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1. Full Text Gut microbiome and serum metabolome alterations in obesity and after weight-loss intervention
by Liu, Ruixin and Hong, Jie and Xu, Xiaoqiang and Feng, Qiang and Zhang, Dongya and Gu, Yanyun and Shi, Juan and Zhao, Shaoqian and Liu, Wen and Wang, Xiaokai and Xia, Huihua and Liu, Zhipeng and Cui, Bin and Liang, Peiwen and Xi, Liuqing and Jin, Jiabin and Ying, Xiayang and Wang, Xiaolin and Zhao, Xinjie and Li, Wanyu and Jia, Huijue and Lan, Zhou and Li, Fengyu and Wang, Rui and Sun, Yingkai and Yang, Minglan and Shen, Yuxin and Jie, Zhuye and Li, Junhua and Chen, Xiaomin and Zhong, Huanzi and Xie, Hailiang and Zhang, Yifei and Gu, Weiqiong and Deng, Xiaxing and Shen, Baiyong and Xu, Xun and Yang, Huanming and Xu, Guowang and Bi, Yufang and Lai, Shenghan and Wang, Jian and Qi, Lu and Madsen, Lise and Wang, Jiqiu and Ning, Guang and Kristiansen, Karsten and Wang, Weiqing
Nature Medicine, ISSN 1078-8956, 07/2017, Volume 23, Issue 7, pp. 859 - 868
Emerging evidence has linked the gut microbiome to human obesity. We performed a metagenome-wide association study and serum metabolomics profiling in a cohort... 
MEDICINE, RESEARCH & EXPERIMENTAL | THETAIOTAOMICRON | RICHNESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | Y GASTRIC BYPASS | MARKERS | DIET-INDUCED OBESITY | CELL BIOLOGY | MONOSODIUM GLUTAMATE | IMPACT | GLUCOSE | GENES | BARIATRIC SURGERY | Humans | Male | Dysbiosis - microbiology | Obesity - microbiology | Bacteroides thetaiotaomicron - genetics | Case-Control Studies | Young Adult | Adiposity | Adult | DNA, Bacterial - analysis | Female | Weight Gain | Bariatric Surgery | Obesity - surgery | Metabolome | Fusobacterium - genetics | Gastrectomy | Gastrointestinal Microbiome - genetics | Dysbiosis - metabolism | Obesity - metabolism | Bacteroides - genetics | Animals | Glutamic Acid - blood | Mice | Metagenome | Obesity | Usage | Microbiota (Symbiotic organisms) | Patient outcomes | Analysis | Surgery | Weight loss | Glutamate | Health aspects | Intervention | Glutamic acid | Metabolomics | Stomach | Profiling | Adipose tissue | Amino acids | Abundance | Alterations | Microorganisms | Weight control | Metabolites | Intestine | Human performance | Rodents | Weight reduction | Links | Gastrointestinal surgery | Intestinal microflora | Digestive tract
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2. Full Text Long noncoding RNA NORAD, a novel competing endogenous RNA, enhances the hypoxia-induced epithelial-mesenchymal transition to promote metastasis in pancreatic cancer
by Li, Hongzhe and Wang, Xinjing and Wen, Chenlei and Huo, Zhen and Wang, Weishen and Zhan, Qian and Cheng, Dongfeng and Chen, Hao and Deng, Xiaxing and Peng, Chenghong and Shen, Baiyong
Molecular Cancer, ISSN 1476-4598, 11/2017, Volume 16, Issue 1, pp. 169 - 14
Background: Pancreatic cancer, one of the top two most fatal cancers, is characterized by a desmoplastic reaction that creates a dense microenvironment,... 
MIGRATION | INVASION | ONCOLOGY | DUCTAL ADENOCARCINOMA | PUMILIO PROTEINS | COLORECTAL-CANCER | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | PROLIFERATION | IDENTIFICATION | RENAL-CELL CARCINOMA | MIR-125A-3P | Immunohistochemistry | Care and treatment | Prognosis | Analysis | Pancreatic cancer | Metastasis | Diagnosis | Research | Binding proteins | Gene expression
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3. Full Text Downregulation of gas5 increases pancreatic cancer cell proliferation by regulating CDK6
by Lu, Xiongxiong and Fang, Yuan and Wang, Zhengting and Xie, Junjie and Zhan, Qian and Deng, Xiaxing and Chen, Hao and Jin, Jiabin and Peng, Chenghong and Li, Hongwei and Shen, Baiyong
Cell and Tissue Research, ISSN 0302-766X, 12/2013, Volume 354, Issue 3, pp. 891 - 896
Recent studies have revealed that long non-coding RNAs (lncRNAs) play important roles in cancer biology and that lncRNA gas5 (growth arrest-specific 5)... 
Human Genetics | Biomedicine | CDK6 | lncRNA | Pancreatic cancer | Proteomics | Cell cycle | Molecular Medicine | Gas5 | BLADDER-CANCER | METASTASIS | GENOME | CELL BIOLOGY | H19 CONTRIBUTES | LONG NONCODING RNA | GROWTH-ARREST | EXPRESSION | Pancreatic Neoplasms - metabolism | Down-Regulation | Humans | Middle Aged | Pancreatic Neoplasms - pathology | Cyclin-Dependent Kinase 6 - genetics | Gene Expression Regulation, Neoplastic | Cell Growth Processes - physiology | Pancreatic Neoplasms - genetics | Cyclin-Dependent Kinase 6 - metabolism | RNA, Long Noncoding - genetics | Cell Line, Tumor | Aged | RNA, Long Noncoding - metabolism | Development and progression | Cell growth | Gene expression | Ribonucleic acid--RNA
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4. Full Text Whole-exome and targeted gene sequencing of gallbladder carcinoma identifies recurrent mutations in the ErbB pathway
by Li, Maolan and Zhang, Zhou and Li, Xiaoguang and Ye, Junyi and Wu, Xiangsong and Tan, Zhujun and Liu, Chang and Shen, Baiyong and Wang, Xu-An and Wu, Wenguang and Zhou, Daizhan and Zhang, Di and Wang, Ting and Liu, Bingya and Qu, Kai and Ding, Qichen and Weng, Hao and Ding, Qian and Mu, Jiasheng and Shu, Yijun and Bao, Runfa and Cao, Yang and Chen, Peizhan and Liu, Tianyu and Jiang, Lin and Hu, Yunping and Dong, Ping and Gu, Jun and Lu, Wei and Shi, Weibin and Lu, Jianhua and Gong, Wei and Tang, Zhaohui and Zhang, Yong and Wang, Xuefeng and Chin, Y Eugene and Weng, Xiaoling and Zhang, Hong and Tang, Wei and Zheng, Yonglan and He, Lin and Wang, Hui and Liiu, Yun and Liu, Yingbin
Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 8, pp. 872 - 876
Individuals with gallbladder carcinoma (GBC), the most aggressive malignancy of the biliary tract, have a poor prognosis. Here we report the identification of... 
MUTAGENESIS | BILIARY-TRACT | HUMAN CANCERS | GENOME | GENETICS & HEREDITY | Cell Line | Receptor, Epidermal Growth Factor - genetics | Humans | Middle Aged | Gallbladder Neoplasms - genetics | Male | Signal Transduction - genetics | Neoplasm Recurrence, Local - enzymology | Exome | Gallbladder Neoplasms - enzymology | Carcinoma - enzymology | HEK293 Cells | Aged, 80 and over | Cell Line, Tumor | Adult | Carcinoma - genetics | Female | Neoplasm Recurrence, Local - genetics | Aged | Mutation | High-Throughput Nucleotide Sequencing - methods | Usage | Genetic aspects | Research | Nucleotide sequencing | Tumor proteins | Health aspects | Gallbladder cancer | Risk factors | DNA sequencing | Pathogenesis | Science | Kinases | Confidence intervals | Cell growth | Mutagenesis | Rodents | Medical prognosis | Technological change | Bioinformatics | Tumors | Cancer | Index Medicus
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5. Full Text The association between Toll-like receptor 2 single-nucleotide polymorphisms and hepatocellular carcinoma susceptibility
by Junjie, Xie and Songyao, Jiang and Minmin, Shi and Yanyan, Song and Baiyong, Shen and Xiaxing, Deng and Jiabin, Jin and Xi, Zhan and Hao, Chen
BMC Cancer, ISSN 1471-2407, 02/2012, Volume 12, Issue 1, pp. 57 - 57
Background: Toll-like receptors (TLR) are key innate immunity receptors participating in an immune response. Growing evidence suggests that mutations of... 
CELLS | ONCOLOGY | GASTRIC-CANCER | CHRONIC HEPATITIS-B | SILENCE | GROWTH | RISK | TLR | GENE POLYMORPHISMS | EXPRESSION | Haplotypes | Genetic Predisposition to Disease - genetics | Toll-Like Receptor 2 - genetics | Liver Neoplasms - genetics | Humans | Middle Aged | Male | Case-Control Studies | Linkage Disequilibrium | Young Adult | Carcinoma, Hepatocellular - genetics | Adolescent | Polymerase Chain Reaction | Aged, 80 and over | Polymorphism, Single Nucleotide - genetics | Adult | Female | Aged | Physiological aspects | Genetic aspects | Hepatoma | Research | Single nucleotide polymorphisms | TLR2
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6. Full Text Analysis of Differentially Expressed lncRNAs and mRNAs for the Identification of Hypoxia-Regulated Angiogenic Genes in Colorectal Cancer by RNA-Seq
by Han, Yi and Wang, Xiaoyan and Mao, Enqiang and Shen, Baiyong and Huang, Liang
Medical science monitor : international medical journal of experimental and clinical research, ISSN 1643-3750, 03/2019, Volume 25, pp. 2009 - 2015
BACKGROUND Hypoxia is an important feature of solid tumors and related to a perturbed blood supply in pathophysiologies. The aim of our research was to analyze... 
Oligonucleotide Array Sequence Analysis | Gene Expression Regulation - genetics | Colorectal Neoplasms - genetics | Humans | RNA, Messenger - genetics | RNA, Messenger - analysis | Gene Expression Profiling - methods | RNA, Long Noncoding - genetics | Transcriptome - genetics | RNA, Messenger - metabolism | Sequence Analysis, RNA - methods | Tumor Hypoxia - genetics | RNA, Long Noncoding - analysis | Hypoxia - genetics | China | Angiogenesis Modulating Agents - metabolism | Cell Line, Tumor | RNA, Long Noncoding - metabolism | Gene Ontology | Index Medicus
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7. Full Text CQ sensitizes human pancreatic cancer cells to gemcitabine through the lysosomal apoptotic pathway via reactive oxygen species
by Fu, Zhiping and Cheng, Xi and Kuang, Jie and Feng, Haoran and Chen, Lingxie and Liang, Juyong and Shen, Xiaonan and Yuen, Stanley and Peng, Chenghong and Shen, Baiyong and Jin, Zhijian and Qiu, Weihua
Molecular Oncology, ISSN 1574-7891, 04/2018, Volume 12, Issue 4, pp. 529 - 544
As an established anticancer drug, gemcitabine (GEM) is an effective systemic treatment for advanced pancreatic cancer (PC). However, little is known about the... 
chloroquine | lysosomal membrane permeabilization | apoptosis | gemcitabine | reactive oxygen species | OXIDATIVE STRESS | ADENOCARCINOMA | DEATH | AUTOPHAGY | COMBINATION | CHEMOTHERAPY | HEPATOCELLULAR-CARCINOMA | ONCOLOGY | DOUBLE-EDGED-SWORD | ACCUMULATION | Pancreatic Neoplasms - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Pancreatic Neoplasms - pathology | Deoxycytidine - pharmacology | Male | Neoplasm Proteins - metabolism | Pancreatic Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Chloroquine - pharmacology | Animals | Lysosomes - metabolism | Mice, Nude | Cell Line, Tumor | Lysosomes - pathology | Mice | Mice, Inbred BALB C | Deoxycytidine - analogs & derivatives
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8. Full Text Survey of tyrosine kinase signaling reveals ROS kinase fusions in human cholangiocarcinoma
by Gu, Ting-Lei and Deng, Xiaxing and Huang, Feizhou and Tucker, Meghan and Crosby, Katherine and Rimkunas, Victoria and Wang, Yi and Deng, Gang and Zhu, Lei and Tan, Zhiping and Hu, Yerong and Wu, Chunlin and Nardone, Julie and MacNeill, Joan and Ren, Jianmin and Reeves, Cynthia and Innocenti, Gregory and Norris, Brett and Yuan, Jin and Yu, Jian and Haack, Herbert and Shen, Baiyong and Peng, Chenghong and Li, Hongwei and Zhou, Xinmin and Liu, Xunyang and Rush, John and Comb, Michael J
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 1, p. e15640
Cholangiocarcinoma, also known as bile duct cancer, is the second most common primary hepatic carcinoma with a median survival of less than 2 years. The... 
PATHOGENESIS | GLIOBLASTOMA | PROTEIN | PHOSPHORYLATION | PATHWAY | LIVER-REGENERATION | GENES | BIOLOGY | CANCER | EXPRESSION | Proto-Oncogene Proteins - metabolism | Immunoassay | Oncogene Proteins, Fusion - metabolism | Phosphorylation | Signal Transduction | Protein-Tyrosine Kinases - metabolism | Humans | Proto-Oncogene Proteins - genetics | Bile Duct Neoplasms - enzymology | Protein-Tyrosine Kinases - genetics | Animals | Mice, Nude | Oncogene Proteins, Fusion - genetics | Cell Line, Tumor | Bile Ducts, Intrahepatic | Mice | Protein-Tyrosine Kinases - analysis | Cholangiocarcinoma - enzymology | Surveys | Proteins | Tyrosine | Lung cancer | Phenols | Development and progression | Health aspects | Phosphotransferases | Heart surgery | Pathogenesis | Glioblastoma | Identification | Kinases | Phosphatase | Liver cancer | Signal transduction | Rodents | Protein-tyrosine kinase | Chromosomes | Cholangiocarcinoma | EphA2 protein | Epidermal growth factor receptors | Mass spectroscopy | Patients | Bile duct | Medicine | Signaling | Molecular modelling | Scientific imaging | Diagnostic systems | Mass spectrometry | Cancer | Tumors
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9. Full Text MiR-17-5p enhances pancreatic cancer proliferation by altering cell cycle profiles via disruption of RBL2/E2F4-repressing complexes
by Zhu, Youwei and Gu, Jiangning and Li, Ying and Peng, Chenghong and Shi, Minmin and Wang, Di and Wang, Xuelong and Wei, Gang and Ge, Ouyang and Zhang, Bosen and Wu, Jian and Zhong, Yiming and Shen, Baiyong and Chen, Hao
Cancer Letters, ISSN 0304-3835, 01/2018, Volume 412, pp. 59 - 68
The members of the miR-17-92 cluster are upregulated in various cancers and function as a cluster of oncogenic miRNA. Our study characterized a new function of... 
RBL2 | miR-17-5p | E2F4 | Pancreatic cancer | Cell cycle | RB2/P130 | TRANSCRIPTIONAL REPRESSION | RETINOBLASTOMA PROTEIN | GENE | ONCOLOGY | PRB2/P130 | E2F | TUMOR-SUPPRESSOR | DIFFERENTIATION | CLUSTER | EXPRESSION | E2F4 Transcription Factor - genetics | Adenocarcinoma - pathology | Cell Proliferation | Retinoblastoma-Like Protein p130 - physiology | Cell Cycle | E2F4 Transcription Factor - physiology | Humans | Pancreatic Neoplasms - pathology | Cell Line, Tumor | MicroRNAs - physiology | Carcinoma, Pancreatic Ductal - pathology | Retinoblastoma-Like Protein p130 - genetics | Analysis | Cell proliferation | Adenocarcinoma | Repressing | Transcription factors | Cloning | MiRNA | Genomes | Gene expression | Cell adhesion & migration | Proteins | E2F protein | Plasmids | Medical prognosis | Clusters | Tumor suppressor genes | Retinoblastoma | Deoxyribonucleic acid--DNA | Cancer | Tumors
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10. Full Text Corrigendum to “GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mir-17-5p in pancreatic cancer” [Cancer Lett. 380 (2) (2016 Oct 1) 434–41]
by Gu, Jiangning and Wang, Di and Wang, Xuelong and Zhang, Jiaqiang and Zhu, Yi and Li, Ying and Chen, Hao and Shi, Minmin and Shen, Baiyong and Deng, Xiaxing and Zhan, Qian and Wei, Gang and Peng, Chenghong
Cancer Letters, ISSN 0304-3835, 06/2019, Volume 452, pp. 270 - 270
Tumor suppressor genes | Chromosome 5 | Pancreatic cancer | PTEN protein | Chemoresistance | Cancer
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11. Full Text Copy number instability (CNI) combined with CA19-9 as a biomarker for postoperative prognostic prediction of pancreatic cancer
by Zhan, Qian and Ren, Dandan and Mao, Beibei and Song, Xue and Ma, Yanna and Zhang, Henghui and Shen, Baiyong
Journal of Clinical Oncology, ISSN 0732-183X, 05/2019, Volume 37, Issue 15_suppl, pp. e15730 - e15730
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12. Full Text miR-150-5p inhibits hepatoma cell migration and invasion by targeting MMP14
by Li, Tao and Xie, Junjie and Shen, Chuan and Cheng, Dongfeng and Shi, Yuan and Wu, Zhichong and Zhan, Qian and Deng, Xiaxing and Chen, Hao and Shen, Baiyong and Peng, Chenghong and Li, Hongwei and Zhu, Zhecheng
PLoS ONE, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, p. e115577
Hepatocellular carcinoma (HCC) is one of the leading causes of cancer-related mortality worldwide. Despite progress in diagnostics and treatment of HCC, its... 
OVEREXPRESSION | HEPATOCELLULAR-CARCINOMA | CANCER INVASION | METASTASIS | ANGIOGENESIS | POOR-PROGNOSIS | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | MICRORNAS | EXPRESSION | SNAIL | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Matrix Metalloproteinase 14 - biosynthesis | Carcinoma, Hepatocellular - genetics | Adult | Female | Liver Neoplasms - pathology | Neoplasm Proteins - genetics | Liver Neoplasms - genetics | Neoplasm Invasiveness | Neoplasm Proteins - biosynthesis | MicroRNAs - biosynthesis | Hep G2 Cells | Gene Expression Regulation, Enzymologic | RNA, Neoplasm - biosynthesis | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Matrix Metalloproteinase 14 - genetics | RNA, Neoplasm - genetics | Aged | MicroRNAs - genetics | Carcinoma, Hepatocellular - metabolism | Cell Movement | Target recognition | MiRNA | Hepatocellular carcinoma | Chromosome 5 | Hepatoma | Matrix metalloproteinase | Gene expression | Tissues | Kinases | Ovarian cancer | Cell adhesion & migration | Metastases | Liver cancer | Molecular modelling | DNA microarrays | Rodents | Metalloproteinase | Inhibition | Cell migration | Cancer
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13. Full Text Apatinib Inhibits Angiogenesis Via Suppressing Akt/GSK3β/ANG Signaling Pathway in Anaplastic Thyroid Cancer
by Jin, Zhijian and Cheng, Xi and Feng, Haoran and Kuang, Jie and Yang, Weiping and Peng, Chenghong and Shen, Baiyong and Qiu, Weihua
Cellular Physiology and Biochemistry, ISSN 1015-8987, 12/2017, Volume 44, Issue 4, pp. 1471 - 1484
Background/Aims: Anaplastic thyroid carcinoma (ATC) is one of the most lethal human malignancies, and there is no efficient method to slow its process.... 
Original Paper | Apatinib | GSK3β | Akt | Angiogenin | Agiogenesis | Anaplastic thyroid carcinoma | Human Umbilical Vein Endothelial Cells | Neovascularization, Physiologic - drug effects | Apoptosis - drug effects | Humans | Male | Transplantation, Heterologous | Recombinant Proteins - biosynthesis | Antineoplastic Agents - toxicity | Thyroid Carcinoma, Anaplastic - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Ribonuclease, Pancreatic - genetics | Glycogen Synthase Kinase 3 beta - antagonists & inhibitors | Recombinant Proteins - genetics | Recombinant Proteins - pharmacology | Glycogen Synthase Kinase 3 beta - metabolism | Cell Movement - drug effects | Animals | Pyridines - toxicity | Ribonuclease, Pancreatic - metabolism | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Thyroid Carcinoma, Anaplastic - pathology | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | G1 Phase Cell Cycle Checkpoints - drug effects | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Thyroid Neoplasms - metabolism | Thyroid Neoplasms - pathology | Angiogenesis | Thyroid cancer | Cold | Cell cycle | Cytotoxicity | Metastasis | Medical screening | Kinases | Vascular endothelial growth factor | Cancer therapies | Apoptosis
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14. Full Text An 8-year single-center study: 170 cases of middle pancreatectomy, including 110 cases of robot-assisted middle pancreatectomy
by Shi, Yusheng and Jin, Jiabin and Huo, Zhen and Wang, Jian and Weng, Yuanchi and Zhao, Shulin and Deng, Xiaxing and Shen, Baiyong and Peng, Chenghong
Surgery, ISSN 0039-6060, 10/2019
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15. Full Text Genomic signatures of pancreatic adenosquamous carcinoma (PASC)
by Fang, Yuan and Su, Zhe and Xie, Jing and Xue, Ruidong and Ma, Qi and Li, Yanmeng and Zhao, Yifan and Song, Zeshi and Lu, Xiongxiong and Li, Hongwei and Peng, Chenghong and Bai, Fan and Shen, Baiyong
The Journal of Pathology, ISSN 0022-3417, 10/2017, Volume 243, Issue 2, pp. 155 - 159
In pancreatic cancer, pancreatic adenosquamous carcinoma (PASC) containing both ductal adenocarcinoma and squamous carcinoma in the same tumour represents ∼4%... 
mixed cancer | pancreatic adenosquamous carcinoma (PASC) | 3p loss | TP53 | ONCOLOGY | STATISTICS | PATHOLOGY | ABERRATIONS | CANCER | Humans | Pancreatic Neoplasms - genetics | Carcinoma, Adenosquamous - genetics | Mutation - genetics | Neoplasm Proteins - genetics | Genes, Neoplasm - genetics | Genome, Human - genetics | Yuan (China) | Squamous cell carcinoma | Gene mutations | Analysis | Pancreatic cancer | Oncology, Experimental | Genomics | Genetic aspects | Research | Tumor proteins | Cancer | Adenocarcinoma | Pancreatic carcinoma | p53 Protein | Genomes | Adenosquamous | Mutation | Pancreas | Tumors
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