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Journal Article
Infection and Immunity, ISSN 0019-9567, 03/2012, Volume 80, Issue 3, pp. 1088 - 1097
Journal Article
Journal Article
Journal Article
by Qian, KC and Du, LL and Zhu, XH and Liang, SP and Chen, S and Kobayashi, H and Yan, XQ and Xu, M and Dai, YH and Li, RH
JOURNAL OF MATERIALS CHEMISTRY A, ISSN 2050-7488, 06/2019, Volume 7, Issue 24, pp. 14592 - 14601
Molecular oxygen activation (MOA) into reactive oxygen species (ROS) is extremely crucial in numerous catalytic processes, while precise control of ROS... 
AQUEOUS-SOLUTIONS | TUNGSTEN-OXIDE | ENERGY & FUELS | PERFORMANCE | MATERIALS SCIENCE, MULTIDISCIPLINARY | LIGHT | CHEMISTRY, PHYSICAL | NANOSHEETS | PHOTOCATALYST | ELECTROCATALYTIC ACTIVITY | SPECTROSCOPY | MOLYBDENUM OXIDES | WATER
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 139, Issue 3, pp. 904 - 917.e6
Background & Aims Factors that regulate enterocyte apoptosis in necrotizing enterocolitis (NEC) remain incompletely understood, although Toll-like receptor-4... 
Gastroenterology and Hepatology | TLR4 | NEC | NOD2 | TRANSLOCATION | ACTIVATION | NECROTIZING ENTEROCOLITIS | ENTEROCYTE MIGRATION | PATHOGENESIS | NOD-LIKE RECEPTORS | HOST-DEFENSE | RESTITUTION | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Intestinal Mucosa - metabolism | Nod2 Signaling Adaptor Protein - deficiency | Age Factors | Endotoxemia - genetics | Enterocytes - metabolism | Enterocolitis, Necrotizing - genetics | Enterocolitis, Necrotizing - metabolism | Humans | NF-kappa B - metabolism | Nod2 Signaling Adaptor Protein - genetics | Endotoxemia - pathology | Intestinal Mucosa - drug effects | Mitochondrial Proteins - metabolism | Nod2 Signaling Adaptor Protein - agonists | Enterocolitis, Necrotizing - pathology | Disease Models, Animal | Animals, Newborn | Severity of Illness Index | Cell Line | Transduction, Genetic | Enterocolitis, Necrotizing - prevention & control | Mice, Inbred C57BL | Rats | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Acetylmuramyl-Alanyl-Isoglutamine - pharmacology | Animals | Carrier Proteins - metabolism | Nod2 Signaling Adaptor Protein - metabolism | Signal Transduction - drug effects | Protein Array Analysis | Mice | Endotoxemia - metabolism | Apoptosis | Cell Movement | Intestinal Mucosa - pathology | Oligomers | Fluorescence | Mitogens | Nitric oxide
Journal Article
Journal of Materials Chemistry. A, Materials for Energy and Sustainability, ISSN 2050-7488, 01/2019, Volume 7, Issue 24, pp. 14592 - 14601
Molecular oxygen activation (MOA) into reactive oxygen species (ROS) is extremely crucial in numerous catalytic processes, while precise control of ROS... 
Protons | Chemisorption | Oxygen | Reactive oxygen species | Vacancies | Free radicals | Catalysts | Hydrogen evolution | Electron transfer | Activation | Superoxide | Reforming | Tungsten oxides | Radicals | Coupling (molecular) | Organic chemistry | Dehydrogenation | Tungsten | Catalysis | Clean energy | Nanorods | Electrons
Journal Article
Frontiers in Microbiology, ISSN 1664-302X, 2011, Volume 1, p. 144
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 09/2008, Volume 295, Issue 3, pp. G559 - G569
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 2007, Volume 294, Issue 1, pp. G109 - G119
Enterocytes exist in close association with tissue macrophages, whose activation during inflammatory processes leads to the release of nitric oxide (NO).... 
Enterocyte migration | Necrotizing enterocolitis | Inflammation | Intestinal restitution | PHYSIOLOGY | PERMEABILITY | PHOSPHORYLATION | CARDIAC MYOCYTES | intestinal restitution | INTESTINAL INFLAMMATION | S-NITROSYLATION | necrotizing enterocolitis | MULTIPLE ORGAN DYSFUNCTION | inflammation | EPITHELIAL RESTITUTION | enterocyte migration | GASTROENTEROLOGY & HEPATOLOGY | COMMUNICATION | WOUND REPAIR | Gap Junctions - metabolism | Phosphorylation | Enterocytes - metabolism | Coculture Techniques | Male | Time Factors | Nitric Oxide Synthase Type II - antagonists & inhibitors | Nitric Oxide Donors - pharmacology | Cell Line | Lysine - analogs & derivatives | Connexin 43 - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Rats | Mice, Transgenic | Macrophages - enzymology | Protein Transport | Gastroenteritis - enzymology | Macrophages - metabolism | Animals | Lysine - pharmacology | Enterocytes - drug effects | Nitric Oxide Synthase Type II - genetics | Paracrine Communication - drug effects | Gastroenteritis - metabolism | Lipopolysaccharides - pharmacology | Interferons - metabolism | Macrophages - drug effects | Mice | Gap Junctions - drug effects | Macrophage Activation - drug effects | Nitric Oxide - metabolism | S-Nitroso-N-Acetylpenicillamine - pharmacology | Cell Movement | Nitric Oxide Synthase Type II - metabolism
Journal Article
Infection and Immunity, ISSN 0019-9567, 03/2012, Volume 80, Issue 3, pp. 1088 - 1097
Francisella tularensisis a remarkably infectious facultative intracellular pathogen that causes the zoonotic disease tularemia. Essential to the pathogenesis... 
Journal Article
Journal Article