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Cell, ISSN 0092-8674, 2000, Volume 103, Issue 7, pp. 1009 - 1017
Circadian oscillations in mammalian physiology and behavior are regulated by an endogenous biological clock. Here we show that loss of the PAS protein MOP3... 
Journal Article
Carcinogenesis, ISSN 0143-3334, 5/2014, Volume 35, Issue 5, pp. 1067 - 1077
Journal Article
Molecular Cell, ISSN 1097-2765, 2000, Volume 6, Issue 6, pp. 1389 - 1399
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 1/2013, Volume 91, Issue 1, pp. 59 - 67
In Chuvash polycythemia, a homozygous 598C>T mutation in the von Hippel–Lindau gene (VHL) leads to an R200W substitution in VHL protein, impaired degradation... 
Human Genetics | Hypoxia-inducible factors | Biomedicine | Glycolysis | Internal Medicine | Molecular Medicine | VHL | Glucose | Insulin | Gluconeogenesis | MEDICINE, RESEARCH & EXPERIMENTAL | HYPOXIA-INDUCIBLE FACTOR | CELLS | PROMOTES GLYCOGEN ACCUMULATION | THROMBOSIS | ERYTHROPOIESIS | DEFICIENCY | GENETICS & HEREDITY | MICE | OXYGEN HOMEOSTASIS | EXPRESSION | Glucose Transport Proteins, Facilitative - metabolism | Polycythemia - genetics | Glucose-6-Phosphatase - genetics | Glycated Hemoglobin A - metabolism | Humans | Middle Aged | Polycythemia - blood | Male | Muscle, Skeletal - metabolism | Pyruvate Dehydrogenase Complex - genetics | Insulin - blood | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Adult | Female | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Glucose Transport Proteins, Facilitative - genetics | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Pyruvate Dehydrogenase Complex - metabolism | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Liver - metabolism | Gene Expression Regulation | Genotype | Glucose-6-Phosphatase - metabolism | Homozygote | Hypoxia - blood | Hypoxia - genetics | Animals | Alleles | Hypoxia - physiopathology | Mice | Polycythemia - physiopathology | Mutation | Blood Glucose - metabolism | Glucose metabolism | Transcription factors | Polycythemia | Physiological aspects | Development and progression | Genetic aspects | Research | High altitude | Hypoxia-inducible factor 1 | Transcription | Liver | Genes | Inactivation | Degradation | Rodents | Hemoglobin | Skeleton | Glucose transporter | Enzymes | Fasting | Deactivation | Diabetes mellitus | Glycerol | Pharmacology | Hypoglycemia | Gene expression | Metabolism | Patients | Citric acid | Skeletal muscle | Homozygotes | Hippel-Lindau gene | Hypoxia | Neoplasia | glucose | hypoxia inducible factors | insulin | gluconeogenesis | glycolysis
Journal Article
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 06/2000, Volume 273, Issue 1, pp. 231 - 238
The Hypoxia-Inducible Factor-1 (HIF-1) activates the transcription of many genes required for cellular and organismal responses to oxygen deprivation. The... 
ARNT2 | HIF-1 | hypoxia | angiogenesis | VEGF | neuron | gene expression | Angiogenesis | Neuron | Hypoxia | Gene expression | HUMAN BRAIN-TUMORS | EMBRYONIC-DEVELOPMENT | INDUCIBLE FACTOR 1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | TISSUE-SPECIFIC EXPRESSION | GLUCOSE DEPRIVATION | HIPPOCAMPAL-NEURONS | PAS DOMAIN PROTEIN-1 | BIOPHYSICS | CLOSE SEQUENCE SIMILARITY | TRANSCRIPTION FACTOR | ENDOTHELIAL GROWTH-FACTOR | Antibody Specificity | Endothelial Growth Factors - metabolism | Vascular Endothelial Growth Factors | Transcriptional Activation | PC12 Cells | Hypoxia-Inducible Factor 1, alpha Subunit | Stem Cell Transplantation | Aryl Hydrocarbon Receptor Nuclear Translocator | Gene Deletion | Lymphokines - metabolism | Neovascularization, Pathologic - physiopathology | Neurons - metabolism | Dimerization | Basic Helix-Loop-Helix Transcription Factors | Teratocarcinoma - metabolism | Hypoxia-Inducible Factor 1 | Liver - metabolism | Rats | Teratocarcinoma - pathology | Mice, Nude | Stem Cells - pathology | Liver - cytology | Mice | Neovascularization, Pathologic - metabolism | Vascular Endothelial Growth Factor A | Transcription Factors - deficiency | RNA, Messenger - analysis | Neurons - cytology | Stem Cells - metabolism | Hypoxia - metabolism | Neovascularization, Pathologic - pathology | DNA-Binding Proteins - metabolism | Lymphokines - genetics | Nuclear Proteins - genetics | Teratocarcinoma - genetics | RNA, Messenger - genetics | Cells, Cultured | Gene Expression Regulation | Nuclear Proteins - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Sequence Homology, Amino Acid | Transcription Factors - metabolism | Hypoxia - genetics | Animals | Endothelial Growth Factors - genetics | Protein Binding | Neovascularization, Pathologic - genetics | Receptors, Aryl Hydrocarbon
Journal Article
Immunity, ISSN 1074-7613, 2000, Volume 13, Issue 3, pp. 397 - 407
The c-fes protooncogene encodes a nonreceptor tyrosine kinase (Fes) implicated in cytokine receptor signal transduction, neutrophil survival, and myeloid... 
SIGNAL-TRANSDUCTION | PROTOONCOGENE PRODUCT | COLONY-STIMULATING FACTOR | C-FOS PROMOTER | BINDING PROTEINS | MYELOID DIFFERENTIATION | PROTEIN-TYROSINE KINASE | IMMUNOLOGY | FES-RELATED PROTEIN | CELL-POPULATIONS | TRANSGENIC MICE | Lymphoid Tissue - enzymology | Immunologic Deficiency Syndromes - pathology | Bone Marrow Cells - enzymology | Immunity, Innate - genetics | Hematopoietic Stem Cells - pathology | Male | Lymphoid Tissue - immunology | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | Signal Transduction - immunology | Hematopoietic Stem Cells - immunology | Milk Proteins | Cell Division - immunology | Bone Marrow Cells - immunology | Female | Trans-Activators - biosynthesis | Macrophages - immunology | Cell Division - genetics | Gene Targeting | Immunologic Deficiency Syndromes - enzymology | Cell Adhesion - genetics | Mutagenesis, Site-Directed | Macrophages - pathology | Mice, Inbred C57BL | Bone Marrow Cells - pathology | Cells, Cultured | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Homeostasis - immunology | Proto-Oncogene Proteins - genetics | Signal Transduction - genetics | Proto-Oncogene Proteins - deficiency | Proto-Oncogene Proteins c-fes | Macrophages - enzymology | Mice, Knockout | Cell Adhesion - immunology | Cell Differentiation - immunology | Lymphoid Tissue - pathology | Animals | STAT5 Transcription Factor | Immunologic Deficiency Syndromes - genetics | Immunologic Deficiency Syndromes - metabolism | Trans-Activators - metabolism | Mice | STAT3 Transcription Factor | Protein-Tyrosine Kinases | DNA-Binding Proteins - biosynthesis | Hematopoietic Stem Cells - enzymology | Homeostasis - genetics
Journal Article
Journal Article
Journal Article