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by Yamagami, Keitaro and Kurogi, Ryota and Kurogi, Ai and Nishimura, Kunihiro and Onozuka, Daisuke and Ren, Nice and Kada, Akiko and Nishimura, Ataru and Arimura, Koichi and Ido, Keisuke and Mizoguchi, Masahiro and Sakamoto, Tetsuya and Kayama, Takamasa and Suzuki, Michiyasu and Arai, Hajime and Hagihara, Akihito and Iihara, Koji and Masayoshi, Takigami and Kenji, Kamiyama and Kiyohiro, Houkin and Shougo, Nishi and Tetsuyuki, Yoshimoto and Sadao, Kaneko and Koji, Oka and Hiroshi, Ooyama and Kyousuke, Kamada and Kenichi, Makino and Naoki, Tokumitsu and Kazuhiro, Sako and Susumu, Suzuki and Nozomi, Suzuki and Naoto, Izumi and Kazumi, Nitta and Masahumi, Ootaki and Masanori, Isobe and Mikio, Nishiya and Takaaki, Yamazaki and Syouji, Mabuchi and Kuniaki, Ogasawara and Naohiko, Kubo and Yukihiko, Shimizu and Keiichi, Saito and Tatumi, Yamanome and Atsuo, Yoshino and Mitsuyuki, Fujitsuka and Masaaki, Takami and Hirotoshi, Ohtaka and Teruyuki, Hirano and Yosiaki, Shiokawa and Takaharu, Okada and Ichiro, Suzuki and Michihiro, Kohno and Jou, Haraoka and Yoshinori, Arai and Noriyoshi, Kawamura and Akira, Isoshima and Masaharu, Yasue and Mitsuhiko, Hokari and Takayoshi, Kobayashi and Kensuke, Kawai and Taketoshi, Maehara and Hajime, Arai and Makoto, Noguchi and Haruhiko, Hoshino and Hirofumi, Hiyama and Kensaku, Yoshida and Osamu, Utsugi and Yasuaki, Takeda and Kouichi, Tamaki and Hirohide, Karasudani and Takao, Urabe and Shiro, Kobayashi and Michio, Nakamura and Yorio, Koguchi and Junichi, Ono and Sumio, Suda and Hiromu, Hadeishi and Toshio, Fukutake and Kenji, Wakui and Hirokazu, Tanno and Naoki, Ishige and Takashi, Ohasi and Hideki, Sakai and Yasuaki, Nishimura and Takayuki, Watanabe and Takashi, Matsumoto and Naoki, Koketsu and Yuichi, Hirose and Manabu, Doyu and Toshinori, Hasegawa and Naoto, Kuwayama and Shinichi, Terao and Nobuhiko, Mizutani and Noriyuki, Suzaki and Satoshi, Okuda and Keizo, Yasui and Yukio, Seki and Yasuhiro, Hasegawa and Akira, Ikeda and Youtarou, Takeuchi and ... and J-ASPECT Study Collaborators
World Neurosurgery, ISSN 1878-8750, 2019
Background: The epidemiology of patients with traumatic brain injury (TBI) has changed dramatically over recent decades as a result of rapid advances in aging... 
Aging | Hospital mortality | Prognosis | Traumatic brain injury | Subdural hematoma | Index Medicus
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 07/2019, Volume 9, Issue 1, pp. 1 - 14
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2008, Volume 118, Issue 1, pp. 29 - 39
Adrenomedullin (AM) is a peptide involved both in the pathogenesis of cardiovascular diseases and in circulatory homeostasis. The high-affinity AM receptor is... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVITY-MODIFYING PROTEINS | ENDOGENOUS ADRENOMEDULLIN | CARDIAC-HYPERTROPHY | ENDOTHELIAL-CELLS | GENE-EXPRESSION | SMOOTH-MUSCLE-CELLS | ANGIOTENSIN-II | KNOCKOUT MICE | INDUCED AIRWAY HYPERRESPONSIVENESS | TRANSGENIC MICE | Capillary Permeability - physiology | Embryonic Stem Cells - metabolism | Edema - genetics | Receptors, Calcitonin - metabolism | Tight Junctions - genetics | Humans | Male | Receptors, Calcitonin - genetics | Calcitonin Receptor-Like Protein | Cardiovascular Diseases - pathology | Cardiovascular Diseases - genetics | Receptors, Peptide - genetics | Arteries - metabolism | Embryo Loss - metabolism | Embryo Loss - pathology | Female | Embryo Loss - genetics | Intracellular Signaling Peptides and Proteins - genetics | Receptor Activity-Modifying Protein 2 | Tight Junctions - metabolism | Cardiovascular Diseases - metabolism | Membrane Proteins - genetics | Cells, Cultured | Receptors, Adrenomedullin | Adrenomedullin - metabolism | Arteries - pathology | Adrenomedullin - genetics | Mice, Knockout | Pregnancy | Edema - metabolism | Neovascularization, Physiologic - physiology | Membrane Proteins - biosynthesis | Animals | Endothelium, Vascular - metabolism | Homeostasis - physiology | Embryonic Stem Cells - pathology | Endothelium, Vascular - pathology | Receptor Activity-Modifying Proteins | Mice | Edema - pathology | Tight Junctions - pathology | Receptors, Peptide - metabolism | Proteins | Physiological aspects | Research | Peptides | Cardiovascular diseases | Index Medicus | Abridged Index Medicus
Journal Article
Endocrinology, ISSN 0013-7227, 05/2017, Volume 158, Issue 5, pp. 1194 - 1206
Journal Article
Journal Article
Journal of Pharmacological Sciences, ISSN 1347-8613, 2013, Volume 121, Issue suppl, pp. 27 - 27
Journal Article
Endocrinology, ISSN 0013-7227, 05/2017, Volume 158, Issue 5, pp. 1359 - 1372
Neointimal hyperplasia is the primary lesion underlying atherosclerosis and restenosis after coronary intervention. We previously described the essential... 
PROGENITOR CELLS | ARTERIAL INJURY | VASCULAR INJURY | ACCELERATES REENDOTHELIALIZATION | NAD(P)H OXIDASE | HYPOTENSIVE PEPTIDE | ENDOCRINOLOGY & METABOLISM | ACTIVATED-RECEPTOR-GAMMA | NEOINTIMAL HYPERPLASIA | NECROSIS-FACTOR-ALPHA | SMOOTH-MUSCLE-CELLS | Femoral Artery - pathology | Vascular System Injuries - genetics | Humans | Mice, Inbred C57BL | Cells, Cultured | Male | Signal Transduction - genetics | Vascular System Injuries - prevention & control | Neointima - metabolism | Adrenomedullin - genetics | Mice, Knockout | Femoral Artery - injuries | Neointima - genetics | Animals | Adrenomedullin - physiology | Neointima - pathology | Femoral Artery - metabolism | Receptor Activity-Modifying Protein 2 - genetics | Cytoprotection - genetics | Mice | Endothelial Cells - physiology | Receptor Activity-Modifying Protein 2 - physiology | Cell proliferation | Oxidative stress | Leukocyte migration | Syngeneic grafts | Hyperplasia | Bone marrow transplantation | Adrenomedullin | Smooth muscle | Macrophages | Receptor activity modifying proteins | Angiogenesis | Vascular diseases | Clonal deletion | Rodents | Atherosclerosis | Bone marrow | Deletion | Injuries | Medical treatment | Muscles | Blood vessels | Transplants | Inflammation | Endothelial cells | Restenosis | Arteriosclerosis | Stem cells | Cells (biology) | Infiltration | Bone | Cell migration | Index Medicus | Abridged Index Medicus
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 2019, Volume 1874, pp. 257 - 271
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, pp. e87667 - e87667
Various bioactive peptides have been implicated in the homeostasis of organs and tissues. Adrenomedullin (AM) is a peptide with various bioactivities.... 
UNFOLDED PROTEIN RESPONSE | HYPOTENSIVE PEPTIDE | GENE | INDUCED APOPTOSIS | PLASMA-LEVELS | MULTIDISCIPLINARY SCIENCES | ENDOGENOUS ADRENOMEDULLIN | VASCULAR INTEGRITY | ENDOPLASMIC-RETICULUM STRESS | RENAL INJURY | CISPLATIN NEPHROTOXICITY | Antibiotics, Antineoplastic - pharmacology | eIF-2 Kinase - metabolism | Humans | Antibiotics, Antineoplastic - adverse effects | Kidney Glomerulus - injuries | Kidney Diseases - genetics | Cell Death - genetics | Kidney Diseases - chemically induced | Kidney Glomerulus - metabolism | Cell Death - drug effects | Streptozocin - adverse effects | Kidney Diseases - metabolism | eIF-2 Kinase - genetics | Cell Line | Kidney Tubules, Proximal - pathology | Kidney Diseases - pathology | Kidney Tubules, Proximal - injuries | Receptor Activity-Modifying Protein 2 - metabolism | Adrenomedullin - metabolism | Kidney Glomerulus - pathology | Streptozocin - pharmacology | Adrenomedullin - genetics | Mice, Knockout | Animals | Endoplasmic Reticulum Stress | Kidney Tubules, Proximal - metabolism | Receptor Activity-Modifying Protein 2 - genetics | Mice | Proteins | Blood sugar | Cell death | Genetically modified organisms | Biochemistry | Kidney diseases | Protein binding | Peptides | Syngeneic grafts | Epithelial cells | Streptozocin | Tunicamycin | Homeostasis | Adrenomedullin | Homology | Lethality | Glucose | Receptor activity modifying proteins | Ischemia | Rodents | Animal tissues | Calcitonin | Damage | Genetic modification | Injuries | University graduates | Chronic illnesses | Glucose transporter | Stresses | Stroke | Kidneys | Mortality | Organs | Tubules | Ribonucleic acid--RNA | Insulin | Embryos | Biological activity | Stress | Medicine | Ramps | Signaling | Protein kinase | Diabetes | Endoplasmic reticulum | Transporter | Kidney transplantation | Apoptosis | Index Medicus | RNA | Ribonucleic acid
Journal Article
Chemistry – A European Journal, ISSN 0947-6539, 07/2019
Journal Article
Organic Letters, ISSN 1523-7060, 07/2019
Herein, we report a double deprotonation method used for the preparation of ynolates starting from nonbrominated 2,6-di-tert-butylphenyl esters. The current... 
Journal Article
Journal Article