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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2018, Volume 38, Issue 9, pp. 2184 - 2190
OBJECTIVE—Unreliable antibodies often hinder the accurate detection of an endogenous protein, and this is particularly true for the cardiac and smooth muscle... 
smooth muscle | CRISPR | antibody | Myocardin
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 01/2019, Volume 116, Issue 2, pp. 546 - 555
SENCR is a human-specific, vascular cell-enriched long-noncoding RNA (lncRNA) that regulates vascular smooth muscle cell and endothelial cell (EC) phenotypes.... 
Shear stress | Endothelial cell | Long-noncoding RNA | Adherens junction | PROTEIN | PERMEABILITY | endothelial cell | MULTIDISCIPLINARY SCIENCES | long-noncoding RNA | MECHANISMS | BLOOD-TUMOR BARRIER | IDENTIFICATION | ORGANIZATION | shear stress | adherens junction | SHEAR-STRESS | VE-CADHERIN | ENDOPLASMIC-RETICULUM | LONG | Adherens Junctions - genetics | Cadherins - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Membrane Proteins - genetics | Humans | Shear Strength - physiology | RNA, Long Noncoding - genetics | Antigens, CD - genetics | Catenins - genetics | Adherens Junctions - metabolism | Antigens, CD - metabolism | Catenins - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Protein Domains | Membrane Proteins - metabolism | Cadherins - genetics | RNA, Long Noncoding - metabolism | Physiological aspects | Vascular endothelium | Muscle cells | Smooth muscle | Membrane proteins | Phenotypes | Immunoprecipitation | Membrane permeability | Homeostasis | Muscles | Mass spectroscopy | Adherens junctions | Cell membranes | Shear stresses | Permeability | Ribonucleic acid--RNA | Endothelial cells | Proteins | Genotype & phenotype | Cellular biology | Internalization | Aorta | Cytoskeleton | Mass spectrometry | Structure-function relationships | Integrity | Biological Sciences | PNAS Plus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2018, Volume 38, Issue 9, pp. 2184 - 2190
OBJECTIVE—Unreliable antibodies often hinder the accurate detection of an endogenous protein, and this is particularly true for the cardiac and smooth muscle... 
myocardin | muscle, smooth | epitope | mice | allele
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 2/2015, Volume 35, Issue 2, pp. 312 - 315
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 2, p. e0171262
Journal Article
Journal Article
Lab Animal, ISSN 0093-7355, 04/2012, Volume 41, Issue 4, pp. 102 - 107
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 03/2017, Volume 114, Issue 13, pp. E2739 - E2747
textabstractMegacystis microcolon intestinal hypoperistalsis syndrome (MMIHS) is a congenital visceral myopathy characterized by severe dilation of the urinary... 
Genetics | Smooth muscle | Leiomodin | CRISPR-Cas9 | Myopathy | LIGHT-CHAIN KINASE | NEMALINE MYOPATHY | MEMBERS | CONTRACTILITY | myopathy | MULTIDISCIPLINARY SCIENCES | TROPOMODULIN | smooth muscle | LACKING | genetics | GENE | ACTA2 MUTATION | EXPRESSION | Physiological aspects | Genetic disorders | Health aspects | Muscle contraction | Biological Sciences | PNAS Plus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 03/2017, Volume 114, Issue 13, p. E2739
  Megacystis microcolon intestinal hypoperistalsis syndrome (MMIHS) is a congenital visceral myopathy characterized by severe dilation of the urinary bladder... 
Rodents | Genes | Cytoskeleton | Smooth muscle | Genomes | Mutation
Journal Article
Proceedings of the National Academy of Science of the United States of America, ISSN 0027-8424, 03/2017, Volume 114, Issue 13, pp. E2739 - E2747
Megacystis microcolon intestinal hypoperistalsis syndrome (MMIHS) is a congenital visceral myopathy characterized by severe dilation of the urinary bladder and... 
LIGHT-CHAIN KINASE | NEMALINE MYOPATHY | myopathy | MOUSE EMBRYOGENESIS | MYOSIN | TROPOMODULIN | smooth muscle | FAMILY | genetics | GENE | CRISPR-Cas9 | Leiomodin | ACTA2 MUTATION | EXPRESSION
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2011, Volume 31, Issue 10, pp. 2172 - 2180
OBJECTIVE—Smooth muscle calponin (CNN1) contains multiple conserved intronic CArG elements that bind serum response factor and display enhancer activity in... 
vascular muscle | serum response factor | transgenic models | gene expression | genetically altered mice | MYOCARDIN | ACTIN GENE | CONTRACTILE PHENOTYPE | DNA-SEQUENCE | CELL DIFFERENTIATION | PERIPHERAL VASCULAR DISEASE | PROMOTER | HEMATOLOGY | EXPRESSION | MASTER REGULATOR | TRANSGENIC MICE | Carotid Arteries - metabolism | Immunohistochemistry | Cell Proliferation | Luciferases - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Myocytes, Smooth Muscle - pathology | Luciferases - genetics | Transfection | Chromosomes, Artificial, Bacterial | Carotid Artery Injuries - genetics | Microfilament Proteins - metabolism | Binding Sites | Microfilament Proteins - genetics | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Genes, Reporter | Carotid Artery Injuries - pathology | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Cell Line | Promoter Regions, Genetic | Microfilament Proteins - deficiency | Serum Response Element | Introns | Serum Response Factor - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Rats | Calcium-Binding Proteins - deficiency | Mice, Transgenic | Blotting, Western | Mice, Knockout | Tunica Intima - metabolism | Muscle, Smooth, Vascular - pathology | Animals | Tunica Intima - pathology | Carotid Arteries - pathology | Mice | Calcium-Binding Proteins - genetics | Lac Operon | smooth muscle | transgenic mice | transcription | calponin
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 4, p. e18538
Retinoids are used clinically to treat a number of hyper-proliferative disorders and have been shown in experimental animals to attenuate vascular occlusive... 
KINASE-C SUBSTRATE | PROTEIN-KINASE | INTIMAL HYPERPLASIA | NUCLEAR RECEPTORS | BIOLOGY | SERUM RESPONSE FACTOR | BALLOON ANGIOPLASTY | PROLIFERATION IN-VITRO | NEOINTIMA FORMATION | ANGIOTENSIN-II | CYTOSKELETAL ARCHITECTURE | Cyclic AMP-Dependent Protein Kinases - metabolism | Cell Line | Muscle, Smooth, Vascular - metabolism | Humans | Cell Cycle Proteins - metabolism | Rats | Male | Muscle, Smooth, Vascular - cytology | Promoter Regions, Genetic - genetics | Gene Expression Regulation - drug effects | A Kinase Anchor Proteins - genetics | Animals | Protein Isoforms - metabolism | Cell Cycle Proteins - genetics | Cell Proliferation - drug effects | Mice | Response Elements - genetics | Genes, Tumor Suppressor | A Kinase Anchor Proteins - metabolism | Retinoids - pharmacology | Muscle, Smooth, Vascular - drug effects | Protein Isoforms - genetics | Tretinoin - pharmacology | Immunohistochemistry | RNA | Analysis | Genes | Smooth muscle | Retinoids | Protein kinases | Tumors | Protein kinase A | Actinomycin | Genomes | Thyroid gland | mRNA | Regulatory sequences | Kinases | Carotid arteries | Proteins | Signal transduction | Receptors | Cell growth | Acetylation | Lesions | Anchoring | Antigens | Cloning | Blood vessels | RNA polymerase | Dentistry | Gene expression | Nuclear receptors | Medicine | Microscopy | Tumor suppressor genes | Kinetics | Immunofluorescence | Retinoic acid | A kinase-anchoring protein
Journal Article
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