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Journal Article
American Journal of Clinical Pathology, ISSN 0002-9173, 01/2018, Volume 149, Issue suppl_1, pp. S122 - S122
Abstract Introduction Henoch-Schönlein purpura (HSP) is an acute IgA-mediated vasculitic disorder of predominantly pediatric population. It is characterized by... 
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 11/2016, Volume 68, Issue 11, pp. 2686 - 2696
ObjectiveIncreased expression of type I interferon (IFN) and a broad signature of type I IFN-induced gene transcripts are observed in patients with systemic... 
PATHOGENESIS | HUMAN TISSUES | ACTIVATION | PRIMARY SJOGRENS-SYNDROME | PERIPHERAL-BLOOD CELLS | AICARDI-GOUTIERES SYNDROME | TREX1 | GENE-EXPRESSION | LUPUS-ERYTHEMATOSUS | RHEUMATOLOGY | IFN-ALPHA | Immunohistochemistry | Interferon-alpha - drug effects | Interferon-beta - immunology | Toll-Like Receptor 7 - antagonists & inhibitors | Dendritic Cells - immunology | Humans | Long Interspersed Nucleotide Elements - genetics | Middle Aged | Lupus Erythematosus, Systemic - metabolism | Sjogren's Syndrome - immunology | Male | Monocytes - immunology | Sjogren's Syndrome - metabolism | Interferon Type I - immunology | Interferon-beta - drug effects | Autoimmune Diseases - genetics | DNA Methylation | Kidney - metabolism | Toll-Like Receptor 8 - antagonists & inhibitors | I-kappa B Kinase - antagonists & inhibitors | Leukocytes, Mononuclear - immunology | Lupus Erythematosus, Systemic - immunology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Interferon Type I - metabolism | Long Interspersed Nucleotide Elements - immunology | Adult | Female | Lupus Nephritis - metabolism | Interferon Type I - drug effects | Sjogren's Syndrome - genetics | Interferon-alpha - metabolism | Leukocytes, Mononuclear - drug effects | Salivary Glands, Minor - metabolism | Toll-Like Receptor 9 - antagonists & inhibitors | Autoimmune Diseases - immunology | Lupus Nephritis - immunology | Blotting, Western | Interferon-alpha - immunology | Immunity, Innate - immunology | Interferon-beta - metabolism | Lupus Erythematosus, Systemic - genetics | Aged
Journal Article
American Journal of Clinical Pathology, ISSN 0002-9173, 09/2019, Volume 152, Issue Supplement_1, pp. S42 - S43
Journal Article
Human Pathology, ISSN 0046-8177, 2014, Volume 45, Issue 9, pp. 1918 - 1927
Summary Recently, cancer therapies have been supplemented by vascular endothelial growth factor (VEGF) inhibitors as anti-angiogenic agents. However,... 
Pathology | Thrombotic microangiopathy | Vascular endothelial growth factor inhibitor | Acute tubular injury | Renal failure | Proteinuria | PREECLAMPSIA | PATHOLOGY | RENAL-CELL CARCINOMA | OVEREXPRESSION | SUNITINIB | HYPERTENSION | ANTI-VEGF THERAPY | UREMIC SYNDROME | GLOMERULAR-DISEASE | Niacinamide - analogs & derivatives | Kidney - pathology | Humans | Middle Aged | Male | Antineoplastic Agents - therapeutic use | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Bevacizumab | Proteinuria - chemically induced | Kidney - metabolism | Antineoplastic Agents - adverse effects | Phenylurea Compounds - adverse effects | Kidney Diseases - chemically induced | Angiogenesis Inhibitors - therapeutic use | Female | Acute Kidney Injury - chemically induced | Retrospective Studies | Angiogenesis Inhibitors - adverse effects | Antibodies, Monoclonal, Humanized - adverse effects | Antibodies, Monoclonal, Humanized - therapeutic use | Thrombotic Microangiopathies - pathology | Acute Kidney Injury - pathology | Kidney Diseases - pathology | Niacinamide - adverse effects | Niacinamide - therapeutic use | Phenylurea Compounds - therapeutic use | Neoplasms - drug therapy | Aged | Thrombotic Microangiopathies - chemically induced | Hypertension | Care and treatment | Cancer patients | Chemotherapy | Chronic kidney failure | Anemia | Vascular endothelial growth factor | Cancer | Drug therapy | Cancer therapies | Rodents | Colleges & universities | Ovarian cancer | Index Medicus
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 12/2015, Volume 309, Issue 12, pp. F1000 - F1002
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e76360
We applied customized targeted next-generation exome sequencing (NGS) to determine if mutations in genes associated with renal malformations, Alport syndrome... 
GLOMERULAR-FILTRATION | MULTIDISCIPLINARY SCIENCES | BASEMENT-MEMBRANE NEPHROPATHY | HEMATURIA | NEPHROTIC SYNDROME | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | CHILDHOOD | REMISSION | Nephritis, Hereditary - pathology | Glomerulosclerosis, Focal Segmental - genetics | Humans | Middle Aged | Molecular Sequence Data | Myosin Heavy Chains - genetics | Nephritis, Hereditary - genetics | Male | Autoantigens - chemistry | Proteinuria - genetics | Proteinuria - pathology | Transcription Factors - genetics | Autoantigens - genetics | Molecular Motor Proteins - genetics | Collagen Type IV - chemistry | Exome - genetics | Phenotype | Animals | Pedigree | Collagen Type IV - genetics | Female | High-Throughput Nucleotide Sequencing | Mutation | Glomerulosclerosis, Focal Segmental - pathology | Immunohistochemistry | Urologic diseases | Analysis | Genes | African Americans | Development and progression | Genetic aspects | Health aspects | Multiplexing | Exons | Genomes | Urinary tract | Gene sequencing | Immunology | Etiology | Rodents | Genetics | Collagen (type IV) | Urogenital system | Deoxyribonucleic acid--DNA | Internal medicine | Abnormalities | Genetic diversity | Nephrotic syndrome | Electron microscopy | Minority & ethnic groups | Patients | Hearing loss | Medicine | Pathology | Biopsy | Alport syndrome | Kidney diseases | Proteinuria | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Pathology, ISSN 0031-3025, 2014, Volume 46, pp. S40 - S41
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 05/2017, Volume 28, Issue 5, pp. 1437 - 1449
Journal Article
Reference
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2010, Volume 107, Issue 7, pp. 3012 - 3017
Glomerulonephritis is a major cause of morbidity in patients with systemic lupus erythematosus. Although substantial progress has been made in the... 
Glomerulonephritis | Nephritis | Kidneys | Systemic lupus erythematosus | Kidney cells | Lupus nephritis | Macrophages | Lesions | Autoimmune diseases | Proteinuria | Kidney disease | Tissue repair | Polyinosinic: Polycytidylic acid | Alternative macrophages | KIDNEY-DISEASE | GLOMERULONEPHRITIS | ACTIVATION | polyinosinic: polycytidylic acid | MULTIDISCIPLINARY SCIENCES | alternative macrophages | MONOCYTES | kidney disease | tissue repair | FEATURES | PATHOGENESIS | MATRIX METALLOPROTEINASES | systemic lupus erythematosus | INFLAMMATION | MICE | EXPRESSION | Immunohistochemistry | Immunoblotting | Osteopontin - metabolism | Metalloproteases - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Proteinuria - chemically induced | Flow Cytometry | Statistics, Nonparametric | Interferon Type I - metabolism | Interleukin-10 - metabolism | Lymphokines - metabolism | Female | Gene Expression Regulation - immunology | Electrophoresis, Polyacrylamide Gel | Lupus Nephritis - immunology | Mice, Inbred NZB | Reverse Transcriptase Polymerase Chain Reaction | Poly I-C - toxicity | Gene Expression Regulation - drug effects | Heparin-binding EGF-like Growth Factor | Lupus Nephritis - enzymology | Macrophages - metabolism | Platelet-Derived Growth Factor - metabolism | Animals | Poly I-C - metabolism | Lupus Nephritis - physiopathology | Lupus Nephritis - chemically induced | Mice | Interferon Type I - toxicity | Development and progression | Interferon | Properties | Metalloproteins | Methods | Immunological research | Biological Sciences
Journal Article