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Nature Communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, p. 1829
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 04/2011, Volume 193, Issue 1, pp. 31 - 39
Nuclei of higher organisms are well structured and have multiple, distinct nuclear compartments or nuclear bodies. Paraspeckles are recently identified... 
NONCODING RNA | CELLS | DOMAIN | RETENTION | E-CADHERIN | EXPRESSION | CELL BIOLOGY | Animals | Cell Nucleolus - physiology | Cell Nucleolus - ultrastructure | Mice, Inbred C57BL | Cells, Cultured | RNA, Small Untranslated - genetics | Heterozygote | Mice | Mice, Knockout | Physiological aspects | Composition | Genetic aspects | Chemical properties | Mammals | Cell nuclei
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2016, Volume 113, Issue 2, pp. 332 - 337
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2013, Volume 110, Issue 23, pp. 9409 - 9414
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2015, Volume 112, Issue 13, pp. 4086 - 4091
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 11/2015, Volume 30, Issue 11, pp. 1980 - 1993
ABSTRACT Activating mutations of calcium‐sensing receptor (CaSR) cause autosomal dominant hypocalcemia (ADH). ADH patients develop hypocalcemia,... 
BONE MODELING AND REMODELING | GENETIC ANIMAL MODELS | PTH | DISORDERS OF CALCIUM/PHOSPHATE METABOLISM | FGF23 | VITAMIN D | HORMONE REPLACEMENT/RECEPTOR MODULATORS | Vitamin D | PARATHYROID-HORMONE 1-34 | TRANSMEMBRANE DOMAIN | FUNCTIONAL-CHARACTERIZATION | DISORDERS OF CALCIUM | OF-FUNCTION MUTATIONS | PHOSPHATE METABOLISM | NEGATIVE ALLOSTERIC MODULATORS | BONE-FORMATION | ENDOCRINOLOGY & METABOLISM | REPLACEMENT THERAPY | RECEPTOR MODULATORS | HORMONE REPLACEMENT | BINDING-SITES | ACTIVATING MUTATIONS | CA2+-SENSING RECEPTOR | Bone and Bones - pathology | Hypocalcemia - physiopathology | Benzoates - therapeutic use | Calcium - metabolism | Humans | Hypoparathyroidism - physiopathology | Hypercalciuria - pathology | Molecular Sequence Data | Hypoparathyroidism - genetics | Hypoparathyroidism - pathology | Bone Remodeling - drug effects | Hypocalcemia - pathology | Bone and Bones - drug effects | Hypocalcemia - drug therapy | Propanolamines - therapeutic use | Base Sequence | HEK293 Cells | Hypercalciuria - genetics | Disease Models, Animal | Hypoparathyroidism - drug therapy | Hypoparathyroidism - congenital | Hypercalciuria - physiopathology | Mutant Proteins - metabolism | Mutation - genetics | Gene Knock-In Techniques | Hypercalciuria - drug therapy | Organ Size - drug effects | Phenotype | Animals | Hypocalcemia - genetics | Benzoates - pharmacology | Receptors, Calcium-Sensing - genetics | Mice | Propanolamines - pharmacology | Phosphates | Bones | Parathyroid hormone | Density | Analysis | Hypocalcemia
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 5801 - 15
Journal Article
Journal Article
Journal Article