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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2015, Volume 112, Issue 9, pp. 2853 - 2858
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 09/2010, Volume 114, Issue 5, pp. 1476 - 1486
J. Neurochem. (2010) 114, 1476–1486. Excessive activation of Na+/H+ exchanger isoform 1 (NHE‐1) plays a role in cerebral ischemic injury. The current study... 
transient cerebral ischemia | fluoromethyl ketone | brain damage | reperfusion | U0126 | Neurology | Signal transduction | Brain damage | Biochemistry | Kinases | FMK
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 10/2011, Volume 119, Issue 1, pp. 124 - 135
Journal Article
Scientific Reports, ISSN 2045-2322, 05/2015, Volume 5, Issue 1, pp. 10224 - 10224
Major characteristics of Alzheimer's disease (AD) include deposits of beta-amyloid (A beta) peptide in the brain, loss of synapses, and cognitive dysfunction.... 
AMPHETAMINE-REGULATED TRANSCRIPT | ACTIVATION | MULTIDISCIPLINARY SCIENCES | GENES | MOUSE | NEURONS | DEFICITS | EXPRESSION | BRAIN | PLASTICITY | PROTECTS | Memory - drug effects | Humans | DNA, Mitochondrial | Synapses - pathology | Case-Control Studies | Alzheimer Disease - pathology | Brain - metabolism | Neurotransmitter Agents - genetics | Amyloid beta-Peptides - metabolism | Nerve Tissue Proteins - administration & dosage | Alzheimer Disease - psychology | Neurites - drug effects | Disease Models, Animal | Synapses - drug effects | Gene Expression | Plaque, Amyloid - pathology | Neurotransmitter Agents - metabolism | Alzheimer Disease - drug therapy | Mice, Transgenic | Mitochondria - metabolism | Neurites - metabolism | Mitochondria - drug effects | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Hippocampus - metabolism | Animals | Alzheimer Disease - metabolism | Brain - pathology | Mice | Neurotransmitter Agents - administration & dosage | DNA Damage | Oxidative Stress - drug effects | Alzheimer Disease - genetics | Potentiation | Reactive oxygen species | Transcription | 4-Hydroxynonenal | Toxicity | Memory | DNA damage | Cognitive ability | Depolarization | Electron transport chain | Mitochondria | Ultrastructure | Rodents | Membrane potential | Localization | Alzheimer's disease | Plaques | NADH-ubiquinone oxidoreductase | Neurodegenerative diseases | Long-term potentiation | Amyloid precursor protein | Amphetamine | β-Amyloid | Cocaine- and amphetamine-regulated transcript protein | Cocaine | Immunoreactivity | Alzheimers disease | Hippocampus | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2016, Volume 113, Issue 25, pp. E3558 - E3567
Journal Article
Scientific Reports, ISSN 2045-2322, 04/2015, Volume 5, Issue 1, p. 9621
Journal Article
Journal Article