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by Klionsky, Daniel J and Abdelmohsen, Kotb and Abe, Akihisa and Abedin, Md Joynal and Abeliovich, Hagai and Acevedo Arozena, Abraham and Adachi, Hiroaki and Adams, Christopher M and Adams, Peter D and Adeli, Khosrow and Adhihetty, Peter J and Adler, Sharon G and Agam, Galila and Agarwal, Rajesh and Aghi, Manish K and Agnello, Maria and Agostinis, Patrizia and Aguilar, Patricia V and Aguirre-Ghiso, Julio and Airoldi, Edoardo M and Ait-Si-Ali, Slimane and Akematsu, Takahiko and Akporiaye, Emmanuel T and Al-Rubeai, Mohamed and Albaiceta, Guillermo M and Albanese, Chris and Albani, Diego and Albert, Matthew L and Aldudo, Jesus and Algül, Hana and Alirezaei, Mehrdad and Alloza, Iraide and Almasan, Alexandru and Almonte-Beceril, Maylin and Alnemri, Emad S and Alonso, Covadonga and Altan-Bonnet, Nihal and Altieri, Dario C and Alvarez, Silvia and Alvarez-Erviti, Lydia and Alves, Sandro and Amadoro, Giuseppina and Amano, Atsuo and Amantini, Consuelo and Ambrosio, Santiago and Amelio, Ivano and Amer, Amal O and Amessou, Mohamed and Amon, Angelika and An, Zhenyi and Anania, Frank A and Andersen, Stig U and Andley, Usha P and Andreadi, Catherine K and Andrieu-Abadie, Nathalie and Anel, Alberto and Ann, David K and Anoopkumar-Dukie, Shailendra and Antonioli, Manuela and Aoki, Hiroshi and Apostolova, Nadezda and Aquila, Saveria and Aquilano, Katia and Araki, Koichi and Arama, Eli and Aranda, Agustin and Araya, Jun and Arcaro, Alexandre and Arias, Esperanza and Arimoto, Hirokazu and Ariosa, Aileen R and Armstrong, Jane L and Arnould, Thierry and Arsov, Ivica and Asanuma, Katsuhiko and Askanas, Valerie and Asselin, Eric and Atarashi, Ryuichiro and Atherton, Sally S and Atkin, Julie D and Attardi, Laura D and Auberger, Patrick and Auburger, Georg and Aurelian, Laure and Autelli, Riccardo and Avagliano, Laura and Avantaggiati, Maria Laura and Avrahami, Limor and Awale, Suresh and Azad, Neelam and Bachetti, Tiziana and Backer, Jonathan M and Bae, Dong-Hun and Bae, Jae-sung and Bae, Ok-Nam and Bae, Soo Han and Baehrecke, Eric H and Baek, Seung-Hoon and Baghdiguian, Stephen and Bagniewska-Zadworna, Agnieszka and ... and Medicinska fakulteten and Region Östergötland and Linköpings universitet and Institutionen för klinisk och experimentell medicin and Diagnostikcentrum and Klinisk patologi och klinisk genetik and Institutionen för medicin och hälsa and Avdelningen för läkemedelsforskning and Avdelningen för cellbiologi
Autophagy, ISSN 1554-8627, 01/2016, Volume 12, Issue 1, pp. 1 - 222
Journal Article
06/2017, Advances in Anatomy, Embryology and Cell Biology, ISBN 9783319524962, Volume 224, 216
Autism spectrum disorder (ASD) affects approximately 1% of the human population and is characterised by a core symptomatology including deficits in social... 
Cytology | Autism spectrum disorders | Brain
eBook
Annals of Anatomy, ISSN 0940-9602, 07/2015, Volume 200, pp. 115 - 117
The Shank family comprises three core postsynaptic scaffold proteins of excitatory synapses in the mammalian brain: Shank1, Shank2 and Shank3. Since mutations... 
Shank2 | Shank1 | Autism | Synapse | ASD | Shank3 | Schizophrenia | ISOFORMS | ANATOMY & MORPHOLOGY | BINDING-PROTEIN | FAMILY | EMERGING ROLE | GENE | MUTATIONS | POSTSYNAPTIC DENSITY | BEHAVIORS | Mental Disorders - genetics | Animals | Humans | Translational Medical Research | Mice | Nervous System Diseases - genetics | Nerve Tissue Proteins - genetics | Mice, Knockout | Proteins | Nervous system diseases | Analysis | Cells | Neurophysiology
Journal Article
2017, Advances in Anatomy, Embryology and Cell Biology, ISBN 3319524968, Volume 224
Autism spectrum disorder (ASD) affects approximately 1 % of the human population and is characterized by a core symptomatology including deficits in social... 
Medicine | Neurosciences | Cell biology | Psychiatry
Web Resource
PLoS Genetics, ISSN 1553-7390, 09/2014, Volume 10, Issue 9, p. e1004580
Journal Article
Trends in Cell Biology, ISSN 0962-8924, 2011, Volume 21, Issue 10, pp. 594 - 603
Intact synaptic homeostasis is a fundamental prerequisite for a healthy brain. Thus, it is not surprising that altered synaptic morphology and function are... 
Pathology | MOLECULAR CHARACTERIZATION | DIFFERENTIAL EXPRESSION | INHIBITORY SYNAPSES | AMYLOID-BETA | ALZHEIMERS-DISEASE | SYNAPTIC-TRANSMISSION | COPY NUMBER VARIATION | SHANK FAMILY | DENSITY PROTEINS | DELETION SYNDROME | CELL BIOLOGY | Chromosomes, Human, Pair 22 - metabolism | Child Development Disorders, Pervasive - metabolism | Schizophrenia - metabolism | Humans | Schizophrenia - pathology | Nervous System Diseases - genetics | Alzheimer Disease - pathology | Intellectual Disability - metabolism | Brain - metabolism | Chromosomes, Human, Pair 22 - genetics | Synapses - metabolism | Child | Nervous System Diseases - pathology | Carrier Proteins - physiology | Chromosome Deletion | Nerve Tissue Proteins - physiology | Child Development Disorders, Pervasive - pathology | Chromosome Disorders - metabolism | Intellectual Disability - pathology | Models, Molecular | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Carrier Proteins - metabolism | Neuronal Plasticity | Alzheimer Disease - metabolism | Mutation | Chromosome Disorders - genetics | Proteins | Alzheimer's disease | Analysis | Brain | Mental disorders | Neurodegenerative diseases | Synaptogenesis | Homeostasis | Schizophrenia | Autism | Signal transduction | Plasticity (synaptic) | Reviews | scaffolds | Plasticity (developmental) | shank protein
Journal Article