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Methods in Molecular Biology, ISSN 1064-3745, 2017, Volume 1522, pp. 73 - 82
Here we report methods of preparation for liposome formulations containing lipophilic drugs. In contrast to the encapsulation of water soluble compounds into... 
Lipophilic ara-C drugs | Duplex drugs | Liposomes | Lipophilic drugs | NOAC | Hydrophobic and Hydrophilic Interactions | Liposomes - chemistry | Dialysis | Filtration | Pressure | Pharmaceutical Preparations - chemistry
Journal Article
Journal Article
Methods in molecular biology (Clifton, N.J.), 2010, Volume 605, pp. 129 - 138
Here, we report methods of preparation for liposome formulations containing lipophilic drugs. In contrast to the encapsulation of water-soluble compounds into... 
Liposomes - chemistry | Nucleosides - chemistry | Nucleosides - administration & dosage | Pharmaceutical Preparations - chemistry | Hydrophobic and Hydrophilic Interactions | Dialysis | Molecular Structure | Pharmaceutical Preparations - administration & dosage | Pressure
Journal Article
PLoS Pathogens, ISSN 1553-7366, 11/2013, Volume 9, Issue 11, p. e1003735
Chronic viral infections lead to CD8(+) T cell exhaustion, characterized by impaired cytokine secretion. Presence of the immune-regulatory cytokine IL-10... 
HELPER-CELL | POSTTRANSCRIPTIONAL REGULATION | ACTIVATION | DENDRITIC CELLS | INTERLEUKIN-10 RECEPTOR | DEPLETION | MICROBIOLOGY | RESPONSES | VIROLOGY | C VIRUS-INFECTION | EXPANSION | GENE-EXPRESSION | PARASITOLOGY | Interleukin-10 | Physiological aspects | Host-parasite relationships | Genetic aspects | Macrophages | T cells | Health aspects | Infections | Immunology | Viral infections | Immune system
Journal Article
PLoS Pathogens, ISSN 1553-7366, 10/2012, Volume 8, Issue 10, p. e1002976
Viruses use a limited set of host pathways for infection. These pathways represent bona fide antiviral targets with low likelihood of viral resistance. We... 
RAT-LIVER | LOW-PH | CELLS | PLECONARIL | VIRUS | ENTRY | HUMAN RHINOVIRUSES | MICROBIOLOGY | NITAZOXANIDE | RESPIRATORY SYNDROME CORONAVIRUS | REPLICATION | VIROLOGY | PARASITOLOGY | Influenza viruses | Physiological aspects | Antiviral agents | Amides | Rhinoviruses | Health aspects | Proteins | Antibiotics | Microscopy | Viruses | Infections | Genomes | FDA approval | Drug dosages | Viral infections
Journal Article
Molecular Cancer, ISSN 1476-4598, 04/2011, Volume 10, Issue 1, pp. 36 - 36
Background: Most bladder cancer patients experience lymphatic metastasis in the course of disease progression, yet the relationship between lymphangiogenesis... 
PROGNOSTIC IMPLICATIONS | NODE METASTASIS | ACTIVATION | INHIBITION | ANGIOGENESIS | ONCOLOGY | FACTOR-C | TRANSITIONAL-CELL CARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | NETWORK FORMATION | TUMOR-ASSOCIATED MACROPHAGES | CHEMOTHERAPY | Immunohistochemistry | Lymph Nodes - pathology | Gene Expression Regulation, Neoplastic | Neovascularization, Pathologic | Antigens, CD - genetics | Antigens, CD - metabolism | CD11b Antigen - genetics | Flow Cytometry | Urinary Bladder Neoplasms - genetics | Urinary Bladder Neoplasms - pathology | Female | Lymphangiogenesis | Vascular Endothelial Growth Factor D - genetics | Urinary Bladder Neoplasms - metabolism | Macrophages - pathology | Signal Transduction | Solubility | Lymph Nodes - metabolism | Lymphatic Metastasis | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Antigens, Differentiation, Myelomonocytic - genetics | Mice, Inbred C3H | Vascular Endothelial Growth Factor C - genetics | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Vascular Endothelial Growth Factor C - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Cell Line, Tumor | Mice | CD11b Antigen - metabolism | Vascular Endothelial Growth Factor Receptor-3 - genetics | Physiological aspects | Development and progression | Metastasis | Research | Growth factor receptors | Neovascularization | Bladder cancer
Journal Article
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 06/2008, Volume 14, Issue 6, pp. 633 - 640
To understand the pathogenesis of chronic inflammatory disease, we analyzed an experimental mouse model of chronic lung disease with pathology that resembles... 
MEDICINE, RESEARCH & EXPERIMENTAL | HYPERREACTIVITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | EXPERIMENTAL ASTHMA | AIRWAY INFLAMMATION | IL-13 | GENETIC SEGREGATION | CELL BIOLOGY | V(ALPHA)14 NKT CELLS | PATHWAY | ALLERGIC-ASTHMA | KILLER T-CELLS | MICE | Immunohistochemistry | Pulmonary Disease, Chronic Obstructive - virology | RNA, Messenger - analysis | Pulmonary Disease, Chronic Obstructive - physiopathology | RNA, Messenger - metabolism | Respirovirus Infections - virology | Case-Control Studies | Antigens, CD - metabolism | CD4-Positive T-Lymphocytes - immunology | Interleukin-13 - biosynthesis | Interleukin-13 - genetics | Time Factors | Mucins - analysis | Mucins - metabolism | Killer Cells, Natural - immunology | Respirovirus Infections - immunology | Macrophages - immunology | Disease Models, Animal | Pulmonary Disease, Chronic Obstructive - genetics | Respirovirus Infections - genetics | Sendai virus - physiology | Killer Cells, Natural - virology | Mucin 5AC | Mice, Inbred C57BL | Cells, Cultured | Respirovirus Infections - physiopathology | Immunity, Innate | Mice, Knockout | Models, Immunological | Animals | Mice | Pulmonary Disease, Chronic Obstructive - immunology | Prevention | Usage | Care and treatment | Immune response | Lung diseases | Models | Research | T cells | Health aspects | Risk factors | Clinical trials | Infections | Immunology | Rodents
Journal Article