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1. Full Text Mycophenolate mofetil versus oral cyclophosphamide in scleroderma-related interstitial lung disease (SLS II): a randomised controlled, double-blind, parallel group trial
by Tashkin, Donald P, Prof and Roth, Michael D, Prof and Clements, Philip J, Prof and Furst, Daniel E, Prof and Khanna, Dinesh, Prof and Kleerup, Eric C, Prof and Goldin, Jonathan, Prof and Arriola, Edgar, PharmD and Volkmann, Elizabeth R, MD and Kafaja, Suzanne, MD and Silver, Richard, Prof and Steen, Virginia, Prof and Strange, Charlie, Prof and Wise, Robert, Prof and Wigley, Fredrick, Prof and Mayes, Maureen, Prof and Riley, David J, Prof and Hussain, Sabiha, MD and Assassi, Shervin, MD and Hsu, Vivien M, MD and Patel, Bela, MD and Phillips, Kristine, MD and Martinez, Fernando, Prof and Golden, Jeffrey, Prof and Connolly, M Kari, Prof and Varga, John, Prof and Dematte, Jane, Prof and Hinchcliff, Monique E, MD and Fischer, Aryeh, MD and Swigris, Jeffrey, DO and Meehan, Richard, MD and Theodore, Arthur, Prof and Simms, Robert, Prof and Volkov, Suncica, MD and Schraufnagel, Dean E, Prof and Scholand, Mary Beth, MD and Frech, Tracy, MD and Molitor, Jerry A, Prof and Highland, Kristin, MD and Read, Charles A, Prof and Fritzler, Marvin J, Prof and Kim, Grace Hyun J, PhD and Tseng, Chi-Hong, PhD and Elashoff, Robert M, Prof and Sclerodema Lung Study II and Sclerodema Lung Study II Investigators
Lancet Respiratory Medicine, The, ISSN 2213-2600, 2016, Volume 4, Issue 9, pp. 708 - 719
Summary Background 12 months of oral cyclophosphamide has been shown to alter the progression of scleroderma-related interstitial lung disease when compared... 
Pulmonary/Respiratory | SURVIVAL | PLACEBO | RESPIRATORY SYSTEM | DYSPNEA | OUTCOMES | SYSTEMIC-SCLEROSIS | CRITICAL CARE MEDICINE | Lung Diseases, Interstitial - etiology | Cyclophosphamide - administration & dosage | Double-Blind Method | Humans | Middle Aged | Male | Treatment Outcome | Lung - physiopathology | Lung Diseases, Interstitial - drug therapy | Disease Progression | Scleroderma, Systemic - complications | Scleroderma, Systemic - physiopathology | Adult | Female | Aged | Lung Diseases, Interstitial - physiopathology | Respiratory Function Tests | Mycophenolic Acid - administration & dosage | Immunosuppressive Agents - administration & dosage
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2. Full Text Genetic Discovery, Rigorous Statistics, and Pandemic Influenza
by Scholand, Mary Beth, MD, FCCP and Liou, Theodore G., MD, FCCP
Chest, ISSN 0012-3692, 2014, Volume 145, Issue 6, pp. 1186 - 1188
Pulmonary/Respiratory | RESPIRATORY SYSTEM | VIRUS | CRITICAL CARE MEDICINE | Humans | Influenza, Human - genetics | Influenza A Virus, H1N1 Subtype | Female | Influenza, Human - virology | Male | Pulmonary Surfactant-Associated Protein B - genetics
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3. Full Text KCNK3 Mutations Are Found in Patients With Pulmonary Arterial Hypertension Associated With Fenfluramine or Dexfenfluramine Ingestion
by Nakayama, Ikue, MD and Scholand, Mary Beth, MD and Best, D., PhD and Sumner, Kelli, PhD and Chung, Wendy, MD and Brown, Lynette, MD and Elliott, C, MD
Chest, ISSN 0012-3692, 2015, Volume 148, Issue 4, pp. 944A - 944B
Pulmonary/Respiratory
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4. Smoking cessation and environmental hygiene
by Pirozzi, Cheryl and Scholand, Mary Beth
Medical Clinics of North America, ISSN 0025-7125, 07/2012, Volume 96, Issue 4, pp. 849 - 867
Although there are nonmodifiable genetic risk factors for chronic obstructive pulmonary disease (COPD), most known risk factors for development and progression... 
Smoking cessation | Air pollution | Environmental hygiene | Occupational exposure | COPD | HOSPITAL ADMISSIONS | PASSIVE SMOKING | LONG-TERM EXPOSURE | LUNG-FUNCTION | RECEPTOR PARTIAL AGONIST | MEDICINE, GENERAL & INTERNAL | OBSTRUCTIVE PULMONARY-DISEASE | TOBACCO-SMOKE | PARTICULATE AIR-POLLUTION | SUSTAINED-RELEASE BUPROPION | RESPIRATORY SYMPTOMS | Smoking - adverse effects | Marijuana Smoking - adverse effects | Air Pollution - prevention & control | Occupational Exposure - adverse effects | Smoke - adverse effects | Humans | Risk Factors | Air Pollution - adverse effects | Smoke - prevention & control | Pulmonary Disease, Chronic Obstructive - etiology | Smoking Cessation - methods | Marijuana Smoking - prevention & control | Tobacco Smoke Pollution - adverse effects | Pulmonary Disease, Chronic Obstructive - prevention & control | Occupational Exposure - prevention & control | Air Pollutants - adverse effects | Hygiene | Tobacco Smoke Pollution - prevention & control
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5. Full Text Relationship of BMPR2 mutations to vasoreactivity in pulmonary arterial hypertension
by Elliott, C. Gregory and Glissmeyer, Eric W and Havlena, Gregory T and Carlquist, John and McKinney, Jason T and Rich, Stuart and McGoon, Michael D and Scholand, Mary Beth and Kim, Miryoung and Jensen, Robert L and Schmidt, Jon W and Ward, Kenneth
Circulation, ISSN 0009-7322, 05/2006, Volume 113, Issue 21, pp. 2509 - 2515
Background - Vasoreactivity tests are fundamental in evaluating pulmonary arterial hypertension (PAH). Mutations of the transforming growth factor-beta type II... 
Genetics | Pulmonary arterial hypertension | Molecular biology | Vasodilation | Vasoconstriction | molecular biology | DIAGNOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | vasodilation | RECEPTOR | CALCIUM-CHANNEL BLOCKERS | INHALED NITRIC-OXIDE | AGENT | genetics | THERAPY | GENE | GERMLINE MUTATIONS | PERIPHERAL VASCULAR DISEASE | vasoconstriction | pulmonary arterial hypertension | Vasoconstriction - genetics | Vasodilator Agents - pharmacology | Bone Morphogenetic Protein Receptors, Type II - genetics | Humans | Middle Aged | Hypertension, Pulmonary - physiopathology | Hypertension, Pulmonary - genetics | Male | Sequence Analysis, DNA | Vasodilation - genetics | DNA Mutational Analysis | Adolescent | Adult | Female | Mutation
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6. Full Text A candidate gene approach identifies the CHRNA5-A3-B4 region as a risk factor for age-dependent nicotine addiction
by Weiss, Robert B and Baker, Timothy B and Cannon, Dale S and Von Niederhausern, Andrew and Dunn, Diane M and Matsunami, Nori and Singh, Nanda A and Baird, Lisa and Coon, Hilary and McMahon, William M and Piper, Megan E and Fiore, Michael C and Scholand, Mary Beth and Connett, John E and Kanner, Richard E and Gahring, Lorise C and Rogers, Scott W and Hoidal, John R and Leppert, Mark F
PLoS Genetics, ISSN 1553-7390, 07/2008, Volume 4, Issue 7, pp. e1000125 - e1000125
People who begin daily smoking at an early age are at greater risk of long-term nicotine addiction. We tested the hypothesis that associations between... 
SMOKING-CESSATION | CIGARETTE-SMOKING | ADULT RATS | CONDITIONED PLACE PREFERENCE | GENETICS & HEREDITY | FAGERSTROM TOLERANCE QUESTIONNAIRE | LUNG-CANCER MORTALITY | SUSCEPTIBILITY LOCUS | POPULATION-SAMPLE | GENOME-WIDE ASSOCIATION | RECEPTOR GENES | Haplotypes | Tobacco Use Disorder - ethnology | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Age Factors | Humans | Middle Aged | Risk Factors | Logistic Models | Male | Smoking - genetics | Nerve Tissue Proteins - genetics | Adolescent | Adult | Female | Tobacco Use Disorder - genetics | Polymorphism, Single Nucleotide | Receptors, Nicotinic - genetics | Cohort Studies | Protein Subunits - genetics | Index Medicus | Studies | Teenagers | Addictions | Smoking | Nicotine
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7. Full Text Hypercapnia increases core temperature cooling rate during snow burial
by Colin K. Grissom and Martin I. Radwin and Mary Beth Scholand and Chris H. Harmston and Mark C. Muetterties and Tim J. Bywater
Journal of Applied Physiology, ISSN 8750-7587, 04/2004, Volume 96, Issue 4, pp. 1365 - 1370
Previous retrospective studies report a core body temperature cooling rate of 3°C/h during avalanche burial. Hypercapnia occurs during avalanche burial... 
Avalanche burial | Avalanche survival | Hypothermia | avalanche survival | LOWERS | SPORT SCIENCES | PHYSIOLOGY | hypothermia | RESPIRATION | ARTIFICIAL AIR POCKET | VICTIMS | COLD | HYPOXIA | avalanche burial | CARBON-DIOXIDE | Hypercapnia - physiopathology | Snow | Humans | Male | Mountaineering | Body Temperature | Carbon Dioxide | Regression Analysis | Rectum - physiopathology | Disasters | Time Factors | Adult | Female | Respiration
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8. Full Text Airway PI3K Pathway Activation Is an Early and Reversible Event in Lung Cancer Development
by Adam M. Gustafson and Raffaella Soldi and Christina Anderlind and Mary Beth Scholand and Jun Qian and Xiaohui Zhang and Kendal Cooper and Darren Walker and Annette McWilliams and Gang Liu and Eva Szabo and Jerome Brody and Pierre P. Massion and Marc E. Lenburg and Stephen Lam and Andrea H. Bild and Avrum Spira
Science Translational Medicine, ISSN 1946-6234, 04/2010, Volume 2, Issue 26, pp. 26ra25 - ra25
Although only a subset of smokers develop lung cancer, we cannot determine which smokers are at highest risk for cancer development, nor do we know the... 
BRONCHIAL EPITHELIUM | TRANSFORMATION | MEDICINE, RESEARCH & EXPERIMENTAL | PROTEIN-KINASE-C | EPITHELIAL GENE-EXPRESSION | SIGNATURES | 3-KINASE | SMOKERS | MUTATIONS | MYOINOSITOL | CELL-LINES | CELL BIOLOGY | Bronchi - enzymology | Epithelial Cells - drug effects | Humans | Middle Aged | Lung Neoplasms - pathology | Phosphatidylinositol 3-Kinases - metabolism | Pulmonary Disease, Chronic Obstructive - pathology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Bronchi - drug effects | Adult | Precancerous Conditions - pathology | Gene Expression Regulation, Neoplastic - drug effects | Bronchi - pathology | Lung Neoplasms - genetics | Inositol - pharmacology | Precancerous Conditions - enzymology | Reproducibility of Results | Lung Neoplasms - enzymology | Enzyme Inhibitors - pharmacology | PTEN Phosphohydrolase - metabolism | Epithelial Cells - pathology | Enzyme Activation - drug effects | Pulmonary Disease, Chronic Obstructive - enzymology | Smoking - metabolism | Epithelial Cells - enzymology | Smoking - pathology | Aged | Cohort Studies
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9. Full Text A defect in myoblast fusion underlies Carey-Fineman-Ziter syndrome
by Di Gioia, Silvio Alessandro and Connors, Samantha and Matsunami, Norisada and Cannavino, Jessica and Rose, Matthew F and Gilette, Nicole M and Artoni, Pietro and De Macena Sobreira, Nara Lygia and Chan, Wai-Man and Webb, Bryn D and Robson, Caroline D and Cheng, Long and Van Ryzin, Carol and Ramirez-Martinez, Andres and Mohassel, Payam and Leppert, Mark and Scholand, Mary Beth and Grunseich, Christopher and Ferreira, Carlos R and Hartman, Tyler and Hayes, Ian M and Morgan, Tim and Markie, David M and Fagiolini, Michela and Swift, Amy and Chines, Peter S and Speck-Martins, Carlos E and Collins, Francis S and Jabs, Ethylin Wang and Bönnemann, Carsten G and Olson, Eric N and Carey, John C and Robertson, Stephen P and Manoli, Irini and Engle, Elizabeth C and Andrews, Caroline V and Barry, Brenda J and Hunter, David G and Mackinnon, Sarah E and Shaaban, Sherin and Erazo, Monica and Frempong, Tamiesha and Hao, Ke and Naidich, Thomas P and Rucker, Janet C and Zhang, Zhongyang and Biesecker, Barbara B and Bonnycastle, Lori L and Brewer, Carmen C and Brooks, Brian P and Butman, John A and Chien, Wade W and Farrell, Kathleen and FitzGibbon, Edmond J and Gropman, Andrea L and Hutchinson, Elizabeth B and Jain, Minal S and King, Kelly A and Lehky, Tanya J and Lee, Janice and Liberton, Denise K and Narisu, Narisu and Paul, Scott M and Sadeghi, Neda and Snow, Joseph and Solomon, Beth and Summers, Angela and Toro, Camilo and Thurm, Audrey and Zalewski, Christopher K and Moebius Syndrome Res Consortium and Moebius Syndrome Research Consortium
Nature Communications, ISSN 2041-1723, 07/2017, Volume 8, Issue 1, p. 16077
Multinucleate cellular syncytial formation is a hallmark of skeletal muscle differentiation. Myomaker, encoded by Mymk (Tmem8c), is a well-conserved plasma... 
MUSCLE-FIBER-TYPE | MEGF10 | CRANIAL MUSCLES | MULTIDISCIPLINARY SCIENCES | WEB TOOL | CONGENITAL MYOPATHIES | MUTATIONS | DISPROPORTION | MYOMAKER | GENOME | ROBIN-SEQUENCE | Humans | Infant | Male | Muscle Proteins - deficiency | Muscle, Skeletal - metabolism | Genetic Complementation Test | Cell Fusion | Membrane Proteins - deficiency | Myoblasts - metabolism | Adult | Female | Child | Pierre Robin Syndrome - pathology | Disease Models, Animal | Amino Acid Sequence | Gene Expression | Morphogenesis - genetics | Muscle, Skeletal - growth & development | Membrane Proteins - genetics | Muscular Diseases - metabolism | Mobius Syndrome - pathology | Mobius Syndrome - metabolism | Zebrafish | Muscular Diseases - pathology | Myoblasts - pathology | Genes, Recessive | Mobius Syndrome - genetics | Muscle Proteins - genetics | Pierre Robin Syndrome - metabolism | Sequence Homology, Amino Acid | Sequence Alignment | Zebrafish Proteins - deficiency | Animals | Pierre Robin Syndrome - genetics | Pedigree | Embryo, Nonmammalian | Muscle, Skeletal - pathology | Muscular Diseases - genetics | Mutation | Zebrafish Proteins - genetics | Myotubes | Coding | Disorders | Differentiation | Cell fusion | Fusion protein | In vitro methods and tests | Skeletal muscle | Myopathy | Membrane proteins
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10. Full Text Comparison of serum, EDTA plasma and P100 plasma for luminex-based biomarker multiplex assays in patients with chronic obstructive pulmonary disease in the SPIROMICS study
by O'Neal, Wanda K and Anderson, Wayne and Basta, Patricia V and Carretta, Elizabeth E and Doerschuk, Claire M and Barr, R G and Bleecker, Eugene R and Christenson, Stephanie A and Curtis, Jeffrey L and Han, Meilan K and Hansel, Nadia N and Kanner, Richard E and Kleerup, Eric C and Martinez, Fernando J and Miller, Bruce E and Peters, Stephen P and Rennard, Stephen I and Scholand, Mary B and Tal-Singer, Ruth and Woodruff, Prescott G and Couper, David J and Davis, Sonia M and Reporting SPIROMICS Investigators and SPIROMICS Investigators and reporting for SPIROMICS Investigators
Journal of Translational Medicine, ISSN 1479-5876, 01/2014, Volume 12, Issue 1, pp. 9 - 9
Background: As a part of the longitudinal Chronic Obstructive Pulmonary Disease (COPD) study, Subpopulations and Intermediate Outcome Measures in COPD study... 
Multiplex assays | Pilot study | Blood analytes | Biomarkers | P100 plasma | Serum | Chronic obstructive pulmonary disease | EDTA plasma | SPIROMICS | COPD | MEDICINE, RESEARCH & EXPERIMENTAL | COLLECTION | ACTIVATION | ANTICOAGULANTS | PROTEOME | BLOOD | Diagnostic Techniques, Respiratory System | Plasma - metabolism | Reproducibility of Results | Edetic Acid - chemistry | Serum - metabolism | Humans | Middle Aged | Protease Inhibitors | Pulmonary Disease, Chronic Obstructive - blood | Female | Male | Biomarkers - blood | Proteins | Medicine | Studies | Plasma | Anticoagulants | Blood platelets | Cytokines | Internal medicine | Pathogenesis
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11. Full Text Genomics and response to long-term oxygen therapy in chronic obstructive pulmonary disease
by Seo, Minseok and Qiu, Weiliang and Bailey, William and Criner, Gerard J and Dransfield, Mark T and Fuhlbrigge, Anne L and Reilly, John J and Scholand, Mary Beth and Castaldi, Peter and Chase, Robert and Parker, Margaret and Saferali, Aabida and Yun, Jeong H and Crapo, James D and Cho, Michael H and Beaty, Terri H and Silverman, Edwin K and Hersh, Craig P
Journal of Molecular Medicine, ISSN 0946-2716, 12/2018, Volume 96, Issue 12, pp. 1375 - 1385
Chronic obstructive pulmonary disease (COPD) is a leading cause of death worldwide, and long-term oxygen therapy has been shown to reduce mortality in COPD... 
Expression quantitative trait loci | Oxygen | Arylsulfatase B | Genome-wide association study | Microarray | Pharmacogenomics | MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | GENETIC SUSCEPTIBILITY | EMPHYSEMA | GENETICS & HEREDITY | MITOCHONDRIAL DYSFUNCTION | RETINOPATHY | EXPRESSION | Pharmacogenetics | Lung diseases, Obstructive | Care and treatment | Usage | Oxygen therapy | Genetic aspects | Research | Respiratory therapy | Therapy | Epithelial cells | Genes | Genomics | Lung diseases | Oxygen enrichment | Single-nucleotide polymorphism | Gene expression | Patients | Loci | Blood | Quantitative trait loci | Mitochondria | Hypoxemia | DNA microarrays | Network analysis | Obstructive lung disease | Peripheral blood | Biomarkers | Arylsulfatase | Chronic obstructive pulmonary disease | expression quantitative trait loci | microarray | pharmacogenomics | oxygen | genome-wide association study
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12. Full Text Preoperative Evaluation of Patients With Interstitial Lung Disease
by Patel, Nina M and Kulkarni, Tejaswini and Dilling, Daniel and Scholand, Mary Beth and Gupta, Nishant and Strek, Mary and Allam, Joanne Shirine and de Andrade, Joao and Lancaster, Lisa and Carbone, Roberto and D’Annunzio, Samantha and Wickramarathne, Avanthika Thanushi and Luckhardt, Tracy and Kershaw, Corey and Interstitial and Diffuse Lung Disease Network Steering Committee
Chest, ISSN 0012-3692, 11/2019, Volume 156, Issue 5, pp. 826 - 833
interstitial lung disease | acute lung injury | surgery | OBSTRUCTIVE SLEEP-APNEA | RISK-FACTORS | RESECTION | BIOPSY | NONPULMONARY SURGERY | GENERAL-ANESTHESIA | ABDOMINAL-SURGERY | RESPIRATORY SYSTEM | IDIOPATHIC PULMONARY-FIBROSIS | ACUTE EXACERBATION | RESPIRATORY-FAILURE | CRITICAL CARE MEDICINE
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13. Full Text Severity of cough in idiopathic pulmonary fibrosis is associated with MUC5 B genotype
by Scholand, Mary B and Wolff, Roger and Crossno, Peter F and Sundar, Krishna and Winegar, Molly and Whipple, Spencer and Carey, Patrick and Sunchild, Nicholas and Coon, Hilary
Cough, ISSN 1745-9974, 05/2014, Volume 10, Issue 1, pp. 3 - 3
A polymorphism (rs35705950) in the promoter region of the mucin MUC5B is associated with both familial and sporadic forms of idiopathic pulmonary fibrosis.... 
Genetic aspects | Pulmonary fibrosis | Analysis | Genetic polymorphisms
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14. Full Text Multistudy Fine Mapping of Chromosome 2q Identifies XRCC5 as a Chronic Obstructive Pulmonary Disease Susceptibility Gene
by Hersh, C.P and Pillai, S.G and Zhu, G and Lomas, D.A and Bakke, P and Gulsvik, A and Demeo, D.L and Klanderman, B.J and Lazarus, R and Litonjua, A.A and Sparrow, D and Reilly, J.J and Agusti, A and Calverley, P.M and Donner, C.F and Levy, R.D and Make, B.J and Pare, P D and Rennard, S.I and Vestbo, J and Wouters, E.F and Scholand, M.B and Coon, H and Hoidal, J and Silverman, E.K
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 01/2010, Volume 182, Issue 5, pp. 605 - 613
Rationale: Several family-based studies have identified genetic linkage for lung function and airflow obstruction to chromosome 2q. Objectives: We hypothesized... 
Single nucleotide polymorphism | Emphysema | Genetic linkage | Metaanalysis | metaanalysis | PROTEIN | single nucleotide polymorphism | SERPINE2 GENE | genetic linkage | RISK | MECHANISMS | emphysema | RESPIRATORY SYSTEM | EARLY-ONSET | SENESCENCE | LINKAGE | EXPRESSION | COPD | CRITICAL CARE MEDICINE | Smoking - adverse effects | Genetic Predisposition to Disease | Humans | Middle Aged | Male | Chromosome Mapping | Case-Control Studies | Chromosomes, Human, Pair 2 | Amyloid beta-Protein Precursor - genetics | Ku Autoantigen | Age of Onset | Protease Nexins | Female | Aged | Polymorphism, Single Nucleotide | DNA Helicases - genetics | Genetic Linkage | Pulmonary Disease, Chronic Obstructive - genetics | Receptors, Cell Surface - genetics | A. Chronic Obstructive Pulmonary Disease
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15. Full Text Laparoscopic anti-reflux surgery for the treatment of idiopathic pulmonary fibrosis (WRAP-IPF): a multicentre, randomised, controlled phase 2 trial
by Raghu, Ganesh and Pellegrini, Carlos A and Yow, Eric and Flaherty, Kevin R and Meyer, Keith and Noth, Imre and Scholand, Mary Beth and Cello, John and Ho, Lawrence A and Pipavath, Sudhakar and Lee, Joyce S and Lin, Jules and Maloney, James and Martinez, Fernando J and Morrow, Ellen and Patti, Marco G and Rogers, Stan and Wolters, Paul J and Yates, Robert and Anstrom, Kevin J and Collard, Harold R
The Lancet Respiratory Medicine, ISSN 2213-2600, 09/2018, Volume 6, Issue 9, pp. 707 - 714
Abnormal acid gastro-oesophageal reflux (GER) is hypothesised to play a role in progression of idiopathic pulmonary fibrosis (IPF). We aimed to determine... 
ACID GASTROESOPHAGEAL-REFLUX | ANTACID THERAPY | RESPIRATORY SYSTEM | PIRFENIDONE | MICROASPIRATION | PREVALENCE | DISEASE PROGRESSION | ACUTE EXACERBATION | TRANSPLANTATION | CRITICAL CARE MEDICINE | Care and treatment | Usage | Pulmonary fibrosis | Laparoscopic surgery | Laparoscopy | Diagnosis | Research
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