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1. Full Text Case of atopic dermatitis concurrent with idiopathic thrombocytopenic purpura, whose serum thymus and activation‐regulated chemokine level remained undetectable
by Ozawa, Maki and Sasahara, Yoji and Aiba, Setsuya
The Journal of Dermatology, ISSN 0385-2407, 05/2018, Volume 45, Issue 5, pp. 606 - 608
We report a 9‐year‐old Japanese female patient with atopic dermatitis associated with idiopathic thrombocytopenic purpura. She demonstrated high serum... 
idiopathic thrombocytopenic purpura | atopic dermatitis | platelet | thymus and activation‐regulated chemokine | biomarker | thymus and activation-regulated chemokine | DISEASES | PLATELETS | TARC | DERMATOLOGY | Thrombocytopenic purpura | Atopic dermatitis | Development and progression | Allergens | Purpura | Immunoglobulin E | Idiopathic thrombocytopenic purpura | Dermatitis | Chemokines | Thymus
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2. Full Text The aryl hydrocarbon receptor AhR links atopic dermatitis and air pollution via induction of the neurotrophic factor artemin
by Hidaka, Takanori and Ogawa, Eisaku and Kobayashi, Eri H and Suzuki, Takafumi and Funayama, Ryo and Nagashima, Takeshi and Fujimura, Taku and Aiba, Setsuya and Nakayama, Keiko and Okuyama, Ryuhei and Yamamoto, Masayuki
Nature Immunology, ISSN 1529-2908, 01/2017, Volume 18, Issue 1, pp. 64 - 73
Atopic dermatitis is increasing worldwide in correlation with air pollution. Various organic components of pollutants activate the transcription factor AhR... 
ITCH | DISEASES | PRURITUS | TRANSCRIPTION | ULTRAFINE PARTICLES | SKIN | IMMUNOLOGY | EXPRESSION | SENSORY NEURONS | THYMIC STROMAL LYMPHOPOIETIN | NC/NGA MICE | Humans | Basic Helix-Loop-Helix Transcription Factors - metabolism | Dermatitis, Atopic - immunology | Receptors, Aryl Hydrocarbon - metabolism | Axon Guidance - genetics | Epidermis - innervation | Pruritus - immunology | Animals, Newborn | Epidermis - pathology | Basic Helix-Loop-Helix Transcription Factors - genetics | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Receptors, Aryl Hydrocarbon - genetics | Receptor, EphB2 - genetics | Receptor, EphB2 - metabolism | Nerve Tissue Proteins - genetics | Keratin-15 - metabolism | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Air Pollutants - adverse effects | Mice | Keratin-15 - genetics | Keratinocytes - physiology | Atopic dermatitis | Aromatic compounds | Physiological aspects | Air pollution | Research | Health aspects | Risk factors
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3. Full Text Distinct Sets of Genetic Alterations in Melanoma
by Curtin, John A and Fridlyand, Jane and Kageshita, Toshiro and Patel, Hetal N and Busam, Klaus J and Kutzner, Heinz and Cho, Kwang-Hyun and Aiba, Setsuya and Bröcker, Eva-Bettina and LeBoit, Philip E and Pinkel, Dan and Bastian, Boris C
The New England Journal of Medicine, ISSN 0028-4793, 11/2005, Volume 353, Issue 20, pp. 2135 - 2147
A genomic study of four groups of melanoma arising at sites with different levels of exposure to ultraviolet light reveals distinct genetic alterations among... 
MEDICINE, GENERAL & INTERNAL | BRAF MUTATIONS | CUTANEOUS MELANOMA | TUMOR | RISK | MALIGNANT-MELANOMA | SEVERE SUNBURN | SKIN | MICROARRAYS | COMPARATIVE GENOMIC HYBRIDIZATION | NEVI | Cyclin-Dependent Kinase 4 - genetics | Humans | Middle Aged | Male | Phosphatidylinositol 3-Kinases - metabolism | Melanoma - genetics | Aged, 80 and over | Adult | Female | DNA, Neoplasm - analysis | Melanoma - metabolism | PTEN Phosphohydrolase - genetics | Nucleic Acid Hybridization | Signal Transduction | Risk Factors | PTEN Phosphohydrolase - metabolism | Ultraviolet Rays - adverse effects | Skin Neoplasms - metabolism | Environmental Exposure - adverse effects | Cyclin D1 - genetics | Proto-Oncogene Proteins B-raf - genetics | Skin Neoplasms - genetics | Aged | Mutation | Genes, ras | Genome, Human | Mitogen-Activated Protein Kinases - metabolism | Genetic aspects | Research | Melanoma | Cancer | Ultraviolet radiation | Heredity | Genetic diversity | Health risk assessment | Skin cancer
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4. Full Text Synergistic Effect of Nod1 and Nod2 Agonists with Toll-Like Receptor Agonists on Human Dendritic Cells To Generate Interleukin-12 and T Helper Type 1 Cells
by Hiroyuki Tada and Setsuya Aiba and Ken-Ichiro Shibata and Toshiaki Ohteki and Haruhiko Takada
Infection and Immunity, ISSN 0019-9567, 12/2005, Volume 73, Issue 12, pp. 7967 - 7976
Classifications Services IAI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit... 
INFECTIOUS DISEASES | HUMAN MONOCYTIC CELLS | LIPOTEICHOIC ACID | MURAMYL DIPEPTIDE | HOST RECOGNITION | WALL COMPONENTS | IFN-GAMMA | ADJUVANT ACTIVITY | DIAMINOPIMELIC ACID | IMMUNOLOGY | BACTERIAL PEPTIDOGLYCAN | CUTTING EDGE | Adjuvants, Immunologic - pharmacology | Diaminopimelic Acid - pharmacology | Dendritic Cells - immunology | Humans | RNA, Messenger - analysis | Intracellular Signaling Peptides and Proteins - metabolism | RNA, Messenger - metabolism | Th1 Cells - immunology | Th1 Cells - metabolism | Dendritic Cells - drug effects | Toll-Like Receptors - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Polylysine - pharmacology | Poly I-C - pharmacology | Cytokines - metabolism | Interleukin-12 - metabolism | Nod2 Signaling Adaptor Protein | Adaptor Proteins, Signal Transducing - agonists | Toll-Like Receptors - agonists | Acetylmuramyl-Alanyl-Isoglutamine - pharmacology | Drug Synergism | Diaminopimelic Acid - analogs & derivatives | Intracellular Signaling Peptides and Proteins - agonists | Toll-Like Receptors - genetics | Adaptor Proteins, Signal Transducing - genetics | Lipid A - pharmacology | Nod1 Signaling Adaptor Protein | Adaptor Proteins, Signal Transducing - metabolism | Oligodeoxyribonucleotides - pharmacology | Oligopeptides - pharmacology | Host Response and Inflammation
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5. Full Text Notch signaling: Its role in epidermal homeostasis and in the pathogenesis of skin diseases
by Okuyama, Ryuhei and Tagami, Hachiro and Aiba, Setsuya
Journal of Dermatological Science, ISSN 0923-1811, 2007, Volume 49, Issue 3, pp. 187 - 194
Summary Skin undergoes self-renewal throughout life. Terminally differentiated keratinocytes, namely the corneocytes, are continually shed from the surface of... 
Dermatology | Keratinocyte | Oncogenesis | Psoriasis | Differentiation | Notch | Cross-talk | STEM-CELLS | differentiation | DROSOPHILA MASTERMIND | CANCER CELLS | cross-talk | CELL-FATE | P53 HOMOLOG | DERMATOLOGY | GENE | keratinocyte | PATHWAY | psoriasis | oncogenesis | TUMOR-SUPPRESSOR | HUMAN HOMOLOG | Skin Diseases - etiology | Skin - cytology | Animals | Cell Proliferation | Humans | Langerhans Cells - physiology | Homeostasis | Receptors, Notch - physiology | Signal Transduction - physiology | Cell Differentiation | Melanocytes - physiology | Squamous cell carcinoma | Formulae, receipts, prescriptions | Melanoma | Dermatologic agents | Skin | Tumor proteins | Skin cancer
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6. Full Text Successful treatment of microcystic adnexal carcinoma on the philtrum with oral S‐1 monotherapy
by Haga, Takahiro and Fujimura, Taku and Aiba, Setsuya
The Journal of Dermatology, ISSN 0385-2407, 06/2019, Volume 46, Issue 6, pp. e208 - e209
DERMATOLOGY | Care and treatment | Cancer
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7. Full Text The Possible Interaction between Receptor Activator of Nuclear Factor Kappa-B Ligand Expressed by Extramammary Paget Cells and its Ligand on Dermal Macrophages
by Kambayashi, Yumi and Fujimura, Taku and Furudate, Sadanori and Asano, Masayuki and Kakizaki, Aya and Aiba, Setsuya
Journal of Investigative Dermatology, ISSN 0022-202X, 10/2015, Volume 135, Issue 10, pp. 2547 - 2550
BREAST-CANCER PATIENTS | REGULATORY T-CELLS | RANKL | DERMATOLOGY | Immunohistochemistry | T-Lymphocytes, Regulatory - metabolism | RANK Ligand - metabolism | Enzyme-Linked Immunosorbent Assay | Humans | Cells, Cultured | Real-Time Polymerase Chain Reaction - methods | Cell Communication - physiology | NF-kappa B - metabolism | Reference Values | Keratinocytes - cytology | Macrophages - cytology | RNA, Messenger - metabolism | Macrophages - metabolism | Matrix Metalloproteinase 7 - metabolism | Keratinocytes - metabolism | Paget Disease, Extramammary - pathology | Paget Disease, Extramammary - genetics | T-Lymphocytes, Regulatory - cytology | Biomarkers, Tumor - metabolism | Biopsy, Needle
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8. Full Text First-in-human clinical study of novel technique to diagnose malignant melanoma via thermal conductivity measurements
by Okabe, Takahiro and Fujimura, Taku and Okajima, Junnosuke and Kambayashi, Yumi and Aiba, Setsuya and Maruyama, Shigenao
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 3853 - 7
Melanoma is an aggressive skin cancer that originates from melanocytes and, especially in the case of early-stage melanoma, is distributed adjacent to the... 
MULTIDISCIPLINARY SCIENCES | SKIN | Temperature requirements | Invasiveness | Nevus | Melanoma | Dermis | Melanocytes | Epidermis | Heat conductivity | Lymph nodes | Skin cancer | Biopsy | Skin diseases | Tumors
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9. Full Text Tumor-associated macrophages in skin: How to treat their heterogeneity and plasticity
by Fujimura, Taku and Kakizaki, Aya and Furudate, Sadanori and Kambayashi, Yumi and Aiba, Setsuya
Journal of Dermatological Science, ISSN 0923-1811, 2016, Volume 83, Issue 3, pp. 167 - 173
Abstract Immunosuppressive tumor-associated macrophages (TAMs) promote an immunosuppressive environment in the tumor-bearing host, together with regulatory T... 
Dermatology | Immunosuppression | Angiogenetic factors | M2 polarization | Regulatory t cells | Tumor-associated macrophages | Chemokines | CANCER-RELATED INFLAMMATION | MYCOSIS-FUNGOIDES | T-CELL LYMPHOMA | DERMATOLOGY | BREAST-CANCER | GROWTH | RECEPTOR ACTIVATOR | THERAPEUTIC TARGETS | SUPPRESSOR-CELLS | ALTERNATIVE ACTIVATION | PROGRESSION | T-Lymphocytes, Regulatory - metabolism | Skin - metabolism | Humans | Tumor Microenvironment | Antineoplastic Agents - therapeutic use | Molecular Targeted Therapy | Th1 Cells - immunology | T-Lymphocytes, Regulatory - immunology | Th1 Cells - metabolism | Lymphocytes, Tumor-Infiltrating - metabolism | Macrophages - immunology | Skin - pathology | Skin - immunology | Melanoma - metabolism | Skin Neoplasms - pathology | Macrophages - pathology | Signal Transduction | Skin Neoplasms - immunology | Cell Communication | Melanoma - pathology | Skin Neoplasms - metabolism | Macrophages - metabolism | Phenotype | Animals | Melanoma - immunology | Macrophages - drug effects | Chemokines - metabolism | Angiogenic Proteins - metabolism | Skin - drug effects | Immunologic Factors - therapeutic use | Lymphocytes, Tumor-Infiltrating - immunology | Medical colleges | Immunotherapy | Melanoma | Skin | T cells | Macrophages | Skin cancer
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10. Full Text Tumor-associated macrophages: Therapeutic targets for skin cancer
by Fujimura, Taku and Kambayashi, Yumi and Fujisawa, Yasuhiro and Hidaka, Takanori and Aiba, Setsuya
Frontiers in Oncology, ISSN 2234-943X, 01/2018, Volume 8, p. 3
Tumor-associated macrophages (TAMs) and regulatory T cells (Tregs) are significant components of the microenvironment of solid tumors in the majority of... 
Immunosuppression | Angiogenetic factors | M2 polarization | Regulatory T cells | Chemokines | Tumor-associated macrophages | LESIONAL SKIN | ANGIOGENESIS | angiogenetic factors | immunosuppression | MYCOSIS-FUNGOIDES | chemokines | CYTOTOXIC-CELLS | tumor-associated macrophages | regulatory T cells | ONCOLOGY | RECEPTOR ACTIVATOR | SUPPRESSOR-CELLS | LIGAND | MELANOMA GROWTH | EXPRESSION | PROMOTES | Care and treatment | Immunotherapy | Innovations | Development and progression | Macrophages | Health aspects | Skin cancer
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11. Full Text Isolated adrenocorticotropic hormone deficiency possibly caused by nivolumab in a metastatic melanoma patient
by Fujimura, Taku and Kambayashi, Yumi and Furudate, Sadanori and Kakizaki, Aya and Hidaka, Takanori and Haga, Takahiro and Hashimoto, Akira and Morimoto, Ryo and Aiba, Setsuya
The Journal of Dermatology, ISSN 0385-2407, 03/2017, Volume 44, Issue 3, pp. e13 - e14
DERMATOLOGY | Skin Neoplasms - pathology | Skin Neoplasms - drug therapy | Enzyme-Linked Immunosorbent Assay | Endocrine System Diseases - chemically induced | Humans | Male | Melanoma - pathology | Nivolumab - adverse effects | Antineoplastic Agents, Immunological - adverse effects | Neoplasm Metastasis | Genetic Diseases, Inborn - chemically induced | Adrenocorticotropic Hormone - deficiency | Melanoma - drug therapy | Aged | Hypoglycemia - chemically induced | ACTH | Melanoma | Metastasis
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12. Full Text A possible interaction between periostin and CD163+ skin‐resident macrophages in pemphigus vulgaris and bullous pemphigoid
by Fujimura, Taku and Kakizaki, Aya and Furudate, Sadanori and Aiba, Setsuya
Experimental Dermatology, ISSN 0906-6705, 12/2017, Volume 26, Issue 12, pp. 1193 - 1198
Pemphigus vulgaris (PV) and bullous pemphigoid (BP) are autoimmune blistering diseases, and substantial numbers of CD163+ tissue‐associated macrophages (TAMs)... 
tissue‐associated macrophages | pemphigus | periostin | CXCL5 | IL‐36 | tissue-associated macrophages | IL-36 | RHEUMATOID-ARTHRITIS | M2 MACROPHAGES | CYTOKINES | LESIONAL SKIN | SOLUBLE CD163 | DERMATOLOGY | DISEASE | RECEPTOR ACTIVATOR | EXPRESSION | T-CELLS | PSORIASIS | Interleukin-1 - metabolism | Pemphigus - blood | Dermis - metabolism | Humans | Middle Aged | Receptors, Cell Surface - metabolism | Male | Chemokine CXCL5 - metabolism | Matrix Metalloproteinase 12 - metabolism | Cell Adhesion Molecules - metabolism | Antigens, CD - metabolism | Macrophages - metabolism | Pemphigoid, Bullous - blood | Antigens, Differentiation, Myelomonocytic - metabolism | Aged, 80 and over | Adult | Female | Pemphigus - immunology | Aged | Pemphigoid, Bullous - immunology | Skin | DNA microarrays | T cells | Macrophages | Pemphigus | Bullous pemphigoid | Pemphigoid | Cytokines | Immunomodulation | Dermis | Pemphigus vulgaris | Lymphocytes T | Serum levels | CD163 antigen | Molecular modelling | Skin diseases | Chemokines
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13. Full Text BRAF kinase inhibitors for treatment of melanoma: developments from early-stage animal studies to Phase II clinical trials
by Fujimura, Taku and Hidaka, Takanori and Kambayashi, Yumi and Aiba, Setsuya
Expert Opinion on Investigational Drugs, ISSN 1354-3784, 02/2019, Volume 28, Issue 2, pp. 143 - 148
Introduction: Approximately, 30.4-66.0% of cutaneous melanomas possess a mutation in the BRAF gene that activates downstream signaling through the... 
BRAF-mutant metastatic melanoma | HDAC inhibitors | BRAF resistance | immune checkpoints inhibitors | BRAF inhibitors | MEK inhibitors | SURVIVAL | MULTICENTER | ADJUVANT DABRAFENIB | OPEN-LABEL | CHEMOTHERAPY | METASTATIC MELANOMA | DABRAFENIB PLUS TRAMETINIB | PI3K | PHARMACOLOGY & PHARMACY | QUALITY-OF-LIFE | VEMURAFENIB | Skin Neoplasms - pathology | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Skin Neoplasms - drug therapy | Humans | Melanoma - pathology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Disease-Free Survival | Protein Kinase Inhibitors - administration & dosage | Animals | Melanoma - genetics | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Skin Neoplasms - genetics | Protein Kinase Inhibitors - pharmacology | Mutation
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14. Full Text Case of late‐onset erythropoietic protoporphyria with myelodysplastic syndrome who has homozygous IVS3‐48C polymorphism in the ferrochelatase gene
by Suzuki, Hiromi and Kikuchi, Katsuko and Fukuhara, Noriko and Nakano, Hajime and Aiba, Setsuya
The Journal of Dermatology, ISSN 0385-2407, 06/2017, Volume 44, Issue 6, pp. 651 - 655
We report the case of a 42‐year‐old man with a 5‐year history of myelodysplastic syndrome and photosensitivity who had developed painful erythema and blisters... 
homozygous IVS3‐48C polymorphism | erythropoietic protoporphyria | myelodysplastic syndrome | photosensitivity | ferrochelatase | homozygous IVS3-48C polymorphism | DIAGNOSIS | CELLS | SIDEROBLASTIC ANEMIA | FEATURES | DELETION | DERMATOLOGY | DERMAL PHOTOSENSITIVITY | PORPHYRIAS ADVANCES | JAPANESE PATIENTS | ERYTHROCYTE PROTOPORPHYRIN | Protoporphyria, Erythropoietic - genetics | Protoporphyria, Erythropoietic - complications | Humans | Age of Onset | Adult | Ferrochelatase - genetics | Male | Photosensitivity Disorders - etiology | Myelodysplastic Syndromes - complications | Genetic research | Genetic aspects | Genes | Genetic polymorphisms | Photosensitivity | Ferrochelatase | Erythrocytes | Blood vessels | Malignancy | Gene polymorphism | Myelodysplastic syndrome | Erythropoietic protoporphyria | Protoporphyrin | Skin | Erythema | Deoxyribonucleic acid--DNA | Porphyrins
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15. Full Text Successful control of phototherapy‐resistant lymphomatoid papulosis with oral bexarotene
by Fujimura, Taku and Furudate, Sadanori and Tanita, Kayo and Sato, Yota and Kambayashi, Yumi and Hidaka, Takanori and Aiba, Setsuya
The Journal of Dermatology, ISSN 0385-2407, 02/2018, Volume 45, Issue 2, pp. e37 - e38
DERMATOLOGY | Skin Neoplasms - pathology | Lymphomatoid Papulosis - pathology | Lymphomatoid Papulosis - therapy | Skin Neoplasms - therapy | Humans | Middle Aged | Female | Treatment Outcome | Antineoplastic Agents - therapeutic use | Bexarotene - therapeutic use | Skin - pathology | Phototherapy | Antimitotic agents | Retinoids | Antineoplastic agents | Lymphomatoid papulosis
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16. Full Text Tumor-associated M2 macrophages in mycosis fungoides acquire immunomodulatory function by interferon alpha and interferon gamma
by Furudate, Sadanori and Fujimura, Taku and Kakizaki, Aya and Hidaka, Takanori and Asano, Masayuki and Aiba, Setsuya
Journal of Dermatological Science, ISSN 0923-1811, 2016, Volume 83, Issue 3, pp. 182 - 189
Highlights • We previously reported that the TAMs stimulated by stromal factors produce the chemokines that affect the formation of CTCL. • Since IFN-α and... 
Dermatology | IFN-α | Mycosis fungoides | IFN-γ | Tumor-associated macrophages (TAMs) | Chemokines | IMMUNITY | T-CELL LYMPHOMA | SEZARY-SYNDROME | IFN-alpha | DERMATOLOGY | IFN-gamma