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1963, Bibliothek der Antike. Griechische Reihe
Book
2017, Wiener Jahrbuch für jüdische Geschichte Kultur & Museumswesen, ISBN 9783706556446, Volume Band 11 - 2015/16, 173
Book
Circulation Research, ISSN 0009-7330, 08/2017, Volume 121, Issue 4, pp. 368 - 375
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2018, Volume 128, Issue 5, pp. 1720 - 1723
Current therapies for pulmonary arterial hypertension (PAH) provide symptomatic relief and improve prognosis but fall short of improving long-term survival.... 
Hypertension | Heart failure | Disease | Cytokines | Pathogenesis | Therapeutic applications | Lung | Smooth muscle | Arthritis | Inflammation | Gene expression | Pulmonary hypertension | Interleukin 6 | Interleukin 6 receptors | Medical prognosis | Fibroblasts | Biomarkers | Hypoxia | Chronic obstructive pulmonary disease
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 03/2009, Volume 296, Issue 3, pp. 442 - 452
Macrophage migration inhibitory factor (MIF) is a pleiotropic proinflammatory cytokine involved in acute lung injury and other processes such as wound repair... 
Macrophage migration inhibitory factor | Epithelial proliferation | Resolution of inflammation | Lung | PHYSIOLOGY | VIRUS-INFECTION | PRIMARY CULTURE | macrophage migration inhibitory factor | CYCLIN D1 EXPRESSION | PROTEIN-KINASE | FACTOR MIF | ADHESION MOLECULES | resolution of inflammation | lung | IN-VITRO | KERATINOCYTE GROWTH-FACTOR | MESSENGER-RNA | RESPIRATORY SYSTEM | epithelial proliferation | GENE-EXPRESSION | Cell Proliferation | Acute Lung Injury - genetics | DNA Primers - genetics | Peptides - metabolism | Transfection | Base Sequence | Pulmonary Alveoli - cytology | Histocompatibility Antigens Class II - metabolism | Cell Differentiation | Epithelial Cells - cytology | Antigens, Differentiation, B-Lymphocyte - genetics | Acute Lung Injury - physiopathology | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Epithelial Cells - classification | Acute Lung Injury - pathology | Macrophage Migration-Inhibitory Factors - pharmacology | Phenotype | Animals | Epithelial Cells - immunology | Macrophage Migration-Inhibitory Factors - physiology | Acute Lung Injury - immunology | Mice | Antigens, Differentiation, B-Lymphocyte - metabolism | Histocompatibility Antigens Class II - genetics | Pulmonary Alveoli - immunology | Lung diseases | Rodents | Gene expression | DNA repair | Cells | Binding sites | Tumors | Cell adhesion & migration | Index Medicus
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 05/2010, Volume 55, Issue 18, pp. 1915 - 1922
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, pp. e79798 - e79798
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 10/2008, Volume 178, Issue 8, pp. 838 - 846
Rationale The molecular pathomechanisms underlying idiopathic pulmonary fibrosis (IPF) are elusive, but chronic epithelial injury has recently been suggested... 
C/EBP-homologous protein | X-box binding protein-1 | Type II alveolar epithelial cell | Interstitial lung disease | Lung fibrosis | LUNG | OXIDATIVE STRESS | ACTIVATION | INTERSTITIAL PNEUMONIA | lung fibrosis | CELL-DEATH | PATHOGENESIS | UNFOLDED PROTEIN RESPONSE | interstitial lung disease | RESPIRATORY SYSTEM | CHOP | MYOFIBROBLASTS | MUTATIONS | type II alveolar epithelial cell | CRITICAL CARE MEDICINE | Immunohistochemistry | Humans | Middle Aged | Oxidative Stress - physiology | Endoplasmic Reticulum - metabolism | Male | Pulmonary Fibrosis - genetics | Pulmonary Disease, Chronic Obstructive - pathology | Activating Transcription Factor 6 - genetics | X-Box Binding Protein 1 | RNA - genetics | Activating Transcription Factor 6 - biosynthesis | Endoplasmic Reticulum - pathology | Pulmonary Alveoli - metabolism | Female | Pulmonary Fibrosis - metabolism | Pulmonary Disease, Chronic Obstructive - metabolism | Pulmonary Disease, Chronic Obstructive - genetics | In Situ Nick-End Labeling | Severity of Illness Index | Gene Expression | Pulmonary Alveoli - pathology | Activating Transcription Factor 4 - genetics | Transcription Factors - biosynthesis | Pulmonary Fibrosis - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Regulatory Factor X Transcription Factors | Blotting, Western | Activating Transcription Factor 4 - biosynthesis | Apoptosis - physiology | DNA-Binding Proteins - biosynthesis | Index Medicus | Abridged Index Medicus | EBP-homologous protein | E. Interstitial Lung Disease
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2009, Volume 119, Issue 4, pp. 772 - 787
Idiopathic pulmonary fibrosis (IPF) is characterized by distorted lung architecture and loss of respiratory function. Enhanced (myo)fibroblast activation, ECM... 
EPITHELIAL-MESENCHYMAL TRANSITION | PATHOGENESIS | MEDICINE, RESEARCH & EXPERIMENTAL | LUNG FIBROSIS | CELLS | APOPTOSIS | TISSUE GROWTH-FACTOR | CCN FAMILY | MECHANISMS | EXPRESSION | INTERSTITIAL PNEUMONIAS | Oncogene Proteins - genetics | Up-Regulation | Idiopathic Pulmonary Fibrosis - genetics | Epithelial Cells - metabolism | Humans | Middle Aged | Male | Pulmonary Fibrosis - genetics | Wnt Proteins - metabolism | Oncogene Proteins - physiology | CCN Intercellular Signaling Proteins | Idiopathic Pulmonary Fibrosis - etiology | Pulmonary Alveoli - metabolism | Adult | Female | Intracellular Signaling Peptides and Proteins - genetics | Pulmonary Fibrosis - etiology | Bleomycin - toxicity | Disease Models, Animal | Pulmonary Alveoli - pathology | Mice, Inbred C57BL | Oncogene Proteins - pharmacology | Epithelial Cells - pathology | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Recombinant Proteins - pharmacology | beta Catenin - metabolism | Animals | Models, Biological | Proto-Oncogene Proteins - physiology | Pulmonary Fibrosis - physiopathology | Cell Proliferation - drug effects | Idiopathic Pulmonary Fibrosis - physiopathology | Mice | Intracellular Signaling Peptides and Proteins - physiology | Cellular proteins | Pulmonary fibrosis | Genes | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Nature medicine, ISSN 1078-8956, 11/2014, Volume 20, Issue 11, pp. 1289 - 1300
Pulmonary hypertension (PH) is characterized by increased proliferation and apoptosis resistance of pulmonary artery smooth muscle cells (PASMCs). Forkhead box... 
SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL PROLIFERATION | NUCLEAR EXPORT | PACLITAXEL | CELL BIOLOGY | MAINTENANCE | ARTERIAL-HYPERTENSION | MOLECULAR PATHOGENESIS | GROWTH-FACTOR | INHIBITORS | MORPHOGENETIC PROTEIN-RECEPTOR | Small Molecule Libraries - pharmacology | Paclitaxel - pharmacology | Muscle, Smooth, Vascular - metabolism | Apoptosis - drug effects | Humans | Hypertension, Pulmonary - physiopathology | Apoptosis - genetics | Male | Quinolones - pharmacology | RNA, Messenger - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Bone Morphogenetic Proteins - metabolism | Forkhead Transcription Factors - metabolism | Gene Deletion | Adult | Female | Bromodeoxyuridine - metabolism | Vascular Remodeling - drug effects | Muscle, Smooth, Vascular - drug effects | Lung - pathology | Cytokines - metabolism | RNA, Messenger - genetics | Rats | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Lung - physiopathology | Signal Transduction - genetics | Forkhead Transcription Factors - genetics | Gene Expression Regulation - drug effects | Muscle, Smooth, Vascular - pathology | Animals | Signal Transduction - drug effects | Vascular Remodeling - genetics | Cell Proliferation - drug effects | Mice | Lung - blood supply | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Transcription factors | Research | Analysis | Pulmonary hypertension | Smooth muscle | Cell growth | Pulmonary arteries | Apoptosis | Index Medicus
Journal Article