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Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. 4236 - 4236
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 12/2015, Volume 14, Issue 12 Supplement 2, pp. A191 - A191
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 12/2015, Volume 14, Issue 12 Supplement 2, pp. B197 - B197
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2016, Volume 22, Issue 8, pp. 851 - 860
Single-agent immunotherapy has achieved limited clinical benefit to date in patients with pancreatic ductal adenocarcinoma (PDAC). This may be a result of the... 
MEDICINE, RESEARCH & EXPERIMENTAL | SKIN WOUND REPAIR | DUCTAL ADENOCARCINOMA | DECREASES GROWTH | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-INFILTRATING MACROPHAGES | 2 KNOCKOUT MICE | ACTIN CYTOSKELETAL REARRANGEMENT | TISSUE STIFFNESS | CELL BIOLOGY | FAK | ANTIGEN RECEPTOR | T-CELLS | Immunohistochemistry | Pancreatic Neoplasms - metabolism | Humans | Carcinoma, Pancreatic Ductal - metabolism | Tumor Microenvironment | Deoxycytidine - pharmacology | Immunoblotting | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Immunotherapy | Antimetabolites, Antineoplastic - pharmacology | Carcinoma, Pancreatic Ductal - immunology | Disease Models, Animal | Tumor Escape - immunology | Immunotherapy, Adoptive - methods | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Pancreatic Neoplasms - pathology | Reverse Transcriptase Polymerase Chain Reaction | Carcinoma, Pancreatic Ductal - pathology | Focal Adhesion Protein-Tyrosine Kinases - antagonists & inhibitors | Disease Progression | Animals | Aminopyridines - pharmacology | Pancreatic Neoplasms - immunology | Fibrosis | Cell Proliferation - drug effects | Mice | CD8-Positive T-Lymphocytes - immunology | Deoxycytidine - analogs & derivatives | Molecular targeted therapy | Care and treatment | Pancreatic cancer | Patient outcomes | Innovations | Genetic aspects | Properties | Phosphotransferases | Methods | Kinases | Cell adhesion & migration | pancreatic cancer | PDAC | CXCL12 | VS-4718 | immunotherapy | fibrosis | tumor microenvironment | PD-1
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 10/2013, Volume 19, Issue 19 Supplement, pp. B13 - B13
Journal Article
Cancer Research, ISSN 0008-5472, 10/2014, Volume 74, Issue 19 Supplement, pp. 4797 - 4797
Journal Article
Blood, ISSN 0006-4971, 12/2015, Volume 126, Issue 23, pp. 3766 - 3766
Abstract Introduction: BCR-ABL1+ B-progenitor acute lymphoblastic leukemia (Ph+ B-ALL) is a highly aggressive disease that is often refractory to currently... 
Journal Article
Cancer Research, ISSN 0008-5472, 04/2013, Volume 73, Issue 8 Supplement, pp. 924 - 924
Journal Article
Science Translational Medicine, ISSN 1946-6234, 05/2014, Volume 6, Issue 237, pp. 237ra68 - 237ra68
Journal Article
PLoS Genetics, ISSN 1553-7390, 08/2011, Volume 7, Issue 8
  Epithelial-mesenchymal transition (EMT), a mechanism important for embryonic development, plays a critical role during malignant transformation. While much... 
Proteins | Cell culture | Genotype & phenotype | Automation | Growth hormones | Breast cancer | Gene expression | Tumors
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, p. e0180181
Cancer stem cells (CSCs) play an important role in the clonogenic growth and metastasis of pancreatic ductal adenocarcinoma (PDAC). A hallmark of PDAC is the... 
EPITHELIAL CANCER | MULTIPLE-MYELOMA | BREAST-CANCER | FAK | TGF-BETA | MULTIDISCIPLINARY SCIENCES | SELF-RENEWAL | I COLLAGEN | MALIGNANT PHENOTYPE | TUMOR-GROWTH | OVARIAN-CARCINOMA | Collagen Type I - metabolism | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Neoplastic Stem Cells - drug effects | Extracellular Matrix - drug effects | Humans | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - enzymology | Extracellular Matrix - metabolism | Carcinoma, Pancreatic Ductal - pathology | Cell Self Renewal - drug effects | Carcinoma, Pancreatic Ductal - enzymology | Integrin beta1 - metabolism | Cell Movement - drug effects | Neoplasm Metastasis | Animals | Neoplastic Stem Cells - metabolism | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Cell Proliferation - drug effects | Clone Cells | Protein Kinase Inhibitors - pharmacology | Aldehyde Dehydrogenase - metabolism | Adenocarcinoma | Care and treatment | Pancreatic cancer | Collagen | Stem cells | Extracellular matrix | Research | Metastasis | Diagnosis | Integrins | Collagen (type I) | Gemcitabine | Stem cell transplantation | Oncology | Kinases | Drug resistance | Metastases | Proteins | Well construction | Allografts | Paclitaxel | Xenografts | Cell cycle | Pancreas | Medical research | Departments | Adhesion | Medicine | Chemotherapy | Inhibitors | In vivo methods and tests | Focal adhesion kinase | In vitro methods and tests | Tumors | Cancer
Journal Article
Oncotarget, ISSN 1949-2553, 02/2017, Volume 8, Issue 31, pp. 51733 - 51747
Because cancer stem cells (CSCs) have been implicated in chemo-resistance, metastasis and tumor recurrence, therapeutic targeting of CSCs holds promise to... 
FAK | VS-6063 | Focal adhesion kinase | Cancer stem cells | VS-4718 | METASTASIS | ALDEHYDE DEHYDROGENASE | BREAST | CHEMOTHERAPY | BETA | CELL BIOLOGY | focal adhesion kinase | cancer stem cells | TUMOR-GROWTH | PROMOTES | EXPRESSION
Journal Article
Cancer research, ISSN 0008-5472, 2/2014, Volume 74, Issue 4, pp. 1261 - 1271
Malignant mesothelioma (MM) is a highly aggressive, asbestos-related cancer frequently marked by mutations of both NF2 and CDKN2A . We demonstrate that... 
mouse models | tumor suppressor genes | cancer stem cells | malignant mesothelioma | metastasis
Journal Article
Clinical Therapeutics, ISSN 0149-2918, 2007, Volume 29, Issue 11, pp. 2374 - 2384
Abstract Background: Modern premixed insulins offer a flexible approach to the initiation of insulin therapy in patients with poorly controlled type 2... 
Internal Medicine | Medical Education | premixed insulin | type 2 diabetes | intensification | oral antidiabetic drugs | therapy | GLYCEMIC CONTROL | DOUBLE-BLIND CROSSOVER | NPH INSULIN | GLARGINE | GLUCOSE | POSTPRANDIAL HYPERGLYCEMIA | PHARMACOLOGY & PHARMACY | COMPLICATIONS | QUALITY-OF-LIFE | TREATMENT SATISFACTION | Hypoglycemia - epidemiology | Metformin - therapeutic use | Glycated Hemoglobin A - metabolism | Humans | Middle Aged | Male | Biphasic Insulins | Metformin - adverse effects | Russia | Insulin, Isophane | Hypoglycemic Agents - administration & dosage | Endpoint Determination | Female | Weight Gain - drug effects | Insulin - analogs & derivatives | Metformin - administration & dosage | Drug Therapy, Combination | Hypoglycemia - chemically induced | Hypoglycemic Agents - therapeutic use | Diabetes Mellitus, Type 2 - psychology | Insulin - administration & dosage | Diabetes Mellitus, Type 2 - blood | Insulin Aspart | Biomarkers | Quality of Life | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Hypoglycemic Agents - adverse effects | Insulin - adverse effects | Insulin - therapeutic use | Type 2 diabetes | Medical colleges | Complications and side effects | Care and treatment | Blood sugar | Glycosylated hemoglobin | Comparative analysis | Insulin | Medicine, Preventive | Diabetes therapy | Preventive health services
Journal Article
JCI INSIGHT, ISSN 2379-3708, 04/2016, Volume 1, Issue 4
BCR-ABL1(+) B progenitor acute lymphoblastic leukemia (Ph+ B-ALL) is an aggressive disease that frequently responds poorly to currently available therapies.... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | PHOSPHORYLATION | IKAROS | ACUTE LYMPHOBLASTIC-LEUKEMIA | MOUSE MODEL | BCR-ABL | IMATINIB | FOCAL ADHESION KINASE | GENETIC ALTERATIONS | HAPLOINSUFFICIENCY
Journal Article
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