X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (12) 12
humans (9) 9
ubiquitin (8) 8
biochemistry & molecular biology (7) 7
hela cells (6) 6
inositol 1,4,5-trisphosphate receptors - metabolism (6) 6
membrane proteins - metabolism (6) 6
animals (5) 5
article (5) 5
autophagy (5) 5
endoplasmic-reticulum (5) 5
mitochondria (5) 5
mutation (5) 5
proteasome (5) 5
reticulum-associated degradation (5) 5
ubiquitin - metabolism (5) 5
ubiquitin-protein ligases - metabolism (5) 5
calcium signaling (4) 4
cells (4) 4
complex (4) 4
down-regulation (4) 4
endoplasmic reticulum - metabolism (4) 4
multidisciplinary sciences (4) 4
proteasome endopeptidase complex - metabolism (4) 4
protein binding (4) 4
ubiquitination (4) 4
activation (3) 3
cell biology (3) 3
endoplasmic reticulum (3) 3
endoplasmic reticulum-associated degradation (3) 3
inositol 1,4,5-trisphosphate receptor (3) 3
inositol 1,4,5-trisphosphate receptors - genetics (3) 3
inositol trisphosphate receptor (3) 3
membrane proteins - genetics (3) 3
mice (3) 3
mitochondria - metabolism (3) 3
mitophagy (3) 3
mutations (3) 3
phosphorylation (3) 3
protein kinases - metabolism (3) 3
rats (3) 3
research (3) 3
signal transduction (3) 3
ubiquitin-protein ligases - genetics (3) 3
ubiquitination - physiology (3) 3
carbohydrates (2) 2
cell line (2) 2
degradation (2) 2
deubiquitinating enzymes (2) 2
disease (2) 2
e3 ubiquitin ligase (2) 2
enzymes (2) 2
erlin2 (2) 2
gene expression (2) 2
genetic aspects (2) 2
inorganic chemicals (2) 2
kinases (2) 2
ligase (2) 2
membrane proteins (2) 2
mitochondrial degradation (2) 2
mitochondrial dna (2) 2
mitochondrial proteins - metabolism (2) 2
molecular biology (2) 2
nervous system diseases (2) 2
optineurin (2) 2
parkinson disease - metabolism (2) 2
parkinson's disease (2) 2
pathway (2) 2
physiological aspects (2) 2
pink1 (2) 2
polyubiquitin chains (2) 2
proteasomal degradation (2) 2
protein (2) 2
protein degradation (2) 2
protein structure, tertiary (2) 2
protein synthesis and degradation (2) 2
protein-serine-threonine kinases - metabolism (2) 2
proteomics (2) 2
quality-control (2) 2
rnf170 (2) 2
transcription factor tfiiia - metabolism (2) 2
activate parkin (1) 1
adaptor proteins, signal transducing - metabolism (1) 1
advanced basic science (1) 1
alarmins - metabolism (1) 1
amino acid sequence (1) 1
amyotrophic-lateral-sclerosis (1) 1
anaphase-promoting complex (1) 1
animal models (1) 1
arginine - chemistry (1) 1
ataxia - congenital (1) 1
ataxia - genetics (1) 1
ataxia - metabolism (1) 1
automation (1) 1
autophagosome biogenesis (1) 1
autophagy - physiology (1) 1
autophagy-related protein-1 homolog (1) 1
autophagy-related proteins (1) 1
bias (1) 1
binding (1) 1
more...
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Mitochondrion, ISSN 1567-7249, 11/2019, Volume 49, pp. 217 - 226
Mitochondrial dynamics and mitophagy are important aspects of mitochondrial quality control, and are linked to neurodegenerative diseases and muscular... 
Fis1 | Mitophagy | Mitochondrial dynamics | Exhaustive exercise | Inflammation response
Journal Article
Journal Article
Nature, ISSN 0028-0836, 09/2018, Volume 561, Issue 7722, pp. 258 - 262
Although serum from patients with Parkinson's disease contains elevated levels of numerous pro-inflammatory cytokines including IL-6, TNF, IL-1 beta, and IFN... 
OXIDATIVE STRESS | ACTIVATION | MUTANTS | MULTIDISCIPLINARY SCIENCES | IN-VIVO MITOPHAGY | DISEASE | NEURONS | DEFICIENT MICE | MUTATIONS | MITOCHONDRIAL-DNA | Protein Kinases - metabolism | Protein Kinases - genetics | Humans | Stress, Physiological | Receptor, Interferon alpha-beta - antagonists & inhibitors | Membrane Proteins - deficiency | Inflammation - metabolism | DNA, Mitochondrial - genetics | Membrane Proteins - metabolism | Parkinson Disease - metabolism | Protein Kinases - deficiency | Physical Conditioning, Animal | Membrane Proteins - genetics | Mice, Inbred C57BL | Alarmins - metabolism | Ubiquitin-Protein Ligases - metabolism | Immunity, Innate | DNA, Mitochondrial - blood | Mitochondrial Degradation | Animals | Receptor, Interferon alpha-beta - immunology | Inflammation - genetics | Ubiquitin-Protein Ligases - deficiency | Inflammation - prevention & control | Mice | Ubiquitin-Protein Ligases - genetics | Inflammation | Genetic aspects | Research | Gene mutations | Risk factors | Ubiquitin | Heart | Cell culture | Oxidative stress | Animal models | Parkinson's disease | Substantia nigra | Parkinsons disease | Innate immunity | Mitochondrial DNA | Kinases | Immunity | Interleukin 6 | Mitochondria | Movement disorders | Ubiquitin-protein ligase | Deoxyribonucleic acid--DNA | Dopamine receptors | Enzymes | Phenotypes | Dopamine | Cytokines | Neurodegenerative diseases | Body temperature | Damage patterns | Insects | Tumor necrosis factor | PTEN-induced putative kinase | Proteomics | Interferon | Parkin protein | Mutation | Methods
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 504, Issue 7479, pp. 291 - 295
Journal Article
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 04/2016, Volume 113, Issue 15, pp. 4039 - 4039
Selective autophagy of damaged mitochondria requires autophagy receptors optineurin (OPTN), NDP52 (CALCOCO2), TAX1BP1, and p62 (SQSTM1) linking ubiquitinated... 
Journal Article
Current Biology, ISSN 0960-9822, 01/2018, Volume 28, Issue 2, pp. 287 - 295.e6
The clearance of mitochondria by autophagy, mitophagy, is important for cell and organism health [ ], and known to be regulated by ubiquitin. During intestine... 
mitophagy | autophagy | mitochondria | Drosophila | fission | Vps13D | ubiquitin | RECOGNITION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENES | DEGRADATION | AUTOPHAGY | FISSION | DROSOPHILA | GENOME | CELL BIOLOGY | Ubiquitin | Enzymes | Nervous system diseases | Cells | Protein binding
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 04/2016, Volume 113, Issue 15, p. 4039
  Selective autophagy of damaged mitochondria requires autophagy receptors optineurin (OPTN), NDP52 (CALCOCO2), TAX1BP1, and p62 (SQSTM1) linking ubiquitinated... 
Phosphorylation | Mitochondria | Membranes | Proteomics | Autophagy
Journal Article
Journal Article
Journal Article
Cell Calcium, ISSN 0143-4160, 09/2009, Volume 46, Issue 3, p. 147
While cell signaling devotees tend to think of the endoplasmic reticulum (ER) as a Ca.sup.2+ store, those who study protein synthesis tend to see it more as... 
Ubiquitin | Inositol | Protein biosynthesis | Membrane proteins | Protein binding
Journal Article
Cell Calcium, ISSN 0143-4160, 2009, Volume 46, Issue 3, pp. 147 - 153
While cell signaling devotees tend to think of the endoplasmic reticulum (ER) as a Ca store, those who study protein synthesis tend to see it more as site for... 
Inositol 1,4,5-trisphosphate receptor | Ubiquitin | Calcium signaling | Endoplasmic reticulum-associated degradation | Proteasome
Journal Article
Cell Calcium, ISSN 0143-4160, 2009, Volume 46, Issue 3, pp. 147 - 153
Abstract While cell signaling devotees tend to think of the endoplasmic reticulum (ER) as a Ca2+ store, those who study protein synthesis tend to see it more... 
Advanced Basic Science | Calcium signaling | Inositol 1,4,5-trisphosphate receptor | Ubiquitin | Endoplasmic reticulum-associated degradation | Proteasome | UBIQUITIN-PROTEASOME SYSTEM | PROTEIN | RETICULUM-ASSOCIATED DEGRADATION | DOWN-REGULATION | QUALITY-CONTROL | CELL BIOLOGY | CALCIUM | ENDOPLASMIC-RETICULUM | CYSTIC-FIBROSIS | ION CHANNELS
Journal Article
Journal Article
The Journal of Biological Chemistry, ISSN 0021-9258, 12/2008, Volume 283, Issue 51, pp. 35319 - 35328
Inositol 1,4,5-trisphosphate (IP 3 ) receptors form tetrameric channels in endoplasmic reticulum membranes of mammalian cells and mediate IP 3 -induced calcium... 
Protein Synthesis, Post-Translational Modification, and Degradation
Journal Article