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Journal Article
Journal Article
Journal Article
Nature, ISSN 0028-0836, 08/2017, Volume 548, Issue 7669, pp. 537 - 542
Somatic gene mutations can alter the vulnerability of cancer cells to T-cell-based immunotherapies. Here we perturbed genes in human melanoma cells to mimic... 
CELL LUNG-CANCER | METASTATIC MELANOMA | DIFFERENTIAL GENE | CTLA-4 BLOCKADE | PD-1 BLOCKADE | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | RECEPTOR | T-CELLS | GENOME | LYMPHOCYTES | Neoplasms - metabolism | Humans | Genome - genetics | Adoptive Transfer | Janus Kinase 1 - metabolism | T-Lymphocytes, Cytotoxic - drug effects | Apelin - metabolism | Neoplasms - therapy | Apelin Receptors - genetics | Neoplasms - genetics | Melanoma - genetics | Immunotherapy | Female | Genes, Essential - genetics | Melanoma - metabolism | T-Lymphocytes, Cytotoxic - immunology | Knowledge Bases | Reproducibility of Results | Histocompatibility Antigens Class I - immunology | Antigen Presentation - genetics | CRISPR-Cas Systems - genetics | Apelin Receptors - metabolism | Animals | T-Lymphocytes, Cytotoxic - metabolism | Melanoma - immunology | Neoplasms - immunology | Interferon-gamma - immunology | Cell Line, Tumor | Mice | Mutation | Melanoma - therapy | Care and treatment | Genetic aspects | Nucleotide sequencing | Methods | DNA sequencing | Cancer | Cytolytic activity | Animal models | CD8 antigen | Genes | Genomics | Effector cells | Genomes | Lymphocytes T | Kinases | Cancer therapies | Lymphocytes | Janus kinase | Antigen presentation | Antigens | CRISPR | Melanoma | T cell receptors | Tumor cell lines | Signaling | Immune checkpoint | γ-Interferon | Interferon | Tumors
Journal Article
PLoS One, ISSN 1932-6203, 2010, Volume 5, Issue 5, p. e10630
Background: Pancreatic ductal adenocarcinoma (PDAC) has a dismal prognosis. The high risk of recurrence following surgical resection provides the rationale for... 
SURVIVAL | CELLS | DUCTAL ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | HUMAN CHOLANGIOCARCINOMA | TUMOR-SUPPRESSOR GENE | PROGNOSTIC MARKERS | ACTIVATED AKT | EXPRESSION | GEMCITABINE | ENDOTHELIAL GROWTH-FACTOR | Multivariate Analysis | Recurrence | Oligonucleotide Array Sequence Analysis | Humans | Middle Aged | Male | MicroRNAs - metabolism | Carcinoma, Pancreatic Ductal - surgery | Carcinoma, Pancreatic Ductal - genetics | Pancreatic Neoplasms - drug therapy | Biomarkers, Tumor - metabolism | Female | Chemotherapy, Adjuvant | Gene Expression Regulation, Neoplastic - drug effects | Korea | Reproducibility of Results | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - surgery | Proportional Hazards Models | Pancreatic Neoplasms - genetics | Treatment Outcome | Carcinoma, Pancreatic Ductal - pathology | Carcinoma, Pancreatic Ductal - drug therapy | Disease-Free Survival | Drug Resistance, Neoplasm - genetics | Italy | Aged | Biomarkers, Tumor - genetics | Cell Proliferation - drug effects | Fluorouracil - pharmacology | MicroRNAs - genetics | Cohort Studies | Drug Resistance, Neoplasm - drug effects | Immunohistochemistry | Medical research | Care and treatment | Prognosis | MicroRNA | Pancreatic cancer | Patient outcomes | Adjuvant treatment | Medicine, Experimental | Cancer | Adenocarcinoma | Therapy | Medical services | Chemoresistance | Cytotoxicity | Oncology | Paraffin | Tissues | Cancer therapies | Anticancer properties | Proteins | Transfection | Surgery | Bioindicators | Vascular endothelial growth factor | Cell survival | Internal medicine | Mortality | MiRNA | Hazards | Gene expression | Ribonucleic acid--RNA | Patients | Medicine | Pathology | Chemotherapy | Hospitals | Statistical models | MicroRNAs | Medical prognosis | Stem cells | Biomarkers | Mutation | Chemokines | Apoptosis | Tumors | RNA | Ribonucleic acid
Journal Article
Lancet Oncology, The, ISSN 1470-2045, 2015, Volume 16, Issue 2, pp. 177 - 186
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 11/2009, Volume 27, Issue 32, pp. 5410 - 5417
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 07/2003, Volume 349, Issue 5, pp. 427 - 434
In this randomized trial, an antibody against vascular endothelial growth factor, bevacizumab, prolonged the time to progression of disease in patients with... 
MEDICINE, GENERAL & INTERNAL | INHIBITION | PROTEIN | THERAPY | ANGIOGENESIS | PRODUCT | TUMOR-SUPPRESSOR GENE | EMBRYONIC LETHALITY | CARCINOMA | EXPRESSION | HYPOXIA-INDUCIBLE FACTORS | Vascular Endothelial Growth Factor A | Kidney Neoplasms - genetics | Vascular Endothelial Growth Factors | Humans | Ligases - genetics | Middle Aged | Antibodies, Monoclonal - adverse effects | Carcinoma, Renal Cell - genetics | Antibodies, Monoclonal - therapeutic use | Male | Bevacizumab | Endothelial Growth Factors - immunology | Antibodies, Monoclonal, Humanized | Lymphokines - antagonists & inhibitors | Female | Carcinoma, Renal Cell - drug therapy | Genes, Tumor Suppressor | Double-Blind Method | Lymphatic Metastasis | Disease Progression | Endothelial Growth Factors - antagonists & inhibitors | Ubiquitin-Protein Ligases | Adenocarcinoma, Clear Cell - secondary | Neovascularization, Pathologic - drug therapy | Carcinoma, Renal Cell - secondary | Lymphokines - immunology | Kidney Neoplasms - pathology | Von Hippel-Lindau Tumor Suppressor Protein | Kidney Neoplasms - drug therapy | Adenocarcinoma, Clear Cell - genetics | Intercellular Signaling Peptides and Proteins - immunology | Tumor Suppressor Proteins | Adenocarcinoma, Clear Cell - drug therapy | Monoclonal antibodies | Drug therapy | Carcinoma, Renal cell | Proteins | Studies | Angiogenesis | Cytokines | Rodents | Mutation | Vascular endothelial growth factor | Cancer
Journal Article
Clinical Infectious Diseases, ISSN 1058-4838, 8/2010, Volume 51, Issue 3, pp. 350 - 358
Journal Article