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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2014, Volume 111, Issue 43, pp. 15538 - 15543
Journal Article
Nature Medicine, ISSN 1078-8956, 09/2014, Volume 20, Issue 10, pp. 1138 - 1146
In solid tumors, resistance to therapy inevitably develops upon treatment with cytotoxic drugs or molecularly targeted therapies. Here, we describe a system... 
PATHWAYS | MEDICINE, RESEARCH & EXPERIMENTAL | HEPATOCELLULAR-CARCINOMA | INTEGRATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENES | SENSITIVITY | KINASE INHIBITORS | MAP KINASES | JAK/STAT | STRATEGIES | CELL BIOLOGY | Niacinamide - analogs & derivatives | RNA, Small Interfering - genetics | Mitogen-Activated Protein Kinase 14 - antagonists & inhibitors | Humans | Male | Liver Neoplasms, Experimental - metabolism | Carcinoma, Hepatocellular - drug therapy | Mitogen-Activated Protein Kinase 14 - metabolism | RNA Interference | Carcinoma, Hepatocellular - genetics | Female | Antineoplastic Agents - pharmacology | Liver Neoplasms, Experimental - genetics | Liver Neoplasms - genetics | Signal Transduction | Mice, Inbred C57BL | Liver Neoplasms - drug therapy | Mitogen-Activated Protein Kinase 14 - genetics | Activating Transcription Factor 2 - metabolism | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | MAP Kinase Signaling System - drug effects | Liver Neoplasms, Experimental - drug therapy | Liver Neoplasms - metabolism | Mice | Phenylurea Compounds - pharmacology | Protein Kinase Inhibitors - pharmacology | Niacinamide - pharmacology | Carcinoma, Hepatocellular - metabolism | Liver cancer | Research | Drug therapy | Drug resistance | Analysis | Biomarkers | Signal transduction | Protein expression | Pharmacology | Ribonucleic acid--RNA
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2015, Volume 21, Issue 10, pp. 1163 - 1171
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2010, Volume 107, Issue 30, pp. 13438 - 13443
Pancreatic cancer is one of the most fatal malignancies lacking effective therapies. Notch signaling is a key regulator of cell fate specification and... 
Acinar cells | Developmental biology | Cell lines | Pancreatic cells | Mice | Lesions | Carcinogenesis | Pancreatic neoplasms | Tumors | Notch receptors | K-Ras | Genetically engineered mice | Myc | Pancreatic cancer | Notch | ACTIVATION | MULTIDISCIPLINARY SCIENCES | MOUSE | C-MYC | ADULT MICE | CANCER | EPITHELIAL-MESENCHYMAL TRANSITION | pancreatic cancer | SIGNALING PATHWAY | GROWTH | ACINAR-CELLS | EXPRESSION | genetically engineered mice | Immunohistochemistry | Adenocarcinoma - pathology | Pancreatic Neoplasms - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Male | Receptor, Notch2 - genetics | Gene Expression Profiling | Carcinoma, Pancreatic Ductal - genetics | Adenocarcinoma - metabolism | Carcinoma in Situ - genetics | Female | Adenocarcinoma - genetics | Proto-Oncogene Proteins p21(ras) - metabolism | Signal Transduction | Carcinoma in Situ - pathology | Pancreatic Neoplasms - pathology | Kaplan-Meier Estimate | Receptor, Notch2 - metabolism | Pancreas - pathology | Pancreatic Neoplasms - genetics | Pancreas - metabolism | Receptor, Notch1 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Carcinoma, Pancreatic Ductal - pathology | Proto-Oncogene Proteins c-myc - metabolism | Blotting, Western | Disease Progression | Mice, Knockout | Animals | Cell Line, Tumor | Proto-Oncogene Proteins c-myc - genetics | Carcinoma in Situ - metabolism | Receptor, Notch1 - genetics | Biological Sciences
Journal Article
Nature, ISSN 0028-0836, 10/2018, Volume 562, Issue 7725, pp. 69 - 75
Primary liver cancer represents a major health problem. It comprises hepatocellular carcinoma (HCC) and intrahepatic cholangiocarcinoma (ICC), which differ... 
PATHOGENESIS | HEPATOCELLULAR-CARCINOMA | INTRAHEPATIC CHOLANGIOCARCINOMA | READ ALIGNMENT | HEPATOCYTES | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | SEQUENCING DATA | EXPRESSION | DISCOVERY | CELL-DEATH | Humans | Tumor Microenvironment | Apoptosis - genetics | Hepatocytes - pathology | Male | Gene Expression Profiling | Genes, myc | Hepatocytes - metabolism | Proto-Oncogene Proteins c-akt - genetics | DNA-Binding Proteins - metabolism | Carcinoma, Hepatocellular - genetics | DNA Transposable Elements - genetics | Female | Liver Neoplasms - pathology | Cell Differentiation | Cell Lineage - genetics | Disease Models, Animal | Cyclin-Dependent Kinase Inhibitor p16 - deficiency | Cytokines - metabolism | Liver Neoplasms - genetics | Carcinogenesis - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Transcription Factors - metabolism | Cholangiocarcinoma - pathology | Animals | Epigenesis, Genetic - genetics | Carcinoma, Hepatocellular - pathology | Cholangiocarcinoma - genetics | Mosaicism | Mice | Necrosis - genetics | Genes, ras | Liver cancer | Research | Carcinogenesis | Oncology, Experimental | Apoptosis | Cancer | Animal models | Cytokines | Liver | Hepatocellular carcinoma | Genomes | Metastasis | Risk analysis | Gene expression | Risk factors | Metastases | Hepatocytes | Morphology | DNA methylation | Tumorigenesis | Bioinformatics | Cholangiocarcinoma | Deoxyribonucleic acid--DNA | Tumors | Life Sciences
Journal Article
Polyhedron, ISSN 0277-5387, 01/2019, Volume 158, p. 117
In the current work, the hyperalkaline complexation processes in the Ca.sup.2+/d-gluconate (Gluc.sup.-) system have been studied under hypersaline conditions... 
Journal Article
Polyhedron, ISSN 0277-5387, 12/2018, Volume 156, p. 89
In the present study, the proton dissociation and calcium complex formation reactions of [alpha]- and [beta]-ketoglutarate (2- and 3-oxopentane-dioate,... 
Journal Article