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The FEBS Journal, ISSN 1742-464X, 10/2017, Volume 284, Issue 19, pp. 3218 - 3229
Bridging integrator 1 (bin1) gene is a genetic determinant of Alzheimer's disease (AD) and has been reported to modulate Alzheimer's pathogenesis through... 
SH3 domain | nuclear magnetic resonance spectroscopy | protein–protein interaction | Tau | BIN1 | Alzheimer's disease | ALZHEIMERS-DISEASE | NMR-SPECTROSCOPY | BIOCHEMISTRY & MOLECULAR BIOLOGY | PATHOLOGY | MODEL | IDENTIFIES VARIANTS | AMPHIPHYSIN | MEMBRANE CURVATURE | protein-protein interaction | BINDING | EXPRESSION | GENOME-WIDE ASSOCIATION | Adaptor Proteins, Signal Transducing - chemistry | Humans | Peptides - genetics | tau Proteins - metabolism | tau Proteins - chemistry | Peptides - metabolism | Protein Isoforms - metabolism | tau Proteins - genetics | Protein Isoforms - chemistry | Tumor Suppressor Proteins - chemistry | Tumor Suppressor Proteins - genetics | Cloning, Molecular | Escherichia coli - metabolism | Nuclear Magnetic Resonance, Biomolecular | Neurons - metabolism | Protein Interaction Domains and Motifs | Nuclear Proteins - genetics | Binding Sites | Recombinant Proteins - metabolism | Protein Conformation, alpha-Helical | Gene Expression | Tumor Suppressor Proteins - metabolism | Neurons - chemistry | Peptides - chemistry | Models, Molecular | Recombinant Proteins - chemistry | Nuclear Proteins - metabolism | Recombinant Proteins - genetics | Nuclear Proteins - chemistry | Amino Acid Motifs | Sequence Homology, Amino Acid | Sequence Alignment | Protein Conformation, beta-Strand | Escherichia coli - genetics | Adaptor Proteins, Signal Transducing - genetics | Protein Binding | Kinetics | Adaptor Proteins, Signal Transducing - metabolism | Protein Isoforms - genetics | Nuclear magnetic resonance spectroscopy | Neurons | Protein-protein interactions | Spectroscopy | Clathrin | Nuclear magnetic resonance--NMR | Peptides | Neurodegenerative diseases | Pathogenesis | Complexity | Proteins | Magnetic resonance spectroscopy | Tau protein | Spectrum analysis | Isoforms | Alzheimers disease | Binding sites | tau Proteins/metabolism | Nuclear Proteins/chemistry | Protein Isoforms/chemistry | Protein Isoforms/genetics | Adaptor Proteins, Signal Transducing/genetics | Recombinant Proteins/metabolism | Peptides/metabolism | Life Sciences | Recombinant Proteins/chemistry | Adaptor Proteins, Signal Transducing/chemistry | Nuclear Proteins/metabolism | Tumor Suppressor Proteins/chemistry | Nuclear Proteins/genetics | Tumor Suppressor Proteins/metabolism | Protein Isoforms/metabolism | Peptides/chemistry | Recombinant Proteins/genetics | Biochemistry, Molecular Biology | Escherichia coli/genetics | Escherichia coli/metabolism | Adaptor Proteins, Signal Transducing/metabolism | Neurons/chemistry | Tumor Suppressor Proteins/genetics | tau Proteins/genetics | Neurons/metabolism | Peptides/genetics | tau Proteins/chemistry
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 01/2011, Volume 300, Issue 1, pp. C42 - C48
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 01/2013, Volume 304, Issue 1, pp. H94 - H103
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 05/2018, Volume 71, Issue 19, pp. 2106 - 2118
Journal Article
American Journal of Physiology, ISSN 0363-6143, 01/2011, Volume 300, Issue 1, p. C42
Through their ion-pumping and non-ion-pumping functions, Na...-K...-ATPase protein complexes at the plasma membrane are critical to intracellular homeostasis... 
Enzymes | Polypeptides | Ischemia | Rodents | Ions | Physiology | Cells | Steroids
Journal Article
EBioMedicine, ISSN 2352-3964, 07/2016, Volume 9, Issue C, pp. 278 - 292
Journal Article
Acta neuropathologica communications, ISSN 2051-5960, 09/2015, Volume 3, Issue 1, p. 58
Journal Article
Physiological Reports, 10/2016, Volume 4, Issue 19, pp. e12991 - n/a
Journal Article
American Journal of Physiology, ISSN 0363-6135, 01/2013, Volume 304, Issue 1, p. H94
  Na+,K+-ATPase and cell survival were investigated in a cellular model of ischemia-reperfusion (I/R)-induced injury and protection by ouabain-induced... 
Survival analysis | Ischemia | Cardiomyocytes | Physiology | Gene expression | Adenosine triphosphatase
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2012, Volume 287, Issue 20, pp. 16390 - 16398
Decreases in cardiac Na/K-ATPase have been documented in patients with heart failure. Reduction of Na/K-ATPase alpha 1 also contributes to the deficiency in... 
UREMIC CARDIOMYOPATHY | OUABAIN | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | GROWTH | NA+/K+-ATPASE | SMOOTH-MUSCLE-CELLS | HYPERTENSION | CALCIUM-OVERLOAD | ENDOGENOUS DIGITALIS | Sodium-Potassium-Exchanging ATPase - genetics | Caspase 9 - genetics | Caspase 9 - metabolism | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Caspase 8 - metabolism | Apoptosis - genetics | Proto-Oncogene Proteins c-akt - genetics | Caspase 8 - genetics | Myocytes, Cardiac - enzymology | TOR Serine-Threonine Kinases - genetics | Mice, Mutant Strains | Muscle Proteins - metabolism | src-Family Kinases - metabolism | Muscle Proteins - antagonists & inhibitors | Heart Diseases - chemically induced | Proto-Oncogene Proteins c-akt - metabolism | Enzyme Inhibitors - adverse effects | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Signal Transduction - genetics | Enzyme Activation - drug effects | Heart Diseases - enzymology | Sirolimus - pharmacology | Muscle Proteins - genetics | Myocytes, Cardiac - pathology | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Signal Transduction - drug effects | Bufanolides - adverse effects | Anti-Bacterial Agents - pharmacology | Bufanolides - pharmacology | Mice | Enzyme Activation - genetics | src-Family Kinases - genetics | Heart Diseases - genetics | Cardiovascular Disease | Molecular Bases of Disease | Cardiotonic Steroids | Signal Transduction | Cardiomyopathy | Na,K-ATPase | Apoptosis
Journal Article
Biochemistry, ISSN 0006-2960, 05/2010, Volume 49, Issue 17, pp. 3602 - 3610
The isoform-specific region (ISR) is a region of structural heterogeneity among the four isoforms of the catalytic α-subunit of the Na,K-ATPase and an... 
86Rb+ uptake | rat | dileucine motif | α subunit | α1 and α2 isoforms
Journal Article
Physiological Reports, ISSN 2051-817X, 10/2016, Volume 4, Issue 19, pp. np - np
Cardiac glycosides (CG) are traditionally known as positive cardiac inotropes that inhibit Na super(+)/K super(+)-ATPase -dependent ion transport. CG also... 
Journal Article
Alzheimer's & Dementia: The Journal of the Alzheimer's Association, ISSN 1552-5260, 2014, Volume 10, Issue 4, pp. P794 - P794
Journal Article
The American Journal of Physiology, ISSN 0002-9513, 01/2011, Volume 300, Issue 1, p. 42
Through their ion-pumping and non-ion-pumping functions, [Na.sup.+]-[K.sup.+]-ATPase protein complexes at the plasma membrane are critical to intracellular... 
Physiological aspects | Ion channels | Cell physiology | Research | Adenosine triphosphatase
Journal Article
The American Journal of Physiology, ISSN 0002-9513, 01/2013, Volume 304, Issue 1, p. H94
[Na.sup.+], [K.sup.+]-ATPase and cell survival were investigated in a cellular model of ischemia-reperfusion (I/R)-induced injury and protection by... 
Ischemia | Ouabain | Physiological aspects | Sodium in the body | Development and progression | Potassium in the body | Health aspects | Adenosine triphosphatase
Journal Article
Acta Diabetologica, ISSN 0940-5429, 06/2017, Volume 54, Issue 6, pp. 551 - 560
Patients who underwent aortic valve replacement for CAVD (n = 43) were included in this study. Patients with coronary artery disease (CAD) without CAVD (n =... 
Calcific aortic valve disease | Leptin | Human valvular interstitial cells | Osteoblast differentiation | RISK-FACTORS | BONE MORPHOGENETIC PROTEIN-2 | VASCULAR CALCIFICATION | TISSUE | ATHEROSCLEROSIS | OSTEOPONTIN | ENDOCRINOLOGY & METABOLISM | CALCIFIC AORTIC-STENOSIS | SMOOTH-MUSCLE-CELLS | EXPRESSION | CORONARY-ARTERY-DISEASE | Biomarkers - metabolism | Heart Valve Diseases - pathology | Humans | Middle Aged | Osteoblasts - drug effects | Osteoblasts - physiology | Aortic Valve - drug effects | Cells, Cultured | Heart Defects, Congenital - pathology | Male | Case-Control Studies | Aortic Valve - pathology | Phenotype | Aortic Valve Stenosis - pathology | MAP Kinase Signaling System - drug effects | Signal Transduction - drug effects | Cell Differentiation - drug effects | Cell Transdifferentiation - drug effects | Aged, 80 and over | Female | Aged | Leptin - pharmacology | Calcinosis - pathology | Oncogene Protein v-akt - metabolism | Medical research | Phosphatases | Cardiac patients | Medicine, Experimental | Development and progression | Cardiology | Coronary heart disease | Cbfa-1 protein | Alkaline phosphatase | Bone morphogenetic protein 2 | Diabetes mellitus | Body weight | Coronary artery | Dyslipidemia | Osteopontin | Extracellular signal-regulated kinase | Interstitial cells | Cardiovascular disease | AKT protein | Hormones | Signal transduction | Osteoblastogenesis | Calcification | Aorta | Rheumatic heart disease | Heart diseases | Aortic valve | Age | Smoking
Journal Article