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Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2005, Volume 102, Issue 34, pp. 12071 - 12076
Journal Article
Nihon rinsho. Japanese journal of clinical medicine, ISSN 0047-1852, 2013, Volume 71, Issue 2, pp. 244 - 248
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, pp. e25231 - e25231
Journal Article
Nihon rinsho. Japanese journal of clinical medicine, ISSN 0047-1852, 2013, Volume 71, Issue 12, pp. 2119 - 2123
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 10/2017, Volume 37, Issue 42, pp. 10052 - 10062
Journal Article
Methods in molecular biology (Clifton, N.J.), ISSN 1064-3745, 2008, Volume 456, pp. 249 - 257
RNA interference (RNAi) is a powerful, quick, and easy technique to reduce the expression of a particular gene. However, investigators need to consider several... 
RNAi | Transfection | Adipocytes | Gene expression | Gene Transfer Techniques | RNA, Small Interfering - genetics | Gene Expression | Animals | RNA Interference | Adipocytes - cytology | Mice | Cell Culture Techniques | 3T3-L1 Cells | Adipocytes - physiology | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Journal Article
Molecular Pharmacology, ISSN 0026-895X, 05/2013, Volume 83, Issue 5, pp. 1133 - 1140
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 09/2018, Volume 19, Issue 9, p. 2813
Brain and muscle arnt-like protein 1 (BMAL1), is a transcription factor known to regulate circadian rhythm. BMAL1 was originally characterized by its high... 
Ectopic fat | Circadian rhythm | BMAL1 | Skeletal muscle | ectopic fat | BIOCHEMISTRY & MOLECULAR BIOLOGY | OXIDASE ACTIVITY | IDENTIFICATION | CHEMISTRY, MULTIDISCIPLINARY | EXERCISE | CIRCADIAN CLOCK | ADIPONECTIN | ADIPOSE-SPECIFIC PROTEIN | circadian rhythm | IN-VIVO | GENE-EXPRESSION | MICE | COMPONENT | skeletal muscle | ARNTL Transcription Factors - genetics | Oxidative Stress | Mice, Inbred C57BL | Insulin Resistance | ARNTL Transcription Factors - metabolism | Diet, High-Fat - adverse effects | Fats - metabolism | Male | Muscle, Skeletal - metabolism | Obesity - genetics | Mice, Knockout | Obesity - metabolism | Obesity - pathology | Locomotion | Animals | Gene Deletion | Obesity - etiology | Muscle, Skeletal - pathology | Brain | Energy metabolism | Phosphorylation | Transcription factors | Calcium | Activation | Overloading | NF-AT protein | Kinases | AMP-activated protein kinase | Proteins | Cell activation | Control | Clonal deletion | Acetyl-CoA carboxylase | Lymphocytes | Deletion | Endurance | Circadian rhythms | Nutrition | AMP | Muscles | Energy expenditure | Oxygen consumption | Metabolism | Substrates | Flox | Musculoskeletal system | BMAL1 protein | Mice | Cytochrome | Dehydrogenases | Mitochondrial DNA | Rodents | Cell cycle | Oxidation | Gene expression | Fatty acids | Shift work | Studies | Pharmacy | Diabetes | Binding sites | Index Medicus
Journal Article