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BBA - Molecular Basis of Disease, ISSN 0925-4439, 06/2014, Volume 1842, Issue 6, pp. 817 - 830
Errors in ribosome biogenesis can result in quantitative or qualitative defects in protein synthesis and consequently lead to improper execution of the genetic... 
RPL5 | 5S rRNA | Ribosome biogenesis stress | Ribosomopathies | RPL11 | p53 | P53 | PROTEIN S19 DEFICIENCY | MESSENGER-RNA TRANSLATION | POLYMERASE I TRANSCRIPTION | GENE DELETIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | C-MYC | CELL-CYCLE ARREST | NUCLEOLAR DISRUPTION | BIOPHYSICS | DIAMOND-BLACKFAN ANEMIA | P53-DEPENDENT CHECKPOINT | Protein biosynthesis | Tumor proteins | Analysis | Tumors
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2013, Volume 15, Issue 8, pp. 967 - 977
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2012, Volume 109, Issue 50, pp. 20467 - 20472
Impairment of ribosomal biogenesis can activate the p53 protein independently of DNA damage. The ability of ribosomal proteins L5, L11, L23, L26, or S7 to bind... 
Up regulation | Cell nucleolus | Ribosomal proteins | Ribosomes | Cell cycle | Antibodies | HeLa cells | Small interfering RNA | Cell aggregates | Cellular immunity | Ribosomal stress | Proteasome | HDM2 | proteasome | CELLS | INHIBITION | RNA | PATHWAY | MULTIDISCIPLINARY SCIENCES | GROWTH | 40S | MDM2 | CHECKPOINT | ribosomal stress | RNA, Small Interfering - genetics | Up-Regulation | Tumor Suppressor Protein p53 - antagonists & inhibitors | Humans | Stress, Physiological | Ribosomes - metabolism | RNA, Messenger - metabolism | Tumor Suppressor Protein p53 - genetics | Ribosomal Proteins - metabolism | Base Sequence | Cell Nucleolus - metabolism | Tumor Suppressor Proteins - genetics | Active Transport, Cell Nucleus | Protein Stability | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-mdm2 - metabolism | Ribosomal Proteins - antagonists & inhibitors | Tumor Suppressor Proteins - metabolism | Ribosomal Proteins - genetics | RNA, Messenger - genetics | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Transcription Factors - metabolism | Animals | Models, Biological | Cell Line, Tumor | Mice | Proteasome Endopeptidase Complex - metabolism | Dactinomycin - pharmacology | Promyelocytic Leukemia Protein | DNA damage | Physiological aspects | Biosynthesis | Stress (Physiology) | Research | Health aspects | Biological Sciences
Journal Article
Cell Cycle, ISSN 1538-4101, 01/2018, Volume 17, Issue 2, pp. 143 - 144
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 01/2017, Volume 114, Issue 4, p. E496
  In the noisy cellular environment, gene products are subject to inherent random fluctuations in copy numbers over time. How cells ensure precision in the... 
Proteins | Cellular biology | Genes | Bacteria | Viruses | Stochastic models
Journal Article
Proceedings of the National Academy of Sciences of the United States, ISSN 0027-8424, 01/2017, Volume 114, Issue 4, p. 637
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2012, Volume 109, Issue 50, p. 20467
  Impairment of ribosomal biogenesis can activate the p53 protein independently of DNA damage. The ability of ribosomal proteins L5, L11, L23, L26, or S7 to... 
Proteins | Pathogenesis | DNA damage | Leukemia | Biosynthesis
Journal Article
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