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2017, 1, Methods in signal transduction series, ISBN 149870509X, Volume 1, xv, 257 pages
T cells play a vital role mediating adaptive immunity, a specific acquired resistance to an infectious agent produced by the introduction of an antigen. There... 
Receptors | Immunology | T cells | Secretions | Thymus | Molecular Biology | Cell Biology | Thymus Gland - secretion | Cellular signal transduction
Book
Cell Calcium, ISSN 0143-4160, 06/2019, Volume 80, pp. 101 - 102
Journal Article
Cell Calcium, ISSN 0143-4160, 12/2018, Volume 76, p. 135
In a September 2018 paper published in Nature Communications, Gong et al. identified the domains through which human PMCA1 and neuroplastin (NPTN) interact.... 
Medical colleges | Medical genetics | Molecular biology
Journal Article
Nature Cell Biology, ISSN 1465-7392, 10/2015, Volume 17, Issue 10, pp. 1232 - 1234
Decreases in endoplasmic reticulum calcium content are sensed by resident STIM proteins, which can activate plasma membrane Orai channels to facilitate Ca(2+)... 
ORAI1 | ACTIVATION | OPERATED CALCIUM-ENTRY | MECHANISTIC INSIGHTS | CELL BIOLOGY | Animals | Calcium - metabolism | Humans | Endoplasmic Reticulum - metabolism | Cell Membrane - metabolism | Membrane Proteins - metabolism | Proteome - metabolism | Ion transport | Cellular proteins | Genetic aspects | Properties | Calcium ions
Journal Article
EMBO Journal, ISSN 0261-4189, 03/2012, Volume 31, Issue 5, pp. 1123 - 1133
Journal Article
The EMBO Journal, ISSN 0261-4189, 03/2018, Volume 37, Issue 5
Journal Article
The EMBO Journal, ISSN 0261-4189, 03/2018, Volume 37, Issue 5, p. n/a
STIM1 and STIM2 are endoplasmic reticulum Ca2+ sensors that serve multi‐faceted roles in signal transduction in a wide variety of different cell types. In this... 
CHANNELS | DOMAINS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ORAI | GROWTH | CELL BIOLOGY | Signal transduction | STIM1 protein | Transduction | Endoplasmic reticulum | Melanin | Calcium ions | Calcium (reticular) | Development & Differentiation | Physiology | Views | Signal Transduction
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2006, Volume 103, Issue 44, pp. 16586 - 16591
Journal Article
Science, ISSN 0036-8075, 10/2010, Volume 330, Issue 6000, pp. 105 - 109
Journal Article
JOURNAL OF CLINICAL INVESTIGATION, ISSN 0021-9738, 02/2013, Volume 123, Issue 2, pp. 887 - 902
During sepsis, acute lung injury (ALI) results from activation of innate immune cells and endothelial cells by endotoxins, leading to systemic inflammation... 
UNITED-STATES | MEDICINE, RESEARCH & EXPERIMENTAL | CA2+ INFLUX | ACTIVATION | INOSITOL 1,4,5-TRISPHOSPHATE | PROTEIN | ENDOTHELIAL-CELLS | GENE-EXPRESSION | TNF-ALPHA | CALCIUM SENSORS STIM1 | S-GLUTATHIONYLATION | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | NADPH Oxidases - metabolism | Male | Sepsis - complications | Gene Knockdown Techniques | NADPH Oxidases - deficiency | Membrane Glycoproteins - antagonists & inhibitors | Acute Lung Injury - prevention & control | NADPH Oxidases - genetics | Female | Acute Lung Injury - metabolism | Calcium Signaling | Thiadiazoles - pharmacology | Stromal Interaction Molecule 1 | Lipopolysaccharides - toxicity | Calcium Channels | Signal Transduction | NADPH Oxidases - antagonists & inhibitors | NFATC Transcription Factors - metabolism | Cells, Cultured | Inositol 1,4,5-Trisphosphate Receptors - deficiency | Acute Lung Injury - pathology | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Endothelium, Vascular - metabolism | Models, Biological | Anilides - pharmacology | Endothelium, Vascular - pathology | Mice | Acute Lung Injury - etiology | Inositol 1,4,5-Trisphosphate Receptors - genetics | Membrane Glycoproteins - deficiency | Blood vessels | Physiological aspects | Inflammation | Cellular signal transduction | Research | Risk factors | Lipopolysaccharides
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2006, Volume 103, Issue 11, pp. 4040 - 4045
Receptor-induced Ca²⁺ signals are key to the function of all cells and involve release of Ca²⁺ from endoplasmic reticulum (ER) stores, triggering Ca²⁺ entry... 
T lymphocytes | Proteins | Molecules | Isotypes | HEK293 cells | Small interfering RNA | Pharmacology | Cell membranes | Biochemistry | Sensors | Calcium signaling | Mast cells | Patch-clamp | Calcium channel | calcium channel | CELLS | DOMAIN | PROTEIN | EXOCYTOSIS | patch-clamp | MULTIDISCIPLINARY SCIENCES | calcium signaling | SENSOR | mast cells | CRAC CHANNELS | CAPACITATIVE CALCIUM-ENTRY | BORATE | INFLUX | SURFACE | Biological Sciences
Journal Article