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Neurochemical Research, ISSN 0364-3190, 7/2017, Volume 42, Issue 7, pp. 2011 - 2018
There is growing evidence that ketone bodies (KB)—derived from fatty acid oxidation and produced during fasting or consumption of high-fat diets—can exert... 
Neurochemistry | Biochemistry, general | Neurology | Neuroprotection | Neurosciences | Biomedicine | Epilepsy | Beta-hydroxybutyrate | Ketone bodies | Ketogenic diet | Cell Biology | Acetoacetate | NERVOUS-SYSTEM | K-ATP CHANNELS | BIOCHEMISTRY & MOLECULAR BIOLOGY | EXPERIMENTALLY PRODUCED CONVULSIONS | CHILDHOOD EPILEPSY | RANDOMIZED CONTROLLED-TRIAL | NEUROSCIENCES | GLUTAMATE TRANSPORT | MITOCHONDRIAL PERMEABILITY TRANSITION | RAT-BRAIN | Seizures - diet therapy | Diet, Ketogenic - methods | Humans | Ketone Bodies - metabolism | Seizures - drug therapy | Anticonvulsants - metabolism | Anticonvulsants - therapeutic use | Seizures - metabolism | Brain - drug effects | Brain - metabolism | Ketone Bodies - therapeutic use | Animals | Histone Deacetylase Inhibitors - therapeutic use | Histone Deacetylase Inhibitors - metabolism | Epigenetic inheritance | Medical colleges | Permeability | Glutamate | Fatty acids | Anticonvulsants | Analysis | Physiological aspects | GABA | Seizures (Medicine) | Acetone | Brain | Glutamic acid | Histone deacetylase | Animal models | Biochemistry | Fuels | High fat diet | Mitochondria | Modulation | Attenuation | Oxidation | Inhibition | Seizures | Seizing | Fasting | Ketones | Excitability | Inflammation | Potassium channels | Diet | Epigenetics | Electron transport | ATP | Potassium | Neurotransmission | Adenosine triphosphatase
Journal Article
Experimental Neurology, ISSN 0014-4886, 01/2014, Volume 251, pp. 84 - 90
Mitochondria actively participate in neurotransmission by providing energy (ATP) and maintaining normative concentrations of reactive oxygen species (ROS) in... 
Ascorbic acid | Epilepsy | Pyruvate | Mitochondrial complex I | Mitochondrial uncoupling | Kv1.1 knockout mice | Antioxidant | Field potentials | Paired-pulse ratios | Mitochondria | Seizure-like events | ROS | Alpha-tocopherol | Kcna1-null mice | Kainic acid | Seizures | VITAMIN-E | CARDIAC MYOCYTES | Ascorbic add | ALPHA-KETOGLUTARATE DEHYDROGENASE | HYDROGEN-PEROXIDE | LONG-TERM POTENTIATION | BRAIN | OXIDATIVE STRESS | STATUS EPILEPTICUS | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | OSCILLATIONS | Adenosine Triphosphate - administration & dosage | Electric Stimulation | Reactive Oxygen Species - metabolism | Seizures - prevention & control | Electroencephalography | Hippocampus - drug effects | Electron Transport Complex I - metabolism | Seizures - chemically induced | Ascorbic Acid - therapeutic use | Epilepsy, Temporal Lobe - genetics | Respiration - drug effects | Respiration - genetics | Kainic Acid - toxicity | Epilepsy, Temporal Lobe - prevention & control | Disease Models, Animal | Epilepsy, Temporal Lobe - metabolism | Hippocampus - pathology | Mitochondria - metabolism | Mitochondria - drug effects | Seizures - therapy | Adenosine Triphosphate - analogs & derivatives | Hydrogen Peroxide - metabolism | Mice, Knockout | Pyruvic Acid - therapeutic use | alpha-Tocopherol - therapeutic use | Kv1.1 Potassium Channel - deficiency | Animals | Mice | In Vitro Techniques | Hippocampus - physiopathology | Tocopherols | Temporal lobe epilepsy | Anticonvulsants | Mitochondrial DNA | Seizures (Medicine) | epilepsy | seizure-like events | ascorbic acid | antioxidant | kainic acid | mitochondrial complex I | alpha-tocopherol | field potentials | pyruvate | seizures | mitochondrial uncoupling | paired-pulse ratios
Journal Article
Epilepsia, ISSN 0013-9580, 08/2017, Volume 58, Issue 8, pp. 1440 - 1450
Journal Article
Journal Article
Epilepsia, ISSN 0013-9580, 06/2017, Volume 58, Issue S2, pp. 22 - 31
Summary Hypothalamic hamartomas (HHs) are congenital malformations of the ventral hypothalamus resulting in treatment‐resistant epilepsy and are intrinsically... 
Hypothalamic hamartoma | Basic mechanisms | Cellular model | Epilepsy | Gelastic seizure | TUMOR | PRECOCIOUS PUBERTY | CLINICAL NEUROLOGY | INTRINSIC EPILEPTOGENESIS | SEIZURES | TRANSCALLOSAL RESECTION | NETWORK ACTIVITY | GELASTIC EPILEPSY | ELECTROPHYSIOLOGICAL PROPERTIES | OF-THE-LITERATURE | INTRACTABLE EPILEPSY | Child Behavior Disorders - surgery | Dendrites - pathology | Neurons - pathology | Hypothalamic Diseases - physiopathology | Epilepsies, Partial - surgery | Humans | Hypothalamic Diseases - surgery | Epilepsies, Partial - physiopathology | Hamartoma - pathology | Hypothalamus - surgery | Dendrites - physiology | Hamartoma - surgery | Neurons - physiology | Adult | Child | Child Behavior Disorders - psychology | Cognition Disorders - physiopathology | Hypothalamic Diseases - pathology | Child Behavior Disorders - physiopathology | Cognition Disorders - pathology | Hamartoma - physiopathology | Hypothalamus - pathology | Epilepsies, Partial - pathology | Hypothalamus - physiopathology | Magnetic Resonance Imaging | Patch-Clamp Techniques | Cognition Disorders - surgery | GABA | Brain | Glutamate | Hamartoma | Firing pattern | Neurons | Congenital defects | Glutamate decarboxylase | Hypothalamus | Depolarization | Dendritic spines | γ-Aminobutyric acid | Functional anatomy | Ligands | Neoplasia | Seizures
Journal Article
Experimental Neurology, ISSN 0014-4886, 01/2017, Volume 287, Issue Pt 1, pp. 54 - 64
The ketogenic diet (KD) is an effective therapy primarily used in pediatric patients whom are refractory to current anti-seizure medications. The mechanism of... 
PPARgamma2 | Nutrition | Epilepsy | Kcna1 | Seizure | Peroxisome proliferator activated receptor | Ketogenic diet | Kv1.1 | OXIDATIVE STRESS | TEMPORAL-LOBE EPILEPSY | HIGH-FREQUENCY OSCILLATIONS | VITRO HIPPOCAMPAL SLICES | FATTY-ACIDS | NEUROSCIENCES | KETONE-BODIES | PATHOLOGICAL SHARP WAVES | ACTIVATED-RECEPTOR-GAMMA | PPAR-GAMMA | INDUCED STATUS EPILEPTICUS | Anilides - therapeutic use | Age Factors | Gene Expression Regulation, Developmental - genetics | PPAR gamma - metabolism | Anticonvulsants - pharmacology | Epilepsy - diet therapy | Brain - metabolism | Thiazolidinediones - therapeutic use | Thiazolidinediones - pharmacology | Disease Models, Animal | Gene Expression Regulation, Developmental - physiology | PPAR gamma - genetics | Animals, Newborn | Kv1.1 Potassium Channel - genetics | 3-Hydroxybutyric Acid - blood | Diet, Ketogenic - methods | Mice, Inbred C57BL | Drinking - physiology | Gene Expression Regulation, Developmental - drug effects | Anticonvulsants - therapeutic use | Flurothyl - toxicity | Hypoglycemic Agents - pharmacology | Mice, Knockout | Blood Glucose - drug effects | Brain - drug effects | Kv1.1 Potassium Channel - deficiency | Animals | Epilepsy - chemically induced | Convulsants - toxicity | Anilides - pharmacology | Blood Glucose - genetics | Epilepsy - drug therapy | Drinking - drug effects | Mice | Epilepsy - pathology | Fatty acids | Seizures (Medicine) | nutrition | seizure | ketogenic diet | peroxisome proliferator activated receptor
Journal Article