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1995, Lecture notes in mathematics, ISBN 9783540600169, Volume 1607., 155
Hodge theory is a standard tool in characterizing differ- ential complexes and the topology of manifolds. This book is a study of the Hodge-Kodaira and related... 
Hodge theory | Boundary value problems | Numerical solutions | Decomposition (Mathematics) | Potential theory (Mathematics) | Mathematics | Potential Theory | Manifolds and Cell Complexes (incl. Diff.Topology) | Cell aggregation
Book
Electrochimica Acta, ISSN 0013-4686, 05/2016, Volume 199, pp. 280 - 289
Mediated electrocatalytic voltammetry of human sulfite oxidase (HSO) is demonstrated with synthetic one electron transfer iron complexes... 
enzyme | sulfite | molybdenum | Oxidases | Molybdenum compounds | Sulfites | Wine | Amines | Detectors | Sulfates | Molybdenum | Human | Electrodes | Electrocatalysis | Catalysts | Oxidase | Voltammetry | Catalysis
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 02/2019, Volume 176, Issue 4, pp. 554 - 570
Journal Article
Journal Article
Pediatrics, ISSN 0031-4005, 05/2010, Volume 125, Issue 5, pp. e1249 - e1254
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2017, Volume 127, Issue 12, pp. 4365 - 4378
Molybdenum cofactor deficiency (MoCD) is an autosomal recessive inborn error of metabolism characterized by neurodegeneration and death in early childhood. The... 
ENCEPHALOPATHY | MEDICINE, RESEARCH & EXPERIMENTAL | BIOLOGICAL FUNCTION | GEPHYRIN | METABOLISM | CALCIUM | INJURY | SULFITE OXIDASE DEFICIENCY | MOLECULAR-BASIS | DEHYDROGENASE | INBORN-ERRORS | Cysteine - analogs & derivatives | Metal Metabolism, Inborn Errors - metabolism | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Synapses - pathology | Neurodegenerative Diseases - drug therapy | Tungsten Compounds - toxicity | Synapses - metabolism | HEK293 Cells | Organophosphorus Compounds - pharmacology | Metal Metabolism, Inborn Errors - drug therapy | Cysteine - metabolism | GABAergic Neurons - metabolism | Calcium Signaling | Disease Models, Animal | Memantine - pharmacology | Neurodegenerative Diseases - pathology | Metal Metabolism, Inborn Errors - pathology | Neurodegenerative Diseases - genetics | GABAergic Neurons - pathology | Neurodegenerative Diseases - metabolism | Animals | Pterins - pharmacology | Mice | Neurons | Physiological aspects | GABA | Aspartate | Brain damage | Calpain | Glutamate | Molybdenum | Metabolism, Inborn errors of | Methyl aspartate | Care and treatment | Cellular signal transduction | Research | Inborn errors of metabolism | Glutamic acid receptors (ionotropic) | Homeostasis | Antagonists | Infants | Calcium influx | Proteins | Neurotoxicity | γ-Aminobutyric acid | Metabolites | Neurodegeneration | Rodents | Children | Urine | Enzymes | N-Methyl-D-aspartic acid receptors | Metabolism | Gephyrin | Memantine | Molecular modelling | Excitotoxicity | Mutation | Alzheimers disease | Brain injury | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2014, Volume 9, Issue 1, pp. e86030 - e86030
Journal Article