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Cancer Cell, ISSN 1535-6108, 2006, Volume 10, Issue 3, pp. 175 - 176
Reactive oxygen species and tumor biology are intertwined in a complex web, making it difficult to understand which came first, whether oxidants are required... 
APOPTOSIS | LYMPHOMA | ONCOLOGY | TUMORIGENICITY | Neoplasms - metabolism | Neoplasms - complications | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Thioredoxins - metabolism | Antineoplastic Agents - therapeutic use | Neoplasms - drug therapy | Development and progression | Cancer
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 10/2016, Volume 55, Issue 4, pp. v - vi
Journal Article
Cancer Cell, ISSN 1535-6108, 02/2015, Volume 27, Issue 2, pp. 156 - 157
Reactive oxygen species (ROS) can initiate cancer, but oxidant generation in tumors leaves them vulnerable to further stresses. In this issue of , Harris and... 
CELLS | ONCOLOGY | ROS | CELL BIOLOGY | Animals | Breast Neoplasms - genetics | Antioxidants - metabolism | Humans | Female | Glutamate-Cysteine Ligase - genetics | Mammary Neoplasms, Animal - genetics | Development and progression | Dancing | Cancer
Journal Article
Molecular Aspects of Medicine, ISSN 0098-2997, 02/2016, Volume 47-48, pp. 76 - 89
Mitochondria are responsible for the majority of oxygen consumption in cells, and thus represent a conceptually appealing site for cellular oxygen sensing.... 
oxidant signaling | Hypoxia | electron transport chain | metabolism | Animals | Reactive Oxygen Species - metabolism | Oxidation-Reduction | Signal Transduction | Models, Biological | Humans | Mitochondria - metabolism | Oxygen - metabolism | Disease Models, Animal | Hypoxia - metabolism
Journal Article
AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, ISSN 1044-1549, 07/2019, Volume 61, Issue 1, pp. 1 - 2
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 05/2018, Volume 58, Issue 5, pp. 553 - 554
[...]hydroxyurea may act by augmenting the generation of nitric oxide (NO), which promotes vasodilation and inhibits platelet aggregation by activating soluble... 
RESPIRATORY SYSTEM | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | Pain | Sickle cell anemia | Pulmonary arteries | Nitric oxide | Mortality | Mutation | Sickle cell disease | Guanylate cyclase | Pulmonary hypertension
Journal Article
Immunity, ISSN 1074-7613, 02/2013, Volume 38, Issue 2, pp. 225 - 236
It is widely appreciated that T cells increase glycolytic flux during activation, but the role of mitochondrial flux is unclear. Here, we have shown that... 
COMPLEX-III | SUPEROXIDE | PROTEIN | GLUCOSE-METABOLISM | LYMPHOCYTE ACTIVATION | IMMUNE-RESPONSES | AEROBIC GLYCOLYSIS | TRANSDUCTION | PROLIFERATION | IMMUNOLOGY | GLUTAMINE UPTAKE | Electron Transport Complex III - metabolism | Cell Proliferation | Reactive Oxygen Species - metabolism | Iron-Sulfur Proteins - genetics | Homeodomain Proteins - immunology | Mitochondria - immunology | Interleukin-2 - immunology | NFATC Transcription Factors - immunology | Mitochondria - genetics | T-Lymphocytes - metabolism | Lymphopenia - metabolism | Female | Tumor Necrosis Factor Receptor Superfamily, Member 7 - immunology | Gene Expression | Signal Transduction | Lymphocyte Activation | Mitochondria - metabolism | Tumor Necrosis Factor Receptor Superfamily, Member 7 - genetics | Homeodomain Proteins - genetics | Mice, Knockout | Animals | Iron-Sulfur Proteins - deficiency | T-Lymphocytes - immunology | Lymphopenia - immunology | Mice | Interleukin-2 - biosynthesis | NFATC Transcription Factors - genetics | Antigens | Medical colleges | Glucose metabolism | Cytochrome b | Mitochondrial DNA | Glucose | T cells | Dextrose | Flow cytometry | Oxygen | Lymphatic system | Transcription factors | Laboratories | T cell receptors | Infections | Biosynthesis | Metabolism | Cell growth | Mitochondria | Metabolites | Immune system | Apoptosis
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 10/2016, Volume 55, Issue 4, pp. v - vi
The American Journal of Respiratory Cell and Molecular Biology (AJRCMB), also known as the Red Journal, was established by the American Thoracic Society (ATS)... 
RESPIRATORY SYSTEM | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | Publishing | Funding | Journals | Science | Appeals | Editorials | Scientists | Molecular biology | Investigations | Manuscripts
Journal Article
Respiratory Physiology & Neurobiology, ISSN 1569-9048, 2010, Volume 174, Issue 3, pp. 201 - 211
Journal Article
American Journal of Respiratory Cell and Molecular Biology (Online), 10/2016, Volume 55, Issue 4, pp. V - V
The American Journal of Respiratory Cell and Molecular Biology (AJRCMB), also known as the Red Journal, was established by the American Thoracic Society (ATS)... 
Journal Article
Nature, ISSN 0028-0836, 12/2010, Volume 468, Issue 7324, pp. 696 - 700
Parkinson's disease is a pervasive, ageing-related neurodegenerative disease the cardinal motor symptoms of which reflect the loss of a small group of neurons,... 
PATHOGENESIS | CELLS | SUBSTANTIA-NIGRA NEURONS | MULTIDISCIPLINARY SCIENCES | DYSFUNCTION | MUTATIONS | MITOCHONDRIAL-DNA DELETIONS | CALCIUM-CHANNEL BLOCKERS | PARKINSONS-DISEASE | Oncogene Proteins - genetics | Oxidative Stress | Calcium - metabolism | Iridoid Glycosides - pharmacology | Male | Neurons - cytology | Cyclophilins - metabolism | Calcium Channels, L-Type - pharmacology | Brain - metabolism | Iridoids | Mitochondrial Proteins - metabolism | Gene Deletion | Superoxides - metabolism | Neurons - metabolism | Parkinson Disease - metabolism | Calcium Signaling | Dopamine - metabolism | Brain - cytology | Parkinson Disease - pathology | Purines - pharmacology | Mitochondrial Proteins - antagonists & inhibitors | Oncogene Proteins - metabolism | Mice, Transgenic | Mitochondria - metabolism | Calcium Channel Blockers - pharmacology | Ion Channels - antagonists & inhibitors | Peroxiredoxins | Protein Deglycase DJ-1 | Parkinson Disease - prevention & control | Animals | Biological Clocks - physiology | Ion Channels - metabolism | Dihydropyridines - pharmacology | Oncogene Proteins - deficiency | Mice | Calcium Channels, L-Type - metabolism | Uncoupling Protein 1 | Development and progression | Calcium channels | Parkinson's disease | Dopaminergic mechanisms | Health aspects | Green fluorescent protein | Proteins | Neurotransmitters | Neurons | Parkinsons disease
Journal Article