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PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53657
Tuberculosis (TB) is responsible for death of nearly two million people in the world annually. Upon infection, Mycobacterium tuberculosis (Mtb) causes... 
IMMUNE-RESPONSE | CELLS | LATENT TUBERCULOSIS | CHRONIC MURINE TUBERCULOSIS | MULTIDISCIPLINARY SCIENCES | MYCOBACTERIUM-TUBERCULOSIS INFECTION | PULMONARY TUBERCULOSIS | NECROSIS-FACTOR-ALPHA | MICE | TRIACYLGLYCEROL | TNF | Granuloma - microbiology | Gene Expression | Humans | Bacterial Proteins - genetics | Drug Resistance, Bacterial | Antibodies, Neutralizing - pharmacology | Antibiotics, Antitubercular - pharmacology | Mycobacterium tuberculosis - drug effects | Triglycerides - metabolism | Host-Pathogen Interactions | Granuloma - pathology | Models, Biological | Gene Deletion | Bacterial Proteins - metabolism | Granuloma - immunology | Mutation | Rifampin - pharmacology | Latent Tuberculosis - microbiology | Lipase - deficiency | Mycobacterium tuberculosis - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Lipase - genetics | Mycobacterium tuberculosis - genetics | Granuloma | Tuberculosis | Analysis | Monoclonal antibodies | Models | Triglycerides | Disease susceptibility | B cells | Gene expression | Health aspects | Immunosuppressive agents | Pathogens | Leukocytes | Tumor necrosis factor-α | Kinases | Drug resistance | Immunological tolerance | Deletion mutant | Host-pathogen interactions | Dormancy | White blood cells | Lipid bodies | Clonal deletion | Biomimetics | Tumor necrosis factor-TNF | Rifampin | Resuscitation | Immune system
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2009, Volume 4, Issue 6, p. e6077
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2012, Volume 7, Issue 12, p. e51641
Journal Article
Journal Article
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 09/2017, Volume 377, Issue 12, pp. 1119 - 1131
Patients with myocardial infarction and a high-sensitivity CRP level of 2 mg or more per liter were assigned to one of three canakinumab doses or placebo. The... 
C-REACTIVE PROTEIN | MEDICINE, GENERAL & INTERNAL | HEART-DISEASE | MYOCARDIAL-INFARCTION | STATIN THERAPY | CLONAL HEMATOPOIESIS | INFLAMMATION | CARDIOVASCULAR-DISEASE | INTERLEUKIN-1-BETA INHIBITION | RISK | CORONARY-ARTERY-DISEASE | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Male | Secondary Prevention | Infection - etiology | Incidence | Dose-Response Relationship, Drug | Anti-Inflammatory Agents - adverse effects | C-Reactive Protein - metabolism | Lipids - blood | Cardiovascular Diseases - epidemiology | Cardiovascular Diseases - mortality | Anti-Inflammatory Agents - administration & dosage | Female | Neutropenia - chemically induced | Interleukin-1beta - antagonists & inhibitors | Stroke - prevention & control | Atherosclerosis - drug therapy | Double-Blind Method | Interleukin-1beta - immunology | Atherosclerosis - blood | Myocardial Infarction - drug therapy | Antibodies, Monoclonal - administration & dosage | Aged | Myocardial Infarction - prevention & control | Myocardial infarction | Cerebral infarction | Stroke | Heart attacks | C-reactive protein | Cytokines | Clinical trials | Cardiovascular disease | Innate immunity | Inflammation | Angina | Angina pectoris | Thrombosis | Cholesterol | Disease prevention | Proteins | Arteriosclerosis | Atherosclerosis | Cardiovascular diseases | Health risk assessment | Drug therapy | Drug dosages | Klinisk medicin | Clinical Medicine
Journal Article