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1. Full Text Variants of DENND1B Associated with Asthma in Children
by Sleiman, Patrick M.A and Flory, James and Imielinski, Marcin and Bradfield, Jonathan P and Annaiah, Kiran and Willis-Owen, Saffron A.G and Wang, Kai and Rafaels, Nicholas M and Michel, Sven and Bonnelykke, Klaus and Zhang, Haitao and Kim, Cecilia E and Frackelton, Edward C and Glessner, Joseph T and Hou, Cuiping and Otieno, F. George and Santa, Erin and Thomas, Kelly and Smith, Ryan M and Glaberson, Wendy R and Garris, Maria and Chiavacci, Rosetta M and Beaty, Terri H and Ruczinski, Ingo and Orange, Jordan M and Allen, Julian and Spergel, Jonathan M and Grundmeier, Robert and Mathias, Rasika A and Christie, Jason D and von Mutius, Erika and Cookson, William O.C and Kabesch, Michael and Moffatt, Miriam F and Grunstein, Michael M and Barnes, Kathleen C and Devoto, Marcella and Magnusson, Mark and Li, Hongzhe and Grant, Struan F.A and Bisgaard, Hans and Hakonarson, Hakon
The New England Journal of Medicine, ISSN 0028-4793, 01/2010, Volume 362, Issue 1, pp. 36 - 44
A genomewide association study involving children of Northern European ancestry who had asthma yielded an association with a locus on chromosome 1q31 that was... 
MEDICINE, GENERAL & INTERNAL | CELL SUBSETS | PROTEIN | ORMDL3 EXPRESSION | DENN/MADD | CHILDHOOD ASTHMA | SUSCEPTIBILITY GENES | DROSOPHILA | Receptors, Tumor Necrosis Factor - metabolism | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Guanine Nucleotide Exchange Factors - genetics | Meta-Analysis as Topic | Humans | Male | Death Domain Receptor Signaling Adaptor Proteins - genetics | North America | Asthma - genetics | Case-Control Studies | Chromosomes, Human, Pair 1 | Chromosomes, Human, Pair 17 | Female | Polymorphism, Single Nucleotide | African Continental Ancestry Group - genetics | Odds Ratio | Child | Cytokines | Dendritic cells | Glucocorticoids | Genomics | African Americans | Single-nucleotide polymorphism | Grants | Tumor necrosis factor-α | Chromosome 1 | Asthma | Children & youth | Studies | Killer cells | Software packages | Data collection | Replication | Genetic factors | Children | Natural killer cells | Chromosomes | Chromosome 17 | Age | Hospitals | Genealogy
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2. Full Text ARAF recurrent mutation causes central conducting lymphatic anomaly treatable with a MEK inhibitor
by Li, Dong and March, Michael E and Gutierrez-Uzquiza, Alvaro and Kao, Charlly and Seiler, Christoph and Pinto, Erin and Matsuoka, Leticia S and Battig, Mark R and Bhoj, Elizabeth J and Wenger, Tara L and Tian, Lifeng and Robinson, Nora and Wang, Tiancheng and Liu, Yichuan and Weinstein, Brant M and Swift, Matthew and Jung, Hyun Min and Kaminski, Courtney N and Chiavacci, Rosetta and Perkins, Jonathan A and Levine, Michael A and Sleiman, Patrick M A and Hicks, Patricia J and Strausbaugh, Janet T and Belasco, Jean B and Dori, Yoav and Hakonarson, Hakon
Nature medicine, ISSN 1078-8956, 07/2019, Volume 25, Issue 7, pp. 1116 - 1116
The treatment of lymphatic anomaly, a rare devastating disease spectrum of mostly unknown etiologies, depends on the patient manifestations . Identifying the... 
VASCULAR ANOMALIES | CHYLOTHORAX | SYSTEM | MEDICINE, RESEARCH & EXPERIMENTAL | NOONAN SYNDROME | VESSELS | RAF | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYDROPS | PHENOTYPE | FETUSES | CELL BIOLOGY | Drug therapy | Risk factors | Lymphatic diseases | Edema | Therapy | Phosphorylation | Phenotypes | Lymphatic system | Respiratory function | Sirolimus | MEK inhibitors | Medical treatment | Medical services | Extracellular signal-regulated kinase | Zebrafish | Cadherin | Patients | Oxygen requirements | Inhibitors | Etiology | Actin | Pulmonary functions | Mutation | Dismantling
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3. Full Text Copy number variations in CTNNA3 and RBFOX1 associate with pediatric food allergy
by Li, Jin and Fung, Irene and Glessner, Joseph T and Pandey, Rahul and Wei, Zhi and Bakay, Marina and Mentch, Frank D and Pellegrino, Renata and Wang, Tiancheng and Kim, Cecilia and Hou, Cuiping and Wang, Fengxiang and Chiavacci, Rosetta M and Thomas, Kelly A and Spergel, Jonathan M and Hakonarson, Hakon and Sleiman, Patrick M. A
Journal of Immunology, ISSN 0022-1767, 08/2015, Volume 195, Issue 4, pp. 1599 - 1607
Food allergy is a significant public health concern, especially among children. Previous candidate gene studies suggested a few susceptibility loci for food... 
NEUROGENIC INFLAMMATION | DISEASES | METAANALYSIS | GENE | VARIANTS | DERMATITIS | ASTHMA | ALPHA-T-CATENIN | PEANUT ALLERGY | IMMUNOLOGY | GENOME-WIDE ASSOCIATION | Food Hypersensitivity - genetics | RNA-Binding Proteins - genetics | Genetic Predisposition to Disease | Meta-Analysis as Topic | Reproducibility of Results | Age Factors | Genetic Association Studies | Humans | Middle Aged | RNA Splicing Factors | Child, Preschool | Male | Case-Control Studies | DNA Copy Number Variations | Food Hypersensitivity - immunology | Gene Deletion | Adolescent | Female | Polymorphism, Single Nucleotide | RNA, Small Interfering | alpha Catenin - genetics | Child
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4. Full Text Common genetic variants on 5p14.1 associate with autism spectrum disorders
by Schellenberg, Gerard D and Minshew, Nancy J and Korvatska, Olena and Hakonarson, Hakon and Buxbaum, Joseph D and Hutman, Ted and Alvarez Retuerto, Ana I and Sleiman, Patrick M. A and Coon, Hilary and Nurnberger, John I and Kim, Cecilia E and Pericak-Vance, Margaret A and Dong, Hongmei and Bradfield, Jonathan P and Abrahams, Brett S and Gilbert, John R and Munson, Jeffrey and Sonnenblick, Lisa I and Klin, Ami and Levy, Susan E and Herman, Edward I and Ma, Deqiong and Sakurai, Takeshi and Hou, Cuiping and Lajonchere, Clara M and Miller, Judith and Zhang, Haitao and Cook, Edwin H and Estes, Annette and Wang, Kai and State, Matthew W and Haines, Jonathan L and Sweeney, John A and Wassink, Thomas H and Ozonoff, Sally and Piven, Joseph and Chiavacci, Rosetta and Cuccaro, Michael L and Brune, Camille W and Frackelton, Edward and McMahon, William M and Geschwind, Daniel H and Takahashi, Nagahide and Owley, Thomas and Glessner, Joseph T and Salyakina, Daria and Sutcliffe, James S and Dawson, Geraldine and Bucan, Maja and Bernier, Raphael and Imielinski, Marcin and Rappaport, Eric and Sigman, Marian and Cantor, Rita M and Schultz, Robert T and Grant, Struan F. A
Nature, ISSN 0028-0836, 05/2009, Volume 459, Issue 7246, pp. 528 - 533
Autism spectrum disorders (ASDs) represent a group of childhood neurodevelopmental and neuropsychiatric disorders characterized by deficits in verbal... 
CHROMOSOME | DISCONNECTION | MULTIDISCIPLINARY SCIENCES | LOCI | BIPOLAR DISORDER | LINKAGE | IDENTIFICATION | UNDERCONNECTIVITY | EXPRESSION | GENOME-WIDE ASSOCIATION | FAMILIAL RISK | Autistic Disorder - genetics | Cell Adhesion - genetics | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Reproducibility of Results | Humans | Genotype | Case-Control Studies | Cell Adhesion Molecules, Neuronal - genetics | Brain - metabolism | Genetic Markers - genetics | Polymorphism, Single Nucleotide - genetics | Genetic Variation - genetics | Cadherins - genetics | Chromosomes, Human, Pair 5 - genetics | Cohort Studies | Genetic research | Genetic aspects | Risk factors | Pervasive developmental disorders | Autism | Brain | Genomics | Quality control | Genetics | Software | Kinases | Gene expression | Chromosomes
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5. Full Text Autism genome-wide copy number variation reveals ubiquitin and neuronal genes
by Glessner, Joseph T and Wang, Kai and Cai, Guiqing and Korvatska, Olena and Kim, Cecilia E and Wood, Shawn and Zhang, Haitao and Estes, Annette and Brune, Camille W and Bradfield, Jonathan P and Imielinski, Marcin and Frackelton, Edward C and Reichert, Jennifer and Crawford, Emily L and Munson, Jeffrey and Sleiman, Patrick M. A and Chiavacci, Rosetta and Annaiah, Kiran and Thomas, Kelly and Hou, Cuiping and Glaberson, Wendy and Flory, James and Otieno, Frederick and Garris, Maria and Soorya, Latha and Klei, Lambertus and Piven, Joseph and Meyer, Kacie J and Anagnostou, Evdokia and Sakurai, Takeshi and Game, Rachel M and Rudd, Danielle S and Zurawiecki, Danielle and McDougle, Christopher J and Davis, Lea K and Miller, Judith and Posey, David J and Michaels, Shana and Kolevzon, Alexander and Silverman, Jeremy M and Bernier, Raphael and Levy, Susan E and Schultz, Robert T and Dawson, Geraldine and Owley, Thomas and McMahon, William M and Wassink, Thomas H and Sweeney, John A and Nurnberger, John I and Coon, Hilary and Sutcliffe, James S and Minshew, Nancy J and Grant, Struan F. A and Bucan, Maja and Cook, Edwin H and Buxbaum, Joseph D and Devlin, Bernie and Schellenberg, Gerard D and Hakonarson, Hakon
Nature, ISSN 0028-0836, 05/2009, Volume 459, Issue 7246, pp. 569 - 572
Autism spectrum disorders (ASDs) are childhood neurodevelopmental disorders with complex genetic origins(1-4). Previous studies focusing on candidate genes or... 
MIGRATION | SPECTRUM DISORDER | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY GENE | NEUROLIGINS | MUTATIONS | LINKAGE | ASSOCIATION | Autistic Disorder - genetics | Gene Dosage - genetics | Genetic Predisposition to Disease - genetics | Reproducibility of Results | Gene Regulatory Networks - genetics | Humans | Ubiquitin - metabolism | Genotype | Case-Control Studies | Cell Adhesion Molecules, Neuronal - genetics | Europe - ethnology | Genome, Human - genetics | Polymerase Chain Reaction | Polymorphism, Single Nucleotide - genetics | Genetic Variation - genetics | Neurons - metabolism | Cohort Studies | Ubiquitin | Autism | Genetic aspects | Properties | Neurons | Genomics | Genes | Genetics | Standard deviation | Risk factors | Methods | Data bases | Cell adhesion & migration
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6. Full Text GWAS identifies four novel eosinophilic esophagitis loci
by Sleiman, Patrick M. A and Wang, Mei-Lun and Cianferoni, Antonella and Aceves, Seema and Gonsalves, Nirmala and Nadeau, Kari and Bredenoord, Albert J and Furuta, Glenn T and Spergel, Jonathan M and Hakonarson, Hakon
Nature communications, ISSN 2041-1723, 2014, Volume 5, Issue 1, p. 5593
Eosinophilic esophagitis (EoE) is an allergic disorder characterized by infiltration of the oesophagus with eosinophils. We had previously reported association... 
ATOPIC-DERMATITIS | RISK LOCI | METAANALYSIS | PROTEIN | CALPAIN | ALLERGY | JAPANESE POPULATION | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY LOCI | ASTHMA | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease | Genome-Wide Association Study | Guanine Nucleotide Exchange Factors - genetics | Humans | Repressor Proteins - genetics | Male | STAT6 Transcription Factor - genetics | Calpain - genetics | Case-Control Studies | Adult | Female | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | Eosinophilic Esophagitis - genetics
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7. Full Text Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
by Sawcer, Stephen and Hellenthal, Garrett and Pirinen, Matti and Spencer, Chris C.A and Patsopoulos, Nikolaos A and Moutsianas, Loukas and Dilthey, Alexander and Su, Zhan and Freeman, Colin and Hunt, Sarah E and Edkins, Sarah and Gray, Emma and Booth, David R and Potter, Simon C and Goris, An and Band, Gavin and Oturai, Annette Bang and Strange, Amy and Saarela, Janna and Bellenguez, Céline and Fontaine, Bertrand and Gillman, Matthew and Hemmer, Bernhard and Gwilliam, Rhian and Zipp, Frauke and Jayakumar, Alagurevathi and Martin, Roland and Leslie, Stephen and Hawkins, Stanley and Giannoulatou, Eleni and D'Alfonso, Sandra and Blackburn, Hannah and Boneschi, Filippo Martinelli and Liddle, Jennifer and Harbo, Hanne F and Perez, Marc L and Spurkland, Anne and Waller, Matthew J and Mycko, Marcin P and Ricketts, Michelle and Comabella, Manuel and Hammond, Naomi and Kockum, Ingrid and McCann, Owen T and Ban, Maria and Whittaker, Pamela and Kemppinen, Anu and Weston, Paul and Hawkins, Clive and Widaa, Sara and Zajicek, John and Dronov, Serge and Robertson, Neil and Bumpstead, Suzannah J and Barcellos, Lisa F and Ravindrarajah, Rathi and Abraham, Roby and Alfredsson, Lars and Ardlie, Kristin and Aubin, Cristin and Baker, Amie and Baker, Katharine and Baranzini, Sergio E and Bergamaschi, Laura and Bergamaschi, Roberto and Bernstein, Allan and Berthele, Achim and Boggild, Mike and Bradfield, Jonathan P and Brassat, David and Broadley, Simon A and Buck, Dorothea and Butzkueven, Helmut and Capra, Ruggero and Carroll, William M and Cavalla, Paola and Celius, Elisabeth G and Cepok, Sabine and Chiavacci, Rosetta and Clerget-Darpoux, Françoise and Clysters, Katleen and Comi, Giancarlo and Cossburn, Mark and Cournu-Rebeix, Isabelle and Cox, Mathew B and Cozen, Wendy and Cree, Bruce A.C and Cross, Anne H and Cusi, Daniele and Daly, Mark J and Davis, Emma and De Bakker, Paul I.W and Debouverie, Marc and D'Hooghe, Marie Beatrice and Dixon, Katherine and Dobosi, Rita and Dubois, Bénédicte and Ellinghaus, David and Elovaara, Irina and Esposito, Federica and ... and Wellcome Trust Case Control Consor and Int Multiple Sclerosis Genetics Co and Wellcome Trust Case Control Consortium 2 and International Multiple Sclerosis Genetics Consortium and The International Multiple Sclerosis Genetics Consortium & The Wellcome Trust Case Control Consortium 2 and Medicinska fakulteten and Science for Life Laboratory, SciLifeLab and Medicinska och farmaceutiska vetenskapsområdet and Molekylär medicin and Uppsala universitet and Institutionen för medicinska vetenskaper
Nature, ISSN 0028-0836, 08/2011, Volume 476, Issue 7359, pp. 214 - 219
Multiple sclerosis is a common disease of the central nervous system in which the interplay between inflammatory and neurodegenerative processes typically... 
HETEROGENEITY | ALLELES | VARIANTS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY LOCI | HAPLOTYPES | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Immunity, Cellular - genetics | Major Histocompatibility Complex - genetics | Humans | Multiple Sclerosis - genetics | Europe - ethnology | Genome, Human - genetics | HLA-DR Antigens - genetics | Cell Differentiation - immunology | Immunity, Cellular - immunology | Sample Size | T-Lymphocytes, Helper-Inducer - immunology | HLA-DRB1 Chains | Alleles | HLA-A Antigens - genetics | T-Lymphocytes, Helper-Inducer - cytology | Multiple Sclerosis - immunology | Polymorphism, Single Nucleotide - genetics | Physiological aspects | Multiple sclerosis | Genetic aspects | Immune response | Research | Epidemiology | Genealogy | Disease | Quality control | Genetics | Genomes | Statistical methods | Mutation | Regression analysis | Autoimmune diseases | Meta-analysis | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
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8. Full Text Meta-analysis of genome-wide association studies identifies three new risk loci for atopic dermatitis
by Paternoster, Lavinia and Standl, Marie and Chen, Chih-Mei and Ramasamy, Adaikalavan and Bonnelykke, Klaus and Duijts, Liesbeth and Ferreira, Manuel A and Alves, Alexessander Couto and Thyssen, Jacob P and Albrecht, Eva and Baurecht, Hansjoerg and Feenstra, Bjarke and Sleiman, Patrick M. A and Hysi, Pirro and Warrington, Nicole M and Curjuric, Ivan and Myhre, Ronny and Curtin, John A and Groen-Blokhuis, Maria M and Kerkhof, Marjan and Saaf, Annika and Franke, Ane and Ellinghaus, David and Foelster-Holst, Regina and Dermitzakis, Emmanouil and Montgomery, Stephen B and Prokisch, Holger and Heim, Katharina and Hartikainen, Anna-Liisa and Pouta, Anneli and Pekkanen, Juha and Blakemore, Alexana I. F and Buxton, Jessica L and Kaakinen, Marika and Duffy, David L and Madden, Pamela A and Heath, Anew C and Montgomery, Grant W and Thompson, Philip J and Matheson, Melanie C and Le Souef, Peter and St Pourcain, Beate and Smith, George Davey and Henderson, John and Kemp, John P and Timpson, Nicholas J and Deloukas, Panos and Ring, Susan M and Wichmann, H-Erich and Mueller-Nurasyid, Martina and Novak, Natalija and Klopp, Norman and Roiguez, Elke and McArdle, Wendy and Linneberg, Allan and Menne, Torkil and Nohr, Ellen A and Hofman, Albert and Uitterlinden, Ane G and van Duijin, Cornelia M and Rivadeneira, Fernando and de Jongste, Johan C and van der Valk, Ralf J. P and Wjst, Matthias and Jogi, Rain and Geller, Frank and Boyd, Heather A and Murray, Jeffrey C and Kim, Cecilia and Mentch, Frank and March, Michael and Mangino, Massimo and Spector, Tim D and Bataille, Veronique and Pennell, Craig E and Holt, Patrick G and Sly, Peter and Tiesler, Carla M. T and Thiering, Elisabeth and Illig, Thomas and Imboden, Medea and Nystad, Wenche and Simpson, Angela and Hottenga, Jouke-Jan and Postma, Dirkje and Koppelman, Gerard H and Smit, Henriette A and Soderhall, Cilla and Chawes, Bo and Kreiner-Moller, Eskil and Bisgaard, Hans and Melen, Erik and Boomsma, Dorret I and Custovic, Adnan and Jacobsson, Bo and Probst-Hensch, Nicole M and Palmer, Lyle J and Glass, Daniel and Hakonarson, Hakon and Melbye, Mads and ... and Australian Asthma Genetics Consort and EArly Genetics Lifecourse Epidemio and Genetics Overweight Young Adults G and Genetics of Overweight Young Adults (GOYA) Consortium and Australian Asthma Genetics Consortium (AAGC) and EArly Genetics & Lifecourse Epidemiology (EAGLE) Consortium and The Genetics of Overweight Young Adults (GOYA) Consortium and the EArly Genetics & Lifecourse Epidemiology (EAGLE) Consortium and Sahlgrenska akademin and Göteborgs universitet and Gothenburg University and Institute of Clinical Sciences, Section for the Health of Women and Children, Department of Obstetrics and Gynecology and Sahlgrenska Academy and Institutionen för kliniska vetenskaper, sektionen för kvinnors och barns hälsa, Avdelningen för obstetrik och gynekologi
Nature Genetics, ISSN 1061-4036, 02/2012, Volume 44, Issue 2, pp. 187 - 192
Atopic dermatitis (AD) is a commonly occurring chronic skin disease with high heritability. Apart from filaggrin (FLG), the genes influencing atopic dermatitis... 
SPERMATOGENESIS | CELLULAR MOTILITY | VARIANTS | FILAGGRIN | DISEASE | GENETICS & HEREDITY | ASTHMA | MECHANISMS | DIFFERENTIATION | EXPRESSION | PSORIASIS | Dermatitis, Atopic - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Humans | Male | Risk | Chromosomes, Human, Pair 11 - genetics | Genetic Loci | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Chromosomes, Human, Pair 5 | Cell Differentiation - genetics | Epidermis - immunology | Dermatitis, Atopic - immunology | Intermediate Filament Proteins - genetics | Kinesin - genetics | Female | Polymorphism, Single Nucleotide | Cytokines - genetics | Chromosomes, Human, Pair 20 - genetics | Quantitative trait loci | Reports | Genetic aspects | Genetic susceptibility | Atopic dermatitis | Risk factors | Confidence intervals | Genomes | Mutation | Dermatitis | Allergies | Asthma | Meta-analysis | Klinisk medicin | Clinical Medicine
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9. Full Text Multi-ancestry genome-wide association study of 21,000 cases and 95,000 controls identifies new risk loci for atopic dermatitis
by Paternoster, Lavinia and Standl, Marie and Waage, Johannes and Baurecht, Hansjoerg and Hotze, Melanie and Strachan, David P and Curtin, John A and Bonnelykke, Klaus and Tian, Chao and Takahashi, Atsushi and Esparza-Gordillo, Jorge and Alves, Alexessander Couto and Thyssen, Jacob P and den Dekker, Herman T and Ferreira, Manuel A and Altmaier, Elisabeth and Sleiman, Patrick M. A and Xiao, Feng Li and Gonzalez, Juan R and Marenholz, Ingo and Kalb, Birgit and Pino-Yanes, Maria and Xu, Chengjian and Carstensen, Lisbeth and Groen-Blokhuis, Maria M and Venturini, Cristina and Pennell, Craig E and Barton, Sheila J and Levin, Albert M and Curjuric, Ivan and Bustamante, Mariona and Kreiner-Moller, Eskil and Lockett, Gabrielle A and Bacelis, Jonas and Bunyavanich, Supinda and Myers, Rachel A and Matanovic, Anja and Kumar, Ashish and Tung, Joyce Y and Hirota, Tomomitsu and Kubo, Michiaki and McArdle, Wendy L and Henderson, A. John and Kemp, John P and Zheng, Jie and Smith, George Davey and Rueschendorf, Franz and Bauerfeind, Anja and Lee-Kirsch, Min Ae and Arnold, Aneas and Homuth, Georg and Schmidt, Carsten O and Mangold, Elisabeth and Cichon, Sven and Keil, Thomas and Roiguez, Elke and Peters, Annette and Franke, Ane and Lieb, Wolfgang and Novak, Natalija and Foelster-Holst, Regina and Horikoshi, Momoko and Pekkanen, Juha and Sebert, Sylvain and Husemoen, Lise L and Grarup, Niels and De Jongste, Johan C and Rivadeneira, Fernando and Hofman, Albert and Jaddoe, Vincent W. V and Pasmans, Suzanne G. M. A and Elbert, Niels J and Uitterlinden, Ane G and Marks, Guy B and Thompson, Philip J and Matheson, Melanie C and Robertson, Colin F and Ried, Janina S and Li, Jin and Zuo, Xian Bo and Zheng, Xiao Dong and Yin, Xian Yong and Sun, Liang Dan and McAleer, Maeve A and O'Regan, Grainne M and Fahy, Caoimhe M. R and Campbell, Linda E and Macek, Milan and Kurek, Michael and Hu, Donglei and Eng, Celeste and Postma, Dirkje S and Feenstra, Bjarke and Geller, Frank and Hottenga, Jouke Jan and Middeldorp, Christel M and Hysi, Pirro and Bataille, Veronique and Spector, Tim and Tiesler, Carla M. T and ... and AAGC and EArly Genetics Lifecourse Epidemio and Australian Asthma Genetics Consortium (AAGC) and Sahlgrenska akademin and Göteborgs universitet and Gothenburg University and Institute of Clinical Sciences, Section for the Health of Women and Children, Department of Obstetrics and Gynecology and Sahlgrenska Academy and Institutionen för kliniska vetenskaper, sektionen för kvinnors och barns hälsa, Avdelningen för obstetrik och gynekologi
Nature Genetics, ISSN 1061-4036, 12/2015, Volume 47, Issue 12, pp. 1449 - +
Genetic association studies have identified 21 loci associated with atopic dermatitis risk predominantly in populations of European ancestry. To identify... 
CELLS | METAANALYSIS | VARIANTS | FILAGGRIN | DISEASE | GENETICS & HEREDITY | SUSCEPTIBILITY LOCI | ASTHMA | SKIN | INTERLEUKIN-7 RECEPTOR | IL-7 | Dermatitis, Atopic - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Humans | Risk Factors | Immunity, Innate - genetics | Dermatitis, Atopic - pathology | Genetic Loci | Dermatitis, Atopic - ethnology | Case-Control Studies | Ethnic Groups - genetics | T-Lymphocytes - cytology | T-Lymphocytes - metabolism | Genetic Markers - genetics | Polymorphism, Single Nucleotide - genetics | T-Lymphocytes - immunology | Genome-wide association studies | Immune response | Atopic dermatitis | Development and progression | Genotype | Genetic aspects | Identification and classification | Health aspects | Methods | Studies | Confidence intervals | Inflammatory bowel disease | Genealogy | Disease | Psoriasis | Bias | Eczema | Genomes | Mutation | Dermatitis | Quality of life | Genètica | Grups ètnics | Dermatitis atòpica | GENETIC ARCHITECTURE | ATES OF AMERICA | Genetics & Heredity | PE1761 | Obstetrics, Gynecology and Reproductive Medicine | INFLAMMATORY-BOWEL-DISEASE | JAPANESE POPULATION | Reproduktionsmedicin och gynekologi | V110 | PSORIASIS
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