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Annals of Oncology, ISSN 0923-7534, 11/2016, Volume 27, Issue suppl_7
Journal Article
Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. 3870 - 3870
Journal Article
Nihon rinsho. Japanese journal of clinical medicine, ISSN 0047-1852, 08/2015, Volume 73, Issue 8, pp. 1330 - 1335
Journal Article
Nature, ISSN 0028-0836, 09/2012, Volume 489, Issue 7415, pp. 313 - 317
Cornelia de Lange syndrome (CdLS) is a dominantly inherited congenital malformation disorder, caused by mutations in the cohesin-loading protein NIPBL1,2 for... 
SISTER-CHROMATID COHESION | NIPPED-B | COMPLEX | NIPBL | HUMAN GENOME | RNA-SEQ | MULTIDISCIPLINARY SCIENCES | X-CHROMOSOME-INACTIVATION | S-PHASE | PROTEINS | BINDING | Chromatin - metabolism | Chondroitin Sulfate Proteoglycans - chemistry | Humans | Crystallography, X-Ray | Male | Phosphoproteins - metabolism | Cell Cycle Proteins - chemistry | Chromatin Immunoprecipitation | Repressor Proteins - deficiency | De Lange Syndrome - metabolism | Fibroblasts | Female | Transcription, Genetic | Acetylation | Binding Sites | Repressor Proteins - metabolism | De Lange Syndrome - genetics | Repressor Proteins - chemistry | Chromosomal Proteins, Non-Histone - metabolism | Histone Deacetylases - genetics | Cell Cycle Proteins - metabolism | Chondroitin Sulfate Proteoglycans - metabolism | Histone Deacetylases - chemistry | Histone Deacetylases - deficiency | Mutant Proteins - genetics | Prophase | Models, Molecular | Repressor Proteins - genetics | Histone Deacetylases - metabolism | Mutant Proteins - metabolism | Nuclear Proteins - metabolism | Mutation - genetics | Proteins - genetics | Mutant Proteins - chemistry | Protein Conformation | HeLa Cells | Adaptor Proteins, Signal Transducing - metabolism | Anaphase | Chromatin - genetics | Chromosomal Proteins, Non-Histone - chemistry | Genetics | De Lange syndrome | Genetic aspects | Research | Mutation (Biology) | Proteins | Cell culture | Genes | Cell cycle | Mutation | Females | Chromosomes | Crystal structure | Index Medicus | Chromatin | Repressor Proteins | Life Sciences | Phosphoproteins | Chromosomal Proteins, Non-Histone | Histone Deacetylases | De Lange Syndrome | Chondroitin Sulfate Proteoglycans | Nuclear Proteins | Mutant Proteins | Adaptor Proteins, Signal Transducing | Development Biology | Cell Cycle Proteins
Journal Article
2012, ISBN 364216482X, Volume 7, 3
Reference
Journal Article
Journal Article
Journal Article
Laboratory Investigation, ISSN 0023-6837, 05/2017, Volume 97, Issue 5, pp. 567 - 576
Bladder cancer is the most common malignant tumor of the urothelium and is classified into non-muscle-invasive bladder cancer (NMIBC) and muscle-invasive... 
MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | CARCINOMA CELL-LINES | STEM-CELLS | INVASION | METASTASIS | RESISTANCE | E-CADHERIN | PATHOLOGY | IDENTIFICATION | TUMORS | PROGRESSION
Journal Article
Journal Article
Cancer Science, ISSN 1347-9032, 12/2018, Volume 109, Issue 12, pp. 4003 - 4014
Aberrant activation of Wnt/β‐catenin signaling causes tumorigenesis and promotes the proliferation of colorectal cancer cells. Porcupine inhibitors, which... 
colorectal cancer | mouse xenograft model | poly | tankyrase inhibitor | β‐catenin signaling | Wnt | ADP | ribose) polymerase | Wnt/β-catenin signaling | poly(ADP-ribose) polymerase | AXIN | REPEAT-BINDING FACTOR-1 | SENSITIVITY | COLON | PARP | Wnt/beta-catenin signaling | TRF1 | APC | ONCOLOGY | DEGRADATION | AZD2281 | Adenomatous Polyposis Coli Protein - genetics | Cell Survival - drug effects | Colorectal Neoplasms - enzymology | Administration, Oral | Colorectal Neoplasms - genetics | HCT116 Cells | Humans | Enzyme Inhibitors - pharmacology | Treatment Outcome | Antineoplastic Agents - administration & dosage | Antineoplastic Agents - chemistry | Tankyrases - antagonists & inhibitors | Enzyme Inhibitors - administration & dosage | Xenograft Model Antitumor Assays | Animals | Colorectal Neoplasms - drug therapy | Enzyme Inhibitors - chemistry | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Mice | Mutation | Post-translational modification | Growth | Analysis | Colorectal cancer | T cells | Gene expression | Sugars | Monosaccharides | Cell proliferation | Ubiquitin | Medical research | Animal models | Wnt protein | Research & development--R&D | Secretion | Colorectal carcinoma | Oral administration | Myc protein | Kinases | Polyposis coli | Adenomatous polyposis coli | Xenografts | Tumor suppressor genes | Antitumor activity | Ligands | Tumorigenesis | Catenin | Cancer | Index Medicus | poly(ADP‐ribose) polymerase | Original
Journal Article