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British Journal of Dermatology, ISSN 0007-0963, 04/2019, Volume 180, Issue 4, pp. 955 - 957
Linked Article: Larsabal et al. Br J Dermatol 2019; 180:201–202. 
DERMATOLOGY | Fibroblast growth factor receptor 2 | Acanthosis nigricans | Fibroblast growth factor receptors | Life Sciences
Journal Article
Annals of Oncology, ISSN 0923-7534, 2014, Volume 25, Issue 1, pp. 126 - 131
Background: There is scarce data available about epidermal growth factor receptor (EGFR) mutations other than common exon 19 deletions and exon 21 (L858R)... 
Non-small-cell lung cancer | Exon 20 mutations | Tyrosine-kinase inhibitors | Epidermal growth factor receptor mutations | Exon 18 mutations | epidermal growth factor receptor mutations | GEFITINIB | axon 18 mutations | exon 20 mutations | tyrosine-kinase inhibitors | TKIS | SENSITIVITY | CLASSIFICATION | SOMATIC MUTATIONS | CHEMOTHERAPY | TYROSINE KINASE INHIBITORS | GROWTH-FACTOR RECEPTOR | ONCOLOGY | NEVER SMOKERS | CLINICAL-RESPONSE | non-small-cell lung cancer | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Exons | Lung Neoplasms - mortality | Humans | Middle Aged | Male | Antineoplastic Agents - therapeutic use | Young Adult | Aged, 80 and over | Adult | Female | Adenocarcinoma - genetics | Antineoplastic Agents - pharmacology | Lung Neoplasms - genetics | Genetic Association Studies | Carcinoma, Non-Small-Cell Lung - genetics | Gene Frequency | Proportional Hazards Models | Adenocarcinoma - drug therapy | Carcinoma, Non-Small-Cell Lung - mortality | Disease-Free Survival | Drug Resistance, Neoplasm - genetics | Protein Kinase Inhibitors - therapeutic use | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Aged | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Adenocarcinoma - mortality | Life Sciences | Cancer | exon 18 mutations | Original
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 04/2012, Volume 18, Issue 7, pp. 1992 - 2000
Journal Article
Zoonoses and Public Health, ISSN 1863-1959, 06/2018, Volume 65, Issue 4, pp. 454 - 458
Summary Antibiotic resistance mediated by bacterial production of extended‐spectrum beta‐lactamase (ESBL) is a global threat to public health. ESBL resistance... 
multilocus sequence typing | ESBL‐producing Escherichia coli | plasmid typing | Desmodus rotundus | antibiotic resistance | Peru | ESBL-producing Escherichia coli | MOLECULAR CHARACTERIZATION | PLASMIDS | INFECTIOUS DISEASES | AMERICA | CHIROPTERA | BRAZIL | GULLS | GENES | VETERINARY SCIENCES | DIVERSITY | ENTEROBACTERIACEAE | PATHOGENS | Escherichia coli - drug effects | Humans | Chiroptera - microbiology | Multilocus Sequence Typing | beta-Lactamases - biosynthesis | Disease Reservoirs - veterinary | beta-Lactamases - genetics | Disease Reservoirs - microbiology | Zoonoses - epidemiology | Plasmids - genetics | Animals, Domestic - microbiology | Escherichia coli Infections - veterinary | Livestock - microbiology | Peru - epidemiology | Sheep Diseases - epidemiology | Sheep - microbiology | Animals, Wild - microbiology | Cattle Diseases - microbiology | Escherichia coli - enzymology | Sheep Diseases - microbiology | Escherichia coli Infections - microbiology | Cattle Diseases - epidemiology | Cattle - microbiology | Zoonoses - transmission | Escherichia coli Infections - epidemiology | Animals | Escherichia coli - genetics | Escherichia coli - isolation & purification | Anti-Bacterial Agents - pharmacology | Drug Resistance, Multiple, Bacterial - genetics | Zoonoses - microbiology | Drug resistance in microorganisms | Escherichia coli | Vampire bats | Distribution | Beta lactamases | Genetic aspects | Livestock | Health aspects | Zoonoses | Bats | Nucleotide sequence | Genes | Health risks | Infections | Gene expression | Domestic animals | Molecular chains | Disease resistance | Antibiotics | E coli | Wildlife | Strains | Plasmids | Antibiotic resistance | Bacteria | Children | Multilocus sequence typing | Control resistance | Strains (organisms) | Species | Public health | Life Sciences | Human health and pathology | Short Communications | Short Communication
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 07/2009, Volume 15, Issue 14, pp. 4733 - 4741
Journal Article
Journal Article
PLoS Computational Biology, ISSN 1553-734X, 03/2017, Volume 13, Issue 3, p. e1005432
The ability to build in-depth cell signaling networks from vast experimental data is a key objective of computational biology. The spleen tyrosine kinase (Syk)... 
ACTIVATION | EZRIN | GENE | PHOSPHORYLATION | CORTACTIN | IN-VIVO | GROWTH | BIOCHEMICAL RESEARCH METHODS | MATHEMATICAL & COMPUTATIONAL BIOLOGY | PROTEIN-TYROSINE KINASE | EXPRESSION | MASS-SPECTROMETRY | Syk Kinase - metabolism | MCF-7 Cells | Signal Transduction | Models, Biological | Computer Simulation | Humans | Gene Expression Regulation, Neoplastic | Cell Line, Tumor | Female | Gene Expression Profiling - methods | Neoplasm Proteins - metabolism | Breast Neoplasms - metabolism | Physiological aspects | Breast cancer | Cellular signal transduction | Health aspects | Cancer cells | Networks | Transcription factors | Motility | AKT protein | Patching | Biochemistry | Proteins | Signal transduction | Cell activation | Cell adhesion | Mammary gland | Growth factors | Bioinformatics | Carbon-carbon composites | Spleen | Gels | Neurodegenerative diseases | Data acquisition | Substrates | 1-Phosphatidylinositol 3-kinase | Chemistry | Molecular modelling | Computation | Computer applications | CD81 antigen | Mass spectrometry | Cell migration | Cell proliferation | Phosphorylation | Propagation | Switching theory | Biology | Genomes | Kinases | B-cell receptor | Cell adhesion & migration | Morphogenesis | Biological effects | Immunology | Actin | Analytical chemistry | Cell survival | Experimental data | Zinc | Computer programs | Lymphocytes B | Effectors | Data collection | Breast | Molecular biology | Apoptosis | Plasmas (physics) | Cancer | Life Sciences
Journal Article
Human Mutation, ISSN 1059-7794, 06/2019, Volume 40, Issue 6, pp. 716 - 720
Lynch syndrome (LS) is the most frequent cause of hereditary colorectal cancer. A subset of patients with a history of LS shows no causal germline pathogenic... 
AluYa5 | Lynch‐like syndrome | routine diagnosis | retrotransposon | mismatch repair deficiency | Lynch-like syndrome | GENETICS & HEREDITY | CANCER | Phenotypes | Nucleotide sequence | MLH1 protein | Splicing | Colorectal carcinoma | Exon skipping | Insertion | Genomes | Life Sciences | Human health and pathology
Journal Article
Journal Article
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 02/2017, Volume 18, Issue 2
Journal Article
CLINICAL CANCER RESEARCH, ISSN 1078-0432, 07/2019, Volume 25, Issue 14, pp. 4280 - 4289
Purpose: The detection of preexisting EGFR(T790M) subclones and the assessment of their clinical significance in the pretreatment of patients with EGFR(T790M)... 
TYROSINE KINASE INHIBITORS | EGFR T790M MUTATION | FACTOR-RECEPTOR GENE | MULTICENTER | OSIMERTINIB | NSCLC | ONCOLOGY | ACQUIRED-RESISTANCE | OPEN-LABEL | ACTIVATING MUTATIONS | CHEMOTHERAPY
Journal Article
Expert Review of Proteomics, ISSN 1478-9450, 08/2019, Volume 16, Issue 8, pp. 717 - 726
Introduction: The proteome is a dynamic system in which protein-protein interactions play a crucial part in shaping the cell phenotype. However, given the... 
BioID | mass spectrometry | APEX | proximal labeling by complementation | proximal labeling | protein-protein interactions | BIOTINYLATION | BIOTIN LIGASE | CELLS | BIOCHEMICAL RESEARCH METHODS | AFFINITY PURIFICATION | LARGE-SCALE | IDENTIFICATION | MASS-SPECTROMETRY | FRAGMENT COMPLEMENTATION | INTERACTION NETWORK | IN-SITU
Journal Article