Nature Immunology, ISSN 1529-2908, 06/2005, Volume 6, Issue 6, pp. 565 - 570
The recessive mutation 'Heedless' (hdl) was detected in third-generation N-ethyl-N-nitrosourea-mutated mice that showed defective responses to microbial...
ACTIVATION | RECOGNITION | GENETIC-CONTROL | PATHWAY | ENDOTOXIN | LIPOPOLYSACCHARIDE BINDING-PROTEIN | RECEPTOR | MICE | IMMUNOLOGY | IDENTIFICATION | TLR4 | Receptors, Immunologic - chemistry | Toll-Like Receptor 4 | Multiprotein Complexes | Lipopolysaccharide Receptors - metabolism | Interferon Type I - biosynthesis | Mice, Mutant Strains | Antigens, Differentiation - metabolism | Antigens, Ly - metabolism | Lymphocyte Antigen 96 | Macrophages, Peritoneal - drug effects | Lipopolysaccharides - toxicity | Signal Transduction | Mice, Inbred C57BL | Macrophages, Peritoneal - immunology | Antigens, Ly - chemistry | Myeloid Differentiation Factor 88 | Vesicular stomatitis Indiana virus - pathogenicity | Mice, Inbred C3H | Mice, Knockout | Adaptor Proteins, Signal Transducing | Animals | Lipopolysaccharide Receptors - genetics | Antigens, Differentiation - genetics | Mice | Mutation | In Vitro Techniques | Macrophages, Peritoneal - metabolism | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism
ACTIVATION | RECOGNITION | GENETIC-CONTROL | PATHWAY | ENDOTOXIN | LIPOPOLYSACCHARIDE BINDING-PROTEIN | RECEPTOR | MICE | IMMUNOLOGY | IDENTIFICATION | TLR4 | Receptors, Immunologic - chemistry | Toll-Like Receptor 4 | Multiprotein Complexes | Lipopolysaccharide Receptors - metabolism | Interferon Type I - biosynthesis | Mice, Mutant Strains | Antigens, Differentiation - metabolism | Antigens, Ly - metabolism | Lymphocyte Antigen 96 | Macrophages, Peritoneal - drug effects | Lipopolysaccharides - toxicity | Signal Transduction | Mice, Inbred C57BL | Macrophages, Peritoneal - immunology | Antigens, Ly - chemistry | Myeloid Differentiation Factor 88 | Vesicular stomatitis Indiana virus - pathogenicity | Mice, Inbred C3H | Mice, Knockout | Adaptor Proteins, Signal Transducing | Animals | Lipopolysaccharide Receptors - genetics | Antigens, Differentiation - genetics | Mice | Mutation | In Vitro Techniques | Macrophages, Peritoneal - metabolism | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2004, Volume 101, Issue 10, pp. 3516 - 3521
Several subsets of dendritic cells have been shown to produce type I IFN in response to viral infections, thereby assisting the natural killer cell-dependent...
Biological Sciences | Phenotypes | Receptors | Double stranded RNA | Cytokines | Natural killer T cells | Alleles | Infections | Mice | Viral infections | Genotypes | PLASMACYTOID DENDRITIC CELLS | INTERFERON-ALPHA/BETA | NATURAL-KILLER-CELLS | DOUBLE-STRANDED-RNA | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MURINE CYTOMEGALOVIRUS | NF-KAPPA-B | BACTERIAL CPG-DNA | VIRAL-INFECTION | ACTIVATION RECEPTOR | Toll-Like Receptor 3 | Membrane Glycoproteins - metabolism | DNA, Complementary - genetics | Immunity, Innate - genetics | Molecular Sequence Data | Toll-Like Receptor 9 | Cytomegalovirus Infections - immunology | Cytomegalovirus Infections - genetics | Mutation, Missense | DNA-Binding Proteins - metabolism | Toll-Like Receptors | Mice, Mutant Strains | Oligodeoxyribonucleotides - genetics | Killer Cells, Natural - immunology | Antigens, Differentiation - metabolism | Amino Acid Sequence | Immunity, Innate - drug effects | Signal Transduction | Mice, Inbred C57BL | Receptors, Immunologic - deficiency | Receptors, Cell Surface - metabolism | Myeloid Differentiation Factor 88 | DNA-Binding Proteins - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Sequence Homology, Amino Acid | Point Mutation | Adaptor Proteins, Signal Transducing | Phenotype | Animals | Receptors, Cell Surface - deficiency | Antigens, Differentiation - genetics | Mice, Inbred BALB C | Oligodeoxyribonucleotides - pharmacology | Receptors, Immunologic - genetics | Cytokines - biosynthesis | Membrane Glycoproteins - deficiency | Receptors, Immunologic - metabolism | Receptors, Cell Surface - genetics | Dendritic cells | Research
Biological Sciences | Phenotypes | Receptors | Double stranded RNA | Cytokines | Natural killer T cells | Alleles | Infections | Mice | Viral infections | Genotypes | PLASMACYTOID DENDRITIC CELLS | INTERFERON-ALPHA/BETA | NATURAL-KILLER-CELLS | DOUBLE-STRANDED-RNA | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MURINE CYTOMEGALOVIRUS | NF-KAPPA-B | BACTERIAL CPG-DNA | VIRAL-INFECTION | ACTIVATION RECEPTOR | Toll-Like Receptor 3 | Membrane Glycoproteins - metabolism | DNA, Complementary - genetics | Immunity, Innate - genetics | Molecular Sequence Data | Toll-Like Receptor 9 | Cytomegalovirus Infections - immunology | Cytomegalovirus Infections - genetics | Mutation, Missense | DNA-Binding Proteins - metabolism | Toll-Like Receptors | Mice, Mutant Strains | Oligodeoxyribonucleotides - genetics | Killer Cells, Natural - immunology | Antigens, Differentiation - metabolism | Amino Acid Sequence | Immunity, Innate - drug effects | Signal Transduction | Mice, Inbred C57BL | Receptors, Immunologic - deficiency | Receptors, Cell Surface - metabolism | Myeloid Differentiation Factor 88 | DNA-Binding Proteins - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Sequence Homology, Amino Acid | Point Mutation | Adaptor Proteins, Signal Transducing | Phenotype | Animals | Receptors, Cell Surface - deficiency | Antigens, Differentiation - genetics | Mice, Inbred BALB C | Oligodeoxyribonucleotides - pharmacology | Receptors, Immunologic - genetics | Cytokines - biosynthesis | Membrane Glycoproteins - deficiency | Receptors, Immunologic - metabolism | Receptors, Cell Surface - genetics | Dendritic cells | Research
Journal Article
Nature Immunology, ISSN 1529-2908, 02/2006, Volume 7, Issue 2, pp. 156 - 164
Here we have identified 'triple D' (3d), a recessive N-ethyl-N-nitrosourea-induced mutation and phenotype in which no signaling occurs via the intracellular...
IMMUNOSTIMULATORY CPG-OLIGODEOXYNUCLEOTIDES | MHC CLASS-I | RESPONSES | DOUBLE-STRANDED-RNA | ACTIVATION | CAENORHABDITIS-ELEGANS | GENE | CYTOMEGALOVIRUS-INFECTION | AUTOREACTIVE B-CELLS | IMMUNOLOGY | MUSCLE-CONTRACTION | Toll-Like Receptor 7 - metabolism | Membrane Glycoproteins - metabolism | Infection - immunology | Mice, Inbred C57BL | Endoplasmic Reticulum - metabolism | Antigen Presentation - genetics | Signal Transduction - genetics | Mutation, Missense | Toll-Like Receptor 3 - metabolism | Endosomes - metabolism | Infection - genetics | Phenotype | Animals | Membrane Transport Proteins - genetics | Mice, Mutant Strains | Cloning, Molecular | Histocompatibility Antigens Class II - metabolism | Membrane Transport Proteins - metabolism | Mice | Membrane Transport Proteins - immunology | Toll-Like Receptor 9 - metabolism
IMMUNOSTIMULATORY CPG-OLIGODEOXYNUCLEOTIDES | MHC CLASS-I | RESPONSES | DOUBLE-STRANDED-RNA | ACTIVATION | CAENORHABDITIS-ELEGANS | GENE | CYTOMEGALOVIRUS-INFECTION | AUTOREACTIVE B-CELLS | IMMUNOLOGY | MUSCLE-CONTRACTION | Toll-Like Receptor 7 - metabolism | Membrane Glycoproteins - metabolism | Infection - immunology | Mice, Inbred C57BL | Endoplasmic Reticulum - metabolism | Antigen Presentation - genetics | Signal Transduction - genetics | Mutation, Missense | Toll-Like Receptor 3 - metabolism | Endosomes - metabolism | Infection - genetics | Phenotype | Animals | Membrane Transport Proteins - genetics | Mice, Mutant Strains | Cloning, Molecular | Histocompatibility Antigens Class II - metabolism | Membrane Transport Proteins - metabolism | Mice | Membrane Transport Proteins - immunology | Toll-Like Receptor 9 - metabolism
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 03/2004, Volume 101, Issue 10, pp. 3516 - 3521
Journal Article
Nature, ISSN 0028-0836, 02/2005, Volume 433, Issue 7025, pp. 523 - 527
Toll-like receptor 2 (TLR2) is required for the recognition of numerous molecular components of bacteria, fungi and protozoa. The breadth of the ligand...
TOLL-LIKE RECEPTOR-2 | IMMUNE-RESPONSE | PATTERN-RECOGNITION | LIPOTEICHOIC ACID | GENE | METABOLISM | MULTIDISCIPLINARY SCIENCES | FATTY-ACID | LIPOPOLYSACCHARIDE | IDENTIFICATION | LIPOPROTEINS | Peptidoglycan - metabolism | Toll-Like Receptor 2 | CD36 Antigens - genetics | Membrane Glycoproteins - metabolism | Staphylococcus aureus - physiology | Humans | Membrane Glycoproteins - chemistry | Peptidoglycan - chemistry | Zymosan - pharmacology | Mutagenesis - drug effects | Toll-Like Receptors | CD36 Antigens - metabolism | Gene Deletion | Receptors, Cell Surface - chemistry | Staphylococcal Infections - microbiology | Dimerization | Oligopeptides - chemistry | Staphylococcal Infections - metabolism | Cell Line | Oligopeptides - chemical synthesis | Glycerides - chemistry | Staphylococcal Infections - genetics | Signal Transduction | Staphylococcal Infections - immunology | Mice, Inbred C57BL | Membrane Glycoproteins - agonists | Receptors, Cell Surface - agonists | Receptors, Cell Surface - metabolism | Glycerides - metabolism | Mutation - genetics | Immunologic Deficiency Syndromes - chemically induced | Ethylnitrosourea - pharmacology | Mice, Knockout | Staphylococcus aureus - chemistry | Homozygote | Glycerides - pharmacology | Phenotype | Animals | Aging - physiology | Receptors, Cell Surface - deficiency | Mice | Oligopeptides - pharmacology | Peptidoglycan - pharmacology | Lipopeptides | Tumor Necrosis Factor-alpha - biosynthesis | Membrane Glycoproteins - deficiency | Rodents | Immunology | Sensors
TOLL-LIKE RECEPTOR-2 | IMMUNE-RESPONSE | PATTERN-RECOGNITION | LIPOTEICHOIC ACID | GENE | METABOLISM | MULTIDISCIPLINARY SCIENCES | FATTY-ACID | LIPOPOLYSACCHARIDE | IDENTIFICATION | LIPOPROTEINS | Peptidoglycan - metabolism | Toll-Like Receptor 2 | CD36 Antigens - genetics | Membrane Glycoproteins - metabolism | Staphylococcus aureus - physiology | Humans | Membrane Glycoproteins - chemistry | Peptidoglycan - chemistry | Zymosan - pharmacology | Mutagenesis - drug effects | Toll-Like Receptors | CD36 Antigens - metabolism | Gene Deletion | Receptors, Cell Surface - chemistry | Staphylococcal Infections - microbiology | Dimerization | Oligopeptides - chemistry | Staphylococcal Infections - metabolism | Cell Line | Oligopeptides - chemical synthesis | Glycerides - chemistry | Staphylococcal Infections - genetics | Signal Transduction | Staphylococcal Infections - immunology | Mice, Inbred C57BL | Membrane Glycoproteins - agonists | Receptors, Cell Surface - agonists | Receptors, Cell Surface - metabolism | Glycerides - metabolism | Mutation - genetics | Immunologic Deficiency Syndromes - chemically induced | Ethylnitrosourea - pharmacology | Mice, Knockout | Staphylococcus aureus - chemistry | Homozygote | Glycerides - pharmacology | Phenotype | Animals | Aging - physiology | Receptors, Cell Surface - deficiency | Mice | Oligopeptides - pharmacology | Peptidoglycan - pharmacology | Lipopeptides | Tumor Necrosis Factor-alpha - biosynthesis | Membrane Glycoproteins - deficiency | Rodents | Immunology | Sensors
Journal Article
European Journal of Immunology, ISSN 0014-2980, 03/2006, Volume 36, Issue 3, pp. 701 - 711
Lipopolysaccharide (endotoxin, LPS) is a major recognition marker for the detection of gram-negative bacteria by the host and a powerful initiator of the...
Lipopolysaccharide | Mast cells | CD14 | TLR4 | MURINE MACROPHAGES | BINDING-PROTEIN | IMMUNOLOGY | mast cells | SOLUBLE CD14 LEVELS | CEREBROSPINAL-FLUID | GRAM-NEGATIVE BACTERIA | ENDOTHELIAL-CELLS | TOLL-LIKE RECEPTORS | lipopolysaccharide | NF-KAPPA-B | UP-REGULATION | INNATE IMMUNITY
Lipopolysaccharide | Mast cells | CD14 | TLR4 | MURINE MACROPHAGES | BINDING-PROTEIN | IMMUNOLOGY | mast cells | SOLUBLE CD14 LEVELS | CEREBROSPINAL-FLUID | GRAM-NEGATIVE BACTERIA | ENDOTHELIAL-CELLS | TOLL-LIKE RECEPTORS | lipopolysaccharide | NF-KAPPA-B | UP-REGULATION | INNATE IMMUNITY
Journal Article
European Journal of Immunology, ISSN 0014-2980, 06/2005, Volume 35, Issue 6, pp. 1918 - 1927
Type I interferons (IFN) play a critical role in the Toll‐like receptor (TLR)‐mediated expression of B7 costimulatory family members. For example, LPS‐induced...
Toll‐like receptor | B cells | Macrophages | B7RP‐1 | Type I IFN | B7RP-1 | B cells | Toll-like receptor | ACTIVATED PROTEIN-KINASE | DENDRITIC CELLS | C-FOS | type IIFN | INDUCIBLE COSTIMULATOR LIGAND | IMMUNOLOGY | CUTTING EDGE | GERMINAL CENTER FORMATION | TOLL-LIKE RECEPTOR-2 | macrophages | IMMUNE-RESPONSES | T-CELL-ACTIVATION | B7 FAMILY | toll-like receptor | Up-Regulation | Poly I-C - pharmacology | Adaptor Proteins, Vesicular Transport - physiology | B7-1 Antigen - genetics | Receptors, Immunologic - physiology | Toll-Like Receptor 4 | Mice, Inbred C57BL | Gene Expression Regulation | Myeloid Differentiation Factor 88 | Mice, Inbred C3H | Antigens, Differentiation - physiology | Toll-Like Receptors | Adaptor Proteins, Signal Transducing | Membrane Glycoproteins - physiology | Animals | Inducible T-Cell Co-Stimulator Ligand | Interferon-beta - pharmacology | Lipopolysaccharides - pharmacology | Signal Transduction - physiology | Mice | Receptors, Cell Surface - physiology
Toll‐like receptor | B cells | Macrophages | B7RP‐1 | Type I IFN | B7RP-1 | B cells | Toll-like receptor | ACTIVATED PROTEIN-KINASE | DENDRITIC CELLS | C-FOS | type IIFN | INDUCIBLE COSTIMULATOR LIGAND | IMMUNOLOGY | CUTTING EDGE | GERMINAL CENTER FORMATION | TOLL-LIKE RECEPTOR-2 | macrophages | IMMUNE-RESPONSES | T-CELL-ACTIVATION | B7 FAMILY | toll-like receptor | Up-Regulation | Poly I-C - pharmacology | Adaptor Proteins, Vesicular Transport - physiology | B7-1 Antigen - genetics | Receptors, Immunologic - physiology | Toll-Like Receptor 4 | Mice, Inbred C57BL | Gene Expression Regulation | Myeloid Differentiation Factor 88 | Mice, Inbred C3H | Antigens, Differentiation - physiology | Toll-Like Receptors | Adaptor Proteins, Signal Transducing | Membrane Glycoproteins - physiology | Animals | Inducible T-Cell Co-Stimulator Ligand | Interferon-beta - pharmacology | Lipopolysaccharides - pharmacology | Signal Transduction - physiology | Mice | Receptors, Cell Surface - physiology
Journal Article
Comptes rendus - Biologies, ISSN 1631-0691, 2004, Volume 327, Issue 6, pp. 571 - 580
The innate immune system senses pathogens largely through signals initiated by proteins known as 'Toll-like receptors' (TLRs), of which ten representatives are...
type I interferon | TLR family | costimulatory molecule expression | Toll-like receptors | human genome | adapter proteins | innate immune system | Human genome | Type I interferon | Innate immune system | Adapter proteins | Costimulatory molecule expression | INDUCED MITOGENESIS | costimulatory | DOUBLE-STRANDED-RNA | MULTIDISCIPLINARY SCIENCES | IL-1 RECEPTOR | BACTERIAL LIPOPOLYSACCHARIDE LPS | MYD88-DEFICIENT MICE | SIGNALING PATHWAY | MOUSE SPLEEN-CELLS | DROSOPHILA TOLL | BIOLOGY | SALMONELLA-TYPHIMURIUM | molecule expression | IMMUNOLOGICAL-PROPERTIES | Toll-Like Receptors | Animals | Humans | Membrane Glycoproteins - immunology | Mammals | Antigens, Surface - immunology | Immunity | Receptors, Cell Surface - immunology
type I interferon | TLR family | costimulatory molecule expression | Toll-like receptors | human genome | adapter proteins | innate immune system | Human genome | Type I interferon | Innate immune system | Adapter proteins | Costimulatory molecule expression | INDUCED MITOGENESIS | costimulatory | DOUBLE-STRANDED-RNA | MULTIDISCIPLINARY SCIENCES | IL-1 RECEPTOR | BACTERIAL LIPOPOLYSACCHARIDE LPS | MYD88-DEFICIENT MICE | SIGNALING PATHWAY | MOUSE SPLEEN-CELLS | DROSOPHILA TOLL | BIOLOGY | SALMONELLA-TYPHIMURIUM | molecule expression | IMMUNOLOGICAL-PROPERTIES | Toll-Like Receptors | Animals | Humans | Membrane Glycoproteins - immunology | Mammals | Antigens, Surface - immunology | Immunity | Receptors, Cell Surface - immunology
Journal Article
Blood, ISSN 0006-4971, 11/2004, Volume 104, Issue 11, pp. 649 - 649
Abstract Toll-like receptors (TLRs) are key sensors of the innate immune system, and individual TLRs respond to specific molecules derived from microbes. MyD88...
Journal Article
European Journal of Immunology, ISSN 0014-2980, 03/2006, Volume 36, Issue 3, pp. 701 - 711
Lipopolysaccharide (endotoxin, LPS) is a major recognition marker for the detection of gram‐negative bacteria by the host and a powerful initiator of the...
Lipopolysaccharide | Mast cells | CD14 | TLR4 | Immunity, Innate - drug effects | Species Specificity | Cells, Cultured | Gram-Negative Bacterial Infections - genetics | Immunity, Innate - genetics | Escherichia coli - immunology | Gram-Negative Bacterial Infections - immunology | Salmonella - genetics | Lipopolysaccharide Receptors - immunology | Toll-Like Receptor 4 - immunology | Lipopolysaccharides - immunology | Mice, Knockout | Immunity, Innate - immunology | Salmonella - immunology | Animals | Toll-Like Receptor 4 - deficiency | Escherichia coli - genetics | Lipopolysaccharides - pharmacology | Mice | Lymphocyte Antigen 96 - deficiency | Lymphocyte Antigen 96 - immunology
Lipopolysaccharide | Mast cells | CD14 | TLR4 | Immunity, Innate - drug effects | Species Specificity | Cells, Cultured | Gram-Negative Bacterial Infections - genetics | Immunity, Innate - genetics | Escherichia coli - immunology | Gram-Negative Bacterial Infections - immunology | Salmonella - genetics | Lipopolysaccharide Receptors - immunology | Toll-Like Receptor 4 - immunology | Lipopolysaccharides - immunology | Mice, Knockout | Immunity, Innate - immunology | Salmonella - immunology | Animals | Toll-Like Receptor 4 - deficiency | Escherichia coli - genetics | Lipopolysaccharides - pharmacology | Mice | Lymphocyte Antigen 96 - deficiency | Lymphocyte Antigen 96 - immunology
Journal Article
Comptes Rendus Biologies, ISSN 1631-0691, 06/2004, Volume 327, Issue 6, pp. 571 - 580
The innate immune system senses pathogens largely through signals initiated by proteins known as `Toll-like receptors' (TLRs), of which ten representatives are...
Journal Article
Blood, ISSN 0006-4971, 11/2004, Volume 104, Issue 11, pp. 3441 - 3441
Abstract The mammalian Toll-like receptors (TLRs) comprise a key interface between cells of the host and all classes of microbial pathogen. By sensing nucleic...
Journal Article
1920, United States congressional serial set, Volume no. 728, 790
Seeds | World War I (1914-1918) | Agricultural credit | United States | Poultry | Communicable diseases | Food and drug inspection | Executive Department Publications | Agricultural labor | Animal diseases | Agricultural subsidies | Electric lighting | Federal aid to agriculture | Produce trade | Cooperatives | Lime | Animal breeding | Droughts | Farms | Agricultural production | Disaster relief | Electric power | Farm management | Dept. of Agriculture | Veterinary public health | Tobacco plantations | Meat industry and trade | Fruit | Agricultural colleges | Pink bollworm | Monographs | Agricultural economics | Fertilizers | Drainage | Depreciation | Trapping | Tuberculosis | Horses | Annual Reports | Hog cholera | Livestock | Food supply | Forest reserves | Wheat
eBook
Information Systems Journal, ISSN 1350-1917, 05/2015, Volume 25, Issue 3, pp. 231 - 273
Journal Article
Information Systems Journal, ISSN 1350-1917, 05/2015, Volume 25, Issue 3, pp. 231 - 273
Despite the growing importance of information technology (IT) interruptions for individual work, very little is known about their nature and consequences. This...
IT interruptions | individual performance | taxonomy | interviews | log diaries | Individual performance | Taxonomy | Interviews | Log diaries | OVERLOAD | E-MAIL | TASK-PERFORMANCE | KNOWLEDGE | HUMAN-COMPUTER INTERACTION | INFORMATION SCIENCE & LIBRARY SCIENCE | SYSTEMS | WORK | STRESS | FEEDBACK | COMMUNICATION | Diaries | Interruption | Classification | Continuums | Paper | Product development | Information technology | Information systems | Business administration | Humanities and Social Sciences
IT interruptions | individual performance | taxonomy | interviews | log diaries | Individual performance | Taxonomy | Interviews | Log diaries | OVERLOAD | E-MAIL | TASK-PERFORMANCE | KNOWLEDGE | HUMAN-COMPUTER INTERACTION | INFORMATION SCIENCE & LIBRARY SCIENCE | SYSTEMS | WORK | STRESS | FEEDBACK | COMMUNICATION | Diaries | Interruption | Classification | Continuums | Paper | Product development | Information technology | Information systems | Business administration | Humanities and Social Sciences
Journal Article
1907, 662
Includes bibliographical references (p. 551-570) Includes legislation. Includes index. "... also the acquisition and maintenance of mining rights in the public...
Natural Resources | US
Natural Resources | US
Book
Clinical Chemistry, ISSN 0009-9147, 07/1997, Volume 43, Issue 7, pp. 1203 - 1208
The analytical performance of the Tandem(R)-R free PSA assay available from Hybritech Inc. was evaluated. Comparison of recoveries of purified free (unbound)...
Prostate cancer | Tumor markers | KALLIKREIN | prostate cancer | tumor markers | GENE | MARKER | SERUM | K PW MEDICAL LABORATORY TECHNOLOGY | MEDICAL LABORATORY TECHNOLOGY | CANCER | ALPHA-1-ANTICHYMOTRYPSIN | Immunoradiometric Assay - statistics & numerical data | Humans | Sensitivity and Specificity | alpha 1-Antichymotrypsin - blood | Immunoradiometric Assay - methods | Female | Calibration | Prostate-Specific Antigen - blood | Quality Control | Reagent Kits, Diagnostic - statistics & numerical data | Measurement | Comparative analysis | Antigens
Prostate cancer | Tumor markers | KALLIKREIN | prostate cancer | tumor markers | GENE | MARKER | SERUM | K PW MEDICAL LABORATORY TECHNOLOGY | MEDICAL LABORATORY TECHNOLOGY | CANCER | ALPHA-1-ANTICHYMOTRYPSIN | Immunoradiometric Assay - statistics & numerical data | Humans | Sensitivity and Specificity | alpha 1-Antichymotrypsin - blood | Immunoradiometric Assay - methods | Female | Calibration | Prostate-Specific Antigen - blood | Quality Control | Reagent Kits, Diagnostic - statistics & numerical data | Measurement | Comparative analysis | Antigens
Journal Article
2009, ISBN 9781848009226
Book Chapter