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Journal Article
Cancer Cell, ISSN 1535-6108, 2010, Volume 17, Issue 6, pp. 547 - 559
In mice, deletion and activation of results in lung tumors with a high penetrance of lymph node and distant metastases. We analyzed these primary and... 
CELLCYCLE | SIGNALING | INACTIVATION | SUPPRESSOR | SIGNATURES | ONCOLOGY | SRC | ADENOCARCINOMA | SENSITIVITY | MUTATIONS | LKB1/STK11 | EXPRESSION | TUMORIGENESIS | CELL BIOLOGY | Lung Neoplasms - drug therapy | Protein-Serine-Threonine Kinases - deficiency | Protein-Tyrosine Kinases - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Genomics | Humans | Lung Neoplasms - metabolism | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Cell Movement - genetics | Phosphorylation - genetics | RNA Interference | Gene Expression Regulation, Neoplastic - genetics | MAP Kinase Kinase 1 - antagonists & inhibitors | Carcinoma, Non-Small-Cell Lung - metabolism | Signal Transduction - genetics | Enzyme Inhibitors - therapeutic use | Focal Adhesion Protein-Tyrosine Kinases - antagonists & inhibitors | Focal Adhesion Protein-Tyrosine Kinases - genetics | Focal Adhesions - genetics | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Mice | TOR Serine-Threonine Kinases | src-Family Kinases - genetics | Protein-Tyrosine Kinases - antagonists & inhibitors | ras Proteins - genetics | Lung Neoplasms - pathology | Cell Transdifferentiation - genetics | Protein-Tyrosine Kinases - genetics | Neoplasm Metastasis - drug therapy | Mice, Mutant Strains | Protein-Serine-Threonine Kinases - antagonists & inhibitors | src-Family Kinases - metabolism | Female | Drug Therapy, Combination | Lung Neoplasms - genetics | Cell Adhesion - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | src-Family Kinases - antagonists & inhibitors | Protein-Serine-Threonine Kinases - genetics | Proto-Oncogene Proteins - genetics | Up-Regulation - genetics | Xenograft Model Antitumor Assays | Neoplasm Metastasis - genetics | Animals | MAP Kinase Kinase 2 - antagonists & inhibitors | Protein Kinase Inhibitors - therapeutic use | Focal Adhesions - metabolism | Proteomics | Protein Kinase Inhibitors - pharmacology | Oncology, Experimental | Analysis | Lung cancer | Development and progression | Metastasis | Research | Cancer
Journal Article
Cancer Cell, ISSN 1535-6108, 2006, Volume 9, Issue 6, pp. 485 - 495
Journal Article
Oncogene, ISSN 0950-9232, 03/2017, Volume 36, Issue 9, pp. 1309 - 1314
Exposure of murine and human tissues to ionizing radiation (IR) induces the expression of p16[sup.INK4a], a tumor suppressor gene and senescence/aging... 
Tumor suppressor genes | Ionizing radiation | Genetic aspects | Gene expression | Health aspects | Carcinogenesis | Biomarkers | Tissue | Rodents | senescence | tumor suppressor | cell cycle | p16INK4a | ionizing irradiation
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 05/2015, Volume 21, Issue 9, pp. 2167 - 2176
Journal Article
Journal Article
EXPERIMENTAL GERONTOLOGY, ISSN 0531-5565, 02/2016, Volume 74, pp. 29 - 36
Adults older than 65 years undergo more than 120,000 coronary artery bypass (CAB) procedures each year in the United States. Chronological age alone, though... 
Senescence | STEM-CELLS | PHYSICAL PERFORMANCE | RISK | p16(INK4a) levels | INTERLEUKIN-6 | CELLULAR SENESCENCE | GERIATRICS & GERONTOLOGY | Coronary artery bypass | Length of hospital stay | INTENSIVE-CARE-UNIT | Chronological age | DISEASE | GROWTH | Frailty | Aging biomarker | Peripheral blood T-lymphocytes | EXPRESSION | AGE
Journal Article
Nano Letters, ISSN 1530-6984, 01/2017, Volume 17, Issue 1, pp. 242 - 248
Journal Article
Journal Article
Journal Article
Journal Article
Cancer Discovery, ISSN 2159-8274, 12/2014, Volume 4, Issue 12, pp. 1418 - 1429
Journal Article