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Nature Medicine, ISSN 1078-8956, 12/2015, Volume 21, Issue 12, pp. 1481 - 1490
Journal Article
Cancer Cell, ISSN 1535-6108, 01/2016, Volume 29, Issue 1, pp. 32 - 48
Journal Article
Journal Article
Cell Stem Cell, ISSN 1934-5909, 10/2014, Volume 15, Issue 4, pp. 395 - 397
Journal Article
Cancer Cell, ISSN 1535-6108, 07/2013, Volume 24, Issue 1, pp. 5 - 7
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 02/2018, Volume 115, Issue 9, pp. 1964 - 1966
Journal Article
The Journal of experimental medicine, ISSN 0022-1007, 03/2017, Volume 214, Issue 3, pp. 719 - 735
Journal Article
Cancer Cell, ISSN 1535-6108, 11/2013, Volume 24, Issue 5, pp. 557 - 559
Although CEBPA mutations are among the most common genetic abnormalities in acute myeloid leukemia (AML), the transformation mechanism remains largely obscure.... 
INITIATING CELLS | AML | MYELOID-LEUKEMIA | STEM-CELLS | ONCOLOGY | CELL BIOLOGY | SOXC Transcription Factors - genetics | Animals | CCAAT-Enhancer-Binding Proteins - genetics | Gene Expression Regulation, Leukemic | Humans | Leukemia, Myeloid, Acute - genetics
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e72335
Acute myeloid leukemia (AML) is the most common malignant myeloid disorder of progenitor cells in myeloid hematopoiesis and exemplifies a genetically... 
AML1-ETO | RECRUITMENT | CELLS | DOMAIN | PROMYELOCYTIC LEUKEMIA | MULTIDISCIPLINARY SCIENCES | FUSION PROTEIN | RESISTANCE | DIFFERENTIATION | ESTABLISHMENT | HELICOBACTER-PYLORI | Gene Expression | Oncogene Proteins, Fusion - metabolism | Apoptosis - drug effects | Leukemia, Myeloid, Acute - pathology | Humans | Tumor Suppressor Protein p53 - metabolism | Drug Repositioning | Tumor Suppressor Protein p53 - genetics | Dose-Response Relationship, Drug | Animals | Furazolidone - pharmacology | Retroviridae - genetics | Cell Differentiation - drug effects | Oncogene Proteins, Fusion - genetics | Cell Line, Tumor | Leukemia, Myeloid, Acute - drug therapy | Biological Assay | Antineoplastic Agents - pharmacology | Antitrichomonal Agents - pharmacology | Leukemia, Myeloid, Acute - genetics | Care and treatment | Furazolidone | Tumor proteins | Cell differentiation | Health aspects | Drug approval | Drugs | Flow cytometry | Regulatory agencies | Transformation | Leukemia | p53 Protein | DNA damage | Genes | Cytology | Thyroid gland | Cancer therapies | Proteins | Promyeloid leukemia | Cell growth | Hematopoiesis | Cell cycle | Bone marrow | Bacteria | Drug dosages | Tretinoin | CD11b antigen | Myeloid leukemia | Patients | Hemopoiesis | Medicine | Flow stability | Cytometry | Chemotherapy | AML1 protein | Medical prognosis | Stem cells | Cells (biology) | Tumor suppressor genes | Antitumor activity | Acute myeloid leukemia | Retinoic acid | Apoptosis | Acute promyeloid leukemia
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2009, Volume 106, Issue 8, pp. 2853 - 2858
Journal Article