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1. Full Text Knockout of Amyloid β Protein Precursor (APP) Expression Alters Synaptogenesis, Neurite Branching and Axonal Morphology of Hippocampal Neurons
by Southam, Katherine A and Southam, Katherine A and Stennard, Fiona and Stennard, Fiona and Pavez, Cassandra and Pavez, Cassandra and Small, David H and Small, David H
Neurochemical research, ISSN 0364-3190, 6/2019, Volume 44, Issue 6, pp. 1346 - 1355
Neurochemistry | Biochemistry, general | Neurology | APP | Neurosciences | Biomedicine | Alzheimer’s disease | Axon pathfinding | Amyloid | Neurite outgrowth | Cell Biology | Cell culture | Cell survival | Synaptogenesis | Neurons | Axonogenesis | Abnormalities | L-lysine | Neutrons | Adhesion | Substrates | Cell adhesion & migration | Axons | Poly-L-lysine | Adhesive strength | Lysine | Precursors | Rodents | Cell adhesion | Mice | Dendrites | Alzheimer's disease | Hippocampus | Synapses
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2. Full Text Identifying the primary site of pathogenesis in amyotrophic lateral sclerosis - Vulnerability of lower motor neurons to proximal excitotoxicity
by Blizzard, Catherine A and Southam, Katherine A and Dawkins, Edgar and Lewis, Katherine E and King, Anna E and Clark, Jayden A and Dickson, Tracey C
Disease models & mechanisms, ISSN 1754-8403, 03/2015, Volume 8, Issue 3, pp. 215 - 224
Amyotrophic lateral sclerosis | Excitotoxin exposure | Excitotoxicity | Lower motor neuron | Motor neuron disease | Pathology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cell Line | Spinal Cord - drug effects | Mice, Inbred C57BL | Axons - drug effects | Motor Activity - drug effects | Neurotoxins - toxicity | Motor Neurons - pathology | Nerve Degeneration - pathology | Forelimb - physiopathology | Hindlimb - physiopathology | Amyotrophic Lateral Sclerosis - pathology | Animals | Forelimb - pathology | Spinal Cord - pathology | Axons - pathology | Hindlimb - pathology | Hindlimb - drug effects | Kainic Acid - toxicity | Forelimb - drug effects | Motor Neurons - drug effects | Index Medicus
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3. Full Text Rab1-dependent ER–Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS
by Soo, Kai Y and Halloran, Mark and Sundaramoorthy, Vinod and Parakh, Sonam and Toth, Reka P and Southam, Katherine A and McLean, Catriona A and Lock, Peter and King, Anna and Farg, Manal A and Atkin, Julie D
Acta neuropathologica, ISSN 0001-6322, 11/2015, Volume 130, Issue 5, pp. 679 - 697
Pathology | Neurosciences | Medicine & Public Health | ER–Golgi transport | FUS | TDP-43 | Amyotrophic lateral sclerosis | SOD1 | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | COP-Coated Vesicles - pathology | Neurons - pathology | Superoxide Dismutase - genetics | Spinal Cord - metabolism | Humans | Middle Aged | Endoplasmic Reticulum - metabolism | Cytoplasm - metabolism | Male | Golgi Apparatus - pathology | Cytoplasm - pathology | DNA-Binding Proteins - metabolism | Endoplasmic Reticulum - pathology | Spinal Cord - pathology | Aged, 80 and over | Female | Neurons - metabolism | Superoxide Dismutase - metabolism | rab1 GTP-Binding Proteins - metabolism | COP-Coated Vesicles - metabolism | RNA-Binding Protein FUS - genetics | RNA-Binding Protein FUS - metabolism | DNA-Binding Proteins - genetics | Amyotrophic Lateral Sclerosis - pathology | Animals | Amyotrophic Lateral Sclerosis - metabolism | Cell Line, Tumor | Golgi Apparatus - metabolism | Aged | Biological Transport - physiology | Mice | Mutation | Superoxide Dismutase-1 | Proteins | Analysis | Index Medicus
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4. Full Text A widely distributed hydrogenase oxidises atmospheric H2 during bacterial growth
by Islam, Zahra F and Welsh, Caitlin and Bayly, Katherine and Grinter, Rhys and Southam, Gordon and Gagen, Emma J and Greening, Chris
The ISME Journal, ISSN 1751-7362, 2020, Volume 14, Issue 11, pp. 2649 - 2658
Index Medicus
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5. Full Text Do Microglia Default on Network Maintenance in Alzheimer's Disease?
by Southam, Katherine A and Vincent, Adele J and Small, David H
Journal of Alzheimer's disease, ISSN 1387-2877, 03/2016, Volume 51, Issue 3, pp. 657 - 669
phagocytosis | synapse pruning | network abnormalities | neural networks | Microglia | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Microglia - metabolism | Animals | Alzheimer Disease - immunology | Synapses - physiology | Humans | Index Medicus
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6. Full Text Microfluidic primary culture model of the lower motor neuron–neuromuscular junction circuit
by Southam, Katherine A and King, Anna E and Blizzard, Catherine A and McCormack, Graeme H and Dickson, Tracey C
Journal of neuroscience methods, ISSN 0165-0270, 09/2013, Volume 218, Issue 2, pp. 164 - 169
Cell culture | Motor neuron | Muscle | Neuromuscular junction | Microfluidic | Biochemistry & Molecular Biology | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Biochemical Research Methods | Science & Technology | Immunohistochemistry | Microfluidic Analytical Techniques - methods | Microscopy, Electron, Scanning | Neuroglia - cytology | Neuromuscular Junction - cytology | Animals | Motor Neurons - cytology | Rats | Coculture Techniques - methods | Cell Culture Techniques - methods | Rats, Sprague-Dawley | Muscle Cells - cytology | Muscles | Neurons | Analysis | Microfluidics | Index Medicus
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7. Full Text Excitotoxin-induced caspase-3 activation and microtubule disintegration in axons is inhibited by taxol
by King, Anna E.lizabeth and Southam, Katherine A.driana and Dittmann, Justin and Vickers, James C.lement
Acta neuropathologica communications, ISSN 2051-5960, 2013, Volume 1, Issue 1, pp. 59 - 59
Caspase | Axon degeneration | Microtubule | Taxol | Excitotoxicity | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Dendrites - drug effects | Paclitaxel - pharmacology | Microtubule-Associated Proteins - metabolism | Wallerian Degeneration - physiopathology | Caspase 3 - metabolism | Cells, Cultured | Nerve Degeneration - physiopathology | Axons - drug effects | Axons - physiology | Nerve Degeneration - chemically induced | Cerebral Cortex - physiopathology | Dose-Response Relationship, Drug | Animals | Neuroprotective Agents - pharmacology | Microtubules - drug effects | Dendrites - physiology | Cytoskeleton - physiology | Mice | Cerebral Cortex - drug effects | Kainic Acid - toxicity | Cytoskeleton - drug effects | Nerve Degeneration - drug therapy | Axons | Nervous system | Degeneration | Neurons | Index Medicus
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8. Full Text A Nomogram for Predicting 30-day Mortality in Elderly Patients Undergoing Hemiarthroplasty for Femoral Neck Fractures
by Crawford, Zachary Thomas and Southam, Brendan and Matar, Robert and Avilucea, Frank R and Bowers, Katherine and Altaye, Mekibib and Archdeacon, Michael T
Geriatric orthopaedic surgery & rehabilitation, ISSN 2151-4593, 10/2020, Volume 11, pp. 2151459320960087 - 2151459320960087
Rehabilitation | Life Sciences & Biomedicine | Orthopedics | Surgery | Geriatrics & Gerontology | Science & Technology | Mortality | Fractures
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9. Full Text Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis
by Clark, Jayden A and Southam, Katherine A and Blizzard, Catherine A and King, Anna E and Dickson, Tracey C
Journal of chemical neuroanatomy, ISSN 0891-0618, 10/2016, Volume 76, Issue Pt A, pp. 35 - 47
Biochemistry & Molecular Biology | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Amyotrophic Lateral Sclerosis - genetics | Humans | Hindlimb - innervation | Mice, Transgenic | Synapses - pathology | Disease Progression | Forelimb - innervation | Motor Neurons - pathology | Nerve Degeneration - pathology | Amyotrophic Lateral Sclerosis - pathology | Animals | Forelimb - pathology | Axons - pathology | Hindlimb - pathology | Superoxide Dismutase-1 - genetics | Mice | Muscle, Skeletal - pathology | Neuromuscular Junction - pathology | Index Medicus
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10. Full Text Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1G93A mouse model of amyotrophic lateral sclerosis
by Clark, Jayden A and Southam, Katherine A and Blizzard, Catherine A and King, Anna E and Dickson, Tracey C
Journal of chemical neuroanatomy, ISSN 0891-0618, 10/2016, Volume 76, pp. 35 - 47
Distal 'dying back' degeneration | Amyotrophic lateral sclerosis | Forelimb pathology | Neuromuscular junction | SOD1G93A mouse | mouse | SOD1
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