | Mice | Ubiquitin-Protein Ligases - genetics | Ubiquitin | Colon cancer | Ligases | Stem cells | Homeostasis | Disease susceptibility | Sensors | Cell differentiation | Flow cytometry | Transformation | Wnt protein | Transcription | Epithelial cells | Gene regulation | Variations | Guards | Infections | Activation | Experiments | β-catenin | Clonal deletion | Aromatic compounds | Intestine | Rodents | Cell cycle | Deletion | Bone marrow | Bacteria | Tumorigenesis | Immune system | Pathogens | Medical research | Statistical analysis | Mortality | Inflammation | Environmental factors | Epithelium | Regeneration | Organic chemistry | Diet | Ligands | Cancer | Integrity | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 05/2016, Volume 7, Issue 1, pp. 11627 - 11627
IL-23 is a key driver of pathogenic Th17 cell responses. It has been suggested that the transcription factor T-bet is required to facilitate IL-23-driven... 
DEPENDENT COLITIS | INFLAMMATORY-BOWEL-DISEASE | T(H)17 CELLS | ROR-GAMMA-T | AUTOIMMUNE INFLAMMATION | SCID MICE | MULTIDISCIPLINARY SCIENCES | DIFFERENTIATION | IL-23 | TRANSCRIPTION FACTOR | TH17 CELLS | Index Medicus
Journal Article