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Cancer Biology & Therapy, ISSN 1538-4047, 09/2018, Volume 19, Issue 9, pp. 755 - 762
Apo2 ligand (Apo2L)/tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) is unique to selectively induce apoptosis in tumor cells while sparing... 
prostate cancer | autophagy | flux | p62/SQSTM1 | Apo2L/TRAIL | autophagosome | Apoptosis | ACTIVATION | TRAIL | CANCER-CELL-DEATH | CRYSTAL-STRUCTURE | RECEPTOR 5 | p62 | TNF FAMILY | BREAST-CANCER | Apo2L | APOPTOSIS-INDUCING LIGAND | NECROSIS | ONCOLOGY | TRAIL-INDUCED APOPTOSIS | CASPASE-8 | SQSTM1
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e60408
Exposure to genotoxic agents, such as irradiation produces DNA damage, the toxicity of which is augmented when the DNA repair is impaired. Poly (ADP-ribose)... 
CARCINOMA-CELLS | LYMPHOMA-CELLS | AG014699 | GENOTOXIC STRESS | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | HOMOLOGOUS RECOMBINATION | IONIZING-RADIATION | TUMORS | PROGRESSION | POLY(ADP-RIBOSE) POLYMERASE INHIBITOR | Transcriptional Regulator ERG | Cell Line | Cell Survival - drug effects | PTEN Phosphohydrolase - genetics | Prostatic Neoplasms - metabolism | Humans | Trans-Activators | Cellular Senescence - drug effects | Morpholines - pharmacology | PTEN Phosphohydrolase - metabolism | Male | Cell Survival - radiation effects | Recombinant Fusion Proteins | Poly(ADP-ribose) Polymerases - metabolism | Poly(ADP-ribose) Polymerases - genetics | Cellular Senescence - radiation effects | Fluorescent Antibody Technique | Cell Line, Tumor | Indoles - pharmacology | Serine Endopeptidases | Chromones - pharmacology | DNA damage | Analysis | Genetic aspects | Research | DNA repair | Prostate cancer | Cancer | Cell culture | Protein kinase C | Radiosensitivity | Homologous recombination | Lung cancer | Genotoxicity | Radiation | Galactosidase | Phosphatase | ADP | Mimicry | Proteins | Fusion protein | Deoxyribonucleic acid--DNA | Sensitivity analysis | BRCA1 protein | Gene expression | Survival | Radiation dosage | ERG gene | Inhibitors | Irradiation | Sensitivity enhancement | Lymphomas | Mutation | Radiation damage | ETS protein | Senescence | Toxicity | Homology | Biology | Activation | Ribose | Rodents | Cell cycle | Brachytherapy | BRCA2 protein | Cell survival | Poly(ADP-ribose) polymerase | Breast cancer | Radiation therapy | Polymerase | Sensitivity | DNA-dependent protein kinase | Cell lines | Prostate | PTEN protein | Tumors | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 08/2019, Volume 10, Issue 1, pp. 3668 - 15
Breast cancer stem cells (BCSCs) are unique in their ability to undergo unlimited self-renewal, an essential process in breast cancer recurrence following... 
EPITHELIAL-MESENCHYMAL TRANSITION | GLYCOLYSIS | BREAST-CANCER CELLS | STEM-CELLS | MULTIDISCIPLINARY SCIENCES | GROWTH | MECHANISMS | FRUCTOSE 2,6-BISPHOSPHATE | CHLOROQUINE | CHEMOTHERAPY | PROGRESSION | Activation | Breast cancer | Metastasis | Kinases | Gene expression | Autophagy | Fructose | Metastases | Dormancy | Depletion | DNA microarrays | Stem cells | Breast | Disruption | Lesions | 6-Phosphofructo-2-kinase | Phagocytosis
Journal Article
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 2012, Volume 920, pp. 613 - 626
Conference Proceeding
Autophagy, ISSN 1554-8627, 11/2018, Volume 14, Issue 11, pp. 1976 - 1990
Recent reports have made important revelations, uncovering direct regulation of DNA damage response (DDR)-associated proteins and chromatin ubiquitination... 
RNF168 | USP14 | DNA damage response | Autophagy | deubiquitinase | INDUCED APOPTOSIS | DOUBLE-STRAND BREAKS | CELL BIOLOGY | GENOMIC INSTABILITY | PATHWAY | PROSTATE-CANCER | DEPENDENT UBIQUITINATION | DEGRADATION | HOMOLOGOUS RECOMBINATION | PROMOTES
Journal Article
01/2014, ISBN 9781321477139
Deregulated mTORC1 contributes to tumorigenesis and chemoresistance. However, mTORC1-specific inhibitors (rapalogs) show modest efficacy in the clinic, as they... 
Cellular biology | Biochemistry | Molecular biology
Dissertation
British journal of haematology, ISSN 0007-1048, 10/2013, Volume 163, Issue 1, pp. 72 - 80
Previously, we showed that inhibition of the protein kinase C β (PKCβ)/AKT pathway augments engagement of the histone deacetylase inhibitor (HDI)-induced... 
CLL | Non-Hodgkin Lymphoma | Synergy | PI3K | Apoptosis | HDAC
Journal Article
British Journal of Haematology, ISSN 0007-1048, 10/2013, Volume 163, Issue 1, pp. 72 - 80
Previously, we showed that inhibition of the protein kinase C β ( PKC β)/ AKT pathway augments engagement of the histone deacetylase inhibitor ( HDI )‐induced... 
chronic lymphocytic leukaemia | synergy | phosphatidylinositide 3‐kinase | apoptosis | non | histone deacetylase | odgkin lymphoma | Non-Hodgkin lymphoma | Histone deacetylase | Phosphatidylinositide 3-kinase | Synergy | Chronic lymphocytic leukaemia | Apoptosis | SURVIVAL | ACTIVATION | MECHANISM | SUBEROYLANILIDE HYDROXAMIC ACID | non-Hodgkin lymphoma | AKT | B-CELL LYMPHOMA | ABILITY | DRUGS | HEMATOLOGY | NF-KAPPA-B | phosphatidylinositide 3-kinase | Quinazolinones - toxicity | Apoptosis - drug effects | Humans | Phosphatidylinositol 3-Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Antineoplastic Agents - toxicity | Caspases - metabolism | Purines - toxicity | Inhibitory Concentration 50 | Indoles - pharmacology | Antineoplastic Agents - pharmacology | Hydroxamic Acids - pharmacology | Quinazolinones - pharmacology | Hydroxamic Acids - toxicity | Lymphoma, B-Cell - metabolism | Purines - pharmacology | Indoles - toxicity | Enzyme Activation - drug effects | Drug Synergism | Signal Transduction - drug effects | Histone Deacetylase Inhibitors - toxicity | Models, Biological | Cell Line, Tumor | Histone Deacetylase Inhibitors - pharmacology | Cell Proliferation - drug effects | Analysis | Leukemia | Genetic research | Lymphomas | Phospholipids | Phosphotransferases
Journal Article
Journal Article
PLoS ONE, 04/2013, Volume 8, Issue 4
Exposure to genotoxic agents, such as irradiation produces DNA damage, the toxicity of which is augmented when the DNA repair is impaired. Poly (ADP-ribose)... 
BRCA1 protein
Journal Article
Annals of Neurosciences, ISSN 0972-7531, 01/2006, Volume 13, Issue 1, pp. 1 - 4
The imbalance between oxidants and the antioxidant defense system in Li-pilocarpine induced status epilepticus (SE) was studied by measuring lipid... 
Journal Article
01/2014, ISBN 9781321477139, Volume 76, Issue 5
Deregulated mTORC1 contributes to tumorigenesis and chemoresistance. However, mTORC1-specific inhibitors (rapalogs) show modest efficacy in the clinic, as they... 
Book Chapter
Journal Article
Journal of Biomedicine and Biotechnology, ISSN 1110-7243, 2009, Volume 2009, pp. 1 - 9
Journal Article
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