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Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14 Supplement, pp. 2264 - 2264
Journal Article
International Journal of Cancer, ISSN 0020-7136, 10/2017, Volume 141, Issue 7, pp. 1286 - 1294
Targeting deficient mechanisms of cellular DNA repair still represents the basis for the treatment of the majority of solid tumors, and increased DNA repair... 
“BRCAness,” chemotherapy | radiotherapy | RAD51 | prognosis | SURVIVAL | PROTEIN | "BRCAness" | BRCA1 | chemotherapy | BREAST-CANCER | DAMAGE RESPONSE | EX-VIVO ASSAY | ONCOLOGY | PROGNOSTIC MARKER | HOMOLOGOUS RECOMBINATION | EXPRESSION | PREDICTIVE FACTOR | Neoplasms - metabolism | Rad51 Recombinase - metabolism | Cell Proliferation - genetics | Rad51 Recombinase - genetics | Recombinational DNA Repair - genetics | Humans | Radiation Tolerance - genetics | Male | DNA Repair - genetics | DNA, Neoplasm - drug effects | Poly(ADP-ribose) Polymerase Inhibitors - therapeutic use | Neoplasms - drug therapy | Polymorphism, Genetic | Drug Resistance, Neoplasm - genetics | Neoplasms - genetics | Biomarkers, Tumor - metabolism | Genes, BRCA2 | Female | Neoplasms - radiotherapy | Biomarkers, Tumor - genetics | Genes, BRCA1 | DNA, Neoplasm - radiation effects | Precision Medicine | Medical research | Chemotherapy | DNA | Medicine, Experimental | DNA repair | Sugars | Cancer | Monosaccharides | Drugs | Genetic variability | DNA damage | Homologous recombination | Medical services | Clinical trials | Cytotoxicity | Homology | Radioresistance | ADP | Proteins | Rad51 protein | Ribose | Damage | Repair | Deoxyribonucleic acid--DNA | Poly(ADP-ribose) polymerase | Radiation therapy | Gene expression | Polymerase | Biomarkers | Protein expression | Immunoreactivity | Solid tumors | Tumors
Journal Article
Journal Article
by George, J and Lim, J.S and Jang, S.J and Cun, Y.P and Ozretic, L and Kong, G and Leenders, F and Lu, X and Fernandez-Cuesta, L and Bosco, G and Muller, C and Dahmen, I and Jahchan, N.S and Park, K.S and Yang, D and Karnezis, A.N and Vaka, D and Torres, A and Wang, M.S and Korbel, J.O and Menon, R and Chun, S.M and Kim, D and Wilkerson, M and Hayes, N and Engelmann, D and Putzer, B and Bos, M and Michels, S and Vlasic, I and Seidel, D and Pinther, B and Schaub, P and Becker, C and Altmuller, J and Yokota, J and Kohno, T and Iwakawa, R and Tsuta, K and Noguchi, M and Muley, T and Hoffmann, H and Schnabel, P.A and Petersen, I and Chen, Y and Soltermann, A and Tischler, V and Choi, C.M and Kim, Y.H and Massion, P.P and Zou, Y and Jovanovic, D and Kontic, M and Wright, G.M and Russell, P.A and Solomon, B and Koch, I and Lindner, M and Muscarella, L.A and la Torre, A and Field, J.K and Jakopovic, M and Knezevic, J and Castanos-Velez, E and Roz, L and Pastorino, U and Brustugun, O.T and Lund-Iversen, M and Thunnissen, E and Kohler, J and Schuler, M and Botling, J and Sandelin, M and Sanchez-Cespedes, M and Salvesen, H.B and Achter, V and Lang, U and Bogus, M and Schneider, P.M and Zander, T and Ansen, S and Hallek, M and Wolf, J and Vingron, M and Yatabe, Y and Travis, W.D and Nurnberg, P and Reinhardt, C and Perner, S and Heukamp, L and Buttner, R and Haas, S.A and Brambilla, E and Peifer, M and Sage, J and Thomas, R.K and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Institutionen för immunologi, genetik och patologi and Lungmedicin och allergologi and Uppsala universitet and Institutionen för medicinska vetenskaper
Nature, ISSN 0028-0836, 2015, Volume 524, Issue 7563, pp. 47 - 53
We have sequenced the genomes of 110 small cell lung cancers (SCLC), one of the deadliest human cancers. In nearly all the tumours analysed we found bi-allelic... 
NEUROENDOCRINE TUMORS | PATHWAY | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | IN-VIVO | GENES | CYCLE ARREST | NOTCH | C-KIT | MUTATIONS | CARCINOMA | Neurosecretory Systems - pathology | Receptors, Notch - metabolism | Chromosome Breakpoints | Genomics | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Male | Receptors, Notch - genetics | Gene Expression Profiling | Tumor Suppressor Protein p53 - genetics | Small Cell Lung Carcinoma - metabolism | Tumor Suppressor Proteins - genetics | Neurosecretory Systems - metabolism | Female | Nuclear Proteins - genetics | Disease Models, Animal | Lung Neoplasms - genetics | Signal Transduction - genetics | Small Cell Lung Carcinoma - genetics | DNA-Binding Proteins - genetics | Mutation - genetics | Genome, Human - genetics | Small Cell Lung Carcinoma - pathology | Tumor Protein p73 | Animals | Cyclin D1 - genetics | Retinoblastoma Protein - genetics | Alleles | Cell Line, Tumor | Mice | Gene mutations | Analysis | Exome sequencing | Lung cancer, Small cell | Genetic aspects | Drug therapy | Cancer | Pathogenesis | Lung cancer | Genomes | Mutation | Kinases | Gene expression | Cancer therapies | Tumors | Clinical Medicine | Basic Medicine | Medical Genetics | Medicinsk genetik | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Cancer and Oncology | Klinisk medicin | Cancer och onkologi
Journal Article
Nature Genetics, ISSN 1061-4036, 2012, Volume 44, Issue 10, pp. 1104 - 1110
Journal Article
Journal Article
Human Pathology, ISSN 0046-8177, 2011, Volume 42, Issue 8, pp. 1078 - 1088
Journal Article
Molecular Cancer, ISSN 1476-4598, 2011, Volume 10, Issue 1, pp. 127 - 127
Background: The L1 cell adhesion molecule (L1CAM) is potentially involved in epithelial-mesenchymal transition (EMT). EMT marker expression is of prognostic... 
non-small cell lung cancer | STEM-CELLS | CARCINOMA CELLS | TUMOR PROGRESSION | tissue microarray | BIOCHEMISTRY & MOLECULAR BIOLOGY | THERAPEUTIC ANTIBODIES | E-CADHERIN | MALIGNANT-MELANOMA | OVARIAN-CARCINOMA | L1 cell adhesion molecule | prognostic marker | ONCOLOGY | COLORECTAL-CANCER | tumor-stroma interface | epithelial-mesenchymal transition | ADHESION MOLECULE L1 | Multivariate Analysis | Adenocarcinoma - pathology | Prognosis | Lung Neoplasms - mortality | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Lung Neoplasms - metabolism | Middle Aged | Neural Cell Adhesion Molecule L1 - genetics | Lung Neoplasms - pathology | Male | Gene Knockdown Techniques | Adenocarcinoma - metabolism | Carcinoma, Squamous Cell - mortality | RNA Interference | Epithelial-Mesenchymal Transition | Biomarkers, Tumor - metabolism | Female | Neural Cell Adhesion Molecule L1 - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Neoplasm Invasiveness | Carcinoma, Non-Small-Cell Lung - metabolism | Kaplan-Meier Estimate | Carcinoma, Non-Small-Cell Lung - mortality | Cell Line, Tumor | Aged | Biomarkers, Tumor - genetics | Adenocarcinoma - mortality | Care and treatment | Physiological aspects | Cadherins | Glycoproteins | Genetic aspects | Research | Lung cancer, Non-small cell | Health aspects | Medical research | Thoracic surgery | Motility | Hospitals | Surgery | Colleges & universities | Multivariate analysis | Experiments | Manuscripts
Journal Article
Nature communications, ISSN 2041-1723, 2014, Volume 5, Issue 1, p. 3518
Journal Article